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1  ICP34.5 of herpes simplex virus and NL-S of avian sarcoma virus.
2                                          The avian sarcoma virus 16 (ASV 16) is a retrovirus that ind
3       The retroviral oncogene p3k (v-p3k) of avian sarcoma virus 16 (ASV16) codes for the catalytic s
4 ne is a cell-derived insert in the genome of avian sarcoma virus 31 (ASV 31) and functions as the onc
5         Despite sequence differences between avian sarcoma virus and HIV-1 IN and their recognition s
6 hly preferred in vitro integration sites for avian sarcoma virus and human immunodeficiency virus-1 i
7 tion as seen in the integrase core domain of avian sarcoma virus as well as human immunodeficiency vi
8 etected independently of catalysis with both avian sarcoma virus (ASV) and human immunodeficiency vir
9 es of the core domain of integrase (IN) from avian sarcoma virus (ASV) and its active-site derivative
10          The direct-repeat elements (dr1) of avian sarcoma virus (ASV) and leukosis virus have the pr
11 human immunodeficiency virus type 1 (HIV-1), avian sarcoma virus (ASV) and their close orthologs from
12        Recent studies have demonstrated that avian sarcoma virus (ASV) can transduce cycle-arrested c
13            We have described a reconstituted avian sarcoma virus (ASV) concerted DNA integration syst
14                Using a model system in which avian sarcoma virus (ASV) DNA is epigenetically represse
15 of E3 ubiquitin ligases bind the L domain in avian sarcoma virus (ASV) Gag and facilitate viral parti
16                     Reverse transcription in avian sarcoma virus (ASV) initiates from the 3' end of a
17 nt of HIV-1, simian sarcoma virus (SIV), and avian sarcoma virus (ASV) INs predicted which of these r
18 in Daxx was identified as an interactor with avian sarcoma virus (ASV) integrase (IN) in a yeast two-
19 determined the size and shape of full-length avian sarcoma virus (ASV) integrase (IN) monomers and di
20  (aa) in the C-terminal region of the 286-aa avian sarcoma virus (ASV) integrase (IN) protein has bee
21                  In contrast, integration by avian sarcoma virus (ASV) integrase was more efficient a
22           Here, we report the mapping of 226 avian sarcoma virus (ASV) integration sites in the human
23                     The process by which the avian sarcoma virus (ASV) preintegration complex gains a
24 s comparable to one previously described for avian sarcoma virus (ASV) that was stimulated by the pre
25  the integrase protein of an oncoretrovirus, avian sarcoma virus (ASV), suggesting an active import m
26 netic repression and silencing of integrated avian sarcoma virus (ASV)-based vector DNAs in human HeL
27  same requirement for the simple retrovirus, avian sarcoma virus (ASV).
28 d HeLa cell populations that harbored silent avian sarcoma virus-based green fluorescent protein (GFP
29 of integration catalyzed by HIV-1 to that of avian sarcoma virus by analyzing the effect of defined m
30                 Simple retroviruses, such as avian sarcoma virus, do not encode regulatory proteins t
31 f chicken embryos with replication-competent avian sarcoma virus expressing either FgfR2(C278F), a re
32 es have indicated that in the context of the avian sarcoma virus genome, precise deletion of both ASV
33 m permanganate modification to show that the avian sarcoma virus IN catalytic domain is able to disto
34 he metal preference for in vitro activity of avian sarcoma virus IN is Mn2+ > Mg2+ and that a single
35 at the putative dimer-dimer interface of the avian sarcoma virus IN with its analogue, loop188-194, f
36 ar strategy, the unique amino acids found in avian sarcoma virus IN, rather than HIV-1 or Mason-Pfize
37  domain, or the isolated catalytic domain of avian sarcoma virus IN.
38 phic studies of the catalytic core domain of avian sarcoma virus integrase (ASV IN) have provided the
39 itor complex of the catalytic core domain of avian sarcoma virus integrase (ASV IN) were solved at 1.
40 of human immunodeficiency virus-1 integrase, avian sarcoma virus integrase, and bacteriophage Mu tran
41                                    Using the avian sarcoma virus integrase, we demonstrate that the e
42  of host sequences, as is characteristic for avian sarcoma virus integration.
43  the selection and characterization of novel avian sarcoma virus mutants.
44 in human (HeLa) cells, mediated by either an avian sarcoma virus or a human immune deficiency virus t
45 lected were substrates of HIV protease or of avian sarcoma virus protease, both of which have been re
46 t reduction in the total amount of HIV-1 and avian sarcoma virus retroviral vector DNA that is joined
47  of human immunodeficiency virus (HIV) RT or avian sarcoma virus RT.
48                                          The avian sarcoma virus UR2 codes for an oncogenic Gag-Ros f
49 n embryo fibroblasts (CEF) infected with the avian sarcoma virus UR2, encoding the oncogenic receptor

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