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1 egration site in B cell lymphomas induced by avian leukosis virus.
2 to the novel envelope gene of the subgroup J avian leukosis virus.
3 est of this hypothesis, the requirements for avian leukosis virus A (ALV-A) infection were examined.
4             We have analyzed pol- mutants of avian leukosis virus (ALV) and murine leukemia virus (Mu
5 n to bind CCAAT/enhancer elements within the avian leukosis virus (ALV) and Rous sarcoma virus (RSV)
6                                          The avian leukosis virus (ALV) entry mechanism is controvers
7 day-old chicken embryos with the recombinant avian leukosis virus (ALV) EU-8 induces a high incidence
8                                              Avian leukosis virus (ALV) has been used as a model syst
9                                              Avian leukosis virus (ALV) has endogenized prior to chic
10  we provide new evidence for the presence of avian leukosis virus (ALV) in both CEF supernatants and
11                                              Avian leukosis virus (ALV) induces bursal lymphoma in ch
12                                              Avian leukosis virus (ALV) induces bursal lymphoma in tu
13                                              Avian leukosis virus (ALV) induces tumors by integrating
14 st chicken strains are highly susceptible to avian leukosis virus (ALV) induction of bursal lymphoma,
15                                              Avian leukosis virus (ALV) infection induces bursal lymp
16 but the factors that mediate alpharetroviral avian leukosis virus (ALV) integration are unknown.
17                                              Avian leukosis virus (ALV) is detrimental to poultry hea
18                                          The avian leukosis virus (ALV) long terminal repeat (LTR) co
19 ct with different regions of the RSV and the avian leukosis virus (ALV) LTRs.
20                 In this study, we identified avian leukosis virus (ALV) proviral integration sites in
21 al chromosome-transgenic mice expressing the avian leukosis virus (ALV) receptor TVB, fused to monome
22 monstrated that bridge proteins comprised of avian leukosis virus (ALV) receptors fused to epidermal
23                            A new subgroup of avian leukosis virus (ALV) that includes a unique env ge
24                                           An avian leukosis virus (ALV) was found in some chicken emb
25                            A new subgroup of avian leukosis virus (ALV), designated subgroup J, was i
26  characteristics of a eukaryotic retrovirus, avian leukosis virus (ALV), offers a robust, eukaryotic
27                                              Avian leukosis virus (ALV), previously shown to be nonin
28 ion-deficient retroviral vector based on the avian leukosis virus (ALV), we inserted into the chicken
29                                           An avian leukosis virus (ALV)-based retroviral vector syste
30 interference experiments have indicated that avian leukosis virus (ALV)-E may utilize a cellular rece
31 s is a common retroviral integration site in avian leukosis virus (ALV)-induced B-cell lymphomas orig
32 ptor for the cytopathic subgroups B and D of avian leukosis virus (ALV-B and ALV-D), as a tumor necro
33 us avian retrovirus (EAV) and the endogenous avian leukosis virus (ALV-E), which originate from the c
34                                   Subgroup J avian leukosis virus (ALV-J) is a recently identified av
35 in, is a cellular receptor of the subgroup J avian leukosis virus (ALV-J).
36  cellular receptors for subgroup B, D, and E avian leukosis viruses (ALV) encoded by the s1 allele of
37  Host susceptibility to subgroup B, D, and E avian leukosis viruses (ALV) is determined by specific a
38    TVA, the cellular receptor for subgroup A avian leukosis viruses (ALV-A) can mediate viral entry w
39 ar domain of the TVA receptor for subgroup A avian leukosis viruses (ALV-A), fused to the MR1 single-
40                        Subgroups B, D, and E avian leukosis viruses (ALV-B, -D, and -E) share the sam
41 containing RNA of both subgroup E endogenous avian leukosis viruses (ALV-E) and endogenous avian viru
42 cterized the interactions between subgroup A avian leukosis virus [ALV(A)] envelope glycoproteins and
43      The interactions between the subgroup A avian leukosis virus [ALV(A)] envelope glycoproteins and
44 studied genomic RNAs of wild-type and mutant avian leukosis viruses (ALVs) in an attempt to (i) bette
45  B and D and noncytopathic subgroup E of the avian leukosis viruses (ALVs).
46 ansgene encoding the receptor for subgroup A avian leukosis virus and controlled by the astrocyte-spe
47  By infecting both clones and subclones with avian leukosis virus and using a PCR-based assay to dete
48                                          The avian leukosis virus DeltaLR-9 causes a high frequency o
49 rendering mice susceptible to infection with avian leukosis virus-derived gene vectors.
50 set B-cell lymphomas induced by the nonacute avian leukosis virus EU-8.
51 ar domain of the TVB receptor for subgroup B avian leukosis virus fused to epidermal growth factor (E
52 d at a common retroviral integration site in avian leukosis virus-induced lymphomas and has been impl
53                                              Avian leukosis virus induces lymphoma in chickens after
54 vian retroviral [i.e., replication-competent avian leukosis virus long terminal repeat with splice ac
55 n using the retroviral replication-competent avian leukosis virus long terminal repeat, splice accept
56 binding the A1 CCAAT/enhancer motif from the avian leukosis virus long terminal repeat.
57 or, tv-a, to permit infection by recombinant avian leukosis virus produced by the replication-compete
58                          We have adapted the avian leukosis virus RCAS (replication-competent avian s
59 ryocyte-lineage expression of the subgroup A avian leukosis virus receptor, TVA.
60 mino-terminal domain of the alpharetrovirus, avian leukosis virus, revealing a previously undetected
61 irus A (tva), which encodes the receptor for avian leukosis virus subgroup A (ALV/A), we provide dire
62                              Cell killing by avian leukosis virus subgroup B (ALV-B) in cultures has
63                                              Avian leukosis virus subgroup J (ALV-J) is a simple retr
64 ific region, termed the E element or XSR, of avian leukosis virus subgroup J (ALV-J), a member of avi
65 otransferrin, a serine/threonine kinase, and avian leukosis virus subgroup J glycoprotein.
66 differences in receptor usage among the many avian leukosis virus subgroups.
67 o cell lineages by infection with subgroup A avian leukosis virus vectors in lines of transgenic mice
68 ific transfer of genes carried by subgroup A avian leukosis virus vectors.

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