ライフサイエンスコーパス: basal membrane

1 al localization, with a stark absence at the basal membrane.

2 of caveolae-derived vesicles from apical to basal membrane.

3 control RPTC was limited exclusively to the basal membrane.

4 thelial trafficking, and exocytosis from the basal membrane.

5 red hepatocytes surrounded by a laminin-rich basal membrane.

6 primarily at the junction of the lateral and basal membrane.

7 branes and prominent in stress fibers on the basal membrane.

8 nd alpha 8 expression was accentuated at the basal membrane.

9 systems for L-tryptophan in brush border and basal membranes.

10 on with less distribution on the lateral and basal membranes.

11 R252A (or R252E) mutants showed much higher basal membrane affinity and enzyme activity and faster s

12 m accounts for the fibrous topography of the basal membrane and allows for easy modulation of fiber s

13 ntified junctions that stabilize the forming basal membrane and enable basal cell closure.

14 e formation of junctions between the forming basal membrane and the yolk plasmalemma.

15 of ICAM-1 and caveolin-1 to the endothelial basal membrane and transmigrated through transcellular p

16 plan of any animal, possessing no organs, no basal membrane, and only four different somatic cell typ

17 a(+) transport, relocalization of CBI to the basal membrane, and the disappearance of CBI from the ap

18 population of epithelial cells, localizes to basal membranes, and specifically associates with elemen

19 ordered at the apical membranes compared to basal membranes, and that an inverse situation occurred

20 chanisms of net amino acid efflux across the basal membrane (BM) of the placental syncytiotrophoblast

21 encing pH-sensitive microelectrodes near the basal membrane by approximately 65% and 85% respectively

22 othelial growth factor (VEGF) secretion from basal membrane by inhibiting IGF-1 signaling and VEGF re

23 nction of LIN-2 is to localize LET-23 to the basal membrane domain of vulval precursor cells.

24 tion proteins (such as LET-23) to either the basal membrane domain or the cell junctions, and that mi

25 e show that TJ-mediated separation of apical-basal membrane domains is established prior to equilibra

26 shootwards (apical) (rather than rootwards (basal)) membrane domains.

27 on and bound to various extracellular matrix/basal membrane (ECM/BM) components, including collagen t

28 ity was required for contraction but not for basal membrane extension.

29 specifically on the microvilli of hepatocyte basal membranes, facing the space of Disse, where lipopr

30 t and inhibited by >90% by reduced pH in the basal membrane-facing solution.

31 ctivities and actomyosin cytoskeleton impact basal membrane fluctuations in adherent cells.

32 We show that basal membrane function can be substituted in vitro by e

33 erent proliferative forms of anti-glomerular basal membrane (GBM) GN in rats were induced and whole c

34 The channel was localized on the lateral and basal membranes in crypt cells.

35 ely expressed in neurons and is polarized to basal membranes in intestinal epithelial cells.

36 ial epithelial cells; reduced density of the basal membrane; lower density of keratocytes, increased

37 FNA2 human hearing loss, is expressed in the basal membrane of cochlear outer hair cells where it may

38 emidesmosome, the anchoring structure in the basal membrane of epithelial cells.

39 nsity lipoprotein receptors expressed on the basal membrane of hepatocytes.

40 mains based on lipid-phase separation in the basal membrane of our cultured nonstimulated cells, and

41 ession of fibronectin and collagen IV in the basal membrane of pancreatic ducts and of cell clusters

42 that the IL-6 receptor was restricted to the basal membrane of Paneth cells both in vitro and in vivo

43 ressed tagged Inx7a localizes to the nascent basal membrane of the forming cells in wild-type eggs.

44 smic vesicles, and tubular extensions of the basal membrane of the invaded cells.

45 ptide OATP2B1 (SLCO2B1) are expressed in the basal membrane of the placental syncytiotrophoblast.

46 The isoform MCT3 is restricted to the basal membrane of the retinal pigment epithelium where i

47 ial in the testicular environment, where the basal membranes of adjacent polarized Sertoli cells form

48 O-1, were mislocalized along the lateral and basal membranes of fiber cells.

49 s expression co-localized with Col-IV in the basal membranes of juxtaposed endothelial cells.

50 method for studying dynamics at or near the basal membranes of living cells.

51 use eye, MFRP is localized to the apical and basal membranes of RPE and ciliary epithelium (CE).

52 prestressed mesh parallel to the apical and basal membranes of the cell.

53 (SGTP1 and SGTP4) and localized SGTP1 to the basal membranes of the tegument and the underlying muscl

54 e atypical cadherin Fat2 recruits the WRC to basal membranes of tricellular contacts where a new type

55 Polycystin-1 is highly expressed in the basal membranes of ureteric bud epithelia during early d

56 alysis revealed that XKir6.1 is localized to basal membranes on the blastocoel roof and cell-cell jun

57 pe or IP(3)R-knockout cells did not increase basal membrane permeability, but resulted in a substanti

58 tion of these and other findings is that the basal membrane possesses two systems, one of which is si

59 augments synaptic drive by depolarizing the basal membrane potential close to the action potential t

60 current contributed to the more depolarized basal membrane potential observed in VP neurons in the h

61 , that bound to various extracellular matrix/basal membrane proteins and was required for full virule

62 uced levels of several myelin-associated and basal membrane proteins compared with those of wild-type

63 escued the gland migration, lumen length and basal membrane protrusion defects and partially rescued

64 face is severely impaired, resulting in long basal membrane protrusions but little net movement or br

65 cells, and promotes extension of actin-rich basal membrane protrusions in the distal cells.

66 te, to downregulate E-cadherin and to extend basal membrane protrusions.

67 urfaces but was able to infect cells through basal membranes, replicate, and spread into surrounding

68 duals with the T594 M variant showed similar basal membrane slope conductance, compared with the wild

69 This transformation requires basal membrane-stimulated integrin signaling that coordi

70 ingival overgrowth, resulting in compromised basal membrane structure and increased interactions betw

71 Disrupted basal membrane structure in gingival overgrowth tissues

72 sion, and peristromal lamina separation from basal membrane surfaces, as compared with control indexe

73 ntrolled by the stiffness of the lateral and basal membrane surfaces.

74 Basal membrane transport of L-tryptophan is more complex

75 lear evidence that two systems contribute to basal membrane transport since BCH is (in sodium-free me

76 The disruption of pial basal membranes underlying the heterotopias and poor org

77 However, staining at the basal membrane was weak.

78 asion to bronchial epithelium, and thickened basal membrane were found in same biopsy specimen.

79 is mostly cytosolic and concentrated at the basal membrane, whereas galectin-3 tends to be concentra

80 ized to the apical membrane and TNFR1 to the basal membrane, whereas IFN-gammaR1 was detected on both

81 contracted, the myosin ring moved toward the basal membrane with ZO-1 and Rho-kinase.

82 uctural actin (DCSA), extends from apical to basal membranes, with frequent attachments.

83 an immunologic reaction against conjunctival basal membrane zone (BMZ) antigens.