ライフサイエンスコーパス: bacillary

1 cular proliferative tissue response known as bacillary angiomatosis (BA) and bacillary peliosis (BP)

2 Bacillary angiomatosis (BA), one of the many clinical ma

3 Bacillary angiomatosis and bacillary peliosis are vascul

4 al serious sequelae of infections, including bacillary angiomatosis and bacillary peliosis.

5 Bartonella henselae, the causative agent of bacillary angiomatosis and cat scratch disease, also rec

6 trench fever and a cause of endocarditis and bacillary angiomatosis in humans, has the highest report

7 fever, chronic bacteremia, endocarditis, and bacillary angiomatosis in humans.

8 A case of bacillary angiomatosis infection presenting as a skin no

9 The bacillary angiomatosis responded to 6 months of therapy

10 cterium associated with cat scratch disease, bacillary angiomatosis, and bacillary peliosis.

11 onella henselae, the agents of trench fever, bacillary angiomatosis, and parenchymal peliosis, and in

12 ases such as trench fever, endocarditis, and bacillary angiomatosis, B. quintana must survive and rep

13 duals with B. hensalae infection may develop bacillary angiomatosis, bacillary peliosis, and relapsin

14 y life threatening to the patient, including bacillary angiomatosis, bacillary peliosis, and verruga

15 e and B. quintana, have been associated with bacillary angiomatosis, but culture and speciation are d

16 istology of splenic biopsy was suggestive of bacillary angiomatosis, but immunohistochemistry ruled o

17 nt of trench fever and an etiologic agent of bacillary angiomatosis, has an extraordinarily high hemi

18 vidual, B. henselae-induced angiogenesis, or bacillary angiomatosis, is characterized by vascular pro

19 serious human infections globally, including bacillary angiomatosis, Oroya fever, trench fever, and e

20 limaea) henselae causes cat-scratch disease, bacillary angiomatosis, peliosis hepatis, and fever in h

21 exposures was administered to patients with bacillary angiomatosis-peliosis and to 96 matched contro

22 Of the 49 patients with bacillary angiomatosis-peliosis, 26 (53 percent) were in

23 ae and B. quintana, the organisms that cause bacillary angiomatosis-peliosis, are associated with dif

24 and who had clinical lesions consistent with bacillary angiomatosis-peliosis.

25 ifestations include conjunctival and retinal bacillary angiomatosis.

26 al pathogen, causing cat scratch disease and bacillary angiomatosis.

27 roliferative tumors, a disease process named bacillary angiomatosis.

28 cat scratch disease, bacillary peliosis, and bacillary angiomatosis.

29 to cause endocarditis, peliosis hepatis, and bacillary angiomatosis.

30 rculosis is not invariable and may depend on bacillary as well as host factors.

31 with unique biological adaptations, namely, bacillary band and stichosome, found only in whipworms a

32 l features, including stichocytes and paired bacillary bands.

33 proficient littermates resulted in increased bacillary burden and excessive pulmonary inflammation ch

34 tection results increased progressively with bacillary burden and immunosuppression.

35 e time of BCG vaccination also increased the bacillary burden and reduced T cell responses after chal

36 aracterized by a moderately increased tissue bacillary burden and severe pulmonic histopathological d

37 erferon (IFN) gamma responses in controlling bacillary burden in human immunodeficiency virus (HIV)-a

38 ose in well-nourished controls, although the bacillary burden in the malnourished animals continued t

39 serodiagnostic proteins for the spectrum of bacillary burden in tuberculosis.

40 ole-cell lysate were associated with reduced bacillary burden on sputum smear grade, days to culture

41 relations between human responses and sputum bacillary burden were assessed by quantile and hurdle re

42 hich may provide a dynamic measure of sputum bacillary burden when used longitudinally.

43 We observed decreasing bacillary burden with increasing level of immunosuppress

44 Children differ from adults in bacillary burden, spectrum of disease, the metabolism an

45 lished disease, is associated with very high bacillary burden.

46 ens are associated with reduced tuberculosis bacillary burden.

47 DRplus is directly related to the specimen's bacillary burden.

48 and reactive protein numbers increased with bacillary burden.

49 atients, and (iii) responses correlated with bacillary burden.

50 IL-2 did not enhance bacillary clearance or improvement in symptoms in human

51 enerally by an inability to achieve complete bacillary clearance.

52 rowth, differential macrophage viability, or bacillary clumping.

53 In addition, bacillary control was associated with down-regulation of

54 vity was accompanied by a small reduction in bacillary counts, but this did not affect modeling of ba

55 SS to invade human intestinal cells to cause bacillary dysentery (shigellosis) that is responsible fo

56 Shigella flexneri causes a severe form of bacillary dysentery also known as shigellosis.

57 for understanding the induction mechanism of bacillary dysentery and for evaluating Shigella vaccine

58 ome serotypes of Escherichia coli that cause bacillary dysentery and hemorrhagic colitis, respectivel

59 nge with virulent S. flexneri 2a can provoke bacillary dysentery and severe pathogenesis in adult mic

60 n and nonhuman primates, Shigella spp. cause bacillary dysentery by invading colon epithelium and pro

61 Shigella flexneri causes bacillary dysentery in humans by invading epithelial cel

62 Shigella species cause bacillary dysentery in humans by invasion, intracellular

63 eri is a gram-negative bacterium that causes bacillary dysentery in humans that is characterized by a

64 lla dysenteriae serotype 1, a major cause of bacillary dysentery in humans, can use heme as a source

65 f Shigella dysenteriae, a causative agent of bacillary dysentery in humans.

66 Shigella flexneri causes bacillary dysentery with symptoms resulting from the inf

67 Shigella spp. are the major cause of bacillary dysentery worldwide.

68 Shigella spp. are the major cause of bacillary dysentery worldwide.

69 Shigellosis (previously bacillary dysentery) was the primary diarrhoeal disease

70 Shigellae cause bacillary dysentery, a bloody form of diarrhoea that aff

71 Shigella spp. are causative agents of bacillary dysentery, a human illness with high global mo

72 xneri is responsible for the endemic form of bacillary dysentery, an acute rectocolitis in humans.

73 Shigella spp. cause shigellosis, also called bacillary dysentery, and invade colonic epithelial cells

74 Shigella flexneri, an etiological agent of bacillary dysentery, causes apoptosis in vitro.

75 esponsible for diseases such as diarrhea and bacillary dysentery, commonly afflicting infants and chi

76 mple, Shigella spp., the causative agents of bacillary dysentery, differ from the closely related com

77 Shigella flexneri, the causative agent of bacillary dysentery, injects invasin proteins through a

78 Shigella, the etiological agent of bacillary dysentery, rapidly kills human monocyte-derive

79 immunity to Shigella, the causative agent of bacillary dysentery, requires several episodes of infect

80 gella flexneri, the causative agent of human bacillary dysentery, switches off host sumoylation durin

81 Shigella, the leading cause of bacillary dysentery, uses a type III secretion system (T

82 a bacterial pathogen and causative agent of bacillary dysentery.

83 de of Shigella flexneri 2a, a major cause of bacillary dysentery.

84 ic pathogen that is the predominant cause of bacillary dysentery.

85 at invades the colonic epithelium and causes bacillary dysentery.

86 cultative intracellular organism that causes bacillary dysentery.

87 brook agar are used to calculate the rate of bacillary elimination from sputum collected from patient

88 y associated with faster sterilization phase bacillary elimination from the SSCC model (odds ratio [O

89 In Malawi, we modeled bacillary elimination rates (BERs) from sputum cultures

90 counts, but this did not affect modeling of bacillary elimination.

91 d abundant intra- and extracellular bacilli, bacillary fragments, and granular antigen-staining in me

92 owever, RPE cells are better able to control bacillary growth and RPE cell survival is greater than t

93 and found this gene to be necessary for full bacillary growth and survival.

94 alpha (TNF-alpha) is required for control of bacillary growth and the protective granulomatous respon

95 h1-mediated cellular immunity and control of bacillary growth at one pole to poor Ag-specific T cell

96 2O2, catalase and peroxidase activities, and bacillary growth rates measured both intracellularly in

97 gulatory and metabolic pathways required for bacillary growth restriction and reactivation.

98 y third day for 15 days, but weight loss and bacillary growth resumed when KGF was withdrawn.

99 ice were infected with low doses of BCG-TNF, bacillary growth was controlled, granulomas were small a

100 oth-based method with microscopic reading of bacillary growth, the microscopic observation drug susce

101 luminal phagocytes to remain permissive for bacillary growth.

102 lippine cohort comprising patients with high bacillary indices (BI; average:4,9), 94%(n = 161) of MB

103 <.0001), antibiotic-resistant Gram-negative bacillary infections (2.5 infections/100 admissions vs.

104 Accuracy was assessed with 158 Gram-negative bacillary isolates, including 134 carbapenemase producer

105 inistration of oral chemotherapy resulted in bacillary killing.

106 -secreting murine TNF-alpha (BCG-TNF) led to bacillary killing.

107 her among participants who had higher sputum bacillary load (P < .01).

108 bDeltambtE exhibited a significantly reduced bacillary load and histopathological damage in the organ

109 r Ag-specific T cell immunity with extensive bacillary load and Th2 cytokine-expressing lesions at th

110 m tuberculosis (1:1) also did not reduce the bacillary load but caused increased expression of tumor

111 (BCG-vector) at a low dose led to increased bacillary load in all organs and an extensive granulomat

112 After 2 months, the bacillary load in lungs was reduced from 9.74 log10 at b

113 This group had a significantly lower sputum bacillary load relative to correctly classified smear-po

114 and number of lung lesions, decreases in the bacillary load, and improvements in survival, compared w

115 ed host-protective immune responses, reduced bacillary load, and increased survival compared with ani

116 n, severe meningeal inflammation, persistent bacillary load, and progressive clinical deterioration.

117 TNF neutralization led to increased lung bacillary load, disrupted granuloma architecture with ex

118 mined by immunohistochemistry; intracellular bacillary load, following TLR2 and TLR4 blockade.

119 cavitary TB is associated with higher sputum bacillary load, our findings support the hypothesis that

120 ha-induced inflammation without reducing the bacillary load.

121 accelerated mortality without affecting the bacillary load.

122 A is a good method for measuring the initial bacillary load.

123 tissue granulomatous reaction, and increased bacillary load.

124 e to culture positivity was used to estimate bacillary load.

125 impact of appropriate treatment on cough and bacillary load.

126 ophagy and in the reduction of intracellular bacillary load.

127 The treatment resulted in increased lung bacillary loads and even further reduced survival.

128 ive because it improved survival and reduced bacillary loads in spleen whereas clarithromycin and ami

129 nfection with HN878 or W4 resulted in higher bacillary loads in the cerebrospinal fluid and brain, in

130 +) T cells are generated in response to high bacillary loads occurring during tuberculosis.

131 IFNAR1), displayed marked elevations in lung bacillary loads, accompanied by widespread pulmonary nec

132 c CD8(+) T cells develop in response to high bacillary loads, as occurs during tuberculosis, and are

133 cin prevented death but had little impact on bacillary loads.

134 d murine macrophages with high intracellular bacillary loads.

135 commended for the treatment of gram-negative bacillary meningitis and neonatal meningitis.

136 a is the only species of human origin with a bacillary morphology.

137 characterization of eight strains of another bacillary Neisseria species from human infections.

138 anulomas, the two cell types colocalized and bacillary numbers were substantially lower, suggesting t

139 nse known as bacillary angiomatosis (BA) and bacillary peliosis (BP) in some human hosts.

140 Bacillary angiomatosis and bacillary peliosis are vascular proliferative manifestat

141 Herein, we report the first case of bacillary peliosis hepatis due to systemic Bartonella he

142 Bacillary peliosis hepatis is an uncommon but well recog

143 revious observations in cat scratch disease, bacillary peliosis, and bacillary angiomatosis.

144 nfection may develop bacillary angiomatosis, bacillary peliosis, and relapsing bacteremia with fever

145 e patient, including bacillary angiomatosis, bacillary peliosis, and verruga peruana.

146 ctions, including bacillary angiomatosis and bacillary peliosis.

147 l-lined cystic disease in the liver known as bacillary peliosis.

148 scratch disease, bacillary angiomatosis, and bacillary peliosis.

149 d understanding of the heterogeneity in both bacillary physiology and host immune response that poten

150 Gram-negative bacillary pneumonia is a common and serious illness asso

151 owth ceases because of the depletion of this bacillary population needs to be modified.

152 defined as variabilities in human behavior, bacillary properties, and host physiology that fuel the

153 In contrast, bacillary replication was controlled in live phagocytes.

154 tress protein believed to be involved in the bacillary response to adverse conditions and in non-repl

155 Five weeks after infection with either bacillary strain, the inbred rabbits had significantly l

156 e MTB gene regX3 appears to be essential for bacillary survival during phosphate limitation and in ma

157 , we show here that sigF is not required for bacillary survival under nutrient starvation conditions

158 ient in phosphate-related genes, we assessed bacillary survival under phosphate-limited conditions an

159 peroxides) may be an important mechanism of bacillary survival within the host phagocyte.

160 mpact of aerobic glycolysis on intracellular bacillary survival, demonstrating that infection-induced

161 lammatory IL-10, and increased intracellular bacillary survival.

162 After the inhalation of 220 to 880 bacillary units, all of the rabbits were overtly well un

163 After the inhalation of 3,900 to 5,800 bacillary units, half of the rabbits died of progressive

164 e stages of the IBC pathway, filamentous and bacillary UPEC detach from the biofilm-like IBC, fluxing

165 lls prior to infection resulted in decreased bacillary viability, presumably due to extracellular kil

166 lular CatG expression, resulted in increased bacillary viability.