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1 th QT prolongation induced by dl-sotalol and azimilide.
2 was significantly prolonged in the combined azimilide 100 mg and 125 mg daily dose group compared wi
3 first symptomatic recurrence in the combined azimilide 100 mg and 125 mg dose groups with that in the
6 lation or atrial flutter received placebo or azimilide (35 to 125 mg) once daily for 6 or 9 months in
9 his study was to assess the effectiveness of azimilide, a class III antiarrhythmic drug, in reducing
12 oaches under active investigation, including azimilide and dronedarone, are compounds with multiple e
14 differences were observed between the 100-mg azimilide and placebo groups for all-cause mortality in
15 supportive (as has occurred with sotalol and azimilide), and patients who are to receive amiodarone.
18 cteristics, and risk factors associated with azimilide-associated torsades de pointes (TdP) ventricul
19 ction in asymptomatic atrial fibrillation on azimilide compared with placebo (P=0.03) when repeated o
22 this study was to assess the effect of oral azimilide dihydrochloride (AZ) 100 mg versus placebo on
28 7.96, p = 0.005); the hazard ratio (placebo: azimilide) for this comparison was 1.58 (95% confidence
29 00 cells/microL) were slightly higher in the azimilide group than in the placebo group (0.3% versus 0
30 l epithelium was: Ba2+ > clofilium >>> TEA = azimilide >>> trans-6-cyano-4-(N-ethylsulphonyl-N-methyl
34 multiple ionic channels (e.g., ibutilide and azimilide) in order to prolong atrial and ventricular ac
37 n (AF) in a subpopulation of patients in the Azimilide Postinfarct Survival Evaluation (ALIVE) trial.
39 ed to receive once daily doses of placebo or azimilide; recurrent symptomatic arrhythmias were docume
44 The TdP occurred in 56 patients assigned to azimilide, was dose-related, and tended to occur earlier
45 ical use soon, and it is currently reviewing azimilide (which seems to be devoid of frequency-depende
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