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1 T and magnetic resonance (MR) imaging of the axial skeleton.
2 s patterning of the appendicular but not the axial skeleton.
3 l population, the sclerotome, that forms the axial skeleton.
4 exhibit posterior transformations along the axial skeleton.
5 lopment of the cranial base occurs after the axial skeleton.
6 led signaling pathways in development of the axial skeleton.
7 g pronounced homeotic transformations of the axial skeleton.
8 lopment of both the fin ray (dermal) and the axial skeleton.
9 hogenesis of structural birth defects of the axial skeleton.
10 linear regions throughout the somite-derived axial skeleton.
11 ions of Hox paralogous groups throughout the axial skeleton.
12 results in defects in the development of the axial skeleton.
13 boundaries in the sclerotome and developing axial skeleton.
14 eration of cartilage are not detected in the axial skeleton.
15 lformations in both the craniofacial and the axial skeleton.
16 keleton and frontal and lateral views of the axial skeleton.
17 control anterior/posterior patterning of the axial skeleton.
18 of bones and joints in the limbs, skull, and axial skeleton.
19 chord cells inhibited the development of the axial skeleton.
20 ts in high bone mass in the appendicular and axial skeleton.
21 required for developmental patterning of the axial skeleton.
22 in the calcification of vertebrae within the axial skeleton.
23 ctionally equivalent in the formation of the axial skeleton.
24 s in orchestrating normal development of the axial skeleton.
25 is essential for patterning of the limb and axial skeleton.
26 rior homeotic transformations throughout the axial skeleton.
27 nd the expansion of thoracic identity in the axial skeleton.
29 In newly diagnosed cases, MR surveys of the axial skeleton accurately demonstrate the extent of dise
30 Sd<-->+/+ chimeras with malformations of the axial skeleton also had kidney defects, whereas chimeras
31 hundred eighteen patients had tumors of the axial skeleton and 125 patients had limb primary tumors.
33 topic mineralization in the craniofacial and axial skeleton and encodes a loss-of-function allele of
35 wild-type function in the development of the axial skeleton and male reproductive tract, but served a
36 function in the development of the kidneys, axial skeleton and male reproductive tract, consistent w
38 ithelialization, also display defects in the axial skeleton and peripheral nerves that are consistent
39 cted homeotic transformations throughout the axial skeleton and posterior displacement of the hindlim
40 ur study population, (18)F-FLT uptake in the axial skeleton and proximal limbs assessed by SUVmax cor
41 terminants of bone loss and fractures in the axial skeleton and set the stage for subsequent developm
47 embryos exhibited abnormal patterning of the axial skeleton and spinal ganglia, phenotypes traced to
48 n the region of the somite fated to form the axial skeleton and tendons and is able to direct transcr
54 l femur or proximal humerus vs other limb vs axial skeleton); and presence of metastases (no vs yes o
55 ked osteotropism of MM, particularly for the axial skeleton, and for assessment of in vivo activity o
56 partment of somites, which gives rise to the axial skeleton, and from developing ribs, but were able
57 enchymal condensations of the fetal limb and axial skeleton, and in lateral plate mesoderm giving ris
58 ives of the somitic mesoderm, especially the axial skeleton, are severely disorganized in lunatic fri
59 issue-dependent, with trabecular bone in the axial skeleton being strongly dependent (>80% reduction
60 ortening and deformity of the long bones and axial skeleton but apparently normal tooth eruption and
65 ically in the spinal NT, both NT defects and axial skeleton defects were observed, but neither defect
66 nstrate that the tendons associated with the axial skeleton derive from a heretofore unappreciated, f
71 i.e., hallmarked by major involvement of the axial skeleton (e.g., spine, skull, and pelvis) and freq
73 nes of Prx1-Cre;KL(fl/fl) mice but not their axial skeleton failed to increase FGF23 expression as ob
76 The study of the early involvement of the axial skeleton has dominated the research map in spondyl
77 dy form, with its deregionalized pre-cloacal axial skeleton, has been explained as either homogenizat
79 system, cardiac venous pole, inner ear, and axial skeleton; homozygous null mutant animals die perin
81 Hox genes regulate regionalization of the axial skeleton in vertebrates, and changes in their expr
83 ony insertion of ligaments and tendons); the axial skeleton, including the sacroiliac joints; the lim
84 ts with liver metastases, are usually in the axial skeleton initially, and their detection changes ma
88 gh endochondral ossification of the limb and axial skeleton is relatively well-understood, the develo
89 een assumed that their development, like the axial skeleton, is dependent on Sonic hedgehog (Shh) and
92 various stages during the development of the axial skeleton may play a key role in testing mechanisms
93 aphy scan, liver MRI, bone scintigraphy, and axial skeleton MRI have been proven superior to 18F-FDG
95 PS1 is required for proper formation of the axial skeleton, normal neurogenesis, and neuronal surviv
102 bone marrow stromal cells, but not from the axial skeleton or hypothalamic neurons, using Prx1-Cre.
103 ox genes, which have been implicated in both axial skeleton patterning and hematopoietic development.
104 uency of involvement of the small joints and axial skeleton, poor response to aspirin and other nonst
105 the segmentation of the thoracic and lumbar axial skeleton (primary body formation), but are largely
106 rading assessment score of the uptake in the axial skeleton, proximal and distal limbs, liver, and sp
107 ele of Grem1 exhibit a dramatic reduction in axial skeleton relative to animals mutant for Nog alone.
111 for proper urogenital ridge differentiation, axial skeleton specification and limb patterning in mice
112 f fibrosis correlated with the SUVmax in the axial skeleton (spine and iliac crests) and proximal lim
113 e metastases can occur initially outside the axial skeleton, SRS is the recommended initial localizat
114 hat Shh acts early in the development of the axial skeleton, to induce a prochondrogenic response to
116 ular skeleton but not in cells that form the axial skeleton; we observed that bone properties were al
117 posteriorly directed transformations of the axial skeleton, which contrast with the anteriorly direc
122 ected homeotic transformation throughout the axial skeleton with associated alterations in Hox gene e
123 port the interpretation that elements of the axial skeleton with origins from distinct mesodermal tis
124 ression to cartilage including the limbs and axial skeleton, with similar localization specificity as
125 of notochord and have a predilection for the axial skeleton, with the most common sites being the sac
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