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1 trophil count was not associated with severe bacterial infection.
2 severe G6PD deficiency and susceptibility to bacterial infection.
3 tions induced during the in vivo response to bacterial infection.
4 ) was associated with reduced risk of severe bacterial infection.
5 creases the susceptibility of the airways to bacterial infection.
6 9 were enhanced in DCs and in mice following bacterial infection.
7 tic paralyses of botulism and tetanus during bacterial infection.
8 l types, and the defences they employ during bacterial infection.
9 ality in response to endotoxin challenge and bacterial infection.
10 ion both MAPK and NF-kappaB molecules during bacterial infection.
11 es triggered by flg22 perception and reduces bacterial infection.
12 ful have been developed for the treatment of bacterial infection.
13 utant and overexpression lines of AtRAP upon bacterial infection.
14 mphocytes that provide early defence against bacterial infection.
15 nse and host defense against a gram-positive bacterial infection.
16 ctive function of MGL1 in an acute pneumonic bacterial infection.
17  negative role on IL-1beta production during bacterial infection.
18  human salivary gland dysfunction induced by bacterial infection.
19 apid suppression of JA-related components on bacterial infection.
20 treatment regimens have on the dynamics of a bacterial infection.
21 red for a fully competent immune response to bacterial infection.
22 fication for infants at high risk of serious bacterial infection.
23 l greatly facilitate diagnosis of pathogenic bacterial infection.
24  for markers of the innate immune system and bacterial infection.
25  the current knowledge of the role of MCs in bacterial infection.
26 ed, immune responses against severe invasive bacterial infection.
27 ike T cells and impaired T cell responses to bacterial infection.
28 lammatory and pro-resolving processes during bacterial infection.
29  high selectivity and sensitivity in imaging bacterial infection.
30 ression under basal conditions and following bacterial infection.
31 inical implications as a breath biomarker of bacterial infection.
32 al for the development of treatments against bacterial infection.
33 oncentrations close to biological reality of bacterial infection.
34  the host-pathogen interface during a septic bacterial infection.
35  (Van) on Gram-positive bacteria for imaging bacterial infection.
36 mediated protection against an intracellular bacterial infection.
37 and beta5 activities can be uncoupled during bacterial infection.
38 m infections as well as to systemic and oral bacterial infections.
39 mycin, are promising candidates for treating bacterial infections.
40 be promises a new way for in vivo imaging of bacterial infections.
41  Multidrug-resistant organisms caused 56% of bacterial infections.
42 e immune system controls both Plasmodium and bacterial infections.
43 al susceptibility pattern of external ocular bacterial infections.
44 ognized as important contributors to chronic bacterial infections.
45 erapeutic avenue to ameliorate CF-associated bacterial infections.
46 osed to be used to treat multidrug-resistant bacterial infections.
47 c target for the treatment of drug-resistant bacterial infections.
48 s has therapeutic potential for clearance of bacterial infections.
49 ostic nanodrugs for combating drug-resistant bacterial infections.
50 LIPA14 kd mosquitoes in response to systemic bacterial infections.
51 tool in the rational design of treatment for bacterial infections.
52 need for new approaches to treat and prevent bacterial infections.
53 tive to traditional antibiotics for fighting bacterial infections.
54 l vaccine modalities to combat Gram-positive bacterial infections.
55  is a critical step in host responses during bacterial infections.
56 esolution melt (HRM) for rapid diagnosis for bacterial infections.
57 ve for the treatment of antibiotic-resistant bacterial infections.
58 strategies to mitigate resistance and combat bacterial infections.
59 re clinically important in the fight against bacterial infections.
60 t options for highly resistant gram-negative bacterial infections.
61 ophils, the primary cellular defense against bacterial infections.
62 (SLE) and increased susceptibility to severe bacterial infections.
63 of effective endolysins for the treatment of bacterial infections.
64 s (UTIs) are among the most commonly treated bacterial infections.
65 inary medicine applications against invasive bacterial infections.
66 ship in pioneering studies to combat serious bacterial infections.
67 athway that enhances host resistance against bacterial infections.
68 for developing preventive treatments against bacterial infections.
69 lays major functions in host defense against bacterial infections.
70 ctive treatments against multidrug-resistant bacterial infections.
71  (TCMs) have been historically used to treat bacterial infections.
72 inical exacerbation by exposure to viral and bacterial infections.
73 d using microbiology methods at the sites of bacterial infections.
74 ovel antibacterial for treatment of invasive bacterial infections.
75 , caspase-11 activation during Gram-negative bacterial infections.
76 mprove disease outcomes during intracellular bacterial infections.
77 r simultaneous therapy of diseases caused by bacterial infections.
78 ype I IFNs were shown to be important during bacterial infections.
79 r contexts, or in response to some viral and bacterial infections.
80 ective, role for IL-17A during Gram-positive bacterial infections.
81 y has received little attention in detecting bacterial infections.
82 ign approach to treating multidrug-resistant bacterial infections.
83  novel role of mast cells in defense against bacterial infections.
84 timely diagnostics are critical for managing bacterial infections.
85 ance demands rapid and accurate diagnosis of bacterial infections.
86 d mucociliary clearance, and recurrent acute bacterial infections.
87 is an urgent need for new materials to treat bacterial infections.
88 TP comes at the cost of an increased risk of bacterial infections.
89 ntegral protective role against encapsulated bacterial infections.
90  as a last-line defense in treatment of many bacterial infections.
91 e, predisposing patients to life-threatening bacterial infections.
92  essential in reducing disease burden due to bacterial infections.
93 ne tissue responses to infection by viral or bacterial infections.
94 BS, and of patients with enteritis caused by bacterial infection, 13.8% developed IBS.
95  results in reduced resistance to a systemic bacterial infection [8, 9].
96  and LL-37 in protecting host from pulmonary bacterial infection after a single intra-tracheal instil
97 ps, 46.3% (63/136) were classified as having bacterial infection, although 94.9% (129/136) received a
98  = 2; sepsis n = 1); 15 (40%) had at least 1 bacterial infection and 9 (24%) at least 1 episode of th
99 function to promote T cell immunity during a bacterial infection and are an important source of extra
100 se strategies are often inadequate to detect bacterial infection and are not specific for living bact
101 mr1 mutants exhibit increased sensitivity to bacterial infection and decreased phagocytosis of bacter
102 C. elegans are excellent models for studying bacterial infection and host response, as well as for ge
103  create an environment that favors secondary bacterial infection and may provide previously unidentif
104  to the host immunoprotective effects during bacterial infection and suggest a role for PPARgamma imm
105 ta during the host inflammatory responses to bacterial infection and suggest that the axis in which t
106 tant mice showed increased susceptibility to bacterial infection and were largely resistant to endoto
107                                              Bacterial infection and/or ectopic expression of EspL le
108  mice and humans increases susceptibility to bacterial infections and causes defects in TLR and IL1 l
109 Procalcitonin is a biomarker used to monitor bacterial infections and guide antibiotic therapy.
110 erates physiological heterogeneity in clonal bacterial infections and helps to determine the distinct
111 rotein 2 (NOD2) is essential for controlling bacterial infections and inflammatory disorders.
112 re use of antibiotics is limited to treating bacterial infections and manure is only applied from Apr
113 ts, who frequently suffer from opportunistic bacterial infections and metabolic complications, freque
114           Hemolysis drives susceptibility to bacterial infections and predicts poor outcome from seps
115 immature granulocytes, which reflects severe bacterial infections and septic condition but has not be
116 munoglobulins were detected in patients with bacterial infections and stimulated LILRA2-expressing ce
117  associated with increased susceptibility to bacterial infections and suppression of inflammatory sig
118                               In this model, bacterial infections and TNFalpha enhanced immune activi
119 respiratory syndrome who were likely to have bacterial infections and who would benefit from antibiot
120  infections (UTI) are one of the most common bacterial infections and would greatly benefit from a ra
121 uced the odds of febrile neutropenia, likely bacterial infection, and bloodstream infection by >/=70%
122 ignals in promoting maximal apoptosis during bacterial infection, and demonstrate that heterogeneity
123 active alcoholism/acute alcoholic hepatitis, bacterial infection, and others); (3) severity of SI and
124 ut not GBP2 KO mice were more susceptible to bacterial infection, and small interfering RNA treated-m
125 tant regulatory role in the host response to bacterial infection, and they underscore a role for Sts
126 matory cytokine required for host control of bacterial infections, and its production must be tightly
127 wound dressings absorb wound exudate, reduce bacterial infections, and maintain a moist environment f
128  higher rates of CDC-B events, CDC-C events, bacterial infections, and mortality at lower CD4 cell co
129  AMR, discovery of novel agents to treat AMR bacterial infections, and nonpharmacological strategies
130  the host immune response to prevent serious bacterial infections, and represent a significant advanc
131 ophils are the first line of defense against bacterial infections, and the generation of reactive oxy
132    Accordingly, complications from secondary bacterial infection are a leading cause of influenza-ass
133      Two broad categories of defense against bacterial infection are resistance, control of microbial
134                                    Secondary bacterial infections are a major source of associated mo
135                    Multidrug-resistant (MDR) bacterial infections are a serious threat to public heal
136                                    Recurrent bacterial infections are a significant burden worldwide,
137                         Antibiotic-resistant bacterial infections are a substantial source of morbidi
138            It is estimated that up to 80% of bacterial infections are accompanied by biofilm formatio
139                                More-frequent bacterial infections are accordingly encountered in neut
140                         More than 80% of the bacterial infections are associated with biofilm formati
141                                     Although bacterial infections are commonly identified, other micr
142 knowledge gap is particularly concerning, as bacterial infections are frequently complicated by sever
143 wever, their regulation and functions during bacterial infections are unclear.
144 olutions to effectively reduce the burden of bacterial infections are urgently needed.
145 rgm1 in mice leads to increased lethality to bacterial infections as well as enhanced inflammation to
146 iated molecular pattern during intracellular bacterial infections, as c-di-AMP secretion into the inf
147                                      Chronic bacterial infections associated with biofilm formation a
148  positron emission tomography probe for live bacterial infection based on the small-molecule antibiot
149 at risk of nocardiosis, a rare opportunistic bacterial infection, but prognosis and outcome of these
150 tivation was required for liver repair after bacterial infection, but resulted in fibrosis after peri
151 t infections are among the most common human bacterial infections, but extension to the kidneys is ra
152 termination factor Rho is used to treat some bacterial infections, but its importance varies across b
153 is prominent in murine lupus models and some bacterial infections, but the inhibitors and activators
154 used clinically to treat multidrug-resistant bacterial infections, but their poor physicochemical pro
155 ious approaches have been reported to combat bacterial infection by surface modifications to titanium
156 role in limiting host immunopathology during bacterial infections by controlling effector T cell acti
157 Tg(+)) mice successfully cleared spontaneous bacterial infections by PND22, the SHS-exposed Scnn1b-Tg
158 ells, stimulating the immune system to fight bacterial infections by promoting development of innate-
159 matory lesions and appears to be specific to bacterial infections by targeting the maltodextrin trans
160  particularly beneficial in the treatment of bacterial infections, by reducing the systemic and envir
161  Recent data from human studies suggest that bacterial infections can alter susceptibility to anaphyl
162     These results raise the possibility that bacterial infections can induce sensitivity to viral inf
163                                  Intentional bacterial infections can produce efficacious antitumor r
164                                              Bacterial infections can quickly turn into sepsis, with
165                                              Bacterial infection caused by intracellular pathogens, s
166 eals a deleterious consequence in intestinal bacterial infection caused by PFOS accumulation.
167                        Syphilis is a chronic bacterial infection caused by Treponema pallidum that is
168  peptide SET-M33 has strong activity against bacterial infections caused by Gram-negative bacteria.
169 his condition is characterized by fungal and bacterial infections caused by impaired generation of TH
170                                    Sepsis by bacterial infection causes high mortality in patients in
171 imultaneous detection of the two most common bacterial infections: Chlamydia trachomatis and Neisseri
172 ntially allow the prevention or treatment of bacterial infections circumventing some of the hurdles o
173 was associated with a reduced risk of severe bacterial infection compared with deferred ART (hazard r
174 were consistent when subgroups of the severe bacterial infection composite were analysed separately.
175                                              Bacterial infection confirmed by culture.
176  from uncontrolled inflammatory responses to bacterial infections, continues to cause high morbidity
177 aberrant production of complement C5a during bacterial infection could have broader effect on comprom
178                                              Bacterial infections developed in 4 infants (5%) and vir
179 g from inflammatory stimuli such as viral or bacterial infections during pregnancy serves as a risk f
180 tagging (IT) has revolutionised the study of bacterial infection dynamics in laboratory animal models
181 ive, no vaccines to protect from this severe bacterial infection exist.
182                       Differentiating active bacterial infection from sterile inflammation can be dif
183 gulates macrophage function, but its role in bacterial infection has not been investigated.
184 onventional antibiotics for the treatment of bacterial infections has become increasingly strained du
185 sed surveillance machinery for Gram-negative bacterial infections has been recently discovered.
186                              The increase in bacterial infections has led to urgent demands for simpl
187 wheezing illnesses associated with virus and bacterial infections have been identified as pivotal ris
188 tly greater mortality, an inability to fight bacterial infection, heightened levels of proinflammator
189 ection is to differentiate between viral and bacterial infections, hopefully facilitating clinical de
190  (HR, 0.42; 95% CI, 0.25-0.69; P = .001) and bacterial infections (HR, 0.44; 95% CI, 0.29-0.68; P < .
191  stratify young febrile infants for invasive bacterial infections (IBIs), defined as bacteremia and/o
192 HIV-positive people enrolled, 120 had severe bacterial infections (immediate-initiation group n=34, d
193 diabetes, refractive surgeries, and viral or bacterial infections impairs tear production, the blinki
194 on for active drug delivery to treat gastric bacterial infection in a mouse model using clarithromyci
195 rd injury directly causes increased risk for bacterial infection in mice as well as in patients.
196 te versus deferred ART on the risk of severe bacterial infection in people with high CD4 cell counts
197               High clinical risk factors for bacterial infection in recipients are often found in cri
198 er donors were significantly associated with bacterial infection in recipients.
199 unctional immunity capable of combating live bacterial infections in a mouse model.
200 ctively prevents not only the development of bacterial infections in all these indications but also f
201 macrophage oxidative responses in persistent bacterial infections in CF and create new opportunities
202 iae has virtually eliminated the concern for bacterial infections in children aged 3 to 36 months.
203 diate ART reduces the risk of several severe bacterial infections in HIV-positive people with high CD
204 s passeckerianus are important in combatting bacterial infections in human and veterinary medicine.
205 fault inflammasome response during viral and bacterial infections in human myeloid cells.
206 s a broad-spectrum antibiotic used to combat bacterial infections in humans and animals.
207 trated in both acute viral and intracellular bacterial infections in mice.
208 ococcus, GBS) is a leading cause of invasive bacterial infections in newborns, typically acquired ver
209  of antiretroviral therapy on risk of severe bacterial infections in people with high CD4 cell counts
210  that distinguished infants with and without bacterial infections in the test set with 87% (95% CI, 7
211 d infants with bacteremia from those without bacterial infections in the test set with 94% (95% CI, 7
212 py can effectively prevent the recurrence of bacterial infections in this context, SPAD should be con
213 d enable more favorable outcomes elicited by bacterial infections in tumor-bearing subjects.
214 infections (UTIs) are one of the most common bacterial infections in women.
215 odern patient is increasingly susceptible to bacterial infections including those due to multidrug-re
216 eostasis changes dramatically in response to bacterial infections, including production of metal sequ
217 ity and the healthcare costs associated with bacterial infections, including the implementation of lo
218                            Here we show that bacterial infection induces the cellular immune response
219                                 For example, bacterial infection is a key challenge in the treatment
220 od gene expression to identify subjects with bacterial infection is a promising strategy.
221                         Antibiotic-resistant bacterial infection is a serious threat to public health
222                      Rapid identification of bacterial infection is essential to ensure early appropr
223 rstanding of how the course of opportunistic bacterial infection is influenced by the microenvironmen
224 articipation of PTEN in host defense against bacterial infection is less well understood.
225 eive unnecessary antibiotic treatment, while bacterial infection is missed in others.
226                A broader approach to address bacterial infection is needed.
227 ontrol immune responses in the resolution of bacterial infections is critical for the development of
228  role of type I IFNs in host defense against bacterial infections is enigmatic.
229  RAGE signaling in response to opportunistic bacterial infections is largely unknown.
230 ole in regulating macrophage function during bacterial infections is not well characterized.
231 tes secrete proinflammatory cytokines during bacterial infection leading to biliary inflammation and
232 HFD-fed atERalphaKO involving severe uterine bacterial infections likely resulting from a previously
233 de/galactosamine) hepatitis and in models of bacterial infection (Listeria monocytogenes, lipopolysac
234  a potential radiopharmaceutical for imaging bacterial infection longitudinally in the lung.
235                     Here we show that during bacterial infection, lysozyme is rerouted via secretory
236 ed lung could be used to track the degree of bacterial infection measured by BLI, with a minimum dete
237 antibacterial activity in vitro and in acute bacterial infection models, but their activity in chroni
238                                              Bacterial infection modulates miRNA expression to subver
239 tentially be used to image and localize most bacterial infections, much like (18)F-FDG has been used
240  genes as optimal markers for discriminating bacterial infection (naive AUC = 0.94; nested CV-AUC = 0
241  that, at normal levels of expression during bacterial infection, NleB1/NleB(CR) antagonizes death re
242  pneumonia (MCPP), cases without a confirmed bacterial infection (nonconfirmed), cases confirmed for
243 pontaneous bacterial peritonitis is an acute bacterial infection of ascitic fluid; it has a high inci
244                          We demonstrate that bacterial infection of Drosophila females reduces ovipos
245                                              Bacterial infection of hepatocytes, which do not express
246                                       During bacterial infection of macrophages, RECON antagonized ST
247 of beclin-1-dependent autophagy during acute bacterial infection of the liver and spleen.
248 pace (apoplast) is another important step in bacterial infection of the phyllosphere.
249 ERalphaKO mice associated with a destructive bacterial infection of the uterus driven by commensal mi
250 , antibiotics are frequently used to prevent bacterial infections of larval bees, but the impact of a
251                                      Chronic bacterial infections of the lung are the leading cause o
252  months; 62% male) included 52 with definite bacterial infection, of whom 36 (69%) required intensive
253  lipopolysaccharide (LPS), which simulates a bacterial infection, on postnatal day 9.
254 s five [9.3%, 3.1-20.3], p=0.226), and brain bacterial infections (one [2.4%, 0.1-12.6] vs zero [0%,
255                                         Upon bacterial infection, one of the defense mechanisms of th
256                  We find that in response to bacterial infection or lipopolysaccharide (LPS) stimulat
257 test result, and the incidence of concurrent bacterial infections or the presence of multiple viruses
258    Toll-like receptor 4 (TLR4) activation by bacterial infection, or by sterile inflammatory insult i
259 tween-group difference in the rate of severe bacterial infection (P=0.32).
260 ing compounds for the treatment of ureolytic bacterial infections, particularly infections resulting
261 d inflammatory cytokine responses to LPS and bacterial infection, POP2 transgenic mice are more resis
262 ifying transplantation recipients at risk of bacterial infection prior to surgery and may contribute
263 for outpatient treatment of possible serious bacterial infection (PSBI) in neonates and young infants
264               Iron is a limiting nutrient in bacterial infection putting it at the centre of an evolu
265             Recent studies also suggest that bacterial infections regulate pain through direct action
266 crophage Nrp1 in host immune defense against bacterial infection remain unknown.
267                                       Occult bacterial infections represent a worldwide health proble
268  Application of ICL-1 in a model of systemic bacterial infection reveals increased iron accumulation
269 t a highly variable surface antigen prolongs bacterial infection sufficiently to exhaust the immune r
270  become hypersusceptible to fungal, and even bacterial infections, suggesting that Avr2 targets a con
271 , POP2 transgenic mice are more resistant to bacterial infection than wild-type mice.
272 ation is a promising strategy for addressing bacterial infection that focuses on removing the harmful
273 nza infection might predispose to subsequent bacterial infections that cause severe pneumonia.
274 sting possible protection against subsequent bacterial infections that influenza infection might pred
275 pensated cirrhosis, are at increased risk of bacterial infections that may further precipitate other
276                  The surge in drug-resistant bacterial infections threatens to overburden healthcare
277 oblasts in contributing to responses against bacterial infection through activation of bacteria-speci
278 ntimicrobial, M1 macrophage responses during bacterial infections through histone modifications and a
279 ophila the Imd pathway detects Gram-negative bacterial infections through recognition of diaminopimel
280 otic cell death, in the context of viral and bacterial infections, tissue damage, and inflammation.
281 mps to treat a wide variety of diseases from bacterial infection to cancer, due to the vast presence
282 growth, which can extend over days and allow bacterial infections to advance before effective antibio
283                    The recalcitrance of many bacterial infections to antibiotic treatment is thought
284 tic impact on persistent, biofilm-associated bacterial infection treatments.
285 t that silencing of AtRAP by AtlsiRNA-1 upon bacterial infection triggers defense responses through r
286         Importantly, a significantly reduced bacterial infection was observed in the middle ear of th
287                           Likewise, systemic bacterial infection was worsened by heightened IL-4 acti
288  A random sample of infants with and without bacterial infections was selected for RNA biosignature a
289       Constrained logistic models to predict bacterial infection were fit using screened LASSO.
290                     Genes discriminatory for bacterial infection were identified using the Bonferroni
291 mples from patients with Still's disease and bacterial infections were used also.
292 aly, such as genetic disorders and viral and bacterial infections, were negative, and the ZIKV genome
293 ncreased susceptibility to several viral and bacterial infections, whereas moderate alcohol consumpti
294 atment since the mid-twentieth century, this bacterial infection, which is caused by Treponema pallid
295 l hazards regression to model time to severe bacterial infection, which was defined as a composite en
296 y for new opportunities in the prevention of bacterial infections, which is crucial in times when ant
297 helium impairs mucus removal and facilitates bacterial infection with increased cough, sputum product
298 nt for the sensitive and specific imaging of bacterial infection with strong translational potential.
299 us pneumoniae is a leading cause of invasive bacterial infections, with nasal colonization an importa
300 myositis model, [(18)F]FPTMP identified live bacterial infection without demonstrating confounding in

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