ライフサイエンスコーパス: basal transcription factor

1 tor IIB (TFIIB) and the TATA binding protein basal transcription factor.

2 x, probably through direct interactions with basal transcription factors.

3 cells, suggesting that TEF-1 interacts with basal transcription factors.

4 a complex of proteins that includes Sp1 and basal transcription factors.

5 8 for viability, we identified a plethora of basal transcription factors.

6 enes requires the participation of a host of basal transcription factors.

7 rged kinetoplastids possess a reduced set of basal transcription factors.

8 a single subunit RNA polymerase and a set of basal transcription factors.

9 e nucleus occurs subsequent to TBP and other basal transcription factors.

10 kinases-7 (Cdk7) and Cdk9, components of two basal transcription factors.

11 FR gene expression through interactions with basal transcription factors.

12 d eukaryotic RNAPII is assisted by conserved basal transcription factors.

13 anscriptional activity and with occupancy by basal transcription factors.

14 stable targeting may require the presence of basal transcription factors.

15 lear ribonucleoproteins (snRNPs) and certain basal transcription factors.

16 ould contact transcriptional coactivators or basal transcription factors.

17 tivates p53 by blocking its interaction with basal transcription factors.

18 ore, consistent with direct interaction with basal transcription factors, addition of recombinant TFI

19 en that LC8 has never been identified with a basal transcription factor and that T. brucei relies on

20 at the ATM protein interacted with the TFIIH basal transcription factor and the XPG protein of the NE

21 in developmental transcription for the TFIID basal transcription factors and for the DNA core promote

22 ), termed coactivator, that is distinct from basal transcription factors and is critical for efficien

23 with biochemically purified and recombinant basal transcription factors and RNA polymerase II.

24 merase II complex containing a subset of the basal transcription factors and the human homologues of

25 there is no knowledge about the function of basal transcription factors, and there is an apparent ra

26 findings suggest that the core promoter and basal transcription factors are important yet mostly une

27 ty suggesting that interaction with the same basal transcription factors are involved in both functio

28 monstrate the following: 1) pol II and other basal transcription factors are recruited to LCR core hy

29 hat TAF(II)250, TFIID, and potentially other basal transcription factors are targets for regulation b

30 with as much RNA polymerase II (Pol II) and basal transcription factors as present at the active Gat

31 ling can circumvent the need for a subset of basal transcription factors at specific promoters.

32 oiety itself interferes with the assembly of basal transcription factors at the promoter.

33 zation of coactivator p300, as well as other basal transcription factors, at the nucleosomes for regu

34 e suppressed by mutations that do not affect basal transcription factor binding-DNA contacts.

35 -coupled repair as an integral member of the basal transcription factor BTF2/TFIIH complex.

36 2/-25), in assembly of a myeloid-restricted, basal transcription factor complex.

37 They also provide an example of a basal transcription factor containing a Myb DNA binding

38 ogen receptor alpha (ERalpha) interacts with basal transcription factors, coregulatory proteins, and

39 0, the spliceosome assembly factor SC35, and basal transcription factors defines spatially and tempor

40 the RNAP and the molecular mechanisms of the basal transcription factors E (TFE) and Spt4/5 through c

41 gical function for acetylation in regulating basal transcription factors has not been reported.

42 Kin28 is a subunit of the basal transcription factor holo-TFIIH and its trimeric s

43 the SMA region, encoding the p44 subunit of basal transcription factor II (BTF2p44).

44 e 1 (BPV-1), has been shown to interact with basal transcription factor IIB (TFIIB) and the TATA bind

45 r TAF(II)250 is the largest component of the basal transcription factor IID (TFIID).

46 The basal transcription factor IIE (TFIIE) is thought to be

47 impeding the entry of RNA polymerase II and basal transcription factor IIF into a competent preiniti

48 here were to develop procedures for studying basal transcription factors in the cytosol of M. mazeii

49 e expression by facilitating the assembly of basal transcription factors into a stable preinitiation

50 The overall fold of these two basal transcription factors is essentially the same as t

51 regulates transcription as part of the TFIIH basal transcription factor, is an attractive target for

52 ity that altered levels of sequence-specific basal transcription factors may contribute to neurologic

53 o transcription/translation we show that the basal transcription factor NF-Y or a related factor bind

54 r AP-1 activation, on the DNA binding of the basal transcription factor Oct-1, or on hydrogen peroxid

55 d transcription in a reporter assay, and the basal transcription factors OCT1 and SP1 were shown to b

56 TATA-binding protein (TBP) is common to the basal transcription factors of all three RNA polymerases

57 It does not include basal transcription factors or chromatin-associated prot

58 lectively phosphotylates RAP74 but not other basal transcription factors or common phosphoacceptor pr

59 Capping enzyme is not stably associated with basal transcription factors or the RNA polymerase II (Po

60 ecific functional interactions with both the basal transcription factor OsTFIIB and the accessory tra

61 gene and a somewhat lesser fraction for the basal transcription factor, p44 subunit (BTF2p44) gene.

62 be required to maintain the normal levels of basal transcription factors required for immediate respo

63 tion and architecture, promoter elements and basal transcription factors required for the initiation

64 rRNA by inactivation of the RNA polymerase I basal transcription factor RRN3/TIF-IA.

65 differential binding of a sequence-specific basal transcription factor(s) to the Inr.

66 report that TAF1B, a subunit of human Pol I basal transcription factor SL1, is structurally related

67 The basal transcription factor SNAPc binds to the PSE, a cor

68 l sequence element (PSE), which recruits the basal transcription factor SNAPc, and a distal sequence

69 bind the POU domain activator Oct-1 and the basal transcription factor SNAPc, respectively.

70 we report that Atgl is down-regulated by the basal transcription factor Sp1 in preadipocytes and that

71 e promoter lacks canonical binding sites for basal transcription factors such as TATA and CCAAT boxes

72 mal activator Sp1, and the components of the basal transcription factors such as TBP, TFIIB, and Cdk7

73 ntial for the DNA looping and recruitment of basal transcription factors such as TFIIB and Cdk7 but n

74 ORE)) and is stimulated by promoter-specific basal transcription factors, such as two human TFIIB fam

75 omain protein Brd2 is closely related to the basal transcription factor TAF(II)250, which is essentia

76 ct effect on the interaction of p53 with the basal transcription factor TAF(II)31.

77 Specifically, basal transcription factor Taf4b is down-regulated in th

78 e promoter DNA is recognized and bent by the basal transcription factor TATA-binding protein (TBP).

79 matin remodeling factor component BRG-1, and basal transcription factors TATA-binding protein (TBP) a

80 This domain has been reported to bind the basal transcription factors TATA-binding protein and TFI

81 e in specific interactions in vitro with the basal transcription factors TATA-binding protein, TFIIB,

82 ar transcription factors Fos and Jun and the basal transcription factor TBP (TATA binding protein).

83 Our results suggest that the basal transcription factor TBP/TFIID represents an impor

84 iency virus (HIV) type I Tat protein and the basal transcription factor TBP/TFIID.

85 complex multisubunit RNA polymerase and the basal transcription factors TBP and TF(II)B, closely res

86 allosteric component and is mitigated by the basal transcription factor TFEalpha/beta.

87 thway that involves its interaction with the basal transcription factor TFIIB.

88 e EICP0 protein interacted directly with the basal transcription factors TFIIB and TBP and that the E

89 assay, ICP4 was unable to interact with the basal transcription factors, TFIIB, TFIIE, TFIIF, and TF

90 Basal transcription factor TFIID comprises the TATA-box-

91 The basal transcription factor TFIID consists of the TATA-bi

92 The basal transcription factor TFIID is composed of the TATA

93 No significant effect was noted on the basal transcription factor TFIID recognition of TATA-con

94 ing the interaction of these proteins is the basal transcription factor TFIID, which recognizes the c

95 the TATA binding protein, a component of the basal transcription factor TFIID.

96 ing protein (TBP), which is a subunit of the basal transcription factor TFIID.

97 form a TAF-like function in complex with the basal transcription factor TFIID.

98 system and interacted specifically with the basal transcription factor (TFIIE) in HeLa nuclear extra

99 y HSF-4a occurs through interaction with the basal transcription factor TFIIF.

100 between a transcriptional repressor with the basal transcription factor TFIIF.

101 mino acids 142-485) selectively bound to the basal transcription factors TFIIF and the TATA-box-bindi

102 preliminary studies, we have identified the basal transcription factor TFIIH as the potential target

103 Further, we show that the basal transcription factor TFIIH is constitutively recru

104 Basal transcription factor TFIIH phosphorylates the RNA

105 The human basal transcription factor TFIIH plays a central role in

106 nscription by disrupting the assembly of the basal transcription factor TFIIH through sequestration o

107 interaction between SCL and a subunit of the basal transcription factor TFIIH, suggesting a potential

108 Pol II CTD is phosphorylated at Ser 5 by the basal transcription factor TFIIH.

109 hat SCL interacts with p44, a subunit of the basal transcription factor TFIIH.

110 een the repairosome and the RNA polymerase H basal transcription factor TFIIH.

111 -dependent kinase 7 (CDK7), a subunit of the basal transcription factor TFIIH.

112 ed in E. coli, and is not a component of the basal transcription factor, TFIIH.

113 nts represented the first demonstration of a basal transcription factor that binds, in an activation-

114 eveal that NC2 (Dr1-Drap1) is a bifunctional basal transcription factor that differentially regulates

115 s same region of p53 is able to bind several basal transcription factors that appear to be important

116 The known basal transcription factors that support TATA-dependent

117 atinized templates suppressed recruitment of basal transcription factors, thereby amplifying the effe

118 ng protein (TBP), RVFV appears to target the basal transcription factor THIIH to induce shut-off of h

119 A polymerase II and recruit it and the other basal transcription factors to a promoter.

120 er, the Tat protein could then interact with basal transcription factors to activate transcription.

121 RNA polymerases (RNAPs) require basal transcription factors to assist them during transc

122 It interacts with variety of basal transcription factors to initiate and elongate tra

123 own but is thought to involve recruitment of basal transcription factors to the promoter.

124 The basal transcription factors, which act in conjunction wi

125 TFIIH is a 10-subunit RNA polymerase II basal transcription factor with a dual role in DNA repai

126 DNA, there are several reports of RNAPII and basal transcription factors within silenced regions.