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1 individual responses to this common songbird bacterial pathogen.
2 ts(-/-) mice to infection by a Gram-negative bacterial pathogen.
3 nes is a major intracellular human foodborne bacterial pathogen.
4 with the evolution of niche adaptation in a bacterial pathogen.
5 nse of the intact animal against an ingested bacterial pathogen.
6 56.5%) specificity for identification of any bacterial pathogen.
7 s infection than from infection by any other bacterial pathogen.
8 tiated by Yersinia pestis, a highly virulent bacterial pathogen.
9 izations and deaths than any other foodborne bacterial pathogen.
10 uired for the inducible immune response to a bacterial pathogen.
11 enes dictate the immune pathogenesis of this bacterial pathogen.
12 stant and susceptible to infection with oral bacterial pathogens.
13 re activities and to examine the samples for bacterial pathogens.
14 (NOG1), functions for plant immunity against bacterial pathogens.
15 infectious diseases, such as those caused by bacterial pathogens.
16 ted with increased pilus production in other bacterial pathogens.
17 ming proteins secreted by many Gram-positive bacterial pathogens.
18 asion strategy that may be employed by other bacterial pathogens.
19 (Vibrio cholerae and Staphylococcus aureus) bacterial pathogens.
20 vices in the virulence of many Gram-negative bacterial pathogens.
21 onal response of primary macrophages to live bacterial pathogens.
22 ipulation of host cell signaling in favor of bacterial pathogens.
23 aling and early plant defense in response to bacterial pathogens.
24 an is integral to detection of gram-positive bacterial pathogens.
25 critical first-line defense against invading bacterial pathogens.
26 (AR) and spread of multidrug-resistant (MDR) bacterial pathogens.
27 ntial for mucosal immunity against pulmonary bacterial pathogens.
28 aining proteins represent an iron source for bacterial pathogens.
29 ribute to the virulence and pathogenicity of bacterial pathogens.
30 ctin structures induced by various viral and bacterial pathogens.
31 tivation of htrA has been described for many bacterial pathogens.
32 w these states affect and can be affected by bacterial pathogens.
33 hages could promote within-host evolution of bacterial pathogens.
34 he most important and most virulent invasive bacterial pathogens.
35 elucidation of disease-causing mutations in bacterial pathogens.
36 nts enabling detection and quantification of bacterial pathogens.
37 regulatory function for ANRs among different bacterial pathogens.
38 rate phages drive genomic diversification in bacterial pathogens.
39 rest as an approach to suppress virulence in bacterial pathogens.
40 ors of oxidative stress responses in several bacterial pathogens.
41 l as well as the destruction of a variety of bacterial pathogens.
42 ed 10 peptides and tested them against known bacterial pathogens.
43 e development and evolution of resistance of bacterial pathogens.
44 a harsh environment for clinically important bacterial pathogens.
45 with the intracellular survival of multiple bacterial pathogens.
46 toxin virulence factors from important human bacterial pathogens.
47 cluding those that regulate cell division in bacterial pathogens.
48 tiology that includes a variety of viral and bacterial pathogens.
49 in membrane protrusions or at the surface of bacterial pathogens.
50 play a role in healing and host response to bacterial pathogens.
51 in which bees were exposed to opportunistic bacterial pathogens.
52 o combat multidrug- and carbapenem-resistant bacterial pathogens.
53 new mechanisms in other medically important bacterial pathogens.
55 mpylobacter jejuni is a helix-shaped enteric bacterial pathogen and a common cause of gastroenteritis
58 is the single leading cause of death from a bacterial pathogen and estimated to be the leading cause
59 Burkholderia pseudomallei, an intracellular bacterial pathogen and the causative agent of human meli
60 with higher affinity, lower Kd, than several bacterial pathogens and competitively excluded them from
61 agnostic tests include culture-dependent for bacterial pathogens and culture-independent methods for
62 st multi-drug resistant clinical isolates of bacterial pathogens and include three novel scaffolds, w
63 o macrophage function in response to enteric bacterial pathogens and is a potential therapeutic targe
64 y, the discovery of GTase toxins produced by bacterial pathogens and secreted into the host cells has
65 ecent increase in extensively drug-resistant bacterial pathogens and the associated increase of morbi
66 ensitive RNA-based analysis of intracellular bacterial pathogens and their hosts without physical sep
68 the cell surface to function in adhesion for bacterial pathogens, and are critical for virulence.
69 studying the interaction of the environment, bacterial pathogens, and the host in disease dynamics.
76 oupled to the capsular polysaccharide of the bacterial pathogen, are the most effective strategy for
77 Borrelia, and perhaps other tick-transmitted bacterial pathogens, are uniquely adapted to survive in
78 Pseudomonas aeruginosa is a Gram-negative bacterial pathogen associated with acute and chronic inf
80 ctural moieties of its natural Gram negative bacterial pathogen-associated molecular pattern (PAMP) c
82 nables Ca(2+) -signalling specificity during bacterial pathogen attack and is required for a complete
83 highlight how successful, highly infectious bacterial pathogens avoid or subvert host autophagy mech
84 molecule for flow-resistant adhesion of the bacterial pathogen B. garinii on human endothelial cells
85 es a universal platform for the detection of bacterial pathogens based on engineered peptides, as alt
87 viding rapid and sensitive discrimination of bacterial pathogens both individually, and within a mult
88 , the extreme virulence of two opportunistic bacterial pathogens, Burkholderia thailandensis and Chro
89 hanges present in successful host-restricted bacterial pathogens but never before observed on this ti
90 ld perfectly discriminated between viral and bacterial pathogens, but higher procalcitonin strongly c
91 recognition of both viral and intracellular bacterial pathogens, but its regulation remains unclear.
92 se RNase E in shaping the transcriptome of a bacterial pathogen by functioning as both a degradative
93 complexes that initiate host defense against bacterial pathogens by activating caspase-1-dependent cy
97 ction in the relative abundance of potential bacterial pathogens capable of causing disease in humans
100 TcdA) and toxin B (TcdB) are produced by the bacterial pathogen Clostridium difficile and are respons
102 antimicrobial resistance (AMR) mechanisms in bacterial pathogens, coupled with the dwindling number o
103 fp), which are key virulence factors in many bacterial pathogens, define a large group of multipurpos
104 efense against a highly antibiotic-resistant bacterial pathogen depends on crosstalk between inflamma
108 and colleagues uncover a strategy whereby a bacterial pathogen disables the plant immune system with
110 ated platform enabling the identification of bacterial pathogen DNA sequences in complex samples in l
111 Haemophilus influenzae (NTHi) is the leading bacterial pathogen during chronic obstructive pulmonary
114 is a crucial pathway for growth of the human bacterial pathogen Enterococcus faecalis The final enzym
118 Upon entry into host cells, intracellular bacterial pathogens establish a variety of replicative n
120 sceptible to pulmonary infections, including bacterial pathogens, even after hematopoietic reconstitu
123 f this primitive TLR and identification of a bacterial pathogen for N. vectensis reveal ancient TLR f
125 hich may represent a general mechanism among bacterial pathogens for activating cell association.
126 ty had increased resistance to the cytosolic bacterial pathogen Francisella novicida Genetic deletion
128 Tularemia is caused by the Gram-negative bacterial pathogen Francisella tularensis Infection of m
129 lecular mechanisms used by the intracellular bacterial pathogens Francisella spp. and Brucella spp. t
131 sociated virulence factors of the widespread bacterial pathogen group A Streptococcus, are antigenica
138 Immunization strategies against commensal bacterial pathogens have long focused on eradicating asy
139 lays a dominant role in the evolution of the bacterial pathogen Helicobacter pylori, but its dynamics
140 way has been shown to suppress the growth of bacterial pathogens; however, the identification and mod
141 tics have been highly effective against most bacterial pathogens; however, the increasing resistance
146 and blood culture volume affect detection of bacterial pathogens in children with pneumonia and shoul
147 hes for the rapid and sensitive detection of bacterial pathogens in crude food samples utilizing a th
149 When model SPR detections of food-borne bacterial pathogens in homogenized foods are used, it is
150 difficile has become one of the most common bacterial pathogens in hospital-acquired infections in t
151 hat invade and kill a range of Gram-negative bacterial pathogens in natural environments and in vitro
154 ens tested, 1603 (37.3%) were positive for a bacterial pathogen, including 1147 (71.5%) that were pos
156 ng problem of antimicrobial resistance among bacterial pathogens, including methicillin-resistant Sta
157 n emerging understanding that the ability of bacterial pathogens, including multidrug-resistant organ
158 action and have activity against a range of bacterial pathogens, including strains resistant to fluo
160 of infections caused by multidrug-resistant bacterial pathogens increases with hospital length of st
162 Work investigating gammaherpesviruses and bacterial pathogens indicates that microbial pathogens d
163 the colony that helps protect the brood from bacterial pathogen infection, resulting in a lower colon
167 arsenal of effector proteins is injected by bacterial pathogens into the host cell or its vicinity t
168 tration (MIC) of an antimicrobial drug for a bacterial pathogen is used as a measure of the bacterial
172 identification of the sequence type (ST) of bacterial pathogens is critical for epidemiological surv
174 t the prevalence of antibiotic resistance in bacterial pathogens is important for public health.
175 agents that target the virulence factors of bacterial pathogens is one way to begin to address the i
184 olic growth of the facultative intracellular bacterial pathogen Listeria monocytogenes Two days after
185 ransmission of the facultative intracellular bacterial pathogen Listeria monocytogenes, most of the b
186 s of polysaccharide deacetylases (PDAs) from bacterial pathogens, modifying the protein backbone at t
187 mechanism by which an obligate intracellular bacterial pathogen modulates ERAD to satisfy its nutriti
189 For example, tuberculosis, caused by the bacterial pathogen Mycobacterium tuberculosis, requires
190 one of the world's most prevalent and deadly bacterial pathogens, Mycobacterium tuberculosis (Mtb) in
191 e system, house finches and the conjunctival bacterial pathogen Mycoplasma gallisepticum (MG), to exp
192 nd tolerance of infection with B. cepacia, a bacterial pathogen of rising importance in hospital-acqu
195 and Streptococcus pneumoniae, Gram-positive bacterial pathogens of significant clinical concern, use
198 or prevention of acute lung injury caused by bacterial pathogens or excessive mechanical ventilation.
200 ed that ILK1 contributes to plant defense to bacterial pathogens, osmotic stress sensitivity, and cel
201 ilar selection pressures on mutation rate as bacterial pathogens, particularly during long periods of
203 gly correlated with increased probability of bacterial pathogens, particularly typical bacteria.
204 ADP-ribosyltransferase TccC3 from the insect bacterial pathogen Photorhabdus luminescence modifies ac
205 ied that, following perception of fungal and bacterial pathogens, plant cells initially close their P
208 oodborne illnesses have shown that foodborne bacterial pathogens present a significant threat to publ
209 AMR and predict the resistance phenotypes of bacterial pathogens prior to culturing could inform clin
212 reak caused by a naturally occurring mutator bacterial pathogen provides a dramatic example of the po
215 is required for proper basal immunity to the bacterial pathogen Pseudomonas syringae Although SARD4 k
216 ts showed enhanced disease resistance to the bacterial pathogen Pseudomonas syringae and the oomycete
217 ere we show that miR863-3p is induced by the bacterial pathogen Pseudomonas syringae carrying various
219 , ultimately affecting susceptibility to the bacterial pathogen Pseudomonas syringae Glucose-6-phosph
220 n with virulent and avirulent strains of the bacterial pathogen Pseudomonas syringae pv tomato DC3000
221 1-1 showed a similar disease response to the bacterial pathogen Pseudomonas syringae pv. tomato (Pst)
222 c acid, and show increased resistance to the bacterial pathogen Pseudomonas syringae These results su
223 mutants show increased susceptibility to the bacterial pathogen Pseudomonas syringae, consistent with
235 ar Typhi is a human-restricted Gram-negative bacterial pathogen responsible for causing an estimated
237 nt profiling by sequencing (Grad-seq) in the bacterial pathogen Salmonella enterica, partitioning its
238 ential virulence factor of the human-adapted bacterial pathogen Salmonella Typhi (6,7) , the cause of
240 treptococcus intermedius is an opportunistic bacterial pathogen secreting a human-specific cytolysin
242 ion with extracellular microbes, such as the bacterial pathogen Staphylococcus aureus Recruitment and
243 IsdB is a receptor on the surface of the bacterial pathogen Staphylococcus aureus that extracts h
244 icrobe, Grayczyk et al. (2017) show that the bacterial pathogen Staphylococcus aureus unexpectedly se
245 lamuthia mandrillaris and Acanthamoeba), six bacterial pathogens (Streptococcus pneumoniae, Haemophil
246 I.8 is required for basal resistance against bacterial pathogens, substantiating a role for eNAD(+) i
249 defense against infections by intracellular bacterial pathogens, such as virulent Francisella tulare
250 n this review, we focus on how intracellular bacterial pathogens target innate immune signaling, the
251 t Enterobacteriaceae (CRE) are high-priority bacterial pathogens targeted for efforts to decrease tra
254 , or group A Streptococcus (GAS), is a human bacterial pathogen that can manifest as a range of disea
255 Francisella tularensis in an intracellular bacterial pathogen that causes a potentially lethal dise
257 during infection with Helicobacter pylori, a bacterial pathogen that causes persistent inflammation a
262 ui is a multihost, facultative intracellular bacterial pathogen that primarily causes pneumonia in fo
263 Legionella pneumophila is an intracellular bacterial pathogen that replicates in alveolar macrophag
264 Francisella tularensis is a facultative bacterial pathogen that replicates intracellularly withi
265 aphylococcus aureus is a medically important bacterial pathogen that, during infections, acquires iro
266 ota is unable to prevent colonization by two bacterial pathogens that cause mortality in neonates.
267 ers the potential of enhancing resistance to bacterial pathogens that have evolved to evade FLS2-medi
268 rming MACPF domain, reduces the viability of bacterial pathogens that reside within membrane-bound co
271 evolutionarily convergent mechanism used by bacterial pathogens to escape host immune clearance and
272 A type 3 secretion system is used by many bacterial pathogens to inject proteins into eukaryotic c
273 scusses RNA-mediated mechanisms exploited by bacterial pathogens to successfully infect and colonize
274 the outer membrane proteome is critical for bacterial pathogens to survive environmental changes, su
275 hindered simultaneous profiling of host and bacterial pathogen transcripts from the same sample.
278 Shigella, like many other Gram-negative bacterial pathogens, uses a type III secretion system to
279 le matrix followed by specifically detecting bacterial pathogens using SERS active nanoparticles func
281 for the isolation and detection of multiple bacterial pathogens via magnetic separation and SERS.
286 ical feature, but a high positive result for bacterial pathogens was observed among patients with dac
287 the primary host response to infection by a bacterial pathogen, we evaluated the response of Sts(-/-
288 resolve environmental enteropathy and clear bacterial pathogens, we aimed to assess whether antibiot
290 s are classical virulence factors of several bacterial pathogens which disrupt epithelial barrier fun
292 proteins contributes to the defence against bacterial pathogens, which need iron for their metabolis
293 nts were also more susceptible to fungal and bacterial pathogens, while infection and colonization by
295 facilitated rapid and sensitive detection of bacterial pathogens with a detection limit of 10(2)CFU/m
300 ector-triggered immunity." The Gram-negative bacterial pathogen Yersinia inactivates critical protein
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