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   1 cesses such as LTP, LTD, spine motility, and axonal pathfinding.                                     
     2 o help direct olfactory sensory neuron (OSN) axonal pathfinding.                                     
     3 and that this antagonism is important during axonal pathfinding.                                     
     4 lls either to specify cell fate or to direct axonal pathfinding.                                     
     5 last proliferation, neuronal positioning and axonal pathfinding.                                     
     6  direct evidence that they are essential for axonal pathfinding.                                     
     7 otactin, a cell surface receptor involved in axonal pathfinding.                                     
     8 from proliferation to synaptic modulation to axonal pathfinding.                                     
     9 s that govern specification of cell fate and axonal pathfinding.                                     
    10 , and that the space cadet gene functions in axonal pathfinding.                                     
    11 e neuronal cell body and is not required for axonal pathfinding.                                     
    12 germinal center kinase family play a role in axonal pathfinding.                                     
    13 ility and that NMD acts locally to influence axonal pathfinding.                                     
  
    15  suggest that APP overexpression may perturb axonal pathfinding and circuit formation in developing D
  
    17    Null mutation of the GAP-43 gene disrupts axonal pathfinding and is generally lethal shortly after
    18 the repulsive effect of the Eph receptors in axonal pathfinding and may facilitate tumor cell invasio
  
    20 ion along the dorso-ventral axis but also in axonal pathfinding and organisation of the axonal scaffo
  
    22 s) help control neuronal fate determination, axonal pathfinding and synaptic communication and plasti
    23  in the expression of proteins essential for axonal pathfinding and/or neuronal differentiation in bo
    24 is known to promote neuronal survival, guide axonal pathfinding, and participate in activity-dependen
    25 les in vertebrate tissue boundary formation, axonal pathfinding, and stem cell regeneration by steeri
    26 , however, so it is unknown which aspects of axonal pathfinding are controlled by their activities.  
  
    28 the first to show that ADCY8 is required for axonal pathfinding before axons reach their targets.    
    29 vely studied for their role in dendritic and axonal pathfinding, but less is known about their potent
    30 ral vascular development but is required for axonal pathfinding by several populations of neurons in 
    31 ranscripts in vivo coincides temporally with axonal pathfinding by spinal neurons, supporting a role 
  
    33 gest that EphA8/Eek receptors play a role in axonal pathfinding during development of the mammalian n
    34 that morphogen gradients also serve to guide axonal pathfinding during development of the nervous sys
    35 em for studying the mechanisms that underlie axonal pathfinding during development, little is known a
  
  
  
    39 rmore, there is a long-lasting alteration in axonal pathfinding in secondary motoneurons that is not 
  
  
    42  communication during development, including axonal pathfinding in the nervous system and cell-cell i
  
  
    45 ith particularly well-characterized roles in axonal pathfinding, in the healing of damaged epithelia 
    46  as well as mutants with specific defects in axonal pathfinding, including exit from the spinal cord 
    47  and signaling through this isoform mediates axonal pathfinding, independent of the MuSK downstream c
    48  establish a previously unknown mechanism of axonal pathfinding involving vascular-derived endothelin
  
    50 ts of neuronal development such as embryonic axonal pathfinding, neuroblast proliferation in the larv
  
  
    53 d that embryonic exposure to nicotine alters axonal pathfinding of spinal secondary motoneurons in ze
    54  in the optic nerve and retina, and abnormal axonal pathfinding of the ganglion cell axons at the opt
  
    56 le abnormalities, suggesting that defects in axonal pathfinding or synapse formation may cause the ob
    57 ocesses during axonal development, including axonal pathfinding, orientation of axons in chemotactic 
  
  
  
  
    62 neration after injury depends on accuracy of axonal pathfinding, which is primarily believed to be in
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