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1 erative lung injury, pulmonary infection, or barotrauma).
2  lung stretch associated with volutrauma and barotrauma.
3 ury and pneumonia, and to reduce the risk of barotrauma.
4 tric respiratory failure, without increasing barotrauma.
5 f 718 patients with ALI/ARDS and no baseline barotrauma.
6 lation-perfusion mismatching with shunt, and barotrauma.
7  CI, 0.0% to 4.0%; P = .03), and the risk of barotrauma (5.6% vs 1.6%; difference, 4.0%; 95% CI, 1.5%
8                            It can occur with barotrauma and after chest tube placement.
9 er pulmonary blast injury is associated with barotrauma and the use of lung protective strategies pre
10 ns, acute sinusitis, systemic abnormalities, barotrauma and valsalva maneuver.
11 ive treatment to minimize ventilator-induced barotrauma and volutrauma during severe respiratory fail
12 f stay in the ICU and hospital, incidence of barotrauma, and hospital mortality.
13                     Perflubron distribution, barotrauma, and inability to discern catheters were eval
14 ght infants, associated with oxygen therapy, barotrauma, and/or infections.
15 n in reducing airway pressures and, perhaps, barotraumas are cited.
16      In some cases it seems to be related to barotrauma due to a rapid increase in pressure in the up
17 n cause middle ear and sinus injury and lung barotrauma due to lung overexpansion during ascent from
18 strointestinal hemorrhage; c) bacteremia; d) barotrauma; e) venous thromboembolic disease; and f) cho
19 e intervals: baseline, one day preceding the barotrauma event (one-day lag), and concurrent with the
20 event (one-day lag), and concurrent with the barotrauma event.
21 atients tolerate HFOV and increased rates of barotrauma have been reported in some studies.
22 etween treatment groups in the prevalence of barotrauma, hemodynamic instability, or mucus plugging.
23 tween airway pressures and the risk of early barotrauma in a cohort of 718 patients with ALI/ARDS and
24 er PEEP may increase the likelihood of early barotrauma in ALI/ARDS.
25                          The determinants of barotrauma in mechanically ventilated patients with acut
26 cidence of EOM following NP colonization and barotrauma in the animal model.
27 ifference, -2.4%, 95% CI, -7.1% to 2.2%) and barotrauma incidence (0% in the intensive group vs 0.6%
28 del, no other airway pressure was related to barotrauma, including plateau pressure.
29                 The precise role of arterial barotrauma-mediated apoptosis in causing restenosis is u
30 r PEEP nor PLV reduced the high incidence of barotrauma observed in high-PIP animals.
31 ) above 35 cm H(2)O may increase the risk of barotrauma or volutrauma.
32 s, oxygenation failure, ventilation failure, barotraumas, or mucus plugging between treatment groups.
33 s associated with an increased risk of early barotrauma (relative hazard [RH] 1.67 per 5-cm H2O incre
34  spared prolonged ventilation and consequent barotrauma, resulting in improved respiratory function.
35 r PEEP was associated with a greater risk of barotrauma (RH 1.38 per 5-cm H2O increment; 95% CI 1.09-
36 her PEEP was related to an increased risk of barotrauma (RH 1.50; 95% CI 0.98- 2.30).
37 t PEEP was also related to a greater risk of barotrauma (RH 1.93; 95% CI 1.44-2.60), controlling for
38                               HFOV increased barotrauma risk compared with conventional ventilation (
39 mits low pressure lung ventilation, avoiding barotrauma to lungs made friable by Panton-Valentine leu
40 aused by minor trauma or are associated with barotrauma to the orbit due to sneezing, coughing, or vo
41 four study days, the cumulative incidence of barotrauma was 13% (95% confidence interval [CI] 10.6 to
42 eumothorax requiring drainage within 7 days; barotrauma within 7 days; and ICU, in-hospital, and 6-mo

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