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1 ompaction, which is associated with worsened axonal pathology.
2 tions in cerebral white matter structure and axonal pathology.
3 ervous system but with additional peripheral axonal pathology.
4  in CMT disease primarily associated with an axonal pathology.
5 ures consistent with both motor neuronal and axonal pathology.
6 rotein (PLP) gene by oligodendrocytes causes axonal pathology.
7 ppear to be caused by the reduction of acute axonal pathology.
8 of substantia nigra dopaminergic neurons and axonal pathology, a cardinal pathological hallmark of PD
9  results demonstrate a pattern of widespread axonal pathology after lateral FP brain injury in the ra
10 ce of morphologically detectable synaptic or axonal pathology and again displayed regional selectivit
11                                     Although axonal pathology and loss in MS has been recognized for
12                         Close to WM lesions, axonal pathology and microglial activation may explain s
13   We report that ZPR1-deficient mice exhibit axonal pathology and neurodegeneration.
14 arly and late AnkG mutants is accompanied by axonal pathology and neurological dysfunction.
15 f EAE, with decreased demyelination, reduced axonal pathology, and decreased central nervous system (
16          Tissues with more severe myelin and axonal pathology are associated with greater texture het
17 hagy in neurons and its role in neuronal and axonal pathology are not yet understood.
18 he result of primary FMRP-deficiency-related axonal pathology, as opposed to secondary connectional d
19 acrophages, which is usually associated with axonal pathologies, astrocytes selectively remove large
20                   This finding suggests that axonal pathology caused by CNS myelin defects may underl
21                    Lmx1a/b deficiency caused axonal pathology characterized by alpha-synuclein(+) inc
22 reased autophagy, shrinkage of soma size and axonal pathology even in the pons region.
23                                              Axonal pathology has been highlighted as a cause of neur
24                                Although many axonal pathologies have been described in AD, studies ha
25  may therefore represent one of the earliest axonal pathologies in SOD1(G93A) mice, which worsens at
26 s establishing a foundation for ameliorating axonal pathology in AD.
27 bute to neurodegenerative diseases linked to axonal pathology in adults.
28                                  We analyzed axonal pathology in archival brain biopsy and autopsy sa
29     Our findings point to a wider pattern of axonal pathology in bipolar disorder than previously tho
30 ediated axonal injury that can contribute to axonal pathology in MS.
31 best and most specific noninvasive marker of axonal pathology in multiple sclerosis.
32 amyotrophic lateral sclerosis (ALS), display axonal pathologies including abnormal accumulations of p
33                                              Axonal pathology is a major cause of neurological disabi
34                          We wondered whether axonal pathology is also an early pathological feature i
35                        Similarly, myelin and axonal pathology occurred with elevated ERK1/2 activity
36 al. report that the core motor phenotype and axonal pathology of HSPs are recapitulated in mice lacki
37 ociated with diminished dopamine release and axonal pathology of nigrostriatal dopaminergic projectio
38 e normal-appearing white matter reveal early axonal pathology outside inflammatory demyelinating lesi
39 d neurofilament compaction (NFC), two common axonal pathology processes involved in traumatic axonal
40 alpha-syn antibodies attenuated synaptic and axonal pathology, reduced the accumulation of CT-truncat
41 sociated with cavitation, necrosis and acute axonal pathology (spheroids), in both grey and white mat
42 gro-striatal axonal terminals leads to early axonal pathology, synaptic disruption, dysfunction of do
43      Many neurodegenerative diseases exhibit axonal pathology, transport defects, and aberrant phosph
44       Furthermore, RIPK1- and RIPK3-mediated axonal pathology was commonly observed in SOD1(G93A) tra
45                                              Axonal pathology was more common in the MS material than
46 oduced a concentration-dependent increase in axonal pathology within the striatum reflected by the am

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