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1 trocytic ascites is a variant of spontaneous bacterial peritonitis.
2 s due to Entamoeba histolytica as well as to bacterial peritonitis.
3 enteric infection, and possibly spontaneous bacterial peritonitis.
4 lammation and sepsis that are symptomatic of bacterial peritonitis.
5 e a mortality rate comparable to spontaneous bacterial peritonitis.
6 f peritoneal macrophages, and (iii) clearing bacterial peritonitis.
7 t worsen vasodilatation, such as spontaneous bacterial peritonitis.
8 hallenge with other TLR agonists, as well as bacterial peritonitis.
9 ophilus influenzae rarely causes spontaneous bacterial peritonitis.
10 contribute to optimal survival during acute bacterial peritonitis.
11 from hospitalized patients with spontaneous bacterial peritonitis.
12 al in murine models of acute endotoxemia and bacterial peritonitis.
13 the host's systemic inflammatory response to bacterial peritonitis.
14 vity significantly increase the lethality of bacterial peritonitis.
15 significantly increased in a mouse model of bacterial peritonitis after Cx43 inhibition and in Cx43
17 utrophils are recruited normally to sites of bacterial peritonitis and chemical dermatitis, indicatin
18 scites and hepatorenal syndrome, spontaneous bacterial peritonitis and hepatic encephalopathy, as wel
19 duces mortality in patients with spontaneous bacterial peritonitis and improves outcome following lar
20 neal dialysis patients presenting with acute bacterial peritonitis and monitoring individuals before
21 vo response to a well-characterized model of bacterial peritonitis and sepsis induced by cecal ligati
22 ovement in c-statistic: <.01 for spontaneous bacterial peritonitis and variceal hemorrhage to ascites
23 mRNA levels were increased in mice following bacterial peritonitis, and blocking an endogenous tumor
24 of cirrhosis, including ascites, spontaneous bacterial peritonitis, and esophageal variceal hemorrhag
26 al bleeding, serious infections, spontaneous bacterial peritonitis, and hepatorenal syndrome (RR = 0.
27 ly community-acquired pneumonia, spontaneous bacterial peritonitis, and IgE-mediated food allergy.
28 with interrupted uteroplacental blood flow, bacterial peritonitis, and older age but not in fetuses
29 xis, acute bacterial meningitis, spontaneous bacterial peritonitis, and outpatient gonococcal infecti
30 lbumin, blood urea, ascites, and spontaneous bacterial peritonitis as factors significantly predictiv
31 l of normal C57BL/6 mice in a model of acute bacterial peritonitis, cecal ligation and puncture (CLP)
32 dict clinical outcomes (ascites, spontaneous bacterial peritonitis, Child-Turcotte-Pugh score >or=7 o
33 enteric infection, and possibly spontaneous bacterial peritonitis, community-acquired pneumonia, and
34 factor were analyzed in patients with overt bacterial peritonitis during the first 5 days of infecti
36 ld-Turcotte-Pugh score, ascites, spontaneous bacterial peritonitis, hepatic encephalopathy, variceal
37 iagnoses as symptomatic ascites, spontaneous bacterial peritonitis, hepatorenal syndrome, or fulminan
38 revalence and natural history of spontaneous bacterial peritonitis in asymptomatic patients with asci
39 sment of diagnostic criteria for spontaneous bacterial peritonitis in outpatients may be required.
41 NLRP3 inflammasome is activated during acute bacterial peritonitis in patients on PD, and this activa
44 We describe a typical case of spontaneous bacterial peritonitis in which the causative organism wa
45 ations (ascites, encephalopathy, spontaneous bacterial peritonitis) increased with severity of AKI.
46 OFA score, bleeding, female sex, spontaneous bacterial peritonitis, intermediate increases in serum c
51 om individuals with a history of spontaneous bacterial peritonitis, known to have defective cellular
53 the cells that produced IFN-gamma in a mouse bacterial peritonitis model and determine the effects of
55 ysis, antibiotic prophylaxis for spontaneous bacterial peritonitis (odds ratio [OR] 8.3, P=0.002), po
56 ermined by diagnoses of ascites, spontaneous bacterial peritonitis, or esophageal variceal hemorrhage
57 utpatient diagnoses for ascites, spontaneous bacterial peritonitis, or esophageal variceal hemorrhage
58 tion, renal replacement therapy, spontaneous bacterial peritonitis, positive blood culture, and infec
60 act infection (UTI) (28.5%), and spontaneous bacterial peritonitis (SBP) (22.5%) were the most preval
61 e major infectious diseases were spontaneous bacterial peritonitis (SBP) (645, 49.8%), urinary tract
62 D) score over 20 points and with spontaneous bacterial peritonitis (SBP) as a precipitating event was
64 own to decrease the incidence of spontaneous bacterial peritonitis (SBP) in patients with cirrhosis a
68 , urinary tract infection (UTI), spontaneous bacterial peritonitis (SBP), and sepsis without specific
72 Urinary tract infections (52%), spontaneous bacterial peritonitis (SBP, 23%) and spontaneous bactere
73 dition, malignancy-related ascites (13%) and bacterial peritonitis specimens (5.8%) occasionally yiel
74 , ascites, hepatorenal syndrome, spontaneous bacterial peritonitis, the cardiopulmonary complications
76 30% for secondary prophylaxis of spontaneous bacterial peritonitis, to 90% for assays for cell number
77 k of infection: prior episode of spontaneous bacterial peritonitis, upper gastrointestinal bleeding,
78 ly; P < 0.0001), particularly in spontaneous bacterial peritonitis, urinary tract infection, and pneu
79 lications of cirrhosis (ascites, spontaneous bacterial peritonitis, varices, variceal hemorrhage, enc
80 techniques for the diagnosis of spontaneous bacterial peritonitis, we prospectively compared three b
81 arcinoma, variceal bleeding, and spontaneous bacterial peritonitis were associated with a higher mort
82 blood vessels, restricted dietary intake, or bacterial peritonitis were compared with tissues from TM
83 involved in the pathogenesis of spontaneous bacterial peritonitis, which is a common complication of
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