1 icles, as indicated by plaque formation in a bacterial lawn.

2 rains kill these amoebae and leave an intact bacterial lawn.

3 n prior to application to the phage-infected bacterial lawn.

4  in cell migration, forming large plaques on bacterial lawns.

5  on these bacteria and form plaques in their bacterial lawns.

6 enter development and display slow growth on bacterial lawns.

7           Solitary foragers move slowly on a bacterial lawn and disperse across it, while social fora

8 e atg1-1 mutant, which does not aggregate on bacterial lawns and arrests as loose mounds on nitrocell

9 r primarily clear circular zones of lysis on bacterial lawns at the site of gamma phage inoculation a

10 phagy mutants do not aggregate in plaques on bacterial lawns, but they do proceed further in developm

11 ses of bacteria, form visible lesions within bacterial lawns (called plaques), which are employed ubi

12                         When developing on a bacterial lawn, cells of the mutant strain (dtfA- cells)

13  The DdPPK1 mutant formed smaller plaques on bacterial lawns compared with those of the WT.

14  bodies that do form when cells are grown on bacterial lawns lack the one- and two-dimensional symmet

15               The carbohydrate is present in bacterial lawns prior to addition of nematodes, indicati

16 , and atg8(-) is more aberrant in plaques on bacterial lawns than on nitrocellulose filters.

17                                In the assay, bacterial lawns were grown on agar plates, harvested wit

18 y evaluation showed improved robustness when bacterial lawns were tested with high- and low-density i