ライフサイエンスコーパス: axonin-1

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1 hat would have an asymmetric distribution of axonin-1.

2 phacan had no effect on its binding to TAG-1/axonin-1.

3 e shape in the related molecules hemolin and axonin-1.

4 s of 12 missense mutations on binding to L1, axonin-1 and F11 and shown for the first time that where

5 ther CAMs of the immunoglobulin superfamily, axonin-1 and F11.

6 Therefore, axonin-1 and NrCAM are crucial for the contact-mediated

7 Axonin-1 and NrCAM were previously shown to be involved

8 Thus, axonin-1 appears to become redistributed within the memb

9 ule (NgCAM), which binds heterophilically to axonin-1, appears uniformly distributed on even those ax

10 gand in axon growth by a mechanism involving axonin-1 as a neuronal receptor and suggest that dynamic

11 the central neurons expressed high levels of axonin-1 both in vitro and in vivo.

12 Although the levels of L1 and axonin-1, both of which bind L1, were markedly different

13 lting from treatment with antibodies against axonin-1, confirming that axonin-1/Nr-CAM interactions a

14 dsRNA targeting choline acetyltransferase or axonin-1, did not exhibit this phenotype.

15 f close axonal apposition are accompanied by axonin-1 expression, suggesting that such contacts are i

16 been implicated as a ligand for another CAM, axonin-1, in guidance of commissural axons across the fl

17 Moreover, antibodies against axonin-1 inhibited Nr-Fc-induced neurite outgrowth, indi

18 c-induced neurite outgrowth, indicating that axonin-1 is a neuronal receptor for Nr-CAM on these peri

19 les are typically partly labelled, such that axonin-1 is expressed on membranes apposing other labell

20 binding of neurocan and phosphacan to TAG-1/axonin-1 is therefore the opposite of that previously ob

21 hat N-CAM is a high-affinity ligand of TAG-1/axonin-1 (Kd approximately 1 nM), and specific binding o

22 Axonin-1,L1, BEN, and N-cadherin were expressed more abu

23 antibodies against axonin-1, confirming that axonin-1/Nr-CAM interactions are important for guidance

24 control conditions, perturbations of either axonin-1 or NrCAM interactions prevented the growth cone

25 Thus, the localization of axonin-1 strongly suggests that it plays an important ro

26 sed on the structure of the Ig1-4 domains of axonin-1, suggests that Glu-33 and Tyr-418 hydrogen-bond

27 in vitro studies indicating that the Nr-CAM-axonin-1/TAG-1 interaction is involved in peripheral axo

28 dase staining for the cell adhesion molecule axonin-1 to label sensory axons.

29 imately 1 nM), and specific binding of TAG-1/axonin-1 to tenascin-C was also observed (Kd approximate

30 showed an overlapping localization of TAG-1/axonin-1 with all four of these ligands, further support

31 s of the neural cell adhesion molecule TAG-1/axonin-1, with dissociation constants of 0.3 nM and 0.04

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