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1 d the expiry period showed no marked drop in bacterial count.
2 sive care unit-acquired pneumonia and higher bacterial count.
3 ly at different time points for quantitative bacterial counts.
4 er standardization of IL-8 concentrations to bacterial counts.
5 l one associated with higher intraperitoneal bacterial counts.
6 USA300 strains caused reduced pathology and bacterial counts.
7 r system (in vivo imaging system [IVIS]) and bacterial counts.
8 only moderately larger with minimally higher bacterial counts.
9 ntal endocarditis infections by target organ bacterial counts.
10 ng compared to wild-type 104, as assessed by bacterial counts.
11 cterial cells directly from samples with low bacterial counts (10(4) cfu/mL) using a custom-designed
15 ctions in corneal pathology and also lowered bacterial counts after infection with six different labo
17 te (IA3902) resulted in significantly higher bacterial counts and a significantly longer duration of
19 WT mice, which was associated with increased bacterial counts and elevated inflammatory cytokine and
21 1alpha, developed larger lesions with higher bacterial counts and had decreased neutrophil recruitmen
22 in was accompanied by a >50-fold increase in bacterial counts and higher numbers of viable intracellu
23 ubstantially larger skin lesions with higher bacterial counts and impaired neutrophil recruitment com
27 c FasL(-/-) exhibited significantly elevated bacterial counts and polymorphonuclear leukocyte numbers
30 topathology, myeloperoxidase (MPO) activity, bacterial counts, and ELISA analysis were used to assess
34 ry reaction, abscess wall formation, abscess bacterial counts, and peritoneal bacterial counts, were
36 Clinical score, slit lamp, histopathology, bacterial counts, and polymorphonuclear neutrophil (PMN)
37 tion, slit lamp examination; clinical score; bacterial counts; and myeloperoxidase (MPO), RT-PCR, ELI
39 M1 showed decreased survival and higher lung bacterial counts, as well as increased dissemination of
40 metry and microscopy and by determining live bacterial counts associated with B cells both in vivo an
43 ase chain reaction was used to measure total bacterial counts, Bacteroides/Prevotella (herein referre
45 was demonstrated in animal models, reducing bacterial counts by six orders of magnitude, and contrib
46 s, we found that G. vaginalis and M. hominis bacterial counts, Candida vaginitis, and herpes simplex
47 early inflammation and a delayed increase in bacterial counts compared to animals infected with NTHi
48 y E. coli had significantly higher pulmonary bacterial counts compared with animals that received E.
49 xhibited lower mortality and decreased brain bacterial counts compared with mice infected with the co
50 ice display persistently elevated peritoneal bacterial counts compared with wild-type mice, reduced p
52 o approximately 7.6 log(10) by 18 hours, but bacterial count declined to approximately 6.4 log(10) CF
55 revealed rapid and significant decreases in bacterial counts for protegrin-1-treated groups compared
56 ifferences occurred despite the finding that bacterial counts for the strain pairs were indistinguish
59 hoc exploratory analyses of UTIs with higher bacterial counts (>/=10(5) colony-forming units per mL),
60 sera were collected after each challenge for bacterial counts, histological evaluation, cytokine prof
61 tes (HCTLR4KO) and then determined survival, bacterial counts, host inflammatory responses, and organ
63 PVL(+) strains also had significantly higher bacterial counts in abscesses compared with mice given n
64 helial and lymphocyte apoptosis and systemic bacterial counts in animals given iron supplementation a
67 e had a six- to eight-fold increase in total bacterial counts in comparison with sham and control mic
70 ory CD8+ T cells can reduce spleen and liver bacterial counts in IFN-gamma-deficient mice 3 d after L
72 stant C57BL/6 mice, the mutant achieved high bacterial counts in lung and spleen that persisted in ti
73 (TNF) and interferon (IFN)-gamma, increased bacterial counts in lungs and blood, and early lethality
74 ed lung levels of TNF and IFN-gamma, reduced bacterial counts in lungs and plasma 40 h after the inoc
78 resulted in significantly lower total viable bacterial counts in moderate-to-deep pockets when compar
79 tiserum to mice produced significantly lower bacterial counts in organs than did normal rabbit serum
81 d was quantified in terms of change in total bacterial counts in pore throats in low permeability reg
82 n resulted in elevated PMN levels and viable bacterial counts in the cornea 3 and 5 days after infect
85 nt (MyD88(-/-)) mice had dramatically higher bacterial counts in the lungs, with decreased neutrophil
88 ility to fluorescein isothiocyanate dextran, bacterial counts in the mesenteric lymph nodes complex,
92 erleukin-12 (IL-12) antibodies, resulting in bacterial counts in the spleens and livers of anti-IL-12
93 cholera toxin (CT), had significantly lower bacterial counts in their kidneys ( P = 0.001) and splee
94 e that lacked both T-cell subsets had higher bacterial counts in their livers 15 to 18 days after inf
95 d no significant effect on individual plaque bacterial counts in unadjusted models or those adjusted
96 mice, serum levels of IL-6 and TNF-alpha and bacterial counts in various organs were significantly re
98 se (i.e., cachexia, diarrhea, and high fecal bacterial counts) is preceded by a lengthy subclinical s
99 s developed an inflammatory lesion with high bacterial counts, marked neutrophil infiltration, and hi
101 rs were recorded, and total and quantitative bacterial counts of Aggregatibacter actinomycetemcomitan
102 marcescens, 3 Acps significantly reduced the bacterial counts of infected females, suggesting a prote
104 There was no change in the total aerobic bacterial count or total mould count as a result of trea
105 ts (P=.006; inverse association), M. hominis bacterial counts (P=.0001; positive association), Candid
106 In multivariate analysis, only lactobacilli bacterial counts (P=.006; inverse association), M. homin
109 he control sites indicated that 90.6% of the bacterial counts remained the same, 6% increased, and 3%
110 se of recombinant IL-17 had lesion sizes and bacterial counts resembling those of WT mice, demonstrat
111 GBS) showed 100% detection, but at the lower bacterial counts, SBCB and IGLB were more sensitive than
113 as associated with a significant decrease in bacterial counts, suppressed bacterial production of PVL
114 nt, proteolytic activities and psychrophilic bacterial count than did samples treated with soybean an
115 ceptibility more rapidly, with lower initial bacterial counts than existing commercial systems, and p
116 ontal diseases sites, as well as lower total bacterial count, than all the other groups at 180 days.
117 This phenotype was accompanied by higher bacterial counts, the formation of extracellular bacteri
119 1 by 24-48 hours of infection, and increased bacterial counts up to 5 days after infection, compared
120 g 173 positive BAL sample pairs, significant bacterial counts were detected exclusively in 6.4% of le
121 aerodynamics were evaluated; neutrophil and bacterial counts were determined in bronchoalveolar lava
128 in CapG(+/+) mice at days 5 to 9, while the bacterial counts were identical on day 5 for Salmonella-
130 rviving animals were sacrificed at 72 h, and bacterial counts were performed on their kidneys, livers
132 on, abscess bacterial counts, and peritoneal bacterial counts, were all similar, but blood bacterial
133 WT mice, Kit(W-sh/W-sh) mice showed reduced bacterial counts with less bacterial dissemination to di
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