ライフサイエンスコーパス: back pain

1 scores indicating more disability related to back pain).

2 oung and middle-aged adults with chronic low back pain.

3 eatment option for patients with chronic low back pain.

4 ial activation, in patients with chronic low back pain.

5 rheumatologist, with symptoms of the chronic back pain.

6 ute or chronic nonradicular or radicular low back pain.

7 aches, vague upper abdominal pain, and lower back pain.

8 degeneration is the leading cause of chronic back pain.

9 a 3-month history of fatigue and unremitting back pain.

10 d hot flushes, alopecia, abdominal pain, and back pain.

11 ute or chronic nonradicular or radicular low back pain.

12 tern United States with a 3-month history of back pain.

13 respiratory tract infection, influenza, and back pain.

14 n of muscle fat content in patients with low back pain.

15 and to ameliorate IVD-associated chronic low back pain.

16 generation are believed to contribute to low back pain.

17 line addressed pharmacologic options for low back pain.

18 controlled trial of acupuncture for chronic back pain.

19 the relationships of BMI and height with low back pain.

20 sociated with modest effects for chronic low back pain.

21 tability of acupuncture to patients with low back pain.

22 and they can be responsible for chronic low back pain.

23 ty can identify patients prone to persistent back pain.

24 a clinically effective treatment for chronic back pain.

25 as they considered prognosis studies of low back pain.

26 nal injections of methylprednisolone for low back pain.

27 (46.8%) were classified to have inflammatory back pain.

28 67% specific for a diagnosis of inflammatory back pain.

29 ditions owing to non-specific arthralgia and back pain.

30 s and the development of symptomatic chronic back pain.

31 degeneration is a major cause of chronic low back pain.

32 ctive treatment for chronic or recurrent low back pain.

33 the effectiveness of massage for chronic low back pain.

34 ars (QALYs) and health-care costs related to back pain.

35 mbar facet joints has been implicated in low back pain.

36 , acute myocardial infarction, and acute low back pain.

37 practice for the evaluation of inflammatory back pain.

38 ing that the acute pain relieves the ongoing back pain.

39 and the outcome, the incident occurrence of back pain.

40 following acute, experimentally induced low back pain.

41 nical care without immediate imaging for low-back pain.

42 adults with acute, subacute, or chronic low back pain.

43 and harms of SMT for acute (</=6 weeks) low back pain.

44 nd the role of MBSR in the management of low back pain.

45 ommendations on noninvasive treatment of low back pain.

46 ogic and nonpharmacologic treatments for low back pain.

47 al disc degeneration (IVDD) is linked to low back pain.

48 cacy and safety of MBSR in patients with low back pain.

49 ty as a primary outcome in patients with low back pain.

50 therapy, she reported 3 months of worsening back pain.

51 d nonpharmacologic treatment options for low back pain.

52 nditions, including osteoarthritis and lower back pain.

53 ercise program for patients with chronic low back pain.

54 anagement practice for patients with chronic back pain.

55 be a lifelong task for patients with chronic back pain.

56 aminations by rheumatologists due to chronic back pain.

57 rug treatments for patients with chronic low back pain?

58 ith indigestion (0.74, 0.58-0.95; p=0.0018), back pain (0.76, 0.58-0.99; p=0.044), diabetes (0.63, 0.

59 ith indigestion (0.71, 0.56-0.89; p=0.0033), back pain (0.77, 0.59-0.99; p=0.040), diabetes (0.71, 0.

60 ere lower than patients with no inflammatory back pain (0.82, SD 0.16, P = .0048).

61 nificantly after MR imaging for inflammatory back pain (14% vs 76%, before vs after; P < .001), mecha

62 s or older with a new primary care visit for back pain (2011-2013) in 3 US health care systems.

63 /wrist pain (17% vs 7%; P = 0.03), and lower back pain (26% vs 9%; P<0.01).

64 38 [20%] of 189 in the bicalutamide group), back pain (35 [19%] vs 34 [18%]), and hot flush (27 [15%

65 [5%] vs 1 [1%]), fatigue (3 [4%] vs 3 [4%]), back pain (4 [5%] vs 3 [4%]), arthralgia (4 [5%] vs 1 [1

66 76%, before vs after; P < .001), mechanical back pain (4% vs 49%, P < .001), spondylitis (7% vs 76%,

67 puncture syndrome (3/8 [38%] vs 8/24 [33%]), back pain (4/8 [50%] vs 4/24 [17%]), and nausea (0/8 [0%

68 3, and October 24, 2014, and had chronic low back pain, a positive diagnostic block at the facet join

69 erative disc disease often causes severe low-back pain, a public health problem with huge economic an

70 Non-specific low back pain affects people of all ages and is a leading co

71 ty were divided according to symptoms of low back pain alone and symptoms of low back pain with objec

72 and disability in patients with chronic low back pain, although this difference became nonsignifican

73 link between antecedent depression and later back pain among female military nurses.

74 tistically-significant predictor of incident back pain among female subjects (odds ratio [OR]: 1.75,

75 le of depression as a potential predictor of back pain among nurses appears understudied.

76 skeleton was performed on 174 patients with back pain and 11 control subjects.

77 c disease, which is the major cause of lower back pain and affects a large proportion of the populati

78 ewly diagnosed AIDS presented with months of back pain and fever.

79 ual care, resulted in greater improvement in back pain and functional limitations at 26 weeks, with n

80 The yoga and usual care groups had similar back pain and general health scores at 3, 6, and 12 mont

81 ationship between self-management of chronic back pain and health-related quality of life (HRQoL); (2

82 y in patients presenting with acute chest or back pain and high blood pressure.

83 ebral disc degeneration (IDD) causes chronic back pain and is linked to production of proinflammatory

84 onditions, such as fibromyalgia, chronic low back pain and myofascial pain.

85 ety, musculoskeletal disorders including low back pain and neck pain, diabetes, and cirrhosis--increa

86 ng on clinical outcomes in patients with low-back pain and no indication of serious underlying condit

87 positive affect, the affective component of back pain and pain tolerance.

88 20-year-old man presented with 1 week of low back pain and progressive lower extremity weakness.

89 mages, intracranial pressure processing, low back pain and real-time tumour tracking; (3) outcome pre

90 rapid progression to coma preceded by lower back pain and recurrent falls.

91 ntensity and disability in patients with low back pain and sciatica after lumbar disc herniation.

92 (MR) imaging in patients with persistent low back pain and sciatica effectively demonstrates spine an

93 SOE, moderate) are effective for chronic low back pain and strengthens previous findings regarding th

94 most important factor leading to chronic low back pain and subsequent disability after discectomy.

95 e sacroiliac joints in patients with chronic back pain and suspected axial spondyloarthritis.

96 portant to remember about rare causes of low back pain and to perform detailed physical examination,

97 aging in patients with acute or subacute low-back pain and without features suggesting a serious unde

98 patients) undergoing lumbar spine CT for low back pain and/or radiculopathy.

99 y local DRG inflammation (a rat model of low back pain) and by a peripheral paw inflammation model.

100 imaging for patients with uncomplicated low back pain) and using the results for public reporting an

101 ic resonance imaging (MRI) (for headache and back pain), and referrals to other physicians (for all 3

102 ed, duloxetine was effective for chronic low back pain, and benzodiazepines were ineffective for radi

103 uloskeletal pain, nasopharyngitis, headache, back pain, and diarrhea.

104 Leg pain, back pain, and disability were converted to common scale

105 t acetaminophen is ineffective for acute low back pain, and duloxetine is associated with modest effe

106 50 mg dose (one subject) and abdominal pain, back pain, and eczema after multiple doses of 800 mg avo

107 Grade 3 deep venous thrombosis, grade 3 back pain, and grade 3 vomiting were each noted once in

108 Whereas symptoms of SEAs can include fever, back pain, and neurological dysfunction, the presentatio

109 se events (six vaginal symptoms, one case of back pain, and one case of abdominal pain) and one unexp

110 occurred in nine (3%) patients, and anaemia, back pain, and pain in extremities, each of which occurr

111 es such as spine bone mineral density (BMD), back pain, and the presence of vertebral fractures at ba

112 1.10-1.42), and preoperative pain disorders (back pain: aOR, 1.57; 95% CI, 1.42-1.75; neck pain: aOR,

113 Several systemic medications for low back pain are associated with small to moderate, primari

114 acologic therapies for primarily chronic low back pain are associated with small to moderate, usually

115 n-related conditions, but its effects on low back pain are uncertain.

116 e events in evolocumab-treated patients were back pain, arthralgia, headache, muscle spasms, and pain

117 orty patients with recent-onset inflammatory back pain, as assessed by the Calin criteria, HLA-B27 po

118 emselves in different ways such as joint and back pain, as well as deficiencies in skeletal bone qual

119 are most applicable to acute or subacute low-back pain assessed in primary-care settings.

120 fect on troublesome subacute and chronic low-back pain at a low cost to the health-care provider.

121 ire [RDQ]; range, 0-23) and in self-reported back pain bothersomeness (scale, 0-10) at 26 weeks.

122 and assessment of disease progression in low back pain, brain tumours and primary epilepsy; (2) enhan

123 tive for short-term pain relief in acute low back pain but caused sedation.

124 height are linked to the pathogenesis of low back pain, but evidence-based confirmation is lacking.

125 is a commonly used treatment for chronic low back pain, but high-quality evidence for its effectivene

126 brain representation for a constant percept, back pain, can undergo large-scale shifts in brain activ

127 ns, across different frequencies, in chronic back pain (CBP) patients (n = 15) as compared to healthy

128 e of the acute stress response of 16 chronic back pain (CBP) patients and 18 healthy individuals expo

129 ious thermal stimuli in controls and chronic back pain (CBP) patients.

130 d the volume of human hippocampus in chronic back pain (CBP), complex regional pain syndrome (CRPS),

131 Patients with chronic low back pain (cLBP) or amyotrophic lateral sclerosis (ALS)

132 s effective for mild to moderate chronic low back pain (cLBP), but its comparative effectiveness with

133 ommendation 2: For patients with chronic low back pain, clinicians and patients should initially sele

134 pain in 16 patients with chronic nonspecific back pain (CNBP) and in 16 age- and gender-matched healt

135 (D2/D3R) function in chronic non-neuropathic back pain (CNBP) by comparing CNBP patients and healthy

136 nically important improvement in chronic low back pain compared with a standardized exercise program

137 er prevalence of neck, hand/wrist, and lower back pain compared with family medicine physicians; repe

138 iotics (for URIs), radiography (for URIs and back pain), computed tomography (CT) or magnetic resonan

139 We randomized 24 patients with chronic back pain diagnosed with lumbar disk degeneration and un

140 fferent imaging methods, and duration of low-back pain did not affect the results, but analyses were

141 e events, mostly mild self-limited joint and back pain, did not differ between the yoga and PT groups

142 Secondary outcomes were self-reported low back pain, disability, global improvement, satisfaction,

143 Because non-specific low back pain does not have a known pathoanatomical cause, t

144 h exercise but not with rest; awakening from back pain during the second half of the night only; and

145 der adults with a new primary care visit for back pain, early imaging was not associated with better

146 All participants received a back pain education booklet.

147 adults aged 20 to 70 years with chronic low back pain enrolled between September 2012 and April 2014

148 study, individuals who developed an intense back pain episode were followed over a 1-year period, du

149 proximately 1.5), and a history of prior low back pain episodes and demographic variables were not us

150 eturn to work, global improvement, number of back pain episodes or time between episodes, patient sat

151 Subjects with recent onset of back pain exhibited emergence of kD only when the pain b

152 rdless of attribution, were cough, headache, back pain, fever, and chills.

153 , hydronephrosis (three [2%] vs seven [4%]), back pain (five [3%] vs three [2%]), pathological fractu

154 up, n = 94), to subjects who have lived with back pain for >10 years (chronic back pain group, n = 59

155 roximately 2 months with no prior history of back pain for 1 year (early, acute/subacute back pain gr

156 d for prediction of persistent disabling low back pain for findings attainable during the clinical ev

157 e 36 years or older was each associated with back pain for male and female nurses.

158 n adverse events within the first month were back pain (four of 70 in the kyphoplasty group and five

159 iofrequency denervation to treat chronic low back pain from these sources.

160 s of patients with fewer than 8 weeks of low back pain from which likelihood ratios (LRs) were calcul

161 y for back pain in the early, acute/subacute back pain group is limited to regions involved in acute

162 me, whereas in the persistent acute/subacute back pain group, activity diminished in acute pain regio

163 volved in acute pain, whereas in the chronic back pain group, activity is confined to emotion-related

164 In the recovered acute/subacute back pain group, brain activity diminished in time, wher

165 those who recover (recovered acute/sub-acute back pain group, n = 19) and those in which the back pai

166 ck pain persists (persistent acute/sub-acute back pain group, n = 20; based on a 20% decrease in inte

167 lived with back pain for >10 years (chronic back pain group, n = 59).

168 back pain for 1 year (early, acute/subacute back pain group, n = 94), to subjects who have lived wit

169 Patients with inflammatory back pain had worse quality of life than those without.

170 were aged 18 and over, suffered from chronic back pain, had opted in to the clinic and had sufficient

171 We found that patients with back pain have alterations in brain dopamine function th

172 wever, significant associations remained for back pain (hazard ratio, 1.13 [99% CI, 1.03-1.24]), migr

173 2.5%), and proteinuria, hyperbilirubinemia, back pain, hyperkalemia, and anorexia (n = 1 each).

174 Patients were classified with inflammatory back pain if they had >/=2 positive responses to 4 valid

175 hat most patients with acute or subacute low back pain improve over time regardless of treatment, cli

176 ted included reduction or elimination of low back pain, improvement in back-specific and overall func

177 20; based on a 20% decrease in intensity of back pain in 1 year).

178 al obscurations occurred in 68% of patients, back pain in 53%, and pulse synchronous tinnitus in 52%.

179 The prevalence of inflammatory back pain in a cohort of anterior uveitis patients was f

180 r a history of depression predicted incident back pain in a population of military registered nurses

181 We examined the prevalence of low back pain in adolescents and its association with BMI an

182 ht was associated with increased risk of low back pain in both genders.

183 Low back pain in children and adolescents is a common proble

184 k during 2011-2014 without evidence of prior back pain in clinical records.

185 ed mechanisms of pain regulation, in chronic back pain in human subjects.

186 er BMI was significantly associated with low back pain in males (for overweight, odds ratio = 1.097,

187 nt implications for the future management of back pain in primary care.

188 n to best practice advice in people with low-back pain in primary care.

189 Spondylolysis is a common cause for back pain in the adolescent athlete.

190 We observed that brain activity for back pain in the early, acute/subacute back pain group i

191 Odds ratios for low back pain in the tallest group compared with the shortes

192 to be a further, modifiable risk factor for back pain in this population.

193 e (in 73% of the patients), new or worsening back pain (in 50%), neurologic symptoms (in 48%), nausea

194 ad', 'tumor', 'spine', 'classification' and 'back pain' in the title and abstract of the manuscripts

195 outcomes included the extent of disability, back-pain intensity, and quality-of-life measures at pre

196 Low-back pain is a common and costly problem.

197 Acute low back pain is common and spinal manipulative therapy (SMT

198 Back pain is common in the general population, but only

199 ceptability of acupuncture treatment for low back pain is complex and multifaceted.

200 The most common cause of low back pain is degenerative disease of the intervertebral

201 The incidence of low back pain is extremely high and is often linked to inter

202 The clinical course of low back pain is often favourable, thus many patients requir

203 Observations: Low back pain is rarely seen in youth before they reach scho

204 afternoon in 47 subjects without current low back pain (IVDs = 230; age range, 20-71 years) after obt

205 us, and chronic pain conditions (chronic low back pain, knee osteoarthritis, and fibromyalgia).

206 Low back pain (LBP) in children and adolescents is a common

207 Low back pain (LBP) is a common debilitating condition which

208 Low back pain (LBP) is a widespread debilitating disorder of

209 Low back pain (LBP) is common in children but the prognostic

210 Low back pain (LBP) is common in primary care.

211 Low back pain (LBP) is responsible for more than 2.5 million

212 ted with a specific phenotype of chronic low back pain (LBP).

213 gram to adults with chronic or recurrent low back pain led to greater improvements in back function t

214 ain patients, we followed brain activity for back pain longitudinally over a 1-year period, and compa

215 categorizing musculoskeletal pain into lower back pain, lower extremity (hips, knees, and feet/ankles

216 ng brain activity for rating fluctuations of back pain magnitude.

217 the largest number of YLDs in 2010 were low back pain, major depressive disorder, other musculoskele

218 nfidence for clinical features (inflammatory back pain, mechanical back pain, muscular back pain, rad

219 tain noncancer pain diagnoses, in particular back pain, migraine, and psychogenic pain.

220 noses of pain-related conditions (arthritis, back pain, migraine, neuropathy, headache or tension hea

221 health problems not listed, or complained of back pain, migraines, or digestive problems at baseline.

222 treatments for participants with chronic low back pain (Mint study) were conducted in 16 multidiscipl

223 duced mechanical pain behaviors induced by a back pain model and a model of peripheral inflammatory p

224 features (inflammatory back pain, mechanical back pain, muscular back pain, radicular back pain, spon

225 83 [50%]), skin disorders (n = 81 [48.8%]), back pain (n = 54 [32.5%]), and alopecia (n = 53 [31.9%]

226 acetaminophen was ineffective for acute low back pain, nonsteroidal anti-inflammatory drugs had smal

227 MRI studies of inflammatory back pain of short duration have identified disease star

228 on and at 30 days to grade their severity of back pain on a 10-point numeric rating scale in response

229 c imaging is indicated for patients with low back pain only if they have severe progressive neurologi

230 the patient does not develop the symptoms of back pain or leg pain during the injection.

231 dental but may be found in patients with low back pain or neuromuscular deficits.

232 were half as likely to have work-related low-back pain (OR=0.50, 95% CI 0.26-0.96) and nurses reporti

233 ical trials of participants with chronic low back pain originating in the facet joints, sacroiliac jo

234 rted acute on chronic onset of thoracolumbar back pain over a period of 24 hours.

235 ms, and upper body, lower extremity, and low back pain over six months.

236 She has had back pain over this same period and has been taking acet

237 Falls (p=0.04) and back pain (p=0.05) were more common in the lithium group

238 e general population, but only a subgroup of back pain patients develops a disabling chronic pain sta

239 We tracked brain properties in subacute back pain patients longitudinally for 3 years as they ei

240 er number of healthy individuals and chronic back pain patients were also studied concomitantly, as p

241 In a subset of subacute back pain patients, we followed brain activity for back

242 and affective dimensions of pain in chronic back pain patients.

243 egions predicts placebo analgesia in chronic back pain patients.

244 k pain group, n = 19) and those in which the back pain persists (persistent acute/sub-acute back pain

245 sitive responses to 4 validated inflammatory back pain questions: presence of morning stiffness >30 m

246 ry back pain, mechanical back pain, muscular back pain, radicular back pain, spondylitis, sacroiliiti

247 Occupational back pain rates are substantial among registered nurses,

248 First we compared back pain-related brain activity between subjects who ha

249 Back pain remains a challenge for primary care internati

250 a longitudinal brain imaging study, subacute back pain (SBP) patients were followed over the course o

251 to a person's health status) indicating low back pain severity were divided according to symptoms of

252 For back pain, smaller differences favoring steroids compare

253 on, massage, and acupuncture for chronic low back pain (SOE, low to moderate).

254 puncture is modestly effective for acute low back pain (SOE, low).

255 Among patients with acute low back pain, spinal manipulative therapy was associated wi

256 cal back pain, muscular back pain, radicular back pain, spondylitis, sacroiliitis, and other) and ove

257 y had >/=1 of the following: chest/abdominal/back pain, syncope, perfusion deficit, and if AAS was in

258 y drugs had smaller benefits for chronic low back pain than previously observed, duloxetine was effec

259 rse triage to identify the rare cases of low back pain that are caused by medically serious pathology

260 truction site manager experienced 6 weeks of back pain that was not responsive to over-the-counter no

261 spect to reductions in disability related to back pain, the changes in the Oswestry Disability Index

262 etween cohorts, but for patients with lumbar back pain, they range between 2- and 3-fold.

263 os ranging from 1.33 [99% CI, 1.22-1.45] for back pain to 2.61 [1.82-3.74] for psychogenic pain).

264 ies varied from a low of 31% (n = 8) for low back pain to a high of 68% (n = 23) for fibromyalgia.

265 terviews following acupuncture treatment for back pain to identify, understand and describe the eleme

266 ect patients classified to have inflammatory back pain to refer for early rheumatologic assessment.

267 Ophthalmologists may use these questions on back pain to select patients classified to have inflamma

268 many guidelines allow for older adults with back pain to undergo imaging without waiting 4 to 6 week

269 eviews and RCTs, for RCTs of adults with low back pain treated in ambulatory settings with SMT compar

270 Among adults with chronic low back pain, treatment with MBSR or CBT, compared with usu

271 rials showing modest effects for chronic low back pain; trials were not designed to assess serious ha

272 re pulmonary embolism (three [4%] patients), back pain (two [2%]), and diarrhoea (two [2%]).

273 deviation]; age range, 20-79 years) with low back pain underwent standard 1.5-T MR imaging, which was

274 lude intense vulvar and vaginal itching, low back pain, uterine cramps, fetal distress, and preterm l

275 Prevalence of low back pain was 0.2% for both males and females with objec

276 population, and 35% (4-88) in workers; lower back pain was 18% (14-24), 31% (22-41), and 44% (33-55),

277 The incidence rate of back pain was 18.6 per 100 person-years and the period p

278 rs, 224 (65.7%) were women, mean duration of back pain was 7.3 years (range, 3 months-50 years), 123

279 assessment, a classification of inflammatory back pain was 92% sensitive and 67% specific for a diagn

280 Lower back pain was prevalent (63%), followed by ankle/foot (5

281 Chest or back pain was the most commonly reported presenting symp

282 Mr B, a 60-year-old man with back pain, was not informed of an incidental finding of

283 Seven RCTs involving 864 patients with low back pain were eligible for review.

284 Grade 3 to 4 neutropenia, diarrhea, and back pain were increased in patients treated with gemcit

285 ents for predicting persistent disabling low back pain were maladaptive pain coping behaviors, nonorg

286 lts with troublesome subacute or chronic low-back pain were recruited from 56 general practices and r

287 patients using ice-pack therapy for chronic back pain who developed erythematous, purpuric plaques a

288 commendation 3: In patients with chronic low back pain who have had an inadequate response to nonphar

289 At lower levels of nerve damage (lumbar back pain with disc herniation) association with greater

290 ness >30 minutes in duration; improvement in back pain with exercise but not with rest; awakening fro

291 s of low back pain alone and symptoms of low back pain with objective corroborating findings.

292 Low back pain with or without objective findings was associa

293 1573 adults (aged >/=18 years) with back pain (with or without radiculopathy) consultations

294 py may be effective for treatment of chronic back pain, with benefits lasting at least 6 months.

295 month period because of an acute, severe low back pain, with sphincter dysfunction, partially resembl

296 ting (OP-8) reduced MR imaging rates for low back pain without conservative therapy in either Medicar

297 of MR imaging examinations performed for low back pain without history of conservative therapy.

298 Lumbar imaging for low-back pain without indications of serious underlying cond

299 early diagnostic imaging in older adults for back pain without radiculopathy is uncertain.

300 s and American Pain Society guideline on low back pain, would provide better care to patients, improv