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1  is an important determinant of the arterial baroreceptor reflex.
2 speed would reduce MSNA through the arterial baroreceptor reflex.
3 ypertension and possibly with changes in the baroreceptor reflex.
4 charge frequency of the S-A node through the baroreceptor reflex.
5 ch is critically important for mediating the baroreceptor reflex.
6 994, 5 microM) but this had no effect on the baroreceptor reflex.
7 vation of reflexes, primarily the sinoaortic baroreceptor reflex.
8 he baroreflex, all indicative of an impaired baroreceptor reflex.
9 ch may subserve respiratory chemosensory and baroreceptor reflexes.
10 nd activation of cardiopulmonary, chemo- and baroreceptor reflexes.
11 ractus solitarii (NTS) are necessary for the baroreceptor reflex, a primary mechanism for homeostatic
12 pressure to derive phenotypes related to the baroreceptor reflex, a short-term controller of blood pr
13 odels describing the coupled function of the baroreceptor reflex and mechanics of the circulatory sys
14 obstructive sleep apnoeic events, alters the baroreceptor reflex and this may lead to hypertension.
15 wn that eNOS may contribute to regulation of baroreceptor reflexes and arterial pressure, we examined
16                        This study determined baroreceptor reflex (BR) function in conscious rats whic
17                                          The baroreceptor reflex bradycardia was depressed significan
18     AT1 receptors in the NTS can depress the baroreceptor reflex bradycardia which is independent of
19 sing effect of cocaine because activation of baroreceptor reflexes decreases SNA, the neural stimulus
20 tivity in the NTS for chronically regulating baroreceptor reflex function in conscious rats.
21 iated signaling that includes a reduction in baroreceptor reflex function, presumably via a NADPH-ROS
22 d increase in BP may be due to a decrease in baroreceptor reflex function.
23  1 mM, 50 nl) also significantly reduced the baroreceptor reflex gain (63+/-8%, p<0.05).
24 try in freely moving animals and spontaneous baroreceptor reflex gain (sBRG) determined by a time-ser
25 ood pressure, and an increase in spontaneous baroreceptor reflex gain (sBRG).
26 ve within the NTS and is a factor regulating baroreceptor reflex gain and heart rate.
27 etry data revealed a decrease in spontaneous baroreceptor reflex gain following sEH inhibition, indic
28 ype 1 (AT1) receptor antagonist improves the baroreceptor reflex gain in spontaneously hypertensive r
29 diated depression of cardiac and sympathetic baroreceptor reflex gain in the WKY.
30                                              Baroreceptor reflex gain measured by the response in hea
31 artially reduced the effect of Ang II on the baroreceptor reflex gain.
32 pear to be involved in the modulation of the baroreceptor reflex in the dlNTS.
33 utamate receptors (mGluRs) in modulating the baroreceptor reflex in the rat NTS.
34 ular resistance and direct inhibition of the baroreceptor reflex, leading to increased blood pressure
35 kg) in conscious freely moving rats, (2) the baroreceptor reflex-mediated changes in heart rate elici
36  is D-SNC, (2) L- and D-SNC markedly inhibit baroreceptor reflex-mediated tachycardia in conscious ra
37  solutions) into the cNTS did not affect the baroreceptor reflex (P > 0.2) while pentobarbitone (100
38 est that a NOS-cGMP signalling system in the baroreceptor reflex pathway distal to the NTS and closer
39 that form the medullary efferent limb of the baroreceptor reflex pathway.
40 between pulmonary arterial and carotid sinus baroreceptor reflexes; physiological and pathological st
41 B3/Neuregulin signaling pathway but also the baroreceptor reflex response, which have been functional
42                                      Cardiac baroreceptor reflex sensitivity (BRS) was impaired in NP
43 s of ephedrine on cardiac autonomic control, baroreceptor reflex sensitivity (BRS), heart rate (HR) v
44  Possible mechanisms include improvements in baroreceptor reflex sensitivity and renal function, rest
45 at during exercise the reduction of arterial baroreceptor reflex sensitivity at the operating point w
46 llow-up: pharmacological baroreflex testing (baroreceptor reflex sensitivity), short-term spectral an
47  point analysis, microvolt T wave alternans, baroreceptor reflex sensitivity, and SD of all normal-to
48 urons are involved in the origination of the baroreceptor reflex, they suggest that only a modest par
49 ndary effects of hyperventilation and of the baroreceptor reflex to maintain cardiac output and there
50 resistance and asphyxia cause changes in the baroreceptor reflex which could lead to an increase in b

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