ライフサイエンスコーパス: base excess

1 resuscitation, shock index, coagulation, and base excess.

2 pressure, heart rate, Po(2), Pco(2), pH, and base excess.

3 ange in chloride and the degree of change in base excess.

4 ge enough to explain the degree of change in base excess (0.8 +/- 2.3 to -2.7 +/- 2.9).

5 rchloremia by 8-12 hrs (mean sodium-chloride base excess, -10.0 mmol/L).

6 ficantly lower, whereas nonsurvivor standard base excess (-17.9 +/- 5.1 vs. -2.9 +/- 4.4 mEq/L, p < .

7 (more negative) sodium chloride-partitioned base excess, 2) maintained a greater urine output, and 3

8 0 mL/kg (P=0.019, odds ratio [OR]=13.79) and base excess 30-min after reperfusion less than -16 (P=0.

9 ) arterial pH and [H(+)]a, and a significant base excess (-4.5 +/- 2.7 mEq/L), consistent with compen

10 7 degrees C [95% CI, 39.4-39.9]), and higher base excess (-5.9 mEq/L [95% CI, -8.0 to -3.8] vs -11.2

11 The baseline pH was 7.27 +/- 0.11 and base excess -5.9 +/- 5.0 mmol/L.

12 well with a systemic indicator of recovery, base excess, 5.4 +/- 4.7 (MalPEG-Hb), 1.7 +/- 3.8 (SB),

13 d anions at admission (mean unmeasured anion base excess, -9.2 mmol/L) to predominant hyperchloremia

14 ic acidosis was common at presentation (mean base excess, -9.7 mmol/L) and persisted for 48 hrs.

15 ry mechanism, weighted-Revised Trauma Score, base excess and hemoglobin, ACT-predicted red blood cell

16 resuscitation with saline, arterial standard base excess and plasma apparent strong ion difference we

17 In about one-sixth of the patients, base excess and plasma bicarbonate were normal.

18 r 30-day mortality in comparison to standard base excess and strong ion gap.

19 uration (SaO(2)), bicarbonate concentration, base excess, and alveolar-arterial oxygen difference wer

20 lobin, oxygen content, lactate, pH, standard base excess, and arginine vasopressin levels were determ

21 stric intramucosal pH, arterial pH, arterial base excess, and arterial lactate concentrations were me

22 ith Ringer's solution resulted in a standard base excess, and Cl(-) between that of saline and Hexten

23 resuscitation room: age, Glasgow Coma Scale, base excess, and prothrombin time.

24 ric pressure has a substantial effect; PCO2, base excess, and respiratory quotient have small effects

25 to limitations in bedside monitoring tools (base excess, anion gap).

26 They also had lower base excess at admission.

27 We compared the base excess (BE) and anion gap (AG) methods with the les

28 nous fluid resuscitation (r = .44), with the base excess changing, on average, by -0.4 mmol/L for eac

29 ressure, arterial bicarbonate concentration, base excess, fibrinogen concentration, and platelet coun

30 The mean improvement in base excess from 0 to 24 hours was significantly greater

31 The primary outcome was mean change in base excess from 0 to 24 hours.

32 with baseline, acidosis of pH 7.1 decreased base excess from 6.6 +/- 0.5 mM to -12.4 +/- 0.5 mM; red

33 Furthermore, bosentan decreased blood base excess in Nx animals (0.1 +/- 0.3 to -0.12 +/- 0.03

34 correlated with raised serum bicarbonate and base excess, indicating compensated respiratory acidosis

35 mergency department pH < or = 7.26, standard base excess < or = -7.3 mEq/L, lactate > or = 5 mmol/L,

36 Base excess missed serious acid-base abnormalities in ab

37 d venous blood gases; electrolytes; lactate; base excess; oxygen delivery, consumption, and extractio

38 , injury severity score, Glascow Coma Scale, base excess, platelet count and hemoglobin, adrenaline,

39 Glascow Coma Scale, systolic blood pressure, base excess, platelet count, hemoglobin, prehospital pla

40 elated with Scvo2 (r=.80), lactate (r=-.78), base excess (r=.80), and shed blood volume (r=-.75).

41 administered was compared with the change in base excess (r2 = .93; p < .0001).

42 ions by expressing them in terms of standard base excess (SBE in mM), which quantifies the metabolic

43 .90-7.00), whereas 20.5% (31/151) selected a base excess threshold (mean, -15.62+/-0.78; median, -16;

44 M infusion corrected pH to 7.40 +/- 0.02 and base excess to 2.6 +/- 0.9 mM (p < .05).

45 Trends toward lower base excess values and higher occurrences of deaths only

46 normal saline administered and the change in base excess was found (r2 = .86; p < .0001), although no

47 Base excess was partitioned using abridged Stewart equat

48 concentration and "anion gap," the other on "base excess," with a third method based on physicochemic