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1 somadendritic patch-clamp recording to track backpropagated action potentials (APs) in rat AOB primar
2                                              Backpropagated action potentials recorded at the soma we
3                                          The backpropagating action potential (BAP) is hypothesised t
4              The [Ca(2+)](i) increase from a backpropagating action potential (bAP) or subthreshold d
5 es via local active conductances, making the backpropagating action potential a candidate for dendrit
6  amplitudes of 10-70% of that generated by a backpropagating action potential at the same location.
7 K(v)7 channels are strongly activated by the backpropagating action potential to attenuate the afterd
8 al neurons is regulated by the spread of the backpropagating action potential to the synapse.
9 pike, the interaction of this spike with the backpropagating action potential, the mechanism responsi
10 ct on the time course of localized events or backpropagating action potential-evoked Ca(2+) signals i
11 re present in spines and can be activated by backpropagating action potentials (APs).
12                                              Backpropagating action potentials (bAPs) (three at 50 Hz
13                                              Backpropagating action potentials (bAPs) are an importan
14                                              Backpropagating action potentials (bAPs) are indispensab
15                                   Dendritic, backpropagating action potentials (bAPs) facilitate the
16    In layer 5 neocortical pyramidal neurons, backpropagating action potentials (bAPs) firing at rates
17           These studies revealed that single backpropagating action potentials (bAPs) produced more r
18 bles that incorporate fast transients due to backpropagating action potentials (bAPs), or other forms
19                                     However, backpropagating action potentials (BPAPs) have been stud
20 rons, in which dendritic sodium channels and backpropagating action potentials allow somatic spikes t
21 tinguishes LTS from other signals, including backpropagating action potentials and dendritic Ca(2+)/N
22                                      Second, backpropagating action potentials are able to serve as a
23            Pairing synaptic stimulation with backpropagating action potentials enhanced the likelihoo
24 oral state-dependent manner and suggest that backpropagating action potentials faithfully inform prox
25 KC) leads to an increase in the amplitude of backpropagating action potentials in distal dendrites th
26 endritic tree can exert a powerful impact on backpropagating action potentials in hippocampal pyramid
27 s of [Ca(2+)](i) changes that were evoked by backpropagating action potentials in pyramidal neurons i
28 ential from spines demonstrate directly that backpropagating action potentials invade the spines.
29 n lead to dendritic depolarization, and that backpropagating action potentials may be neither necessa
30                              We propose that backpropagating action potentials open glutamate-bound N
31 r dendritic spikes in the same branch, while backpropagating action potentials trigger a widespread r
32 he postsynaptic action potential, carried by backpropagating action potentials, can be strongly degra
33 ic glutamate receptors (mGluRs), paired with backpropagating action potentials, causes large, wave-li
34                        In contrast, actively backpropagating action potentials, typical of wakefulnes
35 through voltage-gated channels, activated by backpropagating action potentials, was detected at all d
36                   Despite the lack of active backpropagating action potentials, we find that trains o
37 nhibition during dendritic depolarization by backpropagating action potentials.
38 tion, which many current models attribute to backpropagating action potentials.
39 m local NMDA application in conjunction with backpropagating action potentials.
40 voked a larger dendritic calcium influx than backpropagating action potentials.
41 aired soma- dendritic whole-cell recordings, backpropagated APs were unattenuated up to approximately
42 cium influx into dendrites and spines during backpropagating APs (average increase, ~40%).
43         As a result SK channel activation by backpropagating APs gated STDP induction during low-freq
44 midal neurons regulate calcium influx during backpropagating APs in a distance-dependent manner, and
45  that activation of SK channels in spines by backpropagating APs plays a key role in regulating both
46                         Voltage waveforms of backpropagating APs were minimally altered in the same d
47 ttle amplitude and time course modulation on backpropagating APs; (2) are strongly invaded by the som
48                       Next, the somatic sink backpropagated at a speed of millimeters per minute into
49 the execution of the sequence and ultimately backpropagated away from stereotyped sequence actions, b
50 nge in the baseline-to-peak amplitude of the backpropagating dendritic action potential (bAP) was not
51 s or by weak synaptic inputs coinciding with backpropagating dendritic action potentials.
52 ma, triggering axonal action potentials that backpropagate into the dendrites.
53 + rise from single action potentials as they backpropagate into the oblique dendrites from the main t
54 iring caused by axosomatic Kv7 channel block backpropagated into distal dendrites affecting their act
55           Somatically generated APs actively backpropagated into the dendritic tree and evoked instan
56 id reduce the amplitude of action potentials backpropagating into the apical dendrite.
57 are always recorded first at the soma before backpropagating into the dendrites while undergoing subs
58 s reduced through the block of Ca(2+) entry, backpropagating Na(+) spikes and synaptically evoked EPS
59                Imaging studies revealed that backpropagating Na(+) spikes and synaptically evoked EPS
60 hippocampus, Na+-dependent action potentials backpropagate over the dendrites in an activity-dependen
61 eport cross-modal dendritic interactions via backpropagating postsynaptic potentials.
62                                         This backpropagating SD current flow resembles that of activi
63                                              Backpropagating spikes caused increases in [Ca2+]i at al
64 oltage-dependent calcium conductances by the backpropagating spikes during sharp wave bursts may be c
65                                   Therefore, backpropagating spikes in these cells can only influence
66 al synaptic stimulation rapidly and actively backpropagated throughout the entire dendritic arbor and
67 siological conditions in rat nerve terminals backpropagated up the axon ( approximately 400-800 micro
68 ikely in the axon initial segment, that then backpropagate with high fidelity into the dendrites, res

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