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1 Schwann cells may suggest the potential for axonal regeneration.
2 plore factors that may differentially affect axonal regeneration.
3 ment of blood vessel growth might facilitate axonal regeneration.
4 peroxidasins as extracellular inhibitors of axonal regeneration.
5 dent synaptic plasticity, and injury-induced axonal regeneration.
6 ons caudal to the injury site as evidence of axonal regeneration.
7 s-induced neurodegeneration and insufficient axonal regeneration.
8 ium levels in a range that is permissive for axonal regeneration.
9 nd associated neurological disorders and for axonal regeneration.
10 l scar forms and becomes a major obstacle to axonal regeneration.
11 Thus, the level of LOTUS function titrates axonal regeneration.
12 l functions, including neurite outgrowth and axonal regeneration.
13 t options for motor neuron disease and motor axonal regeneration.
14 reports on the extent to which they support axonal regeneration.
15 glycoprotein (MAG) is a potent inhibitor of axonal regeneration.
16 NgR signaling limits functional recovery and axonal regeneration.
17 ant step for understanding the regulation of axonal regeneration.
18 that NFs may play a role in the mechanism of axonal regeneration.
19 ays a role in glial-neuronal interaction and axonal regeneration.
20 or manipulating its expression in studies of axonal regeneration.
21 ssential preparatory stage to the process of axonal regeneration.
22 insights and a few surprises in the field of axonal regeneration.
23 erotonergic pathways and corticospinal tract axonal regeneration.
24 les after CNS injury is believed to restrict axonal regeneration.
25 suppressive environment to one that promotes axonal regeneration.
26 morphological repair phenotype that promotes axonal regeneration.
27 nd we identify Collagen XII as a promoter of axonal regeneration.
28 its induction in spinal motor neurons during axonal regeneration.
29 se to CNS injury and limits the capacity for axonal regeneration.
30 s expressed at sites of SCI and may restrict axonal regeneration.
31 ho family small GTPase Rac1 and Rac1-induced axonal regeneration.
32 l cord of the immunosuppressed rat to induce axonal regeneration.
33 synaptic strength, activity, maturation, and axonal regeneration.
34 permissive nature of the adult CNS to induce axonal regeneration.
35 ins from Reticulon 1, 2 and 3 do not inhibit axonal regeneration.
36 microglia, which appear to be important for axonal regeneration.
37 sal columns of the rat spinal cord to induce axonal regeneration.
38 venting axonal degeneration and/or promoting axonal regeneration.
39 overexpressing neurotrophic factors enhance axonal regeneration.
40 ractions between them that lead to increased axonal regeneration.
41 NS), also support a modest degree of central axonal regeneration.
42 s (Muller and Mauthner cells) are capable of axonal regeneration.
43 MAG is a potent inhibitor of axonal regeneration.
44 for treatment of CNS conditions that require axonal regeneration.
45 generation without cell death is followed by axonal regeneration.
46 ebris, in Schwann cell proliferation, and in axonal regeneration.
47 well as a role for macrophages in promoting axonal regeneration.
48 st-injury period and during later peripheral axonal regeneration.
49 vation could have important consequences for axonal regeneration.
50 of mammalian glia and neurons and stimulates axonal regeneration.
51 cable formation, Schwann cell migration, and axonal regeneration.
52 ated' Schwann cells, which are essential for axonal regeneration.
53 th-cone guidance during both development and axonal regeneration.
54 cruitment, a reduction in myelin removal and axonal regeneration.
55 they identify promising agents for promoting axonal regeneration.
56 ntire set of mammalian genes contributing to axonal regeneration.
57 of MT-I/II after optic nerve crush promotes axonal regeneration.
58 , is driven by kinesin-1 and is required for axonal regeneration.
59 n that uncovered novel molecules suppressing axonal regeneration.
60 he adult due to the very limited capacity of axonal regeneration.
61 end themselves to the analysis of successful axonal regeneration.
62 creates an environment that is permissive to axonal regeneration.
63 tein, as a key component of GDA(BMP)-induced axonal regeneration.
64 tes the gene expression necessary to enhance axonal regeneration.
65 plays an essential role in GDA(BMP)-mediated axonal regeneration.
66 t facial motoneurons, Srf deletion inhibited axonal regeneration.
67 xpression of Smad2 also blocked SLPI-induced axonal regeneration.
68 sm, suggesting a cytoplasmic SRF function in axonal regeneration.
69 expressed in neural stem cells and controls axonal regeneration.
70 on at later time points, but no evidence for axonal regeneration.
71 e need for additional control and shaping of axonal regeneration.
72 ne expression signatures indicated attempted axonal regeneration, a metabolic switch to glycolysis, o
75 essentially involved in RGC degeneration or axonal regeneration after acute CNS injury.SIGNIFICANCE
78 euronal Nogo-66 receptor (NgR/NgR1) to limit axonal regeneration after central nervous system injury.
80 go-66 and NgR have central roles in limiting axonal regeneration after CNS injury, and NEP1-40 provid
87 in-derived proteins Nogo, MAG and OMgp limit axonal regeneration after injury of the spinal cord and
97 r BMP inhibition with Noggin, retarded early axonal regeneration after sciatic nerve crush injury.
99 One strategy that has emerged for promoting axonal regeneration after spinal cord injury is the impl
105 Retinal ganglion cell (RGC) death and failed axonal regeneration after trauma or disease, including g
106 PCAF is necessary for conditioning-dependent axonal regeneration and also singularly promotes regener
109 r provides a therapeutic basis for enhancing axonal regeneration and functional recovery after CNS in
110 ta confirm previous reports that ECs promote axonal regeneration and functional recovery after spinal
111 and NI250(Nogo), allows moderate degrees of axonal regeneration and functional recovery after spinal
112 entral nervous system (CNS) pose barriers to axonal regeneration and functional recovery following in
113 onditioning lesions do significantly enhance axonal regeneration and indicate that axotomy rather tha
114 nctionally separated from the role of Wnd in axonal regeneration and injury signaling by the requirem
115 actors in the lesion site, thereby promoting axonal regeneration and locomotor function recovery.
116 ed on Schwann cells that modulate peripheral axonal regeneration and myelination are also recognized
117 hibitory molecule (growth-IM) which inhibits axonal regeneration and neurite regrowth after neural in
118 rug and therapeutic nucleic acids to promote axonal regeneration and plasticity after spinal cord inj
119 al anisotropy have been suggested to reflect axonal regeneration and plasticity, but the direct histo
122 onse in humans are able to induce elongative axonal regeneration and remyelination and restore impuls
124 ccumulate in chronic CNS lesions and inhibit axonal regeneration and remyelination, making them signi
126 of c-Jun specifically in SCs caused impaired axonal regeneration and severely increased neuronal cell
127 SA) and the cell adhesion molecule L1 during axonal regeneration and sprouting after injury to the ad
128 teraction in the eye and spinal cord promote axonal regeneration and sprouting of the optic nerve aft
129 nd and NgR receptor in situations of limited axonal regeneration and support the hypothesis that this
133 FGF2 priming facilitated the DPCs to promote axonal regeneration and to improve locomotor function in
134 nsheathing cells (OECs) are known to enhance axonal regeneration and to produce myelin after transpla
135 debris from degenerated fibers, accelerated axonal regeneration, and earlier reinnervation of neurom
137 s for neurorehabilitation and for the use of axonal regeneration as a therapeutic approach to disorde
138 tation in mediating descending propriospinal axonal regeneration as well as optimizing such regenerat
142 fter spinal cord injury promotes significant axonal regeneration beyond the distal end of a PN bridge
143 distal graft interface allows for functional axonal regeneration by chronically injured neurons.
145 g spinal cord transection in larval lamprey, axonal regeneration by descending brain neurons, rather
146 rategies, whereas immune neuroprotection and axonal regeneration can be achieved by transfer of activ
147 well known growth cone protein that promotes axonal regeneration, can be induced in rat brain astrocy
148 proposed mechanisms include abnormalities of axonal regeneration, collateral sprouting and synaptic p
149 toskeleton components, metabolic enzymes and axonal regeneration enhancers, was increased in the cent
154 ly), but not single mutants, showed enhanced axonal regeneration following retro-orbital optic nerve
155 suggest a practical strategy for stimulating axonal regeneration following spinal cord injury.SIGNIFI
156 up) resulted in a significant enhancement of axonal regeneration for the total numbers of descending
157 rest for their potential capacity to support axonal regeneration, for example, after spinal cord inju
159 of the role of astrogliosis in inhibition of axonal regeneration has been challenged by recent findin
160 wn to mediate remyelination and to stimulate axonal regeneration in a number of in vivo rodent spinal
161 roduct, P0, has been implicated in promoting axonal regeneration in addition to its proposed structur
162 ere, we asked whether axonal degeneration or axonal regeneration in adult nerves might be affected by
163 noic acid receptor beta2 (RARbeta2) promoted axonal regeneration in adult sensory neurones located pe
164 ver, it is not known if RARbeta2 can promote axonal regeneration in cortical neurones of the CNS.
167 he hypothesis that RAGE suppresses effective axonal regeneration in superimposed acute peripheral ner
171 nts and neurotrophins were used to influence axonal regeneration in the adult rat after complete spin
174 s, such as Nogo, may account for the lack of axonal regeneration in the central nervous system (CNS)
177 mising donors for transplantation to promote axonal regeneration in the CNS including the spinal cord
181 ation and proliferation is a prerequisite to axonal regeneration in the injured peripheral nervous sy
183 secondary upregulation of NF message during axonal regeneration in the lamprey CNS may be part of an
185 ssion may be an effective means of promoting axonal regeneration in the presence of diverse inhibitor
190 ) of zebrafish is a suitable system to study axonal regeneration in vivo because of both the superfic
191 Further, daidzein was effective in promoting axonal regeneration in vivo in an optic nerve crush mode
196 ured rat spinal cord resulted in compromised axonal regeneration, indicating that POSTN plays an esse
197 and as long as 15 months, after SCI promote axonal regeneration into and beyond a midcervical lesion
200 2 or NGF, but not L1 or LacZ, induced robust axonal regeneration into normal as well as ectopic locat
202 both neuronal and neuronal-glial, results in axonal regeneration into the SC after dorsal root neurot
211 ercoming these inhibitory cues and promoting axonal regeneration is one of the primary targets in dev
213 After a CNS injury in the adult mammals, axonal regeneration is very limited because of the reduc
215 have implicated c-Jun in neuronal death and axonal regeneration, it is unknown whether Jun function
218 are overexpressed sufficiently to accelerate axonal regeneration, myelination and function are restor
220 g the 'nanoaxotomy' chip, we discovered that axonal regeneration occurs much faster than previously d
222 specific epigenetic mechanism that regulates axonal regeneration of CNS axons, suggesting novel targe
223 herefore, in spinal cord-transected lamprey, axonal regeneration of descending brain neurons probably
227 del studies: acute neuroprotection, enhanced axonal regeneration or plasticity, and treatment of demy
228 icative of a more inhibitory environment for axonal regeneration/plasticity, than MMP-9 null mice.
229 e examined whether their neuroprotective and axonal regeneration potentials can be identify in human
231 Despite the poor potential for mammalian axonal regeneration, primate astrocytes activated by Fgf
232 a cell body responses and enhance subsequent axonal regeneration, probably by reducing the initial de
233 These findings show that RARbeta2 induces axonal regeneration programs within injured neurons and
235 ount for this functional improvement include axonal regeneration, remyelination and neuroprotection.
236 after nerve injury is critical for favorable axonal regeneration, remyelination, and clinical improve
238 s suggest that the age-associated decline in axonal regeneration results from diminished Schwann cell
239 tomized adult rat sensory neurons to undergo axonal regeneration reveals new therapeutic targets to b
241 in Schwann cells during both development and axonal regeneration, suggesting that the developmental p
242 the mpz:egfp transgene was not dependent on axonal regeneration, suggesting that the primary signal
243 ar that changes in gene expression accompany axonal regeneration, the extent of this genomic response
244 he central nervous system as an inhibitor of axonal regeneration, the peripheral roles of Nogo isofor
245 ts into the mechanism by which Nogo inhibits axonal regeneration, this discovery may inspire new trea
246 pigenetic mechanisms that promote endogenous axonal regeneration, this provides possible avenues for
247 t dorsal root ganglion neurons for increased axonal regeneration through a neurotrophin-dependent mec
248 central nervous system (CNS) myelin prevents axonal regeneration through interaction with Nogo recept
249 evidence supports that Luman contributes to axonal regeneration through regulation of the unfolded p
251 ted axon growth to laminas I and II, shaping axonal regeneration toward the normal distribution patte
252 We examined blood vessel, Schwann cell and axonal regeneration using validated axotomy models to st
256 for LRP-mediated chemoattraction to mediate axonal regeneration was examined in vivo in a model of c
258 nerve injury in vivo and is associated with axonal regeneration, we have assayed axotomized adult ra
259 therapeutic potential of UPR manipulation to axonal regeneration, we locally delivered XBP1s or an sh
260 Changes in axonal localization of PKC and axonal regeneration were examined in cultured RGCs by im
261 into the area of the damaged nerve promoted axonal regeneration, which led to functional recovery as
263 fied in the peripheral nervous system during axonal regeneration, with overlap to axonal sprouting af
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