コーパス検索結果 (left1)
通し番号をクリックするとPubMedの該当ページを表示します
1 bFGF and collagen type I synthesis was also increased in
2 bFGF and VEGF monotherapy significantly increased surviv
3 bFGF and VEGF reversed the antiangiogenic activity of As
4 bFGF expression was analyzed in the LH-betaTag transgeni
5 bFGF induced membrane receptor cooperation between integ
6 bFGF levels significantly increased during tumorigenesis
7 bFGF stimulation of MMP-13 was mediated at the transcrip
8 bFGF treatment could not rescue PGC proliferation in the
9 bFGF was localized to vascular and tumor cells and rarel
10 bFGF- and EGF-induced beta1-integrin up-regulation and p
11 bFGF-loaded NP (bFGF-NP) were prepared with Poloxamer 18
12 bFGF-NP/UTMD combined treatment significantly enhanced t
13 bFGF/HS promoted the downregulation of intracellular ker
14 Stat3 signaling and overexpression of MMP-2, bFGF, and VEGF, as well as enhanced invasion and angioge
16 on strength by 68-91% from week 2 after AAV2-bFGF treatment and by 82-210% from week 3 after AAV2-VEG
19 ls of both p38 and JNK were diminished after bFGF stimulation of MT1-MMP knockout cells compared with
20 h factor protein levels were increased after bFGF-stimulation of wild-type fibroblast cells compared
21 these expansion states depended on ambient [bFGF], telencephalic developmental stage, and differenti
27 factor receptor 1-expressing BaF3 cells and bFGF-induced axonal branching in hippocampal cultures.
31 e RFA group, there was a decrease in HGF and bFGF expression at 24 and 72 hours (P = 0.001 and P = 0.
35 focal adhesion complexes on RGD- and RGD and bFGF-immobilized patterns as shown by immunostaining of
36 nti-carcinogenic effect by reducing VEGF and bFGF serum levels and by blocking flk-1 receptors, there
40 suggest that galectin-3 modulates VEGF- and bFGF-mediated angiogenesis by binding via its carbohydra
41 minant-negative galectin-3, reduce VEGF- and bFGF-mediated angiogenesis in vitro and that VEGF- and b
43 ted angiogenesis in vitro and that VEGF- and bFGF-mediated angiogenic response is reduced in galectin
44 encoding the kinase PERK decreased VEGFA and bFGF expression, but neither gene was affected by the in
47 F can mobilize proangiogenic factors such as bFGF from their depot or storage sites on bovine corneal
49 potentiated the activity of amphotericin B. bFGF-containing regimens were associated with reduced ti
50 promised by minimal exposure to bFGF because bFGF reduces responsiveness to bone morphogenetic protei
51 stic insights into the cooperativity between bFGF and alphavbeta3 integrin during angiogenic signalin
54 icient to facilitate extinction, as blocking bFGF and returning rats to their home cage had no effect
65 ctivated corneal stromal cells is induced by bFGF/HS and by TGF-beta1, and it accompanies the downreg
66 ion of multiple MAPKs (ERK, p38, and JNK) by bFGF, and more importantly, bFGF activation of protein k
68 The stimulation of mesodermal precursors by bFGF and activin A switches on very rapidly the hematopo
71 survival factor for neural precursor cells, bFGF was evaluated as a growth and chemoresistance facto
72 ls were cultured in growth medium containing bFGF (20 ng/ml), over-expression of CXCR4 significantly
73 echnique is an effective strategy to deliver bFGF to the heart, and the resulting growth factor thera
78 uced 3 weeks after implantation of high-dose bFGF resulted in a lymphatic vessel-dominant phenotype.
79 GFR-2 or VEGFR-3 RESULTS: Although high-dose bFGF stimulation induced a more potent angiogenic respon
80 F-NP showed good round morphology, efficient bFGF encapsulation and stable bioactivity of bFGF in vit
81 wild-type mouse corneas compared with either bFGF pellet implantation or naked MT1-MMP DNA-injected c
82 h induces EphB receptor activation, enhanced bFGF-induced tube formation in an in vitro aortic ring a
87 d to migrate by migration stimulatory factor bFGF, active Rac1 and cdc42 small GTPase levels were dec
88 vations that basic fibroblast growth factor (bFGF or FGF-2) required binding to a cell-surface hepari
89 ors, such as basic fibroblast growth factor (bFGF or FGF2), are necessary for neuronal survival, grow
90 mulated with basic fibroblast growth factor (bFGF) and 12-O-tetradecanoyl phorbol-13-acetate (TPA).
91 cultured in basic fibroblast growth factor (bFGF) and activin A develop as epiblast-like cells (EpiL
92 r A (VEGFA), basic fibroblast growth factor (bFGF) and angiogenin (ANG) in human kidney epithelial ce
93 stem cells, basic fibroblast growth factor (bFGF) and epidermal growth factor (EGF) promote cell pro
96 or detecting basic fibroblast growth factor (bFGF) and prostate-specific antigen (PSA) in solution we
97 tors such as basic fibroblast growth factor (bFGF) and vascular endothelial cell growth factor (VEGF)
98 e effects of basic fibroblast growth factor (bFGF) and vascular endothelial growth factor (VEGF) on s
99 Binding of basic fibroblast growth factor (bFGF) and vascular endothelial growth factor (VEGF) to t
100 matrix-bound basic fibroblast growth factor (bFGF) as well as inhibited the mitogenic activity of bot
101 e release of basic fibroblast growth factor (bFGF) during loading and/or injury of the cartilage matr
102 tain whether basic fibroblast growth factor (bFGF) enhances axonal branching through alterations in p
103 or (HGF) and basic fibroblast growth factor (bFGF) expression was measured at selected time intervals
104 antly, while basic fibroblast growth factor (bFGF) expression, a known inhibitor of transforming grow
105 ithdrawal of basic fibroblast growth factor (bFGF) from the culture induces neural differentiation of
106 tested, the basic fibroblast growth factor (bFGF) had the most significant effect in promoting the m
108 (IGF-1) and basic fibroblast growth factor (bFGF) have been shown to protect against radiation-induc
110 s exposed to basic fibroblast growth factor (bFGF) in vitro generated four stereotypical clonal expan
114 HB-EGF, and basic fibroblast growth factor (bFGF) messenger RNA was assessed by reverse-transcriptio
116 imulation by basic fibroblast growth factor (bFGF) or vascular endothelial growth factor results in c
117 r (CNTF) and basic fibroblast growth factor (bFGF) production in Muller cells, which may enhance phot
118 oliferation, basic fibroblast growth factor (bFGF) release, collagen type I synthesis, and wound heal
120 or (EGF) and basic fibroblast growth factor (bFGF) to generate neuralized spheres containing primitiv
121 showed that basic fibroblast growth factor (bFGF) was downregulated in MDA-MB-231-injected tibiae fr
124 k embryos by basic fibroblast growth factor (bFGF), and areas of neovascularization were measured.
126 owth factor, basic fibroblast growth factor (bFGF), and platelet-derived growth factor (PDGF) in NIH-
127 e-2 (MMP-2), basic fibroblast growth factor (bFGF), and vascular endothelial growth factor (VEGF) and
128 ogether with basic fibroblast growth factor (bFGF), elicited subsequent neural lineage differentiatio
129 (VEGF) and basic fibroblasts growth factor (bFGF), indicating an intrinsic defect in endothelial cel
131 or (EGF) and basic fibroblast growth factor (bFGF), suggesting that PS1 dominates EFG and bFGF signal
132 r (VEGF) and basic fibroblast growth factor (bFGF), which in turn, inhibits tumor growth and metastas
133 nhibitors on basic fibroblast growth factor (bFGF)- and vascular endothelial growth factor (VEGF)-med
134 (MT1-MMP) on basic fibroblast growth factor (bFGF)-induced corneal neovascularization in vivo and in
137 mplicated in basic fibroblast growth factor (bFGF)-regulated angiogenesis through incompletely unders
144 in 4 (BMP4), basic fibroblast growth factor (bFGF, also known as FGF2), vascular endothelial growth f
145 se in plasma basic fibroblast growth factor (bFGF; P = .04) and increase in plasma interleukin-18 (IL
146 roteins (eg, basic fibroblast growth factor [bFGF] but not vascular endothelial growth factor [VEGF]
147 rs (VEGF and basic fibroblast growth factor [bFGF]) play a role in promoting angiogenesis during SK a
148 eukin-8, and basic fibroblast growth factor [bFGF]) were determined before and during treatment.
149 aling pathways engaged by angiogenic factors bFGF and VEGF in tumor angiogenesis are not fully unders
151 , including basic fibroblast growth factors (bFGF or FGF-2), transforming growth factor-beta (TGF-bet
153 blast growth factor (FGF) family, basic FGF (bFGF) and FGF-18, have been implicated in the regulation
161 p38, and JNK) by bFGF, and more importantly, bFGF activation of protein kinase C (PKC) delta played a
162 al therapy was able to significantly improve bFGF accumulation in brain tissues (p<0.05) including th
164 cretion levels and a significant increase in bFGF were observed following light exposure, compared to
165 actor activity with significant increases in bFGF or VEGF from weeks 4 to 6 in the treated tendons (p
169 ling rate of PDLFs at 12 hours and increased bFGF and collagen type I release from GFs and PDLFs at 2
173 elivery of EMD478761 significantly inhibited bFGF-induced angiogenesis in CAM, as determined by a red
175 stion were plated in serum-free or insulin-, bFGF/heparin sulfate (HS)-, TGF-beta1-, or fetal bovine
179 ies, exogenous low-molecular-weight (18 kDa) bFGF (1 ng) significantly enhanced carboplatin-induced a
180 y 42 by switching EGF-containing SHEM to LIF/bFGF-containing MESCM through transient activation of LI
182 Abl as a key factor differentially mediating bFGF- and VEGF-induced angiogenesis in microvascular end
184 es the very efficient formation of mesoderm; bFGF and activin A induce the differentiation of these m
185 Results show TFP (1 ng/mL TGF-beta1, 5 ng/mL bFGF, 10 ng/mL PDGF) supplementation of serum-free chond
186 support the intranasal use of nanoliposomal bFGF as an efficient, non-invasive means to bypass the b
189 ained 3 weeks after implantation of an 80-ng bFGF micropellet without supplementary modulating agents
192 nt with this, Sema3A disrupted VEGF- but not bFGF-mediated endothelial cell signaling to FAK and Src,
195 ogical role may be related to the ability of bFGF to decrease proteoglycan synthesis and to antagoniz
196 eview, we examined the biological actions of bFGF and FGF-18 in articular and IVD cartilage, the spec
197 ay interfere with the biological activity of bFGF and potentially of other heparin-binding growth fac
198 ndothelial cell growth-promoting activity of bFGF yet had remarkably increased stability (activity su
200 AV2) vector to produce supernormal amount of bFGF or VEGF intrinsically in the tendon, we effectively
201 RB tumors produce significant amounts of bFGF, and the differential production and response to is
202 or obstacle to the widespread application of bFGF is its inherent instability during storage and deli
203 not interfere with high-affinity binding of bFGF to FGFR1 IIIc but can replace heparin as a required
206 gnificantly increased after a combination of bFGF pellet implantation and naked MT1-MMP DNA injection
209 The cellular uptake and cytotoxicity of bFGF-NP were evaluated with primary cultures of the left
216 ent significantly enhanced the efficiency of bFGF cellular uptake (P<0.05) without obvious cytotoxici
217 s resulted in the differential inhibition of bFGF-induced (5%-57%) and VEGF-induced (3%-66%) corneal
218 ntial production and response to isoforms of bFGF may have implications for invasive tumor growth and
219 , but whether drug-induced overexpression of bFGF in this region affects extinction of drug seeking i
220 ggest that cocaine-induced overexpression of bFGF inhibits extinction, as blocking bFGF during extinc
224 s, our findings revealed a neglected role of bFGF in sustaining self-renewal of human PS cells: preve
228 ression of colonic APN overlaps with that of bFGF and HB-EGF, which play a protective role in colitis
229 was also found to increase ubiquitination of bFGF and trypsin-like activity of the 20S proteasome, th
230 portant implications for the clinical use of bFGF and for the stabilization of heparin-binding growth
231 studies suggest the potential usefulness of bFGF and FGFR1 antagonists, as well as FGF-18 and FGFR3
233 27 also showed a strong inhibitory effect on bFGF, PDGF-BB, and serum-induced cell migration and prol
240 were recruited into collagen gel by SDF1 or bFGF than without cytokines in 7 days, whereas BMP7 had
242 ese results suggest that MT1-MMP potentiates bFGF-induced corneal neovascularization, likely by modul
244 n an in vitro aortic ring assay and promoted bFGF-induced corneal angiogenesis in vivo in a corneal p
246 and intravenous injection of the recombinant bFGF to LysM(Cre)/Tgfbr2 KO mice rescued the inhibited m
253 owever, we found that IGF2 cannot substitute bFGF in the TeSR1-supported culture, although endogenous
254 effects of expansion medium supplementation (bFGF, TFP, FBS) and self-assembled construct seeding den
256 nhibitors were more effective at suppressing bFGF-induced angiogenesis than VEGF-induced angiogenesis
258 chemotherapeutic agent doxorubicin, and that bFGF, but not VEGF, neutralizes the death-promoting acti
261 Collectively, these results indicate that bFGF enhances axonal branch formation by augmenting the
264 ly with monomeric JAM-A, which suggests that bFGF induces signaling by triggering JAM-A dimerization.
269 g shows that HKa significantly decreases the bFGF-transactivated phosphorylation of EGFR at Tyr 1173
271 on is a principal rate-limiting event in the bFGF-dependent stimulation of MMP-13 in human adult arti
276 severely compromised by minimal exposure to bFGF because bFGF reduces responsiveness to bone morphog
277 with alphavbeta3 integrin, which responds to bFGF stimulation by JAM-A release to regulate mitogen-ac
278 type c-Abl sensitized angiogenic response to bFGF, but kinase dead mutant c-Abl abolished this activi
281 ression of several angiogenic factors (VEGF, bFGF, IL-8, and ANGPTL-2), as well as ErbB (amphiregulin
282 signaling, p38 MAPK was important for VEGF, bFGF, EGF, IL-6, and other proangiogenic cytokine secret
284 eration rate, as well as expression of VEGF, bFGF, and SDF-1, which was not seen when TGF-beta1 expre
285 ent causing a significant decrease in VEGF-, bFGF-, EGF-, and IL-6-induced endothelial cord formation
287 the UPR is activated in parallel with VEGFA, bFGF, and ANG expression and independently of HIF-1alpha
289 a two-pronged biological mechanism by which bFGF controls the production of catabolic enzymes that a
291 rt a novel molecular mechanism through which bFGF or EGF promotes the proliferation of mouse neuroepi
292 t of malignant or transformed JB6 cells with bFGF is associated with a transient nuclear translocatio
293 wth: treatment of medulloblastoma cells with bFGF prevents them from forming tumors following transpl
296 eatment of cultured hippocampal neurons with bFGF heightens expression of both katanin and spastin, w
297 o cooperate during angiogenic signaling with bFGF and vascular endothelial growth factor (VEGF), resp
298 g experiments, axons of neurons treated with bFGF displayed greater numbers of dynamic free ends of m
301 growth of human embryonic stem cells without bFGF or TGFbeta/Activin/Nodal ligand supplementation.
WebLSDに未収録の専門用語(用法)は "新規対訳" から投稿できます。