ライフサイエンスコーパス: avian leukosis

1 The transmembrane subunit (TM) of the avian leukosis and sarcoma virus (ALSV) envelope glycopr

2 completely defective in mediating subgroup A avian leukosis and sarcoma virus (ALSV-A) entry.

3 Tva is the cellular receptor for subgroup A avian leukosis and sarcoma virus (ALSV-A).

4 The avian leukosis and sarcoma virus long terminal repeat (L

5 h myc in chicken B-cell lymphomas induced by avian leukosis proviral integrations.

6 y define cis-acting determinants involved in avian leukosis sarcoma virus packaging RNA binding to Ga

7 s to mice that express TVA, the receptor for avian leukosis sarcoma virus subgroup A (ALSV-A), under

8 We are using avian leukosis-sarcoma virus (ALSV) vectors to generate

9 The avian leukosis-sarcoma virus (ALV) group of retroviruses

10 rity at the amino acid level) to that of the avian leukosis-sarcoma virus family, it retains several

11 be important for the efficient packaging of avian leukosis-sarcoma virus RNA.

12 In avian leukosis-sarcoma virus, however, we have shown tha

13 sociated with infection by subgroups B and D avian leukosis-sarcoma viruses (ALVs).

14 ptide substrates that are derivatives of the avian leukosis/sarcoma virus nucleocapsid-protease cleav

15 OMMA-1D cells were engineered to express the avian leukosis subtype A receptor, tv-a, to permit infec

16 We have analyzed pol- mutants of avian leukosis virus (ALV) and murine leukemia virus (Mu

17 n to bind CCAAT/enhancer elements within the avian leukosis virus (ALV) and Rous sarcoma virus (RSV)

18 The avian leukosis virus (ALV) entry mechanism is controvers

19 day-old chicken embryos with the recombinant avian leukosis virus (ALV) EU-8 induces a high incidence

20 Avian leukosis virus (ALV) has been used as a model syst

21 Avian leukosis virus (ALV) has endogenized prior to chic

22 we provide new evidence for the presence of avian leukosis virus (ALV) in both CEF supernatants and

23 Avian leukosis virus (ALV) induces bursal lymphoma in ch

24 Avian leukosis virus (ALV) induces bursal lymphoma in tu

25 Avian leukosis virus (ALV) induces tumors by integrating

26 st chicken strains are highly susceptible to avian leukosis virus (ALV) induction of bursal lymphoma,

27 Avian leukosis virus (ALV) infection induces bursal lymp

28 but the factors that mediate alpharetroviral avian leukosis virus (ALV) integration are unknown.

29 Avian leukosis virus (ALV) is detrimental to poultry hea

30 The avian leukosis virus (ALV) long terminal repeat (LTR) co

31 ct with different regions of the RSV and the avian leukosis virus (ALV) LTRs.

32 In this study, we identified avian leukosis virus (ALV) proviral integration sites in

33 al chromosome-transgenic mice expressing the avian leukosis virus (ALV) receptor TVB, fused to monome

34 monstrated that bridge proteins comprised of avian leukosis virus (ALV) receptors fused to epidermal

35 A new subgroup of avian leukosis virus (ALV) that includes a unique env ge

36 An avian leukosis virus (ALV) was found in some chicken emb

37 A new subgroup of avian leukosis virus (ALV), designated subgroup J, was i

38 characteristics of a eukaryotic retrovirus, avian leukosis virus (ALV), offers a robust, eukaryotic

39 Avian leukosis virus (ALV), previously shown to be nonin

40 ion-deficient retroviral vector based on the avian leukosis virus (ALV), we inserted into the chicken

41 An avian leukosis virus (ALV)-based retroviral vector syste

42 interference experiments have indicated that avian leukosis virus (ALV)-E may utilize a cellular rece

43 s is a common retroviral integration site in avian leukosis virus (ALV)-induced B-cell lymphomas orig

44 ptor for the cytopathic subgroups B and D of avian leukosis virus (ALV-B and ALV-D), as a tumor necro

45 us avian retrovirus (EAV) and the endogenous avian leukosis virus (ALV-E), which originate from the c

46 Subgroup J avian leukosis virus (ALV-J) is a recently identified av

47 in, is a cellular receptor of the subgroup J avian leukosis virus (ALV-J).

48 cterized the interactions between subgroup A avian leukosis virus [ALV(A)] envelope glycoproteins and

49 The interactions between the subgroup A avian leukosis virus [ALV(A)] envelope glycoproteins and

50 est of this hypothesis, the requirements for avian leukosis virus A (ALV-A) infection were examined.

51 ansgene encoding the receptor for subgroup A avian leukosis virus and controlled by the astrocyte-spe

52 By infecting both clones and subclones with avian leukosis virus and using a PCR-based assay to dete

53 The avian leukosis virus DeltaLR-9 causes a high frequency o

54 set B-cell lymphomas induced by the nonacute avian leukosis virus EU-8.

55 ar domain of the TVB receptor for subgroup B avian leukosis virus fused to epidermal growth factor (E

56 Avian leukosis virus induces lymphoma in chickens after

57 vian retroviral [i.e., replication-competent avian leukosis virus long terminal repeat with splice ac

58 n using the retroviral replication-competent avian leukosis virus long terminal repeat, splice accept

59 binding the A1 CCAAT/enhancer motif from the avian leukosis virus long terminal repeat.

60 or, tv-a, to permit infection by recombinant avian leukosis virus produced by the replication-compete

61 We have adapted the avian leukosis virus RCAS (replication-competent avian s

62 ryocyte-lineage expression of the subgroup A avian leukosis virus receptor, TVA.

63 irus A (tva), which encodes the receptor for avian leukosis virus subgroup A (ALV/A), we provide dire

64 Cell killing by avian leukosis virus subgroup B (ALV-B) in cultures has

65 Avian leukosis virus subgroup J (ALV-J) is a simple retr

66 ific region, termed the E element or XSR, of avian leukosis virus subgroup J (ALV-J), a member of avi

67 otransferrin, a serine/threonine kinase, and avian leukosis virus subgroup J glycoprotein.

68 differences in receptor usage among the many avian leukosis virus subgroups.

69 o cell lineages by infection with subgroup A avian leukosis virus vectors in lines of transgenic mice

70 ific transfer of genes carried by subgroup A avian leukosis virus vectors.

71 mino-terminal domain of the alpharetrovirus, avian leukosis virus, revealing a previously undetected

72 rendering mice susceptible to infection with avian leukosis virus-derived gene vectors.

73 d at a common retroviral integration site in avian leukosis virus-induced lymphomas and has been impl

74 egration site in B cell lymphomas induced by avian leukosis virus.

75 to the novel envelope gene of the subgroup J avian leukosis virus.

76 cellular receptors for subgroup B, D, and E avian leukosis viruses (ALV) encoded by the s1 allele of

77 Host susceptibility to subgroup B, D, and E avian leukosis viruses (ALV) is determined by specific a

78 TVA, the cellular receptor for subgroup A avian leukosis viruses (ALV-A) can mediate viral entry w

79 ar domain of the TVA receptor for subgroup A avian leukosis viruses (ALV-A), fused to the MR1 single-

80 Subgroups B, D, and E avian leukosis viruses (ALV-B, -D, and -E) share the sam

81 containing RNA of both subgroup E endogenous avian leukosis viruses (ALV-E) and endogenous avian viru

82 studied genomic RNAs of wild-type and mutant avian leukosis viruses (ALVs) in an attempt to (i) bette

83 B and D and noncytopathic subgroup E of the avian leukosis viruses (ALVs).