1 cluding the DosR master regulator of hypoxic bacteriostasis.

2 s in turn increased after antibiotic-induced bacteriostasis.

3 ediate amounts of RpoB knockdown resulted in bacteriostasis and a more substantial depletion led to a

4 in which PknB activity is reduced to achieve bacteriostasis, and elevated when replication resumes.

5 ygen tension is a major host factor inducing bacteriostasis, but the molecular mechanisms driving oxy

6 of IFN-gamma production may be important for bacteriostasis, IL-12 p70 is not necessary for appropria

7 e is characterized at 37 degrees C by either bacteriostasis in Ca(2+)-deficient medium with expressio

8 h DosR regulon expression is correlated with bacteriostasis in vitro and a proxy for latency in vivo.

9 ro activity as a ribonuclease and control of bacteriostasis in vivo.

10  analyses revealed that H2O2 exposure caused bacteriostasis in wild-type cells during which time SCVs

11 and LcrV is necessary but not sufficient for bacteriostasis of Ca(2+)-starved yersiniae and suggest t

12                                              Bacteriostasis of Y. pestis (but not enteropathogenic ye

13 h the toxin proposed as either an inducer of bacteriostasis or a mediator of programmed cell death (P

14              Antibiotics can cause dormancy (bacteriostasis) or induce death (cidality) of the target

15 rV is well defined but little is known about bacteriostasis other than that Na(+) plus l-glutamate pr

16                              Mutagenesis and bacteriostasis resulted.

17 ns may directly contribute to the NO-induced bacteriostasis under anaerobic conditions.

18 mune mechanisms in patient blood resulted in bacteriostasis, whereas administration of oral chemother

19 hages until the host's immune defense causes bacteriostasis, which leads the pathogen to go into nonr