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1 cond T-cell receptor (TCR) that recognizes a bacterial antigen.
2 ibute to the enhanced T-cell response to the bacterial antigen.
3 lar to the aggregation observed for purified bacterial antigen.
4 ming of CD8(+) T cells against a nonsecreted bacterial antigen.
5 not IgG antibody, recognizing T-independent bacterial antigens.
6 unt T-independent antibody responses against bacterial antigens.
7 ells independently of cognate recognition of bacterial antigens.
8 polyreactive and frequently bound to host or bacterial antigens.
9 multiple immune effectors targeting multiple bacterial antigens.
10 l trafficking of CD11b+ cells in response to bacterial antigens.
11 may reduce risk of endothelial activation by bacterial antigens.
12 ulated immune responses against intraluminal bacterial antigens.
13 rently are associated with a Th1 response to bacterial antigens.
14 ntibodies in SLE may be elicited by the same bacterial antigens.
15 a dysregulated immune response to ubiquitous bacterial antigens.
16 predominantly reactive toward Staphylococcal bacterial antigens.
17 and TLR4 by gram-positive and gram-negative bacterial antigens.
18 alpha-defensins in response to bacteria and bacterial antigens.
19 3 days of a primary response to particulate bacterial antigens.
20 bicidal peptides when exposed to bacteria or bacterial antigens.
21 cultured with splenocytes in the presence of bacterial antigens.
22 ripheral blood mononuclear cell responses to bacterial antigens.
25 These results suggested that GEC can take up bacterial antigen and consequently process and present t
26 weak homology with a fragment of a putative bacterial antigen and, like that molecule, binds IgG.
27 onse, which primarily targeted gram-negative bacterial antigens and conferred protection against syst
29 -PC is cross-reactive with a variety of oral bacterial antigens and human antigens such as oxidized l
31 Activation is induced by stimulation from bacterial antigens and inflammatory cytokines derived fr
32 eting NK cells resulted in the expression of bacterial antigens and iNOS outside the granulomas and t
33 ed that exposure to lipopolysaccharide (LPS, bacterial antigen) and polyinosinic-polycytidylic acid (
34 ure, Gram stain, and latex agglutination for bacterial antigen) and qPCR for Streptococcus pneumoniae
35 e to innate immunity by sensing bacteria and bacterial antigens, and discharge microbicidal peptides
36 ed after incubation with cytokines, food and bacterial antigens, and homogenates of small and large b
37 f antigen compartmentalization suggests that bacterial antigens are presented by multiple MHC class I
38 T cells that specifically recognize viral or bacterial antigens are selected to survive and prolifera
39 Abs did not show cross-reactivity with other bacterial antigens as confirmed by IgG ELISA, further va
43 increased inflammatory cell mobilization and bacterial antigen clearance from the inflamed colon to t
44 matory responses to common upper respiratory bacterial antigens compared to those of cells derived fr
46 g cell population that mediated tolerance to bacterial antigens during a defined developmental window
47 Vaccines based on preferential expression of bacterial antigens during human infection have not been
48 d BALB/c mice are Th2 cytokine responsive to bacterial antigen early after challenge with P. aerugino
50 s has only rarely been demonstrated toward a bacterial antigen from a naturally occurring human infec
51 en as mediated by FcRn involves retrieval of bacterial antigens from the lumen and initiation of adap
52 induce mucosal immune responses against key bacterial antigens has been a continuing focus of invest
55 ies identify antigen processing of a natural bacterial antigen in the human CD1c system, indicating t
56 sing human corneal epithelial multilayers to bacterial antigens in a culture supernatant (known to up
57 us peptides as minor H antigens and foreign, bacterial antigens in a defensive immune response to inf
63 pid activation of macrophages in response to bacterial antigens is central to the innate immune syste
64 ve T cells expanded by reacting with luminal bacterial antigens is likely caused by the survival of t
65 s to investigate whether HDM are carriers of bacterial antigens leading to IgE sensitization in patie
67 Candida albicans, mumps, Trichophyton, and a bacterial antigen made from lysed Staphylococcus aureus.
69 of this study was to determine whether these bacterial antigens might be involved in HL60 cells cytol
71 to whole bacterial cells and to heat-stable bacterial antigens of all seven prototypic P. aeruginosa
72 re activated in vivo after exposure to these bacterial antigens or bacteria, and mice that lack NKT c
73 g immunohistochemical analysis, we localized bacterial antigens primarily to lymphoid tissues and liv
75 phic nature of M3 and its ability to present bacterial antigens rapidly and dominantly make it an att
77 ted within human dendritic cells showed that bacterial antigens remained intact, even after delivery
78 gic airway inflammation or the TH17-inducing bacterial antigen Saccharopolyspora rectivirgula in a mo
79 n decreased SC and IgA antibodies to certain bacterial antigens (SAEB) in nasal secretions of patient
81 Colitis was induced by transfer of a cecal bacterial antigen-specific C3H/HeJBir (C3Bir) CD4(+) T-c
82 ation, we characterized disease kinetics and bacterial antigen-specific T-cell responses in ex germ-f
83 ted amounts of IL-12 and TNF-alpha following bacterial antigen stimulation, indicating alternative pa
85 lasting elevation of antibodies to commensal bacterial antigens, suggesting that MCMV infection may h
88 ation under caries appears to be elicited by bacterial antigens that diffuse into the pulp through de
89 direct T-cell expression cloning to identify bacterial antigens that induce a preferential or biased
91 e responses against blood-born T-independent bacterial antigens (TI), but the full scope of their imm
92 gen and consequently process and present the bacterial antigen to CD4(+) T cells by MHC class II in c
94 ells (DCs) phagocytose, process, and present bacterial antigens to T lymphocytes to trigger adaptive
95 IBD results from environmental factors (eg, bacterial antigens) triggering a dysregulated immune res
96 ed for exogenous presentation of nonsecreted bacterial antigens via its capacity to upregulate the ex
97 Plasmacytoid dendritic cells, activated by bacterial antigens via TLR9 or by viral infection, did n
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