ライフサイエンスコーパス: awake mice

1 ation-entrained rhythm in the hippocampus of awake mice.

2 al respiration rhythm" (HRR), also occurs in awake mice.

3 c imaging of cortex in both anesthetized and awake mice.

4 osterior nucleus, and V1 in anesthetized and awake mice.

5 during hyperinsulinemic-euglycemic clamp in awake mice.

6 movements in the superior colliculus (SC) in awake mice.

7 ctional imaging of sensory input activity in awake mice.

8 inhibition of neurons deep in the brains of awake mice.

9 n real time based on single-unit feedback in awake mice.

10 s--in the visual cortex of anaesthetized and awake mice.

11 cross these neuron types in anesthetized and awake mice.

12 maps of retinotopy in both anesthetized and awake mice.

13 iorated isoproterenol-induced arrhythmias in awake mice.

14 s, across all six layers in visual cortex of awake mice.

15 and optical imaging of intrinsic signals in awake mice.

16 odor concentrations and in anesthetized and awake mice.

17 t of activity, determines spine stability in awake mice.

18 rainments of PVBC and OLM cell discharges in awake mice.

19 clic voltammetry in the nucleus accumbens of awake mice.

20 vation sufficient to evoke motor behavior in awake mice.

21 of visual processing by behavioral state in awake mice.

22 losure and monocular retinal inactivation in awake mice.

23 response enhancement in the visual cortex of awake mice.

24 dial perforant path-granule cell synapses in awake mice.

25 and ketamine/xylazine anesthesia than in the awake mice.

26 using chronic two-photon calcium imaging in awake mice.

27 during hyperinsulinemic-euglycemic clamps in awake mice.

28 Oral gavage of lipid in awake mice activated neurones throughout the nucleus of

29 image blood flow in cortical capillaries of awake mice and determine long-range correlations in spee

30 potentials and membrane voltage dynamics in awake mice and flies, resolving fast spike trains with 0

31 r cortical feedback in the olfactory bulb of awake mice and further probe its impact on the bulb outp

32 recorded GCs in the MOB of anesthetized and awake mice and identified state-dependent features of od

33 aged ensembles of Purkinje cell dendrites in awake mice and measured their calcium responses to perio

34 shifted toward higher values in the dLGN of awake mice and responses were more sustained.

35 We examined this issue in brain slices, awake mice, and a computational model.

36 al seizures in primary visual cortex (V1) of awake mice, and compared their propagation to the retino

37 ordings in the dLGN of both anesthetized and awake mice, and found that a surprisingly high proportio

38 d the functional role of VIP interneurons in awake mice, and investigated the underlying circuit mech

39 ike train properties of cerebellar output in awake mice, and strongly supports rate coding in the cer

40 increased V1 visual responses in stationary awake mice, artificially mimicking the effect of locomot

41 In this study, we show in awake mice at rest that spiking activity of Purkinje cel

42 In awake mice, brief repeated odor experience leads to a gr

43 tions from ensembles in the visual cortex of awake mice builds neuronal ensembles that recur spontane

44 rapid eye movement (REM) sleep compared with awake mice but are not elevated in non-REM sleep.

45 e function was normal by echocardiography in awake mice, but the smaller heart and a slower heart rat

46 Notably, PV+ neuron activation in awake mice caused a significant improvement in their ori

47 modeling study using a data set recorded in awake mice containing respiratory rate modulation.

48 Our results in awake mice differed in several respects from previous da

49 rons in upper-layer primary visual cortex of awake mice during locomotion and quiet wakefulness.

50 sponses measured in primary visual cortex of awake mice during passive viewing.

51 yer 2/3 excitatory and inhibitory neurons in awake mice during passive visual stimulation and perform

52 of neurons from the primary visual cortex of awake mice during visual stimulation and spontaneous act

53 y visual cortex of lightly anaesthetized and awake mice, during sensory processing.

54 As measured by nuclear scintigraphy in awake mice, gastric emptying of an ingested whole-egg me

55 onal populations in primary visual cortex of awake mice in the presence and absence of visual stimula

56 FP) activity in the whisker barrel cortex of awake mice is phase locked to respiration.

57 lin action and signaling during the clamp in awake mice lacking IKK-beta.

58 ulated rates of muscle glucose metabolism in awake mice lacking pyruvate dehydrogenase kinase 2 and 4

59 Using calcium imaging in awake mice of both sexes, we show that the spatial frequ

60 In vivo imaging in awake mice revealed that L2 cells had higher bandwidth t

61 Whole-cell recordings in L2/3 of awake mice revealed that the E/I ratio systematically de

62 In vivo calcium imaging in awake mice reveals that PV cells are broadly tuned to od

63 ological recordings in the olfactory bulb of awake mice show that individual cells encode the timing

64 aging activity across the cortical mantle in awake mice, show in this issue of Neuron that touch by a

65 The awake -/- mice tested with reflex modification audiometr

66 roscope we confirmed with calcium imaging in awake mice that hM4D activation by CNO inhibits striatop

67 rties of neurons in primary visual cortex of awake mice that were allowed to run on a freely rotating

68 In awake mice, this cortical decay function predicted the t

69 vivo two-photon population Ca(2+) imaging in awake mice, this study investigated how neural represent

70 d a 7 d intranasal insulin delivery (IND) in awake mice to ascertain the biochemical and behavioral e

71 aging of the indicator GCaMP6 in head-fixed, awake mice to characterize the organization of spontaneo

72 Here, we used two-photon calcium imaging in awake mice to compare visual responses in primary visual

73 ordings in visual cortex of anesthetized and awake mice to measure intracellular activity; we then ap

74 l recording and two-photon Ca(2+) imaging in awake mice to show that lateral inhibition shapes freque

75 o-photon calcium imaging in anesthetized and awake mice to visualize both odorant-evoked excitation a

76 aging of granule cell population activity in awake mice using a cortical window implant that leaves t

77 photon imaging across all cortical layers in awake mice using a microprism attachment to the cranial

78 evoked gamma activity in layers 2/3 of V1 of awake mice using targeted patch-clamp recordings and syn

79 In awake mice, visual evoked potentials (VEPs) recorded in

80 Using large-scale population recordings in awake mice, we find distinct coding strategies facilitat

81 ing calcium imaging of cellular responses in awake mice, we find surprising asymmetries in the spatia

82 In awake mice, we found that auditory responses in entorhin

83 Here, using in vivo two-photon imaging in awake mice, we found that learning-induced spine reorgan

84 oton emission computed tomography imaging in awake mice, we identified brain structures activated dur

85 scence imaging of the barrel cortex in fully awake mice, we reveal that acute COX-1 inhibition reduce

86 y juxtacellular and whole-cell-recordings in awake mice, we show here that in the subiculum a subset

87 Using two-photon calcium imaging in awake mice, we show that the encoding of audiovisual sti

88 single-unit recordings in auditory cortex of awake mice, we show that this may not generally hold tru

89 Although astrocytes in visual cortex of awake mice were rarely engaged when neurons were activat

90 esia with urethane or isoflurane and whether awake mice were stationary or running on a treadmill.

91 rtex to drive spiking and vibrissa motion in awake mice when excited with red light through intact sk

92 xcitability during in vivo EEG recordings in awake mice where the effects of the proconvulsant pentyl

93 ging of dendritic spines in barrel cortex of awake mice while being conditioned.