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1 subjects receiving an inflammatory stimulus (bacterial endotoxin).
2 ry--K1 fibroblasts defective in responses to bacterial endotoxin.
3 mportant role in the innate response against bacterial endotoxin.
4 entified as signal-transducing receptors for bacterial endotoxin.
5 mRNA expression in macrophages is induced by bacterial endotoxin.
6 lenged them with lipopolysaccharide (LPS), a bacterial endotoxin.
7 tly by pathogen-associated molecules such as bacterial endotoxin.
8 imuli, such as proinflammatory cytokines and bacterial endotoxin.
9 appaB pathway during both MI and exposure to bacterial endotoxin.
10 (delayed) systemic inflammatory response to bacterial endotoxin.
11 nto the liver, and increased serum levels of bacterial endotoxin.
12 e were exposed to inhaled cigarette smoke or bacterial endotoxin.
13 MH) challenged with a shock-inducing dose of bacterial endotoxin.
14 saccharide to CD14 and host response to this bacterial endotoxin.
15 ms responsible for altered responsiveness to bacterial endotoxin.
16 n response to distension or stimulation with bacterial endotoxins.
17 d kinases (IRAK) regulates responsiveness to bacterial endotoxins.
18 ns of the following microbiological markers: bacterial endotoxin, 3-hydroxy fatty acids, and muramic
22 ive stress occurs in animals challenged with bacterial endotoxin and can affect the expression of imp
23 ogroup B to facilitate metabolic labeling of bacterial endotoxin and compared interactions of purifie
24 ogic changes, and reduced systemic levels of bacterial endotoxin and concentrations of Pseudomonas ae
26 inflammation by mediating responses to both bacterial endotoxin and multiple endogenous ligands, inc
27 GPI fraction, which is at least as active as bacterial endotoxin and Mycoplasma lipopeptide and, ther
29 h Wnts suppress proinflammatory responses to bacterial endotoxin and to TLR1/2, TLR7, and TLR9 ligand
30 he immune system, potentiates the effects of bacterial endotoxin, and increases the lethality of cert
31 ll as in healthy subjects receiving low-dose bacterial endotoxin, and show that these severe stresses
33 e major producers of IFNgamma in response to bacterial endotoxin but not to interleukin-12, and; (iii
34 opolysaccharide factor (LALF) can neutralize bacterial endotoxin, but its ability to prevent mortalit
36 cells were able to respond to challenge with bacterial endotoxin by mounting an acute inflammatory re
40 rabbits to evaluate the ocular reactivity to bacterial endotoxin contained in Dulbecco's phosphate-bu
42 4-dependent cell activation by gram-negative bacterial endotoxins depends on sequential endotoxin-pro
44 receptor 4 (TLR4), together with MD-2, binds bacterial endotoxins (E) with high affinity, triggering
47 ese enzymes in rats subjected to intraportal bacterial endotoxin exposure (lipopolysaccharide [LPS],
48 erved that animals survived a lethal dose of bacterial endotoxin if they had been previously treated
49 vestigated whether exposure to Gram-negative bacterial endotoxin in early neonatal life can alter neu
50 ed a systemic hyper-inflammatory response to bacterial endotoxin in localized aggressive periodontiti
51 peptidoglycan fragment (muramyl peptide) and bacterial endotoxin in the induction of inflammatory pro
52 liver disease identified increased levels of bacterial endotoxin in the portal circulation, suggestin
54 nd the cell line indicate that gram-positive bacterial endotoxins induce hypoxia-inducible factor 1al
57 iency of HIF-1alpha attenuated gram-positive bacterial endotoxin-induced cellular motility and proinf
58 and display a more pronounced and prolonged bacterial endotoxin-induced febrile response than wild t
59 ted by TLR4 signaling, such as Gram-negative bacterial endotoxin-induced lung injury and HDM-triggere
60 gnificant therapeutic approach in preventing bacterial endotoxin-induced sepsis and tissue damage.
62 ntal protocols that controlled for potential bacterial endotoxin-induced TNF-alpha secretion, the cur
68 In conclusion, we have demonstrated that bacterial endotoxin is internalized and transported to s
72 traperitoneal injection of the Gram-negative bacterial endotoxin lipopolysaccharide (LPS) elicits a r
86 ia in young and aged mice with a low dose of bacterial endotoxin lipopolysaccharide (LPS, 2.5 mg/kg)
87 inic-polyribocytidilic acid [Poly(I:C)], the bacterial endotoxin lipopolysaccharide, the locally acti
88 NOS-2) is induced following stimulation with bacterial endotoxin (lipopolysaccharide (LPS)) and/or pr
96 ultures of rat hepatocytes after exposure to bacterial endotoxin (lipopolysaccharide) in a nitric oxi
97 n repetitive exposures to different doses of bacterial endotoxin (lipopolysaccharide) or other stimul
104 cytokines and chemokines is a key feature of bacterial endotoxin, lipopolysaccharide (LPS)-induced in
105 h-old) mice experimentally challenged with a bacterial endotoxin, lipopolysaccharide (LPS, 1.5 mg/kg
107 rmeability and subsequently translocation of bacterial endotoxin-lipopolysaccharide into the blood.
110 Finally, ChQ prevents mouse PTBs induced by bacterial endotoxin LPS or progesterone receptor antagon
111 are more sensitive to the lethal effects of bacterial endotoxin LPS, and in the experiments reported
116 ated after intraperitoneal administration of bacterial endotoxin (LPS) in murine lung and kidney, but
117 on of NO in the down-regulation of CYP2B1 by bacterial endotoxin (LPS) in rat hepatocytes cultured on
119 ration across endothelial cells activated by bacterial endotoxin (LPS) or IL-1beta (60 and 46%, respe
120 we found that treatment of macrophages with bacterial endotoxin (LPS) or Pseudomonas induced L-PGDS
121 ages are among the most sensitive targets of bacterial endotoxin (LPS), responding to minute amounts
122 reported that increasing core temperature of bacterial endotoxin (LPS)-challenged mice to the normal
126 Thus, innate responses to fibrinogen and bacterial endotoxin may converge at the evolutionarily c
128 chemotactic activity during incubation with bacterial endotoxin or aggregated IgG, (b) to mediate th
129 exposed to mediators of inflammation such as bacterial endotoxin or lipopolysaccharide (LPS) in a var
132 ed in media, with or without the addition of bacterial endotoxin or varying molar concentrations of e
134 e-establish intestinal symbiosis, neutralize bacterial endotoxins, or adsorb gut-derived uremic toxin
135 at can be induced by inflammatory cytokines, bacterial endotoxin, osmotic shock, UV radiation, and hy
137 -1 cells, using diverse cell stimuli such as bacterial endotoxin, proinflammatory cytokines (IL-1 and
138 h had been added one of 5 different doses of bacterial endotoxin ranging from 0.02 to 1.4 endotoxin u
139 eptor 4 (TLR4) is the principal receptor for bacterial endotoxin recognition, and functional variants
140 t mammalian cell activation by Gram-negative bacterial endotoxin requires sequential protein-endotoxi
141 olecular pattern molecules (PAMPs) including bacterial endotoxin, respiratory viruses, and microbial
142 The results indicate that AR mediates the bacterial endotoxin signaling that could damage HLECs by
145 g inquiry into the mechanism of responses to bacterial endotoxin, the abundant lipopolysaccharide com
146 influence cytokine production in response to bacterial endotoxin; the high LBP:BPI ratios observed in
147 h kills meningococci and binds to and clears bacterial endotoxin, these being the primary inducers of
148 ng of lipopolysaccharide (LPS, also known as bacterial endotoxin) to human hemoglobin is known to res
149 eripherally with lipopolysaccharide (LPS), a bacterial endotoxin, to induce an inflammatory episode.
151 Our findings demonstrate that flies detect bacterial endotoxins via a gustatory pathway through TRP
153 athogenesis of sepsis is mediated in part by bacterial endotoxin, which stimulates macrophages/monocy
154 (NK) cells and displayed hypersensitivity to bacterial endotoxin, with their innate immune cells prod
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