ライフサイエンスコーパス: back mutation

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1 tions for large mutation rates and including back mutations.

2 ecise for small mutation rates and excluding back mutations.

3 n colonies of a given size in the absence of back-mutations.

4 Both back mutation and compensatory mutations contributed to

5 in models of interference among forward and back mutations at large numbers of sites on a nonrecombi

6 oquine susceptibility is not the result of a back mutation in a formerly resistant parasite or a new

7 A V144M back mutation in SCaM-1 significantly restored its abili

8 -term dynamics of mutants in the presence of back-mutations is also addressed.

9 By sequence analysis and back mutation, it was determined that the observed antag

10 which all characters are binary and in which back-mutations occur only at hybrid vertices.

11 nd binding affinity to parental MMGZ01 after back mutation of 12 canonical murine residues in the FRs

12 case with primary platinum resistance showed back mutation of BRCA1 in the primary tumor and showed a

13 Fitness recovery did not entail back mutations or compensatory mutations, but instead, w

14 Revertant mosaicism due to true back mutations or second-site mutations has been identif

15 genotypic reversion, most likely because of back mutation, originated in a lymphohematopoietic stem

16 In particular, with equal forward- and back-mutation rates, if division-controlled and a death-

17 merged due to continued evolution of pol and back mutation rather than through emergence of an archiv

18 Back mutations resulting in restoration of wild-type seq

19 ermining the DFE, such as protein stability, back-mutations, species complexity, and mutational robus

20 In some circumstances (ignoring back mutations) stable internal steady-state values for

21 Back mutation to germline sequence provided evidence for

22 If back-mutation to F427 offers an accessible pathway to in

23 hich a compensatory mutation, instead of the back mutation, was acquired to recover fitness.

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