ライフサイエンスコーパス: avian myeloblastosis virus

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1 ural products with the ability to combat the avian myeloblastosis virus.

2 (HIV-2), Moloney murine leukemia virus, and avian myeloblastosis virus.

3 thermal stabilities of wild-type recombinant avian myeloblastosis virus (AMV) and Moloney murine leuk

4 dy, reconstruction experiments with purified avian myeloblastosis virus (AMV) IN and retrovirus-like

5 The v-myb oncogene of the avian myeloblastosis virus (AMV) is unique among known o

6 The nuclear protein v-Myb, encoded by the avian myeloblastosis virus (AMV), can induce acute monob

7 ose of the well characterized retroviral RT, avian myeloblastosis virus (AMV).

8 lf-site and full-site reactions catalyzed by avian myeloblastosis virus and human immunodeficiency vi

9 on similarly inhibits the activities of both avian myeloblastosis virus and human immunodeficiency vi

10 Two acute transforming retroviruses, avian myeloblastosis virus and the E26 leukemia virus, t

11 v-Myb, the transforming protein of avian myeloblastosis virus, causes acute myeloid leukemi

12 The v-myb oncogene of the avian myeloblastosis virus has led to the discovery of a

13 recessed attachment (att) site sequences and avian myeloblastosis virus IN (4 nm) were as competent f

14 Molecular insights into the organization of avian myeloblastosis virus IN at the viral DNA ends were

15 The specific activities of virion-derived avian myeloblastosis virus integrase and bacterial recom

16 Avian myeloblastosis virus integrase can efficiently ins

17 The v-Myb oncoprotein encoded by Avian Myeloblastosis Virus is highly oncogenic, induces

18 ar homologue of the transforming gene of the avian myeloblastosis virus, is preferentially expressed

19 Thus, the v-myb gene encoded by the Avian Myeloblastosis Virus, is truncated at both the 5'

20 inhibit other reverse transcriptases tested (avian myeloblastosis virus, Moloney murine leukemia viru

21 te system, extension of the primer by either avian myeloblastosis virus reverse transcriptase (AMV-RT

22 HIV-1 or avian myeloblastosis virus reverse transcriptase (RT) we

23 tro, where the deleted cDNA was generated by avian myeloblastosis virus reverse transcriptase from a

24 owever, DNA polymerase I Klenow fragment and avian myeloblastosis virus reverse transcriptase incorpo

25 The shortened cDNA was generated by avian myeloblastosis virus reverse transcriptase, but no

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