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1  All eight RNA segments were derived from an avian influenza A virus.
2 iratory tract, a pattern not seen before for avian influenza A viruses.
3  feature characteristic of highly pathogenic avian influenza A viruses.
4                                              Avian influenza A virus A/teal/HK/W312/97 (H6N1) possess
5                                 Recently, an avian influenza A virus (A/Hong Kong/156/97, H5N1) was i
6 blish a new lineage in the human population, avian influenza A viruses (AIV) must overcome the intrac
7                                              Avian influenza A viruses also infected HAE, but spread
8                                              Avian influenza A virus (an orthomyxovirus) is a zoonoti
9                Ducks are the natural host of avian influenza A viruses and display few or no disease
10 ortment and the potential emergence of novel avian influenza A viruses, as well as representing a hig
11 ly bind alpha2,6-linked sialic acids whereas avian influenza A viruses bind alpha2,3-linked sialic ac
12 a suggest that the H2N2 and H9N2 subtypes of avian influenza A viruses can contribute to secondary ba
13                                      Typical avian influenza A viruses do not replicate efficiently i
14                                              Avian influenza A viruses from Asia are recognized as th
15 nabated circulation of the highly pathogenic avian influenza A virus/H5N1 continues to be a serious t
16                   An extremely virulent H3N8 avian influenza A virus has been used to infect both imm
17 r the course of two waves of infection, H7N9 avian influenza A virus has caused 436 human infections
18 due to infection by highly pathogenic (H5N1) avian influenza A virus have raised the specter of a dev
19                                              Avian influenza A viruses have gained increasing attenti
20                   A switch in specificity of avian influenza A viruses' hemagglutinin (HA) from avian
21              To analyze the compatibility of avian influenza A virus hemagglutinins (HAs) and human i
22 at are coated with either LPS or inactivated avian influenza A virus (IAV) mimicking the uptake of ba
23        The potential role of wild mammals in avian influenza A virus (IAV) transmission cycles has re
24                                              Avian influenza A viruses (IAV) of the H9N2 subtype have
25             Building on the observation that avian influenza A viruses (IAVs) have a tropism for the
26 Human infections with highly pathogenic H5N1 avian influenza A viruses in the last decade have legiti
27 stigated the replication and transmission of avian influenza A viruses in two species thought to be i
28  the potential risk of other highly virulent avian influenza A viruses infecting and causing disease
29 ficient and SvEv129 wild-type mice using two avian influenza A viruses isolated from humans, A/Hong K
30  factor receptor 1 (TNFR1) by the use of two avian influenza A viruses isolated from humans, A/Hong K
31 ified multiple subtypes of low pathogenicity avian influenza A viruses isolated primarily from migrat
32  these results suggest that PB1-F2 from H7N9 avian influenza A virus may be a major contributory fact
33 ation, T to N, and T to P, the amino acid in avian influenza A virus NS1 proteins) had no effect.
34 inst lethal challenge with highly pathogenic avian influenza A viruses of the H5 and H7 subtypes.
35                                              Avian influenza A viruses of the H7 subtype have resulte
36 Due to dual susceptibility to both human and avian influenza A viruses, pigs are believed to be effec
37                                              Avian influenza A virus polymerases typically do not fun
38 ns and to poultry; therefore, newly emerging avian influenza A viruses pose a continued threat, not o
39                               For a panel of avian influenza A virus strains, we find evidence for a
40 he localization of PB2 of human from that of avian influenza A virus strains.
41 tokine induction of human, highly pathogenic avian influenza A virus subtype H5N1 and other animal in
42                       During 2013, three new avian influenza A virus subtypes, A(H7N9), A(H6N1), and
43 ted for cases of human infection by emerging avian influenza A virus subtypes, including H7N9 and H10
44  of host range restriction and adaptation of avian influenza A viruses to a new host species is still
45 necessary for introduction and adaptation of avian influenza A viruses to mammalian hosts is importan
46                   Human infections caused by avian influenza A virus type subtype H7N9 have been asso
47 between two viral genomic RNA segments of an avian influenza A virus using in vitro experiments.
48                                      An H5N1 avian influenza A virus was transmitted to humans in Hon
49                                         Most avian influenza A viruses, which preferentially replicat

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