コーパス検索結果 (1語後でソート)
通し番号をクリックするとPubMedの該当ページを表示します
1 curative surgery or radiation or metastatic basal cell carcinoma.
2 um antibodies and cSCC or between betaPV and basal cell carcinoma.
3 therapies for Hh-dependent cancers, such as basal cell carcinoma.
4 clinical practice for patients with advanced basal cell carcinoma.
5 nalling inhibitor, in patients with advanced basal cell carcinoma.
6 CN1 and CCN2 may provide clinical benefit in basal cell carcinoma.
7 US Food and Drug Administration for advanced basal cell carcinoma.
8 nd progression of several cancers, including basal cell carcinoma.
9 smodegib for metastatic and locally advanced basal cell carcinoma.
10 elioma, meningioma, renal cell carcinoma and basal cell carcinoma.
11 at are novel and have not been documented in basal cell carcinoma.
12 in the US and is second in incidence only to basal cell carcinoma.
13 rms of cancer, including medulloblastoma and basal cell carcinoma.
14 seases such as genital warts, keratosis, and basal cell carcinoma.
15 n the proliferation of colorectal cancer and basal cell carcinoma.
16 t (1 malignancy) also developed a periocular basal cell carcinoma.
17 ncluding medulloblastoma, ameloblastoma, and basal cell carcinoma.
18 is approved for use in adults with advanced basal-cell carcinoma.
19 surgical excision in patients with low-risk basal-cell carcinoma.
20 py exists for locally advanced or metastatic basal-cell carcinoma.
21 % response rate among patients with advanced basal-cell carcinoma.
22 d greater than 10% for those with metastatic basal-cell carcinoma.
23 naling are implicated in the pathogenesis of basal-cell carcinoma.
24 patients with locally advanced or metastatic basal-cell carcinoma.
25 but are of unproven benefit, especially for basal-cell carcinoma.
26 H1) and smoothened homologue (SMO), occur in basal-cell carcinoma.
27 r activity in locally advanced or metastatic basal-cell carcinoma.
28 d cords, and ulceration were associated with basal cell carcinomas.
29 ltiple, recurrent, locally aggressive facial basal cell carcinomas.
30 TGF-beta signaling in several cell types in basal cell carcinomas.
31 tions in Ptch1 lead to the formation of skin basal cell carcinomas.
32 UVR in sunlight causes mutations that drive basal cell carcinomas.
33 rowth, with excessive signaling resulting in basal cell carcinomas.
34 nderlie the development of infundibulocystic basal cell carcinomas.
35 as well as renal, breast, lung, gastric, and basal cell carcinomas.
36 ve for select locally advanced or metastatic basal cell carcinomas.
37 onfirmed and at least six clinically evident basal-cell carcinomas.
38 s included reduction in the size of existing basal-cell carcinomas.
39 les taken from sites of clinically regressed basal-cell carcinomas.
40 responses in locally advanced and metastatic basal-cell carcinomas.
41 ib dosing regimens in patients with multiple basal-cell carcinomas.
42 the pivotal molecular abnormality underlying basal-cell carcinomas.
43 ent for squamous-cell carcinoma and for most basal-cell carcinomas.
44 long-term regimens in patients with multiple basal-cell carcinomas.
45 ehog signalling underlies the development of basal-cell carcinomas.
46 confirmed, including 9 melanomas (0.5%), 37 basal cell carcinomas (1.9%), and 1 squamous cell carcin
47 icial basal cell carcinoma, 6 weeks; nodular basal cell carcinoma, 12 weeks) or excisional surgery (4
48 types were represented in this analysis: 166 basal cell carcinomas, 146 squamous cell carcinomas, and
49 up and the placebo group with respect to new basal-cell carcinomas (20% [95% CI, -6 to 39] lower rate
50 3%]), cutaneous SCC (11 of 21 tumors [52%]), basal cell carcinoma (3 of 4 tumors [75%]), and ACC (5 o
51 (17/17) and additional TAs in SCCs (24/32), basal cell carcinomas (30/31) and malignant melanomas (1
52 o imiquimod 5% cream once daily (superficial basal cell carcinoma, 6 weeks; nodular basal cell carcin
54 n lesions (8 nevi, 8 seborrheic keratoses, 7 basal cell carcinomas, 7 melanomas, 4 hemangiomas, 4 der
55 luding 55 melanocytic nevi, 20 melanomas, 15 basal cell carcinomas, 7 solar lentigines or seborrheic
57 f cutaneous squamous cell carcinomas, 95% of basal cell carcinomas, 73% of conjunctival squamous cell
58 treatment option for patients with advanced basal cell carcinoma, a population that is difficult to
59 nown about patients' experiences of advanced basal cell carcinoma (aBCC) and basal cell carcinoma nev
60 now FDA-approved for patients with advanced basal cell carcinoma, acquired mutations in Smo can resu
61 currently in clinical trials for eradicating basal cell carcinoma, actinic keratosis, and squamous ce
63 13), 499 patients (468 with locally advanced basal cell carcinoma and 31 with metastatic basal cell c
64 n clinical benefit in patients with advanced basal cell carcinoma and is approved for treatment of pa
65 ns in Smoothened (SMO) have been reported in basal cell carcinoma and medulloblastoma, but are largel
66 ors are clinically effective in treatment of basal cell carcinoma and medulloblastoma, but fail thera
67 thway is best established in tumors, such as basal cell carcinoma and medulloblastoma, where the path
70 Hh) pathway is known to drive development of basal cell carcinoma and medulloblastomas and to associa
71 re associated with an increased incidence of basal cell carcinoma and melanoma, and a nonstatisticall
72 included intradermal nevus misclassified as basal cell carcinoma and nonmelanocytic lesions (eg, seb
73 Hedgehog signaling, where Ptch1 loss causes basal cell carcinoma and Ptch1;Ptch2 loss disrupts skin
74 up period of at least 13 years, the IRRs for basal cell carcinoma and squamous cell carcinoma remaine
75 ere noted among the 42 with locally advanced basal cell carcinoma and two (15%, 2-45) among the 13 wi
77 d specificity of 89-99% for the detection of basal cell carcinomas and can potentially serve as a rap
79 ed as nonmelanocytic (primarily as pigmented basal cell carcinomas and squamous cell carcinomas).
80 than 20% for patients with locally advanced basal-cell carcinoma and greater than 10% for those with
82 xcluded patients with morphoeic or recurrent basal-cell carcinoma and those with Gorlin syndrome.
83 study, we enrolled patients with metastatic basal-cell carcinoma and those with locally advanced bas
84 y, had serious adverse events, including two basal-cell carcinomas and one major cardiovascular adver
85 e number of new squamous-cell carcinomas and basal-cell carcinomas and the number of actinic keratose
89 ngioma, Hodgkin lymphoma, thyroid carcinoma, basal cell carcinoma, and parotid gland tumor, and 68.5%
90 d incidence rate ratios (IRRs) for melanoma, basal cell carcinoma, and squamous cell carcinoma by usi
91 ablished treatments for specific subtypes of basal-cell carcinoma, and the search for more effective
93 evelopment and some forms of cancer, such as basal cell carcinoma, are transduced by the primary cili
94 es including actinic keratosis, squamous and basal cell carcinoma as well as verruca and psoriasis an
95 ared with surgery for superficial or nodular basal cell carcinoma at low-risk sites in our noninferio
97 ndrome with at least ten surgically eligible basal-cell carcinomas at the Children's Hospital Oakland
99 a genome-wide association study on cutaneous basal cell carcinoma (BCC) among 2045 cases and 6013 con
100 f the variability in NMSC incidence: 82% for basal cell carcinoma (BCC) and 85% for squamous cell car
102 s in human skin biopsy samples diagnosed for basal cell carcinoma (BCC) and compared with healthy ski
104 his method to differentiate two skin tumors, basal cell carcinoma (BCC) and Merkel cell carcinoma (MC
105 ary outcomes were time to development of new basal cell carcinoma (BCC) and new invasive squamous cel
106 We report a case of local recurrence of basal cell carcinoma (BCC) and ocular complications foll
107 Whether susceptible people develop both basal cell carcinoma (BCC) and squamous cell carcinoma (
108 mance of this methodology on differentiating basal cell carcinoma (BCC) and squamous cell carcinoma (
109 f non-melanoma skin cancer (NMSC), including basal cell carcinoma (BCC) and squamous cell carcinoma (
110 avirus (HPV) infection in the development of basal cell carcinoma (BCC) and squamous cell carcinoma (
112 f nonmelanoma skin cancer (NMSC), defined as basal cell carcinoma (BCC) and squamous cell carcinoma (
113 tance: Keratinocyte cancers (KCs), including basal cell carcinoma (BCC) and squamous cell carcinoma (
114 ng DSCMs before and after training to detect basal cell carcinoma (BCC) and squamous cell carcinoma (
115 in cancers using a population-based study of basal cell carcinoma (BCC) and squamous cell carcinomas
118 nacone et al. investigate the role of HPV in basal cell carcinoma (BCC) by assessing the presence of
119 , 285 squamous cell carcinoma (SCC), and 300 basal cell carcinoma (BCC) cases, and 870 common control
122 elimination of superficial and early nodular basal cell carcinoma (BCC) in 2 cases using RCM imaging
123 r et al. (this issue) assess the risk of new basal cell carcinoma (BCC) in the Veterans Affairs topic
137 ith samples taken before diagnosis of SCC or basal cell carcinoma (BCC) of the skin were identified.
139 lent or aggressive, as is the case with skin basal cell carcinoma (BCC) or cerebellar medulloblastoma
140 s between cigarette smoking and incidence of basal cell carcinoma (BCC) or squamous cell carcinoma (S
142 rum 25-hydroxyvitamin D (25(OH)D) levels and basal cell carcinoma (BCC) risk in a nested case-control
144 (HH) signaling pathway, a crucial driver of basal cell carcinoma (BCC) tumorigenesis, and reduces BC
145 apillomavirus (HPV) DNA has been detected in basal cell carcinoma (BCC) tumors, but most epidemiologi
146 n size with squamous cell carcinoma (SCC) or basal cell carcinoma (BCC) were evaluated for the factor
147 melanoma, squamous cell carcinoma (SCC), and basal cell carcinoma (BCC)) in prospective studies simul
148 itor vismodegib is FDA approved for advanced basal cell carcinoma (BCC), and shows promise in clinica
149 a new type of targeted therapy for advanced basal cell carcinoma (BCC), and their long-term effects,
150 ts who develop recurrent or locally advanced basal cell carcinoma (BCC), and will inevitably be integ
151 he INTU gene is aberrantly elevated in human basal cell carcinoma (BCC), coinciding with increased pr
152 cutaneous squamous cell carcinoma (SCC) and basal cell carcinoma (BCC), in part as a result of immun
153 risk estimates to evaluate the risks of SCC, basal cell carcinoma (BCC), keratinocyte cancers (KCs) o
154 ted in a variety of human cancers, including basal cell carcinoma (BCC), medulloblastoma (MB) and emb
155 ry outcomes were time to first appearance of basal cell carcinoma (BCC), squamous cell carcinoma (SCC
156 in various cell processes, is upregulated in basal cell carcinoma (BCC), supporting a possible tumori
157 Importance: Rates of skin cancer, including basal cell carcinoma (BCC), the most common cancer, have
158 g (Hh) signaling leads to the development of basal cell carcinoma (BCC), the most common human cancer
160 tion exposure is the primary risk factor for basal cell carcinoma (BCC), the most common human malign
161 s directly target the genetic basis of human basal cell carcinoma (BCC), the most common of all cance
163 uence variants that confer risk of cutaneous basal cell carcinoma (BCC), we conduct a genome-wide ass
164 particularly evident in medulloblastoma and basal cell carcinoma (BCC), where inhibitors targeting t
172 up Study, we prospectively examined risks of basal cell carcinoma (BCC, 22,786 cases), squamous cell
174 procaspase-8) in association with cutaneous basal-cell carcinoma (BCC) and linked to a germline SNP
185 sting of squamous cell carcinomas (SCCs) and basal cell carcinomas (BCCs), are the most common human
186 hway in a variety of human cancer, including basal cell carcinomas (BCCs), medulloblastomas, leukemia
191 e reveals that dermoscopy can help delineate basal cell carcinomas before surgical removal but that i
192 ns are causative of Gorlin syndrome (naevoid basal cell carcinoma), but detection rates > 70% have ra
193 duced the hedgehog target-gene expression by basal-cell carcinoma by 90% (P<0.001) and diminished tum
196 mean reduced rate of new surgically eligible basal-cell carcinomas compared with patients randomly as
197 d the development of new surgically eligible basal-cell carcinomas compared with placebo (0.4 [SD 0.2
198 on for small low-risk superficial or nodular basal-cell carcinoma dependent on factors such as patien
199 which numerous indolent, well-differentiated basal cell carcinomas develop primarily on the face and
204 ons was reflected by the fact that all human basal cell carcinomas examined have detectable STAT3 pho
205 ma and an adenoma) and as controls on eyelid basal cell carcinomas, eyelid squamous cell carcinomas,
206 oved for treatment of patients with advanced basal cell carcinoma for whom surgery is inappropriate.
207 oma within the tumor bed of locally advanced basal cell carcinoma found during vismodegib treatment.
208 c/hypomelanotic malignant lesions (including basal cell carcinoma) gave a 93% sensitivity and 70% spe
209 ell carcinoma tumour burden and prevents new basal-cell carcinoma growth in patients with basal-cell
211 -71.0) of 453 patients with locally advanced basal cell carcinoma had an overall response (153 comple
212 %; 20.7-57.7) of 29 patients with metastatic basal cell carcinoma had an overall response (two comple
214 basal cell carcinoma and 31 with metastatic basal cell carcinoma) had received study drug and had th
216 (HR = 1.15, 95% CI 1.11-1.19, p < 0.001, for basal cell carcinoma; HR = 1.21, 95% CI 1.17-1.25, p < 0
217 gery remains the best treatment for low-risk basal-cell carcinoma, imiquimod cream might still be a u
218 rovide evidence that up-regulation of YAP in basal cell carcinoma impacts both aberrant keratinocyte
219 f melanoma; overall and by stage and risk of basal cell carcinoma in multivariable logistic regressio
220 ling strategy allowed objective diagnosis of basal cell carcinoma in skin tissue samples excised duri
222 n, inhibit the growth of medulloblastoma and basal cell carcinoma in vivo, and prolong survival of mi
223 tance of repeated biopsy in locally advanced basal cell carcinomas in 2 clinical situations: (1) when
225 ypertelorism in three patients, and multiple basal cell carcinomas in the radiation field after age 1
227 odalities to help delineate tumor margins of basal cell carcinomas in the setting of Mohs micrographi
230 cinoma tumor burden and blocks growth of new basal-cell carcinomas in patients with the basal-cell ne
232 al, we enrolled adult patients with multiple basal-cell carcinomas, including those with basal-cell n
234 ecule and are indicated for locally advanced basal cell carcinoma (laBCC) and metastatic basal cell c
238 mouse model)-derived Hedgehog-driven tumors (basal cell carcinoma, medulloblastoma and atypical terat
239 eic keratosis, lichen planus-like keratosis, basal cell carcinomas) misclassified as melanocytic beca
240 of advanced basal cell carcinoma (aBCC) and basal cell carcinoma nevus syndrome (BCCNS), a rare gene
241 en's Hospital Oakland Research Institute and Basal Cell Carcinoma Nevus Syndrome Life Support Network
243 ose below 75 nmol l(-1) more often developed basal cell carcinoma (odds ratio (OR)=1.51 (95% confiden
244 disorder characterized by multiple cutaneous basal cell carcinomas, odontogenic keratocysts, skeletal
247 th histologically confirmed locally advanced basal cell carcinoma or metastatic basal cell carcinoma
248 ed a diagnosis of and were being treated for basal cell carcinoma or squamous cell carcinoma (cases)
249 as also associated with an increased risk of basal cell carcinoma (OR, 1.19 [95% CI, 1.14-1.25], 9% f
250 ing (mean 0.6 [0.72] new surgically eligible basal-cell carcinomas per patient per year vs 1.7 [1.8]
251 lacebo (0.4 [SD 0.2] new surgically eligible basal-cell carcinomas per patient per year vs 30.0 [7.8]
252 (n=15; two [SD 0.12] new surgically eligible basal-cell carcinomas per patient per year vs 34 [1.32]
253 er year vs 34 [1.32] new surgically eligible basal-cell carcinomas per patient per year, p<0.0001).
254 er year vs 1.7 [1.8] new surgically eligible basal-cell carcinomas per patient per year, p<0.0001).
255 r year vs 30.0 [7.8] new surgically eligible basal-cell carcinomas per patient per year, p<0.0001).
256 umber of new nonmelanoma skin cancers (i.e., basal-cell carcinomas plus squamous-cell carcinomas) dur
257 The recurrence rate of periocular nodular basal cell carcinoma (PNBCC) following treatment with im
258 d in a clinical trial testing vismodegib for basal cell carcinoma prevention (NCT00957229), using pre
259 ignancies and the squamous cell carcinoma-to-basal cell carcinoma ratio increased with time postgraft
263 lleagues and Sharpe and colleagues show that basal cell carcinomas resistant to the Smoothened (SMO)
264 e is that of a patient with locally advanced basal cell carcinoma responsive to vismodegib but with a
265 other hyperproliferative disorders, such as basal cell carcinoma, sAC staining was similar to normal
268 on (aOR, 2.13; 95% CI, 1.31-3.47; P=.002) or basal-cell carcinoma specific criteria (aOR, 9.35; 95% C
269 immunosuppressed = 8.9 years), 135 (27%) had basal cell carcinoma, squamous cell carcinoma or Bowen's
270 or tanning facility and an increased risk of basal cell carcinoma, squamous cell carcinoma, and melan
271 We observed no overall increased risk for basal cell carcinoma, squamous cell carcinoma, or melano
272 d within 1 second each, including melanomas, basal cell carcinomas, squamous cell carcinomas, actinic
274 scopy revealed increased tissue stiffness in basal cell carcinoma stroma compared to normal dermis.
279 dverse events over 24 months, which included basal-cell carcinoma (ten [3%] patients vs two [1%] pati
280 key RCM diagnostic criteria for melanoma and basal cell carcinoma that are reproducibly recognized am
281 with Gorlin syndrome with a locally advanced basal cell carcinoma that was stable while the patient w
282 point was reduction in the incidence of new basal-cell carcinomas that were eligible for surgical re
285 organ transplant population at high risk for basal cell carcinoma, therapeutic options for locally ad
290 the smoothened inhibitor vismodegib reduces basal-cell carcinoma tumour burden and prevents new basa
293 per-patient rate of new surgically eligible basal-cell carcinomas was lower with vismodegib than wit
294 advanced basal cell carcinoma or metastatic basal cell carcinoma were recruited from regional referr
295 lanomas, 29 squamous cell carcinomas, and 38 basal cell carcinomas were diagnosed, with a higher prop
296 h showed negative results for both, and from basal cell carcinomas, which showed positive receptor re
298 ll carcinoma and those with locally advanced basal-cell carcinoma who had inoperable disease or for w
WebLSDに未収録の専門用語(用法)は "新規対訳" から投稿できます。