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1 C attachment to the gut by blocking the FimH bacterial adhesin.
2 ough splitting a domain from a Gram-positive bacterial adhesin.
3 cross-links are widespread in Gram-positive bacterial adhesins.
4 ivity that mediates protein glycosylation of bacterial adhesins.
5 ning the carbohydrate binding specificity of bacterial adhesins.
6 ory epithelia or exhibit similarity to known bacterial adhesins.
7 olved in UTI-induced preterm labor involving bacterial adhesins.
8 ink between the enzyme and the expression of bacterial adhesins.
10 rface involves specific interactions between bacterial adhesins and adsorbed salivary components.
11 pB) that had amino acid sequence homology to bacterial adhesins and structural homology to bacterial
12 To evaluate the relative contributions of bacterial adhesins and toxins to ciliary binding, we use
13 is a member of a diverse family of predicted bacterial adhesins, and although lacking a high degree o
15 ose encoded protein has characteristics of a bacterial adhesin; and implicated Ace in binding to coll
17 r hypotheses that some ligands recognized by bacterial adhesins change their compartmentalization and
18 invasion of heart tissue is dependent on the bacterial adhesin choline-binding protein A that binds t
19 h repeat glycoproteins (SRRPs) are important bacterial adhesins conserved in streptococci and staphyl
20 de O-linked to the serine-rich repeat of the bacterial adhesin, Fap1 of Streptococcus parasanguinis.
23 nters around FimH antagonists that block the bacterial adhesin FimH, which would otherwise mediate bi
24 ike serine-rich proteins are a new family of bacterial adhesins found in a variety of streptococci an
25 lycoproteins (SRRPs) are a growing family of bacterial adhesins found in many streptococci and staphy
26 s identified 19 genes with homology to known bacterial adhesin genes, virulence genes, genes involved
28 ructural studies on a class of Gram-positive bacterial adhesins have revealed an intramolecular Cys-G
29 ns are mediated through the binding of other bacterial adhesins, in particular the Opa family of oute
30 This study tested the hypothesis that the bacterial adhesin intimin contributes to tissue specific
34 ulosis internalization is interaction of the bacterial adhesins invasin and YadA with host cell beta1
36 ng this role is lacking, largely because the bacterial adhesins involved in this host-microbe associa
37 Adherence to host epithelium, mediated by bacterial adhesins, is one of the first steps in NTHi co
38 the identification of a yet uncharacterized bacterial adhesin, LabA, which specifically recognizes l
40 is secreted in significant amounts and that bacterial adhesins may have other activities, prompted a
42 eukaryotic and bacterial viruses as well as bacterial adhesins might have a similar maturation mecha
43 lt both because it is polyvalent and because bacterial adhesins often recognize more than one type of
45 IRF-1 expression depends on the presence of bacterial adhesin, our findings do not preclude the poss
50 an E. faecalis sequence, ace, that encodes a bacterial adhesin similar to the collagen binding protei
51 Collectively, these findings establish a new bacterial adhesin structure that has in effect been hija
53 previously discovered a widespread group of bacterial adhesins, termed Multivalent Adhesion Molecule
54 h repeat glycoproteins (SRRPs) are important bacterial adhesins that are conserved in streptococci an
56 sents a new paradigm for target binding by a bacterial adhesin, the identification of which will info
57 ased at sites of inflammation, and allow the bacterial adhesin to selectively associate with surface-
58 We have developed a mouse model to study the bacterial adhesins which mediate the increased intestina
59 lecules and highlight the first example of a bacterial adhesin with two domains that participate in a
60 y infections initiated by the interaction of bacterial adhesins with carbohydrate receptors can be po
61 is aided by the interaction between numerous bacterial adhesins with components of the extracellular
62 onic acid capsule impedes the interaction of bacterial adhesins with keratinocyte receptors, (iii) mo
63 sruption or modulation of the interaction of bacterial adhesins with LR might engender unexpectedly b
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