1 use of the larger size of the RFOs, prevents back diffusion.

2 ) were calculated and corrected for NO axial back-diffusion.

3 opose reflects amplification of nitric oxide back-diffusion, an increase likely associated with a pre

4 tion of the oxidant delivered likely lost by back-diffusion and not involved in hydrocarbon destructi

5 lume recombination collection efficiency and back diffusion collection efficiency for continuous and

6 hambers considering volume recombination and back diffusion effects; the affecting parameters were st

7 ectrolysis were found to be a result of NaCl back diffusion from the outer side of the membrane due t

8 bility systems to assess the significance of back diffusion from thin layers.

9  the loss of a defined plateau region and by back-diffusion from the bottom of the cell.

10 ell intracellular pH (pHi), injury, and acid back-diffusion in response to a luminal acid challenge i

11 ring contributions of, volume recombination, back-diffusion loss and initial recombination, the later

12 k matrices as well as the rate and extent of back-diffusion may be substantially mitigated in rocks t

13  cell surface, allowing for cell injury from back diffusion of gastric acid.

14 retardation solutes have a greater long-term back diffusion risk.

15 reduced in DeltaF/DeltaF mice, although acid back-diffusion was similar to controls.

16               Bicarbonate secretion and acid back-diffusion were measured in a perfused duodenal loop

17 olecules in this field is counterbalanced by back-diffusion within seconds.