ライフサイエンスコーパス: awakening

1 act tonometer (session with nocturnal hourly awakening).

2 was not measured (session without nocturnal awakening).

3 ime to fall asleep, and frequencies of night awakenings).

4 id not alter Pdc when measured 4 hours after awakening.

5 Symptom onset commonly occurred on awakening.

6 ultiple pathways to presynaptic dormancy and awakening.

7 r subunit contribution after PDBu-stimulated awakening.

8 total population, both before and following awakening.

9 depend on circadian phase or sleep stage at awakening.

10 set, shortly after the usual time of morning awakening.

11 rized by early sleep times and early-morning awakening.

12 as persistent vegetative state or death vs. awakening.

13 voked potential responses have <1% chance of awakening.

14 d sustainable agriculture occurred as a slow awakening.

15 GH every 20 minutes from 9 PM until morning awakening.

16 fore going to bed and immediately on morning awakening.

17 st cigarette of the day within 30 minutes of awakening.

18 chemia persisted (P < .05) for 2 hours after awakening.

19 ng active sleep, and decreased markedly upon awakening.

20 trouble, fatigue, and feeling refreshed upon awakening.

21 e VAS for fatigue and feeling refreshed upon awakening.

22 that sustains the increase in ischemia upon awakening.

23 ntal activities and to postural changes upon awakening.

24 normothermia has a high predictive value for awakening.

25 over the first days of coma is predictive of awakening.

26 rtPA had to be started </=3 hours of patient awakening.

27 rmance as a function of time since entrained awakening.

28 ulses, with a steep rise in amplitude before awakening.

29 prior hospitalizations, and prior nocturnal awakenings.

30 with greater value when combined with daily awakenings.

31 -reported child sleep duration and nocturnal awakenings.

32 effect was observed for the number of night awakenings.

33 ing sleep fragmentation (number of overnight awakenings, 1.51 [1.03] at baseline vs 0.92 [0.97] after

34 nts with clinically evident myoclonus before awakening, 2 expert physicians reviewed and classified a

35 2.5%), and the percentage of nights with no awakenings (23.0 +/- 2.5 vs. 15.5+/-2.4%) compared with

36 able improvement of the predictive value for awakening (93%, with 95% confidence interval: 0.77-0.99)

37 atosensory evoked potential result, rates of awakening (95% confidence interval) were calculated: adu

38 ry flow (P), reliever use (R), symptoms (S), awakenings (A), and threshold values for change from bas

39 The night awakenings (Actigraph) pattern was significantly differe

40 igraine triggers such as perfume, stress, or awakening activate multiple hypothalamic, limbic, and co

41 ved from host flora may play a major role in awakening adaptive immunity in neonates.

42 five patients with tonic-clonic seizures on awakening and 30 control subjects had T1-weighted volume

43 ne of four ischemic strokes are noticed upon awakening and are not candidates for intravenous recombi

44 liance by an interprofessional team with the Awakening and Breathing Coordination, Choice of drugs, D

45 n, we developed, implemented, and refined an awakening and breathing coordination, delirium monitorin

46 e important differences as compared with the awakening and breathing coordination, delirium monitorin

47 In this study of the implementation of the awakening and breathing coordination, delirium monitorin

48 Awakening and Breathing Coordination, Delirium monitorin

49 Critically ill patients managed with the Awakening and Breathing Coordination, Delirium monitorin

50 effectiveness and safety of implementing the Awakening and Breathing Coordination, Delirium monitorin

51 entilation status, patients managed with the Awakening and Breathing Coordination, Delirium monitorin

52 models to quantify the relationship between Awakening and Breathing Coordination, Delirium monitorin

53 The awakening and breathing coordination, delirium monitorin

54 was to identify facilitators and barriers to awakening and breathing coordination, delirium monitorin

55 minimized sedation, paired daily spontaneous awakening and breathing trials, and conservative fluid m

56 pontaneous breathing trials, coordination of awakening and breathing trials, choice of sedation, deli

57 inimizing sedation, paired daily spontaneous awakening and breathing trials, early exercise and mobil

58 orhexidine mouth care, and daily spontaneous awakening and breathing trials.

59 care that combines evidence-based practices: awakening and breathing, coordination with target-based

60 Secondary end points of awakening and discharged alive from hospital trended tow

61 osomal dominant condition with early morning awakening and early sleep times.

62 volatile-based sedation may provide superior awakening and extubation times in comparison with curren

63 short and refreshing naps, few problems with awakening and good response to stimulants, without catap

64 ascular reactivity to hypercapnia on morning awakening and its association with specific sleep-relate

65 and their sequella, including early morning awakening and loss of appetite.

66 asthma exacerbations and decreased nocturnal awakenings and activities limited by asthma.

67 rates and quality of life, reduced nocturnal awakenings and asthma attacks, increased the number of a

68 ght eating symptoms, the number of nocturnal awakenings and ingestions, total daily caloric intake af

69 non-REM sleep episodes and in the number of awakenings and movements.

70 mentation was defined as the total number of awakenings and shifts to Stage 1 sleep divided by the to

71 s suggest common dynamical features of brief awakenings and sleep durations across species and may pr

72 levels were measured at wakeup, 30 min after awakening, and at bedtime each day.

73 analyzed independently at coma onset, after awakening, and at follow-up.

74 asleep, maintaining sleep, and early morning awakening, and is coupled with daytime consequences such

75 of tender points, FIQ fatigue, tiredness on awakening, and stiffness scores, Clinical Global Impress

76 , 1.04 (95% CI, 0.88-1.22) for early-morning awakenings, and 1.24 (95% CI, 1.05-1.46) for nonrestorat

77 tings, frequency of nocturnal ingestions and awakenings, and caloric intake after the evening meal.

78 the stages of NREM sleep, resulting in fewer awakenings, and increase a physiological measure of slee

79 p-onset latency, total sleep time, number of awakenings, and sleep quality were selected as outcomes.

80 hen deeper levels of sedation and more rapid awakening are required.

81 y increased the percentage of nights with no awakenings as compared with placebo, salmeterol, and FP

82 ening, as-needed beta-agonist use, nocturnal awakenings, asthma-specific quality of life, and worseni

83 y expenditure in control mice increased upon awakening at a greater rate than in the narcoleptic mice

84 s were scheduled approximately 4 hours after awakening at baseline and after 1, 5, 10, 14, and 30 day

85 s of breath (OR, 1.6; 95% CI, 1.2 to 2.1) or awakening at night (OR, 1.5; 95% CI, 1.1 to 2.0), and wh

86 s of breath (OR, 2.4; 95% CI, 1.3 to 4.4) or awakening at night in the previous 12 mo (OR, 3.2; 95% C

87 ma with frequent albuterol use and nighttime awakenings at least once weekly.

88 omposer activity and carbon stock: a 'biotic awakening' at depth.

89 omly during the day (random grand mal) or on awakening (awakening grand mal), and juvenile absence ep

90 s reached their circadian peak shortly after awakening, before declining to daytime levels.

91 facilitate/hinder the implementation of the awakening, breathing, coordination, delirium, and early

92 five patients with tonic-clonic seizures on awakening, but in none of the 30 control subjects.

93 g approaches, we uncovered rapid presynaptic awakening by select synaptic modulators.

94 , devised by Sloan and colleagues, and night awakenings by use of the Pittsburgh Sleep Quality Index.

95 eased salivary cortisol concentrations after awakening compared with control subjects.

96 e pooled results were evaluated for rates of awakening, confidence intervals, and the possibility of

97 The total number of night awakenings continued to decrease over the remaining foll

98 morning hours, and the postural change after awakening contributes to the morning increase in ischemi

99 Conversely, stable post-awakening correlations--such as those found between the

100 ents, body mass index (BMI), saliva cortisol awakening curves (area under the curve with respect to t

101 Thus, the torpid/awakening cycle of the hibernating European hamster caus

102 Night awakenings decreased from a mean of 19.5 (SD 14.8) per w

103 Primary outcome measure was awakening, defined as the ability to follow commands bef

104 iii) chronic inflammation is a key factor in awakening dormant malignant cells at the primary site, l

105 wheeze (OR, 5.91; P < 0.01), more nighttime awakening due to cough (OR, 4.20; P = 0.03), increased r

106 , while in a persister cell status, and upon awakening due to exposure to cis-2-decenoic acid (cis-DA

107 onist use for at least 2 days, or night-time awakenings due to asthma for at least one night.

108 that these were spontaneous, as the times to awakening during hypercapnia were much higher than durin

109 Hourly awakening during NCT IOP measurements did not significan

110 Hourly awakening during noncontact tonometer IOP measurements d

111 as difficulty with sleep onset and prolonged awakenings during the night.

112 Epilepsy with grand mal on awakening (EGMA) is a well-defined subtype of idiopathic

113 ur data unveil a CCL5-dependent mechanism of awakening endogenous antitumor immunity triggered by ex

114 sufficient to increase the probability of an awakening event during both slow-wave sleep and rapid ey

115 sufficient to increase the probability of an awakening event in histidine decarboxylase-deficient kno

116 Hcrt neurons increased the probability of an awakening event throughout the entire light/dark period

117 s also recorded the total time and number of awakening for each patient overnight.

118 ms seem to be unaffected by hourly nocturnal awakening for IOP measurements in young healthy individu

119 Hourly awakening for nocturnal IOP measurements increased wake

120 with GSD to sleep through the night without awakening for therapy while enhancing safety.

121 ation between lateral brain displacement and awakening from acute coma.

122 n back pain with exercise but not with rest; awakening from back pain during the second half of the n

123 However, PPI was smaller at awakening from non-REM sleep compared to established wak

124 Reduced PPI of the startle reflex at awakening from non-REM sleep supports the hypothesis tha

125 hase and are more likely to have their final awakening from NREM sleep than younger adults, our findi

126 PPI after awakening from REM sleep (52.8 +/- 17.9%) was not signif

127 Comparisons between periods of awakening from sedation vs. sedative infusion were made.

128 diabetic subjects exhibited markedly reduced awakening from sleep during hypoglycemia.

129 Awakening from sleep entails rapid re-establishment of c

130 ing from Hcrt neurons is sufficient to drive awakening from sleep states or is simply correlated with

131 ce, few studies have focused specifically on awakening from sleep.

132 in regional cerebral blood flow (rCBF) upon awakening from stage 2 sleep.

133 frontal and inferior parietal cortices upon awakening from unconsciousness.

134 Patient 1, who had a history of night awakenings from pain, reported 101 awakenings owing to p

135 amical patterns and functions of these brief awakenings from sleep are not well understood, and they

136 In this article, we hypothesize that brief awakenings from sleep may reflect aspects of the endogen

137 for 11.5 and 17% of the overall arousals and awakenings from sleep, respectively.

138 the day (random grand mal) or on awakening (awakening grand mal), and juvenile absence epilepsy (JAE

139 n 556 children (1.7%) and frequent nocturnal awakenings (>/=3 times) in 1033 children (3.2%) at 18 mo

140 eactivity of the cerebral vessels on morning awakening has been suggested as one of the mechanisms un

141 was no group effect for sleep onset, time of awakening, hours slept, or hours napping.

142 sleep fragmentation (number of arousals and awakenings/hr), but the dedicated noninvasive ventilator

143 brain displacement are associated with acute awakening in comatose patients.

144 rmed approximately 4.5 hours after scheduled awakening in each cycle so that 12 tests in each subject

145 n diurnal IOP variation and IOP changes upon awakening in habitual and supine positions.

146 Upon awakening in the morning, healthy volunteers collected o

147 t-light exposure be scheduled immediately on awakening in the treatment of most patients with seasona

148 mulation of OH cells (at rates that promoted awakening in vivo) with electrical monitoring of MCH cel

149 tion from theta to alpha rhythm (spontaneous awakening), increased by 13.3% (P<0.01); VT increased by

150 on where both the hibernation period and the awakening intensity are taken into account.

151 is distinct, transiently aroused, state upon awakening may serve a protective function, preparing an

152 gh a sequence of CORT pulses--as seen around awakening--may ensure that hippocampal glutamatergic syn

153 d with 2.6% of controls, with blind children awakening much earlier.

154 Awakenings occurred in 19 of 19 trials within 5 min, wit

155 Importantly, this test predicted the awakening of 13 out of 51 patients for which the outcome

156 resulting in a positive predictive value of awakening of 82% (95% confidence interval: 0.65-0.93).

157 Overall our findings indicate that upon awakening of a persister population the cells regain the

158 naling in pfGCs, and these cells trigger the awakening of dormant oocytes and complete the process of

159 We further show that pfGCs trigger the awakening of dormant oocytes through KIT ligand (KITL),

160 rdial follicles and govern the quiescence or awakening of dormant oocytes.

161 growth, energy balance and season, time the awakening of gonadotropin releasing hormone (GnRH) neuro

162 ostate cancer, in particular the apparent re-awakening of key developmental programs that occur durin

163 -nocturnal IOP changes, and IOP changes upon awakening of the converters were significantly different

164 y lipopolysaccharide treatment, triggers the awakening of these cells, which develop into macroscopic

165 in saliva during the first 30 minutes after awakening on a workday and on a nonworkday.

166 m were used to evaluate the effect of hourly awakening on IOP rhythm.

167 To evaluate the effects of hourly awakening on sleep architecture, comparisons of sleep st

168 too short duration of sleep or early morning awakening), or a combination of the previous quantitativ

169 were applied either 3-10 s after spontaneous awakenings, or in established wakefulness (> 30 s).

170 ments of the number of hot flushes and night awakenings over time.

171 of night awakenings from pain, reported 101 awakenings owing to pain while taking placebo during the

172 rease in ischemia occurred immediately after awakening (P < .05), but activity-adjusted increases in

173 ndividuals for the sessions with and without awakening (P < .05).

174 sleep quality (p = 0.003), feeling rested on awakening (p = 0.007), daytime fatigue (p = 0.02), and f

175 m domain, rescue beta-agonist use, nocturnal awakenings, peak expiratory flow diurnal variability, an

176 tion: 44 +/- 6 versus 83 +/- 12 arousals and awakenings per hour (p = 0.02).

177 The number of awakenings per hour of sleep, latency to sleep onset, an

178 e fragmentation index (25 vs 23 arousals and awakenings per hr).

179 e dissipation of sleep inertia effects (post-awakening performance and alertness deficits) is effecte

180 s of regional brain activity across the post-awakening period [in particular, a waning negative corre

181 d every 10 min from healthy males during the awakening period or late afternoon using an automated bl

182 s, operating conditions, various measures of awakening, postoperative nausea and vomiting and dischar

183 epeated IOP measurements requiring nocturnal awakenings, potentially disturbing sleep macrostructure.

184 underpinnings of each of these facets of the awakening process.

185 After awakening, rabbits were followed up during 7 days.

186 en percent of eyes had symptoms of pain upon awakening refractory to conservative treatment.

187 f the cortisol profile, such as the cortisol awakening response and the area under the curve.

188 ted with a time-limited increase in cortisol awakening response and with a sustained improvement in S

189 ntrol were associated with a higher cortisol awakening response the next day, but morning awakening r

190 e Beck Depression Inventory and the cortisol awakening response were measured immediately before and

191 esidual depressive symptoms and the cortisol awakening response) in patients with recurrent depressio

192 s (wakeup level, diurnal slope, and cortisol awakening response) were predicted simultaneously from d

193 risk (Beck Depression Inventory and cortisol awakening response).

194 tionship between job strain and the cortisol awakening response.

195 awakening response the next day, but morning awakening responses did not predict experiences of these

196 cleus (CAUD) activity] suggest that the post-awakening reversal of sleep inertia effects may be media

197 n and 1.57 (95% CI, 1.28-1.93) for nocturnal awakenings; RRs for externalizing problems were 1.77 (95

198 Herein, a new strategy is outlined for awakening silent gene clusters using small molecule elic

199 ht, trouble getting back to sleep, and early awakenings), sleep duration (short sleep 5 hours or less

200 utcomes included sleep efficiency, number of awakenings, sleep quality, and total sleep time.

201 large brief cardio-respiratory activation at awakening suggest that a distinct, transiently aroused,

202 infancy and may be an instrumental factor in awakening the potential of group 14 chemistry for applic

203 and measured TMS-evoked EEG responses before awakening the subjects and asking them if they had been

204 After PDBu-induced awakening, these previously dormant terminals had a syna

205 osture-independent IOP pattern around normal awakening time is different in eyes with early glaucomat

206 Median awakening time was 4 minutes (2.2-5 min).

207 Around normal awakening time, the supine IOP increased in the glaucoma

208 bjects reported no conscious experience upon awakening, TMS evoked a larger negative deflection and a

209 We found time since entrained awakening to be the major predictor of peak performance

210 mitted helminths, the worldwide community is awakening to the importance of these infections.

211 Upon awakening, transgenic and control mice displayed a simil

212 ed wide variation in approach to spontaneous awakening trial performance and patient selection.

213 ality improvement collaborative, spontaneous awakening trial practice varies widely and concerns pers

214 t institutional characteristics, spontaneous awakening trial practice, attitudes and barriers regardi

215 tics and attitudes with reported spontaneous awakening trial use was evaluated using logistic regress

216 of respondents reported regular spontaneous awakening trial use, defined as greater than 75% of mech

217 sitively associated with regular spontaneous awakening trial use, whereas the perception that spontan

218 ficantly associated with regular spontaneous awakening trial use.

219 gatively associated with regular spontaneous awakening trial use.

220 Implementation of daily paired spontaneous awakening trials (daily sedation vacation plus spontaneo

221 reathe protocol that pairs daily spontaneous awakening trials (ie, interruption of sedatives) with da

222 thesized that daily, coordinated spontaneous awakening trials (SATs) and spontaneous breathing trials

223 Spontaneous awakening trials (SATs) improve outcomes in mechanically

224 of 1309) has a written policy on spontaneous awakening trials (SATs), but the minority of respondents

225 to assess a protocol that paired spontaneous awakening trials (SATs)-ie, daily interruption of sedati

226 the effect of concerns regarding spontaneous awakening trials and are associated with increased perfo

227 -6.43]), and the perception that spontaneous awakening trials are hard work (odds ratio, 0.53 [95% CI

228 trials, and the perception that spontaneous awakening trials are hard work were negatively associate

229 ons were less likely to perceive spontaneous awakening trials as hard work (odds ratio, 0.44 [95% CI,

230 s routinely in rounds and having spontaneous awakening trials as part of unit culture were positively

231 y ventilated patients undergoing spontaneous awakening trials each day.

232 d with routine implementation of spontaneous awakening trials in an ICU quality improvement collabora

233 use, whereas the perception that spontaneous awakening trials increased short-term adverse effects, s

234 I, 1.55-5.23]), incorporation of spontaneous awakening trials into unit culture (odds ratio, 3.36 [95

235 ents (44%, 446 of 1019) practice spontaneous awakening trials on more than half of ICU days.

236 attitudes and barriers regarding spontaneous awakening trials, and organizational cultural characteri

237 of attitudes and fears regarding spontaneous awakening trials, and organizational practices associate

238 adverse effects, staff fears of spontaneous awakening trials, and the perception that spontaneous aw

239 irium management strategies with spontaneous awakening trials, spontaneous breathing trials, and earl

240 tion, and delirium management to spontaneous awakening trials, spontaneous breathing trials, and ICU

241 The ABCDE bundle consists of spontaneous awakening trials, spontaneous breathing trials, coordina

242 y and concerns persist regarding spontaneous awakening trials.

243 operative) or deep sedation (patient asleep, awakening upon physical stimulation).

244 r event, and hypertension treatment time (on awakening versus at bedtime; per 1-SD elevation: hazard

245 jective measures of sleep latency, number of awakenings, wake time after sleep onset, total sleep tim

246 Awakening was usually before sunrise.

247 irst night, the baseline rate of spontaneous awakenings was determined by polysomnography.

248 tion, measured as the number of arousals and awakenings, was greater during pressure support than dur

249 ircadian phase and an interval elapsed since awakening were attributed to each data point, and circad

250 ivariate analysis, independent predictors of awakening were younger age (odds ratio [OR] = 1.039, 95%

251 needed medication, and number of nights with awakenings were similar between genotype groups.

252 remission) with severe hot flushes and night awakenings were treated with stellate-ganglion block at

253 f the PPT region during sleep leads to rapid awakening, whereas lesions of the PPT in cats reduce REM

254 d QTc variability reach a peak shortly after awakening, which may reflect increased autonomic instabi

255 placebo during the maintenance period and 32 awakenings while taking carbamazepine.

256 10.3 +/- 2.6 LSN and 2.6 +/- 0.6 hours from awakening with deficits.

257 naps, prolonged night-time sleep, difficult awakening with sleep drunkenness and prominent mood dist