ライフサイエンスコーパス: avian

1 mpacts both the production and perception of avian alarm calls using a combination of lab and field e

2 cteristics, while some indeed constitute the avian amygdala.

3 changes associated with song learning in the avian analog of primary motor cortex (robust nucleus of

4 n = 50) is highly rearranged compared to the avian ancestor.

5 brown bats (LBBs) that were compatible with avian and human IAV binding.

6 erica, could potentially be co-infected with avian and human IAVs, facilitating the emergence of zoon

7 ors could facilitate genetic reassortment of avian and human IAVs.

8 sequence of the RBS naturally varies across avian and human influenza virus subtypes and is also evo

9 ment because of their susceptibility to both avian and human influenza viruses.

10 tructural basis for the similarities between avian and mammalian arcopallial and amygdala subregions

11 same location in the Csf1r locus in reptile, avian and mammalian genomes.

12 as its role in male fertility indicates that avian and mammalian HSPA2 may exhibit distinct evolution

13 Dogs are a a potential mixing vessel for avian and mammalian IAVs and represent a human health co

14 The A allele is found in avian and mammalian viruses, but the B allele is viewed

15 milar neural-crest derived cells in both the avian and non-human primate spleen, showing evolutionary

16 approximately coincides with the ages of the avian and two mammalian sex chromosomes systems.

17 hape and its implications for the origins of avians and flight are not yet fully understood.

18 et provide the basis for assembling numerous avian (and possibly other reptilian) species, while the

19 the delivery process for multiple mammalian, avian, and insect cell lines.

20 Utilizing a panel of 28 distinct human, avian, and swine influenza viruses, we found that only a

21 thereby exposing them to predation risk from avian apex predators (cormorants, Phalacrocorax carbo).

22 entrocaudal pallial sector comparable to the avian arcopallium and to part of the mammalian pallial a

23 ggesting increasing biotic homogenization of avian assemblages throughout the United States.

24 roject to the Field L2 in the forebrain, the avian auditory cortex.

25 nce-discriminant neurons are revealed in the avian auditory cortex.

26 becomes restricted to the neural edge of the avian auditory organ, the basilar papilla, by embryonic

27 aking symmetry across the radial axis of the avian auditory organ.

28 together, these results demonstrate that the avian auditory thalamus is a structurally and functional

29 led to the development of a set of universal avian BAC clones that permit rapid anchoring of multiple

30 This is the first demonstration that avian behavior presents fractal organization that predom

31 that formerly infected the global breadth of avian biodiversity.

32 The avian centrifugal visual system, which projects from the

33 tion, plasticity and functional roles of the avian centrifugal visual system.

34 built an up-to-date and complete database on avian colour polymorphism based on the examination of av

35 dinosaurian ancestors, but the origin of the avian condition of low variation during development is p

36 We find that the avian cranium is highly modular, consisting of seven ind

37 n the 2nd SIA-binding site of NA proteins of avian-derived IAVs that became human pandemic viruses.

38 nificent examples is the transition from non-avian dinosaurs to birds that has created numerous evolu

39 at the climate change fingerprint in studied avian distributions is multidirectional.

40 upied areas of climatic niche space promotes avian diversification, or that diversification promotes

41 nd red coloration is a fundamental aspect of avian diversity and serves as an important signal in mat

42 Comparison of avian diversity at the beginning and end of the temporal

43 bird group, comprising almost half of global avian diversity.

44 d a previously undescribed projection of the avian dorsal pallidum.

45 Avian egg shape is generally explained as an adaptation

46 Avian egg white (EW) provides nutrition for the embryo a

47 sign of bilateral symmetry in mammalian and avian embryos is the appearance of the primitive streak

48 rcollicular nucleus and partly surrounds the avian equivalent of the central nucleus of the inferior

49 smembrane protease, serine 2 (TMPRSS2):v-ets avian erythroblastosis virus E26 oncogene homolog (ERG).

50 ition and lost by deletion during mammal and avian evolution, resulting in genome size equilibrium.

51 that bill diversity expanded early in extant avian evolutionary history, before transitioning to a ph

52 tudy, a microarray platform of 78 species of avian EWs was developed and profiled for glycosylation u

53 induced burst of diversification in kiwi, an avian family distributed within several hundred kilomete

54 relevant to both the genetic architecture of avian feather pigmentation and the evolutionary history

55 t they could be spread more globally via the avian flyways that converge and emanate from this region

56 ment provided by vultures and that mammalian-avian following patterns are consistent with the idea th

57 First, we found the avian fovea is off the retinal center towards the dorso-

58 vicle-clavicle structures, convergent to the avian furculum, and they retain shoulder girdle plesiomo

59 on being the rarest type of rearrangement in avian genome evolution.

60 of multiple scaffolds to chromosomes on all avian genomes.

61 any animal species and is posited to control avian germ cell formation.

62 urope and two fatal human infections with an avian H10N8 virus in China have demonstrated that H10 vi

63 The isolation of avian H15 viruses is, however, quite rare and, until rec

64 were largely resistant to highly pathogenic avian H5 and H7 influenza A viruses, but were almost as

65 iruses, including the 2009 H1N1 pandemic and avian H5N1 strains.

66 omising live viral vector expressing NA from avian (H5N1) or pandemic (H1N1) influenza virus, elicite

67 stigated the safety and immunogenicity of an avian H5N2 live attenuated influenza vaccine (LAIV H5N2)

68 Here, we found that natural avian H5N6 viruses have acquired a high affinity for hum

69 These findings indicate that LP avian H7 influenza A viruses are able to infect and caus

70 Low-pathogenic (LP) avian H7 influenza A viruses are widely distributed in t

71 erefore, the disease-causing potential of LP avian H7 influenza A viruses in mammals may be underesti

72 To address this, we studied 30 LP avian H7 viruses isolated from wild avian species in the

73 Accordingly, an engineered avian H7N7 influenza virus carrying a nucleoprotein with

74 The emergence of avian H7N9 influenza A virus in humans with associated h

75 c acids (SIAs) compared to a closely related avian H7N9 virus from 2008.

76 addition to genome segments derived from an avian H7N9 virus, the H7N3 virus reassorted efficiently

77 uated the infection and tropism of human and avian H9 influenza virus in the human respiratory tract

78 Avian H9N2 and 2009 pandemic H1N1 (pH1N1) influenza viru

79 PB2 sequences showed that the proportion of avian H9N2 or human H7N9 influenza isolates bearing PB2-

80 ffinity are just an early adaptation step of avian H9N2 strains; further mutational changes may be re

81 Avian H9N2 virus with the PA-K356R mutation in human A54

82 ntly described a predominant G57 genotype of avian H9N2 viruses that caused countrywide outbreaks in

83 We then apply it to avian haemosporidian parasite systems and to the pocket

84 ifts have shaped the evolutionary history of avian haemosporidian parasites and have played a minor r

85 account for the inter-specific variation in avian head/eye movement behavior.

86 For the avian hepadnavirus duck hepatitis B virus (DHBV), CTD is

87 an lineages infecting similar assemblages of avian host species.

88 s unlikely to cause Newcastle disease in its avian host, representing an essential step toward moving

89 es of natural, low-intensity infection in an avian host-parasite system: adult male red-winged blackb

90 the H5N1 subtype have been transmitted from avian hosts to humans.

91 ans relative to closely related viruses from avian hosts.

92 owed that only a subset of highly pathogenic avian (HPAI) H5N1 influenza virus strains could producti

93 e front of the telencephalon, resembling the avian hyperpallium.

94 ghly pathogenic (HP) and low-pathogenic (LP) avian influenza (AI) H5N2 and H7N1 were investigated dur

95 fluenza (pdmH1N1) virus or highly pathogenic avian influenza (H5N1) virus elicits robust, cross-react

96 de 2.3.4.4 CVVs.IMPORTANCE Highly pathogenic avian influenza (HPAI) A(H5) viruses have circulated con

97 The highly pathogenic avian influenza (HPAI) H5N1 viruses continue to circulat

98 uses in mammals.IMPORTANCE Highly pathogenic avian influenza (HPAI) H5N1 viruses continue to evolve i

99 Emergence of a highly pathogenic avian influenza (HPAI) H5N8 virus in Asia and its spread

100 Highly Pathogenic Avian Influenza (HPAI) has recently (2014-2015) re-emerg

101 tbreak of clade 2.3.4.4 H5 highly pathogenic avian influenza (HPAI) virus that occurred in the United

102 Eurasian clade 2.3.4.4 H5 highly pathogenic avian influenza (HPAI) virus.

103 nd mortality annually, and highly pathogenic avian influenza (HPAI) viruses along with other emerging

104 Highly pathogenic avian influenza (HPAI) viruses of the H5N1 subtype are e

105 the potential to mutate to highly pathogenic avian influenza (HPAI) viruses, but such viruses' origin

106 In December 2016, a low-pathogenic avian influenza (LPAI) A(H7N2) virus was identified to b

107 s to use suitability maps for Low Pathogenic Avian Influenza (LPAI) to identify areas at high risk fo

108 Introductions of low-pathogenic avian influenza (LPAI) viruses of subtypes H5 and H7 int

109 Human infections with highly pathogenic avian influenza A (H5N1) virus are frequently fatal but

110 fluenza A (H1N1) virus and highly pathogenic avian influenza A (H5N1) virus induce expression of tumo

111 The avian influenza A H7N9 virus has caused infections in hu

112 these results suggest that PB1-F2 from H7N9 avian influenza A virus may be a major contributory fact

113 Avian influenza A virus polymerases typically do not fun

114 ted for cases of human infection by emerging avian influenza A virus subtypes, including H7N9 and H10

115 Avian influenza A viruses (IAV) of the H9N2 subtype have

116 necessary for introduction and adaptation of avian influenza A viruses to mammalian hosts is importan

117 that it was most closely related to various avian influenza A(H10N7) viruses.

118 igenic features related to low pathogenicity avian influenza A(H3N2) viruses and were distinct from A

119 equences of representative highly pathogenic avian influenza A(H5) viruses from Vietnam were generate

120 after the emergence of human infections with avian influenza A(H5N1) and has evolved over time, with

121 he immunogenicity and protective efficacy of avian influenza A(H5N1) vaccine.

122 estigated 2 human cases of highly pathogenic avian influenza A(H5N1) virus infection, detected throug

123 tation and reassortment of highly pathogenic avian influenza A(H5N1) viruses at the animal-human inte

124 (CEFs) for studies on avian viruses such as avian influenza but no comprehensive study has as yet be

125 d approach was to select a low-pathogenicity avian influenza H5 virus that elicited antibodies that c

126 used the ferret model to address this for an avian influenza H5N1 vaccine.

127 The emergence of highly pathogenic avian influenza H5N1 viruses has raised concerns about t

128 Clade 2.2.2.1 highly pathogenic avian influenza H5N1 viruses were isolated from the case

129 Since May 2014, highly pathogenic avian influenza H5N6 virus has been reported to cause si

130 Recently, novel highly pathogenic avian influenza H5Nx viruses (clade 2.3.4.4) caused outb

131 An outbreak of highly pathogenic avian influenza H7N7 virus in Italy during 2013 resulted

132 on the seasonality of H5N1 Highly Pathogenic Avian Influenza in the domestic poultry population of Vi

133 with seasonal variation in the incidence of avian influenza outbreaks in the North of the country, t

134 should be taken into consideration in future avian influenza vaccine trials.

135 ans, the immune correlates of protection for avian influenza vaccines cannot be determined from clini

136 Vs in fruit bats and serological evidence of avian influenza virus (AIV) H9 infection in frugivorous

137 Our data provide a glimpse into avian influenza virus adaptation in mammals, with broad

138 However, information on avian influenza virus evolution and transmission during

139 se of the pathogenicity and low incidence of avian influenza virus infections in humans, the immune c

140 Avian influenza virus reassortants resembling the 1918 h

141 pendent cellular cytotoxicity (ADCC) against avian influenza virus subtypes, including H7N9 and H5N1,

142 14, a Eurasian strain H5N8 highly pathogenic avian influenza virus was detected in poultry in Canada.

143 nction (GOF) research with highly pathogenic avian influenza virus, severe acute respiratory syndrome

144 Several subtypes of avian influenza viruses (AIVs) are emerging as novel hum

145 Our surveillance of avian influenza viruses (AIVs) at Delaware Bay, USA, rev

146 TANCE The frequency of human infections with avian influenza viruses (AIVs) has increased in recent y

147 Phylogenetic analysis of these two novel avian influenza viruses (AIVs) suggested that their geno

148 response to the continuing evolution of H5N1 avian influenza viruses and human infections, new candid

149 hosts for avian influenza viruses.IMPORTANCE Avian influenza viruses are capable of crossing the spec

150 H9N2 avian influenza viruses are enzootic in poultry across A

151 etween antigenic drift and viral fitness for avian influenza viruses as well as the challenges of pre

152 Pathogenic H7N9 avian influenza viruses continue to represent a public h

153 s utility for monitoring the evolution of H9 avian influenza viruses from China between 2005 and 2015

154 nfections in humans, as well as detection of avian influenza viruses in birds in the United States.

155 s of numerous outbreaks of highly pathogenic avian influenza viruses in commercial poultry farms.

156 gated serological profiles against human and avian influenza viruses in the general population using

157 Vaccines against H7 avian influenza viruses may be more effective than HI an

158 Since 1997, highly pathogenic avian influenza viruses of the H5N1 subtype have been tr

159 Avian influenza viruses of the H7 hemagglutinin (HA) sub

160 ction for prepandemic vaccines.IMPORTANCE H7 avian influenza viruses present a serious risk to human

161 Human and avian influenza viruses recognize different sialic acid-

162 We analyzed the adaptation of avian influenza viruses to a mammalian host by passaging

163 between antigenic drift and the potential of avian influenza viruses to infect humans.

164 r zoonotic and pandemic emergence.IMPORTANCE Avian influenza viruses, such as H9N2, cause disease in

165 thought to potentiate antigenic diversity in avian influenza viruses.

166 ring therapeutic protection against human or avian influenza viruses.

167 at human disease caused by highly pathogenic avian influenza viruses.

168 redict the pandemic potential of circulating avian influenza viruses.

169 t commonly considered intermediate hosts for avian influenza viruses.IMPORTANCE Avian influenza virus

170 omosome breakpoint data, we established that avian interchromosomal breakpoints appear in the regions

171 Pigeon has a typical avian karyotype (2n = 80), while falcon (2n = 50) is hig

172 Avian leukosis virus (ALV) has endogenized prior to chic

173 but the factors that mediate alpharetroviral avian leukosis virus (ALV) integration are unknown.

174 Avian leukosis virus (ALV) is detrimental to poultry hea

175 in, is a cellular receptor of the subgroup J avian leukosis virus (ALV-J).

176 cological drivers of geographic variation in avian life-history strategies.

177 extensive co-circulation in pigs of Eurasian avian-like (EA) swine H1N1 and 2009 pandemic (pdm/09) H1

178 iting replication that can be overcome by an avian-like pH of activation for nuclear entry and a yet-

179 rofile confirmed these findings and revealed avian-like receptor-binding specificity.

180 ssays, the H10 viruses preferentially bound "avian-like" alpha2,3-linked sialic acids.

181 H10 viruses preferentially bind "avian-like" sialic acids, although several isolates also

182 ciated genes have unique roles in developing avian limbs.

183 -human transmission of influenza A(H7N2), an avian-lineage influenza A virus, that occurred during an

184 Avian malaria lineages were assigned to species level us

185 Although commonly used in avian medicine, limited pharmacokinetic (PK) data in dom

186 Avian metapneumovirus (AMPV) infects the respiratory and

187 yncytial virus (hRSV), hMPV, bovine RSV, and avian metapneumovirus (aMPV).

188 We focus on the visual part of the avian midbrain, the optic tectum (TeO, counterpart to ma

189 tion of sensory modalities in the TeO of the avian midbrain.

190 According to an avian model of colour discrimination thresholds, we foun

191 -generation sequencing technology in applied avian mycoplasma epidemiology at both local and global l

192 Despite attempts to control avian mycoplasmosis through management, vaccination, and

193 only used in the poultry industry to control avian mycoplasmosis; unfortunately, some vaccines may re

194 Neurons in the avian nidopallium caudolaterale (NCL), an endbrain struc

195 tricular ridge and appears comparable to the avian nidopallium.

196 Understanding of avian nocturnal flight comes mainly from northern hemisp

197 The avian nucleus laminaris (NL) is a brainstem nucleus nece

198 vaccine for pandemic preparedness.IMPORTANCE Avian origin H10 influenza viruses sporadically infect h

199 The recent outbreak of avian origin H10N7 influenza among seals in northern Eur

200 Two subtypes of influenza A virus (IAV), avian-origin canine influenza virus (CIV) H3N2 (CIV-H3N2

201 Since avian-origin H15 HA viruses have been shown to cause enh

202 , and include the equine-origin H3N8 and the avian-origin H3N2 viruses.

203 ges acquired during long-term circulation of avian-origin IAVs in mammals.IMPORTANCE Canine influenza

204 be important in the long-term adaptation of avian-origin influenza viruses to mammals.

205 virus to dogs; and the H3N2 CIV, which is an avian-origin virus that adapted to infect dogs.

206 es, the H3N8 and H3N2 viruses, of equine and avian origins, respectively.

207 za viruses (CIV) H3N8 and H3N2 of equine and avian origins, respectively.

208 ns under which C. gallinacea could act as an avian pathogen and possibly also a zoonotic agent.

209 Avian pathogenic Escherichia coli (APEC) causes one of t

210 or efficient NDV infection and their role in avian pathogenicity.

211 TALEN-mediated gene targeting in avian PGCs is therefore an efficient process.

212 xplored the effects of trophic asynchrony on avian population trends and potential underlying demogra

213 gested that a complex group of nuclei in the avian posterior ventral telencephalon is comparable to t

214 Ranchers also reported that avian predators seem to be the most challenging predator

215 phenology, and related asynchrony to annual avian productivity.

216 rs, we disentangled whether storm influences avian reassembly directly via functional traits (i.e. be

217 ow that the H15 HA has a high preference for avian receptor analogs by glycan array analyses.

218 omosome levels comparable, in continuity, to avian reference genomes.

219 Although we have shown that avian reovirus (ARV) p17-mediated inhibition of Akt lead

220 results in terms of the acoustic control of avian reproductive behavior is discussed, and a comparis

221 In the avian reproductive model of the rock dove (Columba livia

222 ption of formants as honest signals in a non-avian reptile combined with previous evidence from birds

223 read among amniotes (mammals, birds, and non-avian reptiles).

224 enza A viruses are widely distributed in the avian reservoir and are the precursors of numerous outbr

225 However, no studies have quantified avian responses to powerful ground-based light sources i

226 Our study shows heterogeneous avian responses to recent environmental changes.

227 However, natural skylight enters the avian retina unidirectionally, through the cornea and th

228 -kappa markedly inhibited the replication of avian RNA viruses in ovo.

229 Using a well-established retroviral model-avian Rous sarcoma virus (RSV)-we analyzed changes in an

230 We found abundant co-expression of both avian (SA alpha2,3-Gal) and human (SA alpha2,6-Gal) type

231 Avian saccadic head/eye movements have been shown to var

232 e efficiency of acid-dependent fusion of the avian sarcoma and leukosis virus (ASLV), with endosomes.

233 , and ligand binding properties of avian SC, avian SC domain variants, and a human SC (hSC) variant l

234 , dynamics, and ligand binding properties of avian SC, avian SC domain variants, and a human SC (hSC)

235 r mammalian scavengers to utilize particular avian scavenger species using preferred food sources sim

236 Large avian scavengers depend on carcasses which are more like

237 to assess interactions between mammalian and avian scavengers in one of the most diverse scavenging g

238 suggest that ongoing population declines in avian scavengers may have significant impacts on mammali

239 Within scavenging communities, avian scavengers often act as producers and mammalian sc

240 Our knowledge of the avian sensory trigeminal system has been largely restric

241 Here, we analyze mosaic evolution in the avian skull using high-dimensional 3D surface morphometr

242 ritical role in looping morphogenesis of the avian small intestine.

243 In our traditional view of the avian somatosensory system, input from the beak and head

244 Here, using data from 1306 recent avian speciation events, we show that plumage dichromati

245 ys on the recombination rate landscape in an avian speciation model, the Eurasian crow.

246 A viruses that infect numerous mammalian and avian species and are capable of causing severe and leth

247 shed the light on the repertoire of IFNs in avian species and provide functional data on the interac

248 mechanisms of action were identified in two avian species following petcoke extract exposure.

249 ed EC50 values were concordant with domestic avian species from similar species sensitivity categorie

250 ed 30 LP avian H7 viruses isolated from wild avian species in the United States and Canada using the

251 ntial of 30 LP H7 viruses isolated from wild avian species in the United States and Canada using the

252 auditory information in auditory generalist avian species is completely lacking.

253 ry dynamics of new HPAI viruses in different avian species is paramount.

254 g domain to screen for DLC sensitivity among avian species predicted that the gray catbird, a relevan

255 the deep brine layer and elevated mercury in avian species reported prior to causeway sealing.

256 pathogen of a wide variety of mammalian and avian species that threatens public health and food secu

257 Hb-binding proteins, such as PIT54 found in avian species, functionally converged with haptoglobin t

258 t fragmentation reduces fecundity in several avian species, including wood thrush, Hylocichla musteli

259 bitumen-derived material-including those to avian species-have not been characterized.

260 ta is available on the repertoire of IFNs in avian species.

261 male germ cells from five mammalian and one avian species.

262 operating at the onset of life in precocial avian species.

263 reproductive success and survival in several avian species.

264 considered uniquely sensitive to DLCs among avian species; but DLC toxicity in nondomesticated birds

265 ticular, we demonstrate how the ASHCE driven avian-specific expression of gene Sim1 driven by ASHCE m

266 g 48 bird genomes, we identified millions of avian-specific highly conserved elements (ASHCEs) that p

267 egulatory roles of ASHCEs in the creation of avian-specific traits, and further highlight the importa

268 the ecological causes of the advancement in avian spring migration phenology is still a challenge du

269 addressing slope estimates of the timing of avian spring migration regressed on (i) year and (ii) te

270 On average, avian spring migration times have advanced over time and

271 for the physiologic status of the mammal and avian stem groups and contextualizes the independent ori

272 and phylogenetically earliest members of the avian stem lineage (Avemetatarsalia), Teleocrater rhadin

273 as are their inputs, there is evidence from avian studies that GABA may also be involved.

274 I refer to a number of avian studies, particularly in corvids and parrots, whic

275 ior pretectal nucleus) to those described in avian studies.

276 ic disease in cattle caused by Mycobacterium avian subsp. paratuberculosis (MAP).

277 This human-avian system offers a simple refinement model for animal

278 oglobin function in comparisons involving 56 avian taxa that have contrasting altitudinal range limit

279 to develop, and social chemosignals occur in avian taxa.

280 tal-scale database of bird nests, suggesting avian thermal niches might be broadly limited by tempera

281 Relationships between non-avian theropod dinosaurs and extant and fossil birds are

282 erize, at the molecular level, MB in the non-avian theropod Tyrannosaurus rex (MOR 1125), and show th

283 revealing conservation of some features from avians to mammals.

284 ential toxicity pathways being impacted, two avian ToxChip PCR arrays-chicken and double-crested corm

285 elial-mesenchymal transition, acquisition of avian trunk neural crest cell (NCC) polarity is prerequi

286 rred to as human-type (NeuAcalpha2-6Gal) and avian-type (NeuAcalpha2-3Gal), respectively.

287 se viruses exhibited a strong preference for avian-type alpha2,3-linked sialic acids; however, bindin

288 n for the experimental finding that a mutant avian virus gained transmissibility in mammals despite t

289 e, virion stability and poor activity of the avian virus RNA-dependent RNA polymerase in human cells.

290 reviously been suggested to be restricted to avian virus species.

291 s identified 32 independent incursions of an avian virus-derived A allele into mammals, whereas 6 int

292 To test this, a number of clade A and B avian virus-derived NS segments were introduced into hum

293 barrier for aerosol droplet transmission of avian viruses in humans and ferrets.

294 ken embryo fibroblasts (CEFs) for studies on avian viruses such as avian influenza but no comprehensi

295 ons with non-H5N1 influenza viruses or other avian viruses.

296 viewed as being almost exclusively found in avian viruses.

297 Avian vocal learning and associated neural adaptations a

298 part of the neuromechanical control loop of avian vocal motor control.

299 Here, we studied the innervation of the avian vocal organ, the syrinx, in the zebra finch.

300 Avian W chromosomes evolved in parallel with mammalian Y