ライフサイエンスコーパス: bacterial translocation

1 itated by microbial dysbiosis and associated bacterial translocation.

2 There was no bacterial translocation.

3 IL-1alpha significantly reduced the rates of bacterial translocation.

4 y to the intestine, and an increased rate of bacterial translocation.

5 ls prevented an increase in MPO activity and bacterial translocation.

6 ith subsequent increases in permeability and bacterial translocation.

7 eading to increased mucosal permeability and bacterial translocation.

8 intestinal barrier, which leads to increased bacterial translocation.

9 nce, intestinal mucus and mucin content, and bacterial translocation.

10 bowel allograft rejection without increasing bacterial translocation.

11 he density of mucosa-associated bacteria and bacterial translocation.

12 that experimental liver fibrosis depends on bacterial translocation.

13 ores epithelial barrier function and reduces bacterial translocation.

14 sulfonic acid-induced colitis and associated bacterial translocation.

15 al integrity leading to gut permeability and bacterial translocation.

16 at CD4 T cell dysfunction might be caused by bacterial translocation.

17 n intestinal T cell suppression and enhanced bacterial translocation.

18 rvested under aseptic conditions to quantify bacterial translocation.

19 gesting an important role for PAFR-dependent bacterial translocation.

20 poptosis, intestinal barrier disruption, and bacterial translocation.

21 y a single site on this protein required for bacterial translocation.

22 ters and disorders associated with increased bacterial translocation.

23 hat intestinal epithelial hypoxia influences bacterial translocation.

24 mucosal injury, luminal fluid secretion, and bacterial translocation.

25 ent of processed foods and that can increase bacterial translocation across epithelia in vitro, might

26 We hypothesized that bacterial translocation across the intact barrier occurs

27 L-1alpha reduces intestinal permeability and bacterial translocation after burn and sepsis.

28 were examined for viability, apoptosis, and bacterial translocation after exposure to a series of in

29 e reports of leakage of LPS from the gut and bacterial translocation after injury in animal models, t

30 this study was to test whether iNOS mediates bacterial translocation after intestinal ischemia-reperf

31 ups than in controls, supporting the role of bacterial translocation and activation of the inflammato

32 eem to indicate that there is little risk of bacterial translocation and contamination from the conju

33 Advanced liver disease predisposes to bacterial translocation and endotoxaemia which can contr

34 ypothesis that altered gut permeability with bacterial translocation and endotoxaemia would be increa

35 acterial challenge model, was used to assess bacterial translocation and gut colonization.

36 out mice were resistant to endotoxin-induced bacterial translocation and ileal mucosal damage.

37 helial permeability and leads to exaggerated bacterial translocation and increased mortality during p

38 tility may be involved in the development of bacterial translocation and infection in patients with l

39 severe shock states is often associated with bacterial translocation and intestinal barrier dysfuncti

40 Protein malnutrition was not associated with bacterial translocation and measurement of enteroadheren

41 Given the strong association between bacterial translocation and mucosal inflammatory disease

42 t to intestinal ischemia-reperfusion-induced bacterial translocation and mucosal injury than wild-typ

43 hine use or abuse results in significant gut bacterial translocation and predisposes patients to seri

44 of the intestinal epithelial barrier allows bacterial translocation and predisposes to destructive i

45 protected against lipopolysaccharide-induced bacterial translocation and prevented the lipopolysaccha

46 anslocation and that IVIG treatment resolves bacterial translocation and restores CD4 T cell function

47 stance of mice irradiated with gamma-rays to bacterial translocation and subsequent sepsis.

48 Bacterial translocation and systemic endotoxemia have be

49 observed in CVID patients is associated with bacterial translocation and that IVIG treatment resolves

50 al barrier dysfunction may lead to secondary bacterial translocation and the development of the multi

51 ntial regulatory mechanism implicated (i.e., bacterial translocation), and its role in conditions of

52 enhanced intestinal epithelial permeability, bacterial translocation, and elevated colonic lymphocyte

53 y impaired C. difficile clearance, increased bacterial translocation, and elevated levels of endotoxi

54 lantation contributing to graft dysmotility, bacterial translocation, and possibly, acute rejection.

55 al colonization of mucosal surfaces, reduces bacterial translocation, and protects mice from alcohol-

56 Fibrosis, intestinal permeability, bacterial translocation, and serum endotoxemia were meas

57 increased postburn barrier permeability and bacterial translocation associated with deranged neutrop

58 partially explain the increased frequency of bacterial translocation associated with tissue ischemia.

59 e resistant to such therapies as a result of bacterial translocation at the time of transplantation,

60 he membrane cycling of SecA, the cytoplasmic bacterial translocation ATPase, and in the stabilizing o

61 ulation, and its severity has been linked to bacterial translocation (BT) and endotoxemia.

62 Liver cirrhosis is associated with bacterial translocation (BT) and endotoxemia.

63 Bacterial translocation (BT) has been suggested to be re

64 1) and exogenous HMGB1 is able to induce gut bacterial translocation (BT) in normal mice; therefore,

65 Here, we investigated the impact of bacterial translocation (BT) to MLNs on TNF-alpha produc

66 Bacterial translocation (BTL) drives pathogenesis and co

67 in pharmacologic antagonists of PAFr blocked bacterial translocation by as much as 80 +/- 6%.

68 se studies indicate that enhanced intestinal bacterial translocation caused by burn injury could be r

69 Sham operation did not induce bacterial translocation, change cecal bacterial populati

70 The aims of our study were to investigate bacterial translocation, changes in the enteric microbio

71 inal barrier permeability and an increase in bacterial translocation compared to infection with curli

72 c response without increased inflammation or bacterial translocation compared with controls.

73 ileal leakage of horseradish peroxidase, and bacterial translocation compared with food deprivation (

74 The crystal structure of a bacterial translocation complex describes the binding st

75 Intestinal inflammation and bacterial translocation contribute to liver fibrosis via

76 ccurred after SMAO than in the mice in which bacterial translocation did not occur after SMAO.

77 sham SMAO or mice subjected to SMAO in which bacterial translocation did not occur.

78 Bacterial translocation did not reduce TER.

79 HMGB1 neutralization is associated with bacterial translocation during APAP hepatotoxicity.

80 nal epithelial barrier as well as monitoring bacterial translocation during infection.

81 Our data suggest that bacterial translocation, endotoxaemia, inflammation and

82 preserved gut barrier integrity and reduced bacterial translocation, epithelial inflammation, and ox

83 r, it is unclear whether clinically relevant bacterial translocation even occurs in humans, much less

84 In contrast, a second temporally distinct bacterial translocation event resulted in successful hep

85 derlying compromised gut immune function and bacterial translocation following morphine treatment.

86 and closure of alveoli (group 2) facilitated bacterial translocation from the alveoli to the bloodstr

87 has been shown to reduce hemorrhage-induced bacterial translocation from the gut in mice and rats.

88 Bacterial translocation from the gut is considered the k

89 This is potentially associated with bacterial translocation from the gut leading to local an

90 These mice also had increased enteric bacterial translocation from the gut to the mesenteric l

91 at SCI increases intestinal permeability and bacterial translocation from the gut.

92 scites is associated with a high rate of gut bacterial translocation (GBT) and spontaneous bacterial

93 - and NO3- were highest in the mice in which bacterial translocation had occurred.

94 ects from HIV infection, including increased bacterial translocation, immune activation, and presence

95 ral nutrition and elemental diets both cause bacterial translocation, immune dysfunction, and increas

96 Intestinal ischemia-reperfusion caused bacterial translocation in 72% of the iNOS+/+ mice but o

97 Intravenous TPN caused greater bacterial translocation in all small intestinal segments

98 The mechanisms for bacterial translocation in cirrhosis that predisposes pa

99 s by cirrhotic plasma, suggesting a role for bacterial translocation in driving B-cell changes in cir

100 In view of the suggested role played by bacterial translocation in liver disease and obesity, we

101 the role of TLRs, intestinal microbiota and bacterial translocation in liver fibrosis, alcoholic liv

102 ased colonization of the small intestine and bacterial translocation in mice lacking Cd1d, an MHC cla

103 sts may prevent epithelial deterioration and bacterial translocation in patients with impaired bile f

104 Gastrointestinal (GI) bacterial translocation in sepsis is well known, but the

105 y cytokines in vitro and in vivo, aggravated bacterial translocation in TG mice under DSS treatment,

106 The incidence of bacterial translocation in the DuP753-treated animals wa

107 Studies of bacterial translocation in two models of human skin indi

108 s that did not express TLR2 also had reduced bacterial translocation, indicating that TLR2 expression

109 cus within the abdominal cavity, followed by bacterial translocation into the blood compartment, whic

110 ile the molecular mechanisms responsible for bacterial translocation into the heart have been elucida

111 rences in histology, cytokine expression and bacterial translocation into the mesenteric lymph node.

112 In acute pancreatitis (AP) bacterial translocation is considered as the key event l

113 A new component of the bacterial translocation machinery, YidC, has been identi

114 ses, and this work supports the concept that bacterial translocation may adversely affect host defens

115 To determine whether bacterial translocation may contribute to these effects,

116 lunts mastocytosis in ileal villi as well as bacterial translocation, measured as numbers of mesenter

117 n both the iNOS+/+ and iNOS-/- mice in which bacterial translocation occurred after SMAO than in the

118 mesenteric artery occlusion (SMAO) in which bacterial translocation occurred had cecal bacterial pop

119 Bacterial translocation occurred prior to changes observ

120 ation and one (endotoxin challenge) in which bacterial translocation occurs and intestinal morphology

121 hese data suggest a novel mechanism by which bacterial translocation occurs and suggest a critical ro

122 mic sepsis, although the mechanisms by which bacterial translocation occurs remain largely unknown.

123 Bacterial translocation of E. coli C-25 from the mucosal

124 t here a novel model of chemotherapy-induced bacterial translocation of E. coli.

125 Ileal myeloperoxidase activity and bacterial translocation of Enterococcus faecalis were as

126 Model systems that allow bacterial translocation of ExoS have found ExoS to have

127 Bacterial translocation of ExoS into epithelial cells is

128 eceptor (PAFR), which is known to potentiate bacterial translocation of gram-positive bacteria, was s

129 Further analyses of bacterial translocation of HopM1 and AtMIN7 stability in

130 y effect intestinal tissue damage leading to bacterial translocation of indigenous E. coli.

131 inal decontamination also suggest a role for bacterial translocation on TLR-4 activation in PFC after

132 inflammatory cytokine secretion, ameliorated bacterial translocation on treatment with dextran sulfat

133 Zinc supplementation diminished bacterial translocation only in lactose-challenged under

134 a substantially impaired ability to support bacterial translocation, particularly from blood to brai

135 creased enterocyte apoptosis and E. faecalis bacterial translocation (postburn day 3).

136 mice versus CTRL+BDL mice, suggesting higher bacterial translocation rate.

137 ses normal intestinal barrier mechanisms and bacterial translocation results.

138 This eradication prevented bacterial translocation, significantly reduced serum end

139 disruptions of the mucous barrier facilitate bacterial translocation that may contribute to the onset

140 al mucosal surface is an important factor in bacterial translocation, that intestinal mucus modulates

141 al barrier function are thought to result in bacterial translocation, the presence of bacterial produ

142 Gut colonization and bacterial translocation to liver, spleen, and mesenteric

143 We demonstrate significant bacterial translocation to mesenteric lymph node (MLN) a

144 regimen for ethyl pyruvate also ameliorated bacterial translocation to mesenteric lymph nodes and le

145 Twenty-four hours after reperfusion, bacterial translocation to mesenteric lymph nodes, ileal

146 Bacterial translocation to mesenteric lymph nodes, liver

147 d both ileal mucosal permeability to FD4 and bacterial translocation to mesenteric lymph nodes.

148 d to alter microbial composition and promote bacterial translocation to other tissues.

149 mice, epithelial proliferation decreased and bacterial translocation to the liver and spleen was dete

150 s of mucosa-associated bacteria and enhances bacterial translocation to the mesenteric lymph nodes an

151 The incidence of bacterial translocation to the mesenteric lymph nodes wa

152 ic intestinal inflammation, with evidence of bacterial translocation to the mesenteric lymph nodes, m

153 Results were correlated with bacterial translocation to the mesenteric lymph nodes.

154 T1D, which correlated with the abrogation of bacterial translocation to the PLNs.

155 Bacterial translocation to the spleen was demonstrated 2

156 rial adherence, mucus bacterial binding, and bacterial translocation, two models were used.

157 ral nutrition and enteral diets may pre-vent bacterial translocation via the preservation and augment

158 dy, the role of macrophages in rejection and bacterial translocation was evaluated by depleting macro

159 wild-type mice following bile duct ligation; bacterial translocation was facilitated by TNFRI-mediate

160 Bacterial translocation was investigated in the liver, s

161 Bacterial translocation was measured using agar cultures

162 disruption of the intestinal epithelium and bacterial translocation was no longer observed.

163 of enteroadherent bacteria was increased and bacterial translocation was observed.

164 LPS-induced bacterial translocation was reduced by aminoguanidine.

165 Bacterial translocation was significantly enhanced in Ta

166 Bacterial translocation was significantly increased afte

167 Bacterial translocation was significantly increased in t

168 Liver fibrosis and bacterial translocation were assessed in Toll-like recep

169 factor receptor (PAFr) in hypoxia-associated bacterial translocation, wherein pharmacologic antagonis

170 sbiosis, intestinal barrier dysfunction, and bacterial translocation, which trigger the state of pers