ライフサイエンスコーパス: baroreflex sensitivity

1 ither baseline cardiovascular parameters nor baroreflex sensitivity.

2 ence between control and occlusion groups in baroreflex sensitivity.

3 ied Oxford technique to assess cardiac vagal baroreflex sensitivity.

4 in an increase in HRV and an improvement in baroreflex sensitivity.

5 ivity and negatively correlated with cardiac baroreflex sensitivity.

6 heart, leading to hypertension and depressed baroreflex sensitivity.

7 vs. sham-operated SHR) and an improvement in baroreflex sensitivity.

8 trual cycle had no influences on cardiovagal baroreflex sensitivity.

9 ssed according to heart rate variability and baroreflex sensitivity.

10 renal sympathetic nerve activity and to test baroreflex sensitivity.

11 an increase in parasympathetic activity and baroreflex sensitivity.

12 ors with a possible reduction in sympathetic baroreflex sensitivity.

13 ood pressure and a smaller change in cardiac baroreflex sensitivity.

14 ivity to higher pressures without changes in baroreflex sensitivity.

15 , employing heart rate variability (HRV) and baroreflex sensitivity.

16 ympathetic nerve activity without changes in baroreflex sensitivity.

17 r the Fontan operation, with reduced HRV and baroreflex sensitivity.

18 transdermal nor oral ERT had any effects on baroreflex sensitivity.

19 d more impaired autonomic balance, ie, lower baroreflex sensitivity (1.4 +/- 1.3 versus 5.0 +/- 1.5 m

20 hostatic intolerance had lower cardiac vagal baroreflex sensitivity (12+/-1 versus 25+/-4 ms/mm Hg; P

21 antecedent hypoglycemia leads to 1) reduced baroreflex sensitivity (16.7 +/- 1.8 vs. 13.8 +/- 1.4 ms

22 Baroreflex sensitivity (adjusted odds ratio, 0.9; p = 0.

23 We have previously shown that depressed baroreflex sensitivity, an established marker of reduced

24 cal responsiveness to ACTH, but had enhanced baroreflex sensitivity and augmented plasma catecholamin

25 Fortunately, emerging data suggest that baroreflex sensitivity and autonomic function may be res

26 ostatic intolerance have lower cardiac vagal baroreflex sensitivity and marginally lower blood volume

27 Baroreflex sensitivity and the sympathetic response to h

28 goal was to test sympathetic and cardiovagal baroreflex sensitivity and the transduction of sympathet

29 ngiotensin II levels, inflammation, impaired baroreflex sensitivity, and autonomic dysfunction, as we

30 stress, impaired sympathetic and cardiovagal baroreflex sensitivity, and increased inflammation.

31 ormalized indexes of sympathetic outflow and baroreflex sensitivity, and reduced the incidence of apn

32 c nervous system--heart rate variability and baroreflex sensitivity--are reviewed, and the clinical a

33 Cardiovagal baroreflex sensitivity assessed during decreasing BP (i.

34 The spontaneous baroreflex sensitivity at baseline was significantly low

35 m, mean and maximum (+/-s.d.) supine control baroreflex sensitivities averaged 5 +/- 3, 18 +/- 6, and

36 fferences in the cardiovagal and sympathetic baroreflex sensitivities between phases under any condit

37 Similar sympathetic baroreflex sensitivity between sexes and phases was also

38 alysis of heart rate variability [HRV]), and baroreflex sensitivity (bolus phenylephrine method and a

39 systolic blood pressure, cardiac parameters, baroreflex sensitivity (BRS) and hyperinsulinemia in the

40 Although heart rate variability (HRV) and baroreflex sensitivity (BRS) are recognized as independe

41 This study sought to evaluate cardiac baroreflex sensitivity (BRS) as a predictor of response

42 Background- Cardiovagal baroreflex sensitivity (BRS) declines with age but is fa

43 lex function as indicated by the blunting of baroreflex sensitivity (BRS) following the antagonizatio

44 y on autonomic cardiovascular control and on baroreflex sensitivity (BRS) have not been studied.

45 Hemispheric influences on baroreflex sensitivity (BRS) have not yet been evaluated

46 Sympathetic baroreflex sensitivity (BRS) is greater during decreasin

47 ter standard tests of autonomic function and baroreflex sensitivity (BRS) measurement, diabetic parti

48 Sympathetic baroreflex sensitivity (BRS) was assessed.

49 Baroreflex sensitivity (BRS) was quantified from the R-R

50 Changes in heart rate variability (HRV) and baroreflex sensitivity (BRS) were also examined.

51 bute to sympathetic overactivity, attenuated baroreflex sensitivity (BRS), and excessive ventilation

52 ventilatory recruitment threshold (VRT-CO2), baroreflex sensitivity (BRS), blood pressure, and blood

53 Arterial baroreflex sensitivity (BRS), muscle sympathetic nerve a

54 Cardiovagal baroreflex sensitivity (BRS), the arterial baroreflex-me

55 ailure (CHF) results in blunting of arterial baroreflex sensitivity (BRS), which arises from alterati

56 es in sympathetic activity and reductions in baroreflex sensitivity (BRS).

57 mental stress, as well as impaired arterial baroreflex sensitivity (BRS).

58 ise-induced muscle chemoreflex activation on baroreflex sensitivity (BRS).

59 od pressure or heart rate, or to cardiovagal baroreflex sensitivity, but correlated with muscle sympa

60 /DeltaMAP, which does not involve changes in baroreflex sensitivity, but may involve changes in chemo

61 nism for the decrease in spontaneous cardiac baroreflex sensitivity (cBRS) during exercise in humans.

62 Sympathetic and cardiovagal baroreflex sensitivities change during the 28-day course

63 at 0.6 of gestation; however, fetal cardiac baroreflex sensitivity decreased with advancing gestatio

64 ared with baseline euglycemic conditions, 1) baroreflex sensitivity decreases significantly (19.2 +/-

65 Sympathetic baroreflex sensitivity did not differ between sexes (P =

66 d pressure and baroreflex threshold, reduced baroreflex sensitivity, diminished plasma catecholamine

67 MSNA, haemodynamic responses and baroreflex sensitivity during early tilting were not dif

68 athetic neural responses but not sympathetic baroreflex sensitivity during orthostasis, though uprigh

69 x sensitivity with OC differ from changes in baroreflex sensitivity during the normal menstrual cycle

70 We tested sympathetic and cardiovagal baroreflex sensitivity during the placebo or "low-hormon

71 sibly 'steady-state' conditions, human vagal baroreflex sensitivity fluctuates in a major way, at ver

72 t during brief periods of observation, human baroreflex sensitivity fluctuates widely and rhythmicall

73 Fast Fourier transforms indicated that baroreflex sensitivity fluctuations (expressed as percen

74 The periodicity of very low frequency baroreflex sensitivity fluctuations was not influenced s

75 rterial pressure, sympathovagal balance, and baroreflex sensitivity for control of heart rate.

76 ween arrhythmic events and predictive tests (baroreflex sensitivity, heart rate turbulence, heart rat

77 Mean values of baroreflex sensitivity, heart rate variability, intracra

78 mpathetic nerve activity and reduced cardiac baroreflex sensitivity heighten cardiovascular risk, alt

79 In addition, baroreflex sensitivity, hemodynamic responses to bolus i

80 er, intravenous CV-11974 failed to alter the baroreflex sensitivities in area postrema-lesioned SHRs.

81 We measured HRV and baroreflex sensitivity in 22 consecutive patients (8 mal

82 nin activity, aldosterone, urine sodium, and baroreflex sensitivity in both groups.

83 y 5959 affects the control of heart rate and baroreflex sensitivity in conscious dogs with pacing-ind

84 Thus, the calcium promoter restores arterial baroreflex sensitivity in HF.

85 hough studies have examined resting arterial baroreflex sensitivity in older subjects, little attenti

86 mpathetic nerve activity and reduced cardiac baroreflex sensitivity in patients with RA compared to m

87 of ANA-12 into the dmNTS greatly diminished baroreflex sensitivity in sham rats, whereas it had less

88 In addition, EX increased arterial baroreflex sensitivity in the CHF group (heart rate slop

89 s measurements of heart rate variability and baroreflex sensitivity in the neuromonitoring setting of

90 AT(1) receptor blockade enhanced baroreflex sensitivity in the non-EX CHF rabbits but had

91 Sympathetic baroreflex sensitivity increased from supine to upright

92 upled with impairments in renal function and baroreflex sensitivity, increased neuroinflammatory mark

93 To test the hypothesis that arterial baroreflex sensitivity increases during exercise-induced

94 eart rate variability, endothelial function, baroreflex sensitivity, inflammation, and platelet funct

95 Reduced baroreflex sensitivity is associated with nicotinamide a

96 Not only sympathetic but also cardiovagal baroreflex sensitivity is similar between sexes and mens

97 t, as measured by heart rate variability and baroreflex sensitivity, is significantly associated with

98 ns of physiological abnormalities: depressed baroreflex sensitivity low LF/HF low LF/(HF + LF) low al

99 Enhanced arterial baroreflex sensitivity may contribute to this reduction.

100 Thus, individual differences in baroreflex sensitivity may explain the hemodynamic varia

101 function, including decreased cardiac vagal baroreflex sensitivity, may contribute directly to morta

102 ing age, left ventricular ejection fraction, baroreflex sensitivity, mean RR interval, standard devia

103 thetic nerve activity (MSNA) and sympathetic baroreflex sensitivity (MSNA-diastolic pressure relation

104 ivity (n=38), heart rate variability (n=34), baroreflex sensitivity (n=20), and ergoreflex activity (

105 roreflex dysfunction (defined by spontaneous baroreflex sensitivity of <6 ms mm Hg).

106 Hypoxia had no significant effect on baroreflex sensitivity or 'set point' for the control of

107 No significant differences in cardiovagal baroreflex sensitivity or vascular transduction were obs

108 ent of heart rate variability (P<0.0001) and baroreflex sensitivity (P=0.03), and overactive ergorece

109 , it still decreased heart rate and restored baroreflex sensitivity (PI/SAP slope, 12.7+/-2.8 ms/mm H

110 central chemoreceptor sensitivity, arterial baroreflex sensitivity, plasma norepinephrine, epinephri

111 0.15 Hz) (r=-.52, P=.006) and inversely with baroreflex sensitivity (r=-.60, P=.005).

112 ver, their specific contribution to arterial baroreflex sensitivity remains unclear.

113 Spontaneous baroreflex sensitivity (SBR) was evaluated as the slope

114 emetry), autonomic function, and spontaneous baroreflex sensitivity (SBRS) were not significantly dif

115 Baroreflex sensitivity showed no correlation with intrac

116 Sympathetic baroreflex sensitivity was assessed by intravenous doses

117 Sympathetic baroreflex sensitivity was assessed by lowering and rais

118 Baroreflex sensitivity was assessed every 10 seconds usi

119 Baroreflex sensitivity was assessed in control and in HF

120 Baroreflex sensitivity was assessed in the time domain w

121 Cardiovagal baroreflex sensitivity was defined as the slope relating

122 However, sympathetic baroreflex sensitivity was greater and mean arterial pre

123 Furthermore, sympathetic baroreflex sensitivity was greater during the ML than th

124 Similarly, cardiovagal baroreflex sensitivity was greater in the LH than in the

125 Baroreflex sensitivity was lower in patients (12+/-1 ver

126 Baroreflex sensitivity was lower in susceptible dogs (10

127 Baroreflex sensitivity was measured by the alpha-index m

128 Sympathetic baroreflex sensitivity was quantified using the slope of

129 Sympathetic baroreflex sensitivity was quantified using the slope of

130 Baroreflex sensitivity was similar in COI and control su

131 s measurements of heart rate variability and baroreflex sensitivity we aimed to test whether autonomi

132 s of RR interval variability and spontaneous baroreflex sensitivity were also computed.

133 y-four-hour ambulatory BP, SND, and arterial baroreflex sensitivity were measured before and after 8

134 t subjects, moderate ongoing fluctuations of baroreflex sensitivity were punctuated by brief major pe

135 Sympathovagal balance and spontaneous baroreflex sensitivity were restored during vitamin C in

136 Similar changes in baroreflex sensitivity were seen.

137 Analyses of heart rate variability and baroreflex sensitivity were used to assess autonomic bal

138 art rate variability, heart rate turbulence, baroreflex sensitivity) were significant predictors of a

139 stiffness; (2) it is associated with reduced baroreflex sensitivity, which increases blood pressure v

140 Furthermore, changes in baroreflex sensitivity with OC differ from changes in ba

141 We estimated vagal baroreflex sensitivity with systolic pressure and R-R in