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1 roblast growth factor 2 (also referred to as basic fibroblast growth factor).
2 evascularization of the graft bed by agarose-basic fibroblast growth factor.
3 PC12 cells, but not outgrowth stimulated by basic fibroblast growth factor.
4 proliferation in the presence and absence of basic fibroblast growth factor.
5 n, but does not alter fibrocyte responses to basic fibroblast growth factor.
6 which was markedly induced by TGF-beta, and basic fibroblast growth factor.
7 inor (truncated discoidin-I-like domain), or basic fibroblast growth factor.
8 , Del-1 doubled fibrovascular growth, as did basic fibroblast growth factor.
9 ed by vascular endothelial growth factor and basic fibroblast growth factor.
10 ses to vascular endothelial growth factor or basic fibroblast growth factor.
11 (HMVECs), with potency comparable to that of basic fibroblast growth factor.
12 ment with phorbol 12-myristate 13-acetate or basic fibroblast growth factor.
13 EC to vascular endothelial growth factor or basic fibroblast growth factor.
14 upregulated autophagy through IL6, IL8, and basic fibroblast growth factor.
15 d decreased secretion of VEGF-A, VEGF-C, and basic fibroblast growth factor.
16 edium containing epidermal growth factor and basic fibroblast growth factor.
17 s preceded by a local increase in VEGF-A and basic fibroblast growth factor.
18 ated by calcitriol, parathyroid hormone, and basic-fibroblast growth factor.
19 expressed elevated phospho-Akt (1.85-fold), basic fibroblast growth factor (1.44-fold), hepatocyte g
20 lin-like growth factor 1 (IGF-1), as well as basic fibroblast growth factor 2 (bFGF2), reportedly ast
21 xenograft tumor models as well as suppresses basic fibroblast growth factor 2 (FGF-2)-induced neovasc
23 , and we established a role for ASMC-derived basic fibroblast growth factor 2 (FGF2b) and FGF recepto
24 on of vascular endothelial growth factor and basic fibroblast growth factor-2 mRNA transcripts in isc
26 d neurospheres, divided in response to bFGF (basic fibroblast growth factor), activated the neuroepit
29 her angiogenic proteins including acidic and basic fibroblast growth factors (aFGF and bFGF), epiderm
30 photoaptamers 0650 and 0615 cross-link human basic fibroblast growth factor and 0518 cross-links HIV
31 morphogenetic protein-4 in combination with basic fibroblast growth factor and activin A results in
33 We show that NFATc2 is also activated by basic fibroblast growth factor and blocked by the inhibi
34 stimulated by keratinocyte growth factor and basic fibroblast growth factor and growth-inhibited by a
35 ogenic and proinflammatory cytokines such as basic fibroblast growth factor and IL-1, endothelial cel
36 sponges containing 0.4% agarose and IL-6 or basic fibroblast growth factor and implanted into the su
37 r PTKs for insulin, epidermal growth factor, basic fibroblast growth factor and insulin-like growth f
39 t in the absence of serum the combination of basic fibroblast growth factor and platelet-derived grow
40 wth response protein 1 and its target genes, basic fibroblast growth factor and platelet-derived grow
41 vitro angiogenesis assay, c7E3 Fab inhibited basic fibroblast growth factor and platelet-stimulated c
42 sin S- or cystatin C-null mice, tumor tissue basic fibroblast growth factor and serum type 1 insulin
44 sed association with eIF4E and inhibition of basic fibroblast growth factor and vascular endothelial
45 mor and that it directly inhibits binding of basic fibroblast growth factor and vascular endothelial
46 igration and blocked angiogenesis induced by basic fibroblast growth factor and vascular endothelial
47 actors were detected in EGFP+ CBSCs in vivo (basic fibroblast growth factor and vascular endothelial
48 ation, and tube formation, the regulation of basic fibroblast growth factor and vascular endothelial
49 r growth factors identified, including VEGF, basic fibroblast growth factor, and brain-derived neurot
50 HUVECs and the angiogenic response to VEGF, basic fibroblast growth factor, and epidermal growth fac
51 es, including epidermal growth factor (EGF), basic fibroblast growth factor, and hepatocyte growth fa
52 such as vascular endothelial growth factor, basic fibroblast growth factor, and hepatocyte growth fa
54 ncluding vascular endothelial growth factor, basic fibroblast growth factor, and interleukin-8, has b
55 edullin, vascular endothelial growth factor, basic fibroblast growth factor, and platelet-derived gro
56 hemotactic factors including angiotensin II, basic fibroblast growth factor, and platelet-derived gro
57 in the mouse cornea using 80 ng of purified basic fibroblast growth factor, and the neovascular resp
58 angiogenic factors, including interleukin 8, basic fibroblast growth factor, and vascular endothelial
59 ference and neutralization of interleukin-8, basic fibroblast growth factor, and vascular endothelial
60 ole of isoforms of protein kinase C (PKC) in basic fibroblast growth factor- and interleukin-1alpha-m
63 s activated by nerve growth factor (NGF) and basic fibroblast growth factor as well as by the guanine
64 encoding fibroblast growth factor 2 (Fgf2 or basic fibroblast growth factor) as a target gene that is
65 ukin-6 (vIL-6) in vitro and VEGF, vIL-6, and basic-fibroblast growth factor (b-FGF) in mouse xenograf
66 GF activity by the seminal observations that basic fibroblast growth factor (bFGF or FGF-2) required
68 ment with either the angiogenesis stimulator basic fibroblast growth factor (bFGF) (1 microg/g/d intr
70 nt JB6 mouse epidermal cells stimulated with basic fibroblast growth factor (bFGF) and 12-O-tetradeca
71 potent embryonic stem (ES) cells cultured in basic fibroblast growth factor (bFGF) and activin A deve
72 ascular endothelial growth factor A (VEGFA), basic fibroblast growth factor (bFGF) and angiogenin (AN
73 tance contractions with two candidate mRNAs, basic fibroblast growth factor (bFGF) and elongation fac
75 were cultured in the presence or absence of basic fibroblast growth factor (bFGF) and exposed to ion
77 on is associated with elevated production of basic fibroblast growth factor (bFGF) and matrix metallo
79 determined the optimal concentration of both basic fibroblast growth factor (bFGF) and neuregulin-1 b
80 ed whether the heparin-binding growth factor basic fibroblast growth factor (bFGF) and one of its rec
82 heparan sulfate proteoglycans that can bind basic fibroblast growth factor (bFGF) and serum amyloid
87 ions were not changed by surgical delay, but basic fibroblast growth factor (bFGF) and VEGF expressio
88 t YKL40 displaced extracellular matrix-bound basic fibroblast growth factor (bFGF) as well as inhibit
93 pression) was decreased significantly, while basic fibroblast growth factor (bFGF) expression, a know
97 as insulin-like growth factor 1 (IGF-1) and basic fibroblast growth factor (bFGF) have been shown to
98 Cs) (p<0.01) and the angiogenesis induced by basic fibroblast growth factor (bFGF) in a Matrigel plug
100 ascular endothelial growth factor (VEGF) and basic fibroblast growth factor (bFGF) increase SDF-1 exp
101 ascular endothelial growth factor (VEGF) and basic fibroblast growth factor (bFGF) increases the expr
108 ession of APN and its receptors, HB-EGF, and basic fibroblast growth factor (bFGF) messenger RNA was
110 We studied the effect of local delivery of basic fibroblast growth factor (bFGF) on angiogenesis an
111 FXa are inhibited by antibodies neutralizing basic fibroblast growth factor (bFGF) or by heparin.
112 fold differences in responsiveness to either basic fibroblast growth factor (bFGF) or vascular endoth
115 lant contained 0.5, 1.0, or 1.5 microg human basic fibroblast growth factor (bFGF) per milliliter of
116 asing ciliary neurotrophic factor (CNTF) and basic fibroblast growth factor (bFGF) production in Mull
117 broblasts (PDLFs) in terms of proliferation, basic fibroblast growth factor (bFGF) release, collagen
119 n, migration, capillary-like tube formation, basic fibroblast growth factor (bFGF) signaling, and per
122 viously reported that high concentrations of basic fibroblast growth factor (bFGF) support feeder-ind
123 duced with epidermal growth factor (EGF) and basic fibroblast growth factor (bFGF) to generate neural
124 The BMP antagonist noggin synergizes with basic fibroblast growth factor (bFGF) to repress BMP sig
125 This effect was rescued by the addition of basic fibroblast growth factor (bFGF) to the culture med
129 erences in angiogenic factors, the levels of basic fibroblast growth factor (bFGF) were compared with
130 with high-dose (80 ng) or low-dose (12.5 ng) basic fibroblast growth factor (bFGF) were placed in BAL
131 astasis is linked to increased production of basic fibroblast growth factor (bFGF), a powerful mitoge
132 s was induced on 10-day-old chick embryos by basic fibroblast growth factor (bFGF), and areas of neov
133 ransforming growth factor beta1 (TGF-beta1), basic fibroblast growth factor (bFGF), and epidermal gro
134 ntaining vascular endothelial growth factor, basic fibroblast growth factor (bFGF), and heparin to dr
135 rations of leukemia inhibitory factor (LIF), basic fibroblast growth factor (bFGF), and nerve growth
136 ignaling induced by epidermal growth factor, basic fibroblast growth factor (bFGF), and platelet-deri
137 le factor 1alpha (Hif-1alpha), Snail1, Slug, basic fibroblast growth factor (bFgf), and retinaldehyde
138 f vascular endothelial growth factor (VEGF), basic fibroblast growth factor (bFGF), and their recepto
139 tude of this effect to the angiogenic factor basic fibroblast growth factor (bFGF), and to investigat
140 ssion of matrix metalloproteinase-2 (MMP-2), basic fibroblast growth factor (bFGF), and vascular endo
141 so prevented the activation of NF-kappa B by basic fibroblast growth factor (bFGF), angiotensin-II (A
142 SMCs, when stimulated with thrombin, release basic fibroblast growth factor (bFGF), causing phosphory
143 lso the fourth ventricles and, together with basic fibroblast growth factor (bFGF), elicited subseque
144 ascular endothelial growth factor (VEGF) and basic fibroblast growth factor (bFGF), generated enthusi
145 g vascular endothelial growth factor (VEGF), basic fibroblast growth factor (bFGF), insulin-like grow
146 f vascular endothelial growth factor (VEGF), basic fibroblast growth factor (bFGF), interleukin-1 rec
147 nalyses were used to determine the levels of basic fibroblast growth factor (bFGF), neuronal nitric o
148 release polygalactone polymer rod containing basic fibroblast growth factor (bFGF), no growth factor
150 a with stromal-derived factor-1alpha (SDF1), basic fibroblast growth factor (bFGF), or bone morphogen
151 f vascular endothelial growth factor (VEGF), basic fibroblast growth factor (bFGF), placental growth
152 onic (E14) mice were initially cultured with basic fibroblast growth factor (bFGF), Sonic hedgehog, a
153 on factors epidermal growth factor (EGF) and basic fibroblast growth factor (bFGF), suggesting that P
154 and significantly affected the expression of basic fibroblast growth factor (bFGF), vascular endothel
155 etion (24 h) of tumor necrosis factor-alpha, basic fibroblast growth factor (bFGF), vascular endothel
156 or type 1 (FGFR-1), and a minority exhibited basic fibroblast growth factor (bFGF), whereas few expre
157 ascular endothelial growth factor (VEGF) and basic fibroblast growth factor (bFGF), which in turn, in
158 the effects of cyclooxygenase inhibitors on basic fibroblast growth factor (bFGF)- and vascular endo
160 process, integrin alphavbeta3 is specific to basic fibroblast growth factor (bFGF)-induced angiogenes
161 type-1 matrix metalloproteinase (MT1-MMP) on basic fibroblast growth factor (bFGF)-induced corneal ne
162 helial cell apoptosis but strongly inhibited basic fibroblast growth factor (bFGF)-induced endothelia
164 ed that ephrinB1 and EphB1 were expressed in basic fibroblast growth factor (bFGF)-induced vasculariz
165 scular endothelial growth factor (VEGF)- and basic fibroblast growth factor (bFGF)-mediated angiogeni
166 othelial cells, JAM-A has been implicated in basic fibroblast growth factor (bFGF)-regulated angiogen
167 nd latent antithrombins was observed in both basic fibroblast growth factor (bFGF)-stimulated and uns
181 ) reduced angiogenic sprouting stimulated by basic fibroblast growth factor (bFGF); (b) inhibited mat
183 ivin A, bone morphogenetic protein 4 (BMP4), basic fibroblast growth factor (bFGF, also known as FGF2
184 injection of KA alone or in combination with basic fibroblast growth factor (bFGF, intracerebroventri
185 tment was associated with decrease in plasma basic fibroblast growth factor (bFGF; P = .04) and incre
186 involved in cartilage homeostasis, including basic fibroblast growth factors (bFGF or FGF-2), transfo
187 ascular endothelial growth factor (VEGF) and basic fibroblasts growth factor (bFGF), indicating an in
188 es can bind to heparin-binding proteins (eg, basic fibroblast growth factor [bFGF] but not vascular e
189 As multiple angiogenic factors (VEGF and basic fibroblast growth factor [bFGF]) play a role in pr
190 ial growth factor [VEGF], interleukin-8, and basic fibroblast growth factor [bFGF]) were determined b
191 cytokines platelet-derived growth factor or basic fibroblast growth factor, both of which also stimu
192 of the common proangiogenic factors VEGF and basic fibroblast growth factor but rather induced expres
193 ors, vascular endothelial growth factor, and basic fibroblast growth factor by regulating IkappaBalph
194 rowth factor; (b) fibroblast growth factor 2/basic fibroblast growth factor; (c) interleukin (IL)-1al
195 lar endothelial growth factor (VEGF) but not basic fibroblast growth factor can induce gene expressio
197 esis inhibitor), or for thrombospondin-1 and basic fibroblast growth factor, confirms the segregation
198 viously shown that a truncated form of 24-kd basic fibroblast growth factor consisting of the amino t
200 neprilysin, resulted in a slight increase in basic fibroblast growth factor electrophoretic mobility
201 well a robust mitogenic response of ASCs to basic fibroblast growth factor, epidermal growth factor,
202 riments demonstrated that only PDGF, and not basic fibroblast growth factor, epidermal growth factor,
204 ied as a potent inhibitor of the VEGFR-2 and basic fibroblast growth factor (FGF) kinases (IC(50) = 1
206 cells (CECs) are simultaneously treated with basic fibroblast growth factor (FGF)-2 and RhoA inhibito
207 porcine aortic valve leaflets, we show that basic fibroblast growth factor (FGF-2) effectively block
210 ost angiogenic factors, we demonstrated that basic fibroblast growth factor (FGF-2) was a major cytok
211 fter stroke, levels of the 50-kD heparanase, basic fibroblast growth factor (FGF-2), and angiopoietin
212 reas steady-state hematopoiesis is normal in basic fibroblast growth factor (FGF-2)-knockout mice, pa
213 ties were analyzed in saliva samples; NO and basic fibroblast growth factor (FGF-b) levels were analy
214 ophage migration inhibition factor (MIF) and basic fibroblast growth factor (FGF-basic) cytokines tha
215 ulation of beta-chemokines (CCL2 and CCL22), basic fibroblast growth factor (FGF-basic), hepatocyte g
216 zebrafish leukemia inhibitory factor (Lif), basic fibroblast growth factor (Fgf2) and glial-cell-lin
218 ing approach to identify QTLs that influence basic fibroblast growth factor (FGF2) induced neovascula
222 essant treatments increase the expression of basic fibroblast growth factor (FGF2), a polypeptide gro
223 onditions included, but were not limited to, basic fibroblast growth factor (FGF2), heparin-binding E
224 ascular endothelial growth factor (VEGF) and basic fibroblast growth factor (FGF2), were previously i
225 vascular endothelial cell growth factor and basic fibroblast growth factor further decreased miR-223
226 , and vascular endothelial growth factor and basic fibroblast growth factor gene expression that is i
227 Activin A, bone morphogenetic protein 4, and basic fibroblast growth factor) generated CMs with high
228 red with baseline, levels of interleukin-17, basic fibroblast growth factor, granulocyte colony-stimu
229 n > EGF > heparin binding (HB)-EGF > IGF-1 > basic fibroblast growth factor >IL-8 > HGF > IL-1 > KGF>
230 ic factor (BDNF) expression but not VEGF and basic fibroblast growth factor in the ischemic brain com
231 ch as vascular endothelial growth factor and basic fibroblast growth factor in these osteogenic proge
233 hydrogenase II, vascular endothelial factor, basic fibroblast growth factor) in the context of human
234 cular endothelial growth factor (VEGF)-C and basic fibroblast growth factor increased in VR1814-infec
235 hibit vascular endothelial growth factor- or basic fibroblast growth factor-induced angiogenesis at c
236 scular endothelial growth factor-induced and basic fibroblast growth factor-induced angiogenesis at m
237 ir conditional ablation completely abrogated basic fibroblast growth factor-induced angiogenesis in M
238 epressor of NF-kappa B, I kappa B-2A, blocks basic fibroblast growth factor-induced angiogenesis in v
239 administered i.p. by osmotic pump inhibited basic fibroblast growth factor-induced angiogenesis in W
240 28.6 microM, and it significantly inhibited basic fibroblast growth factor-induced angiogenesis on t
241 not mitogenic on its own, CCN3 also enhances basic fibroblast growth factor-induced DNA synthesis.
242 eral injection of Marimastat also suppressed basic fibroblast growth factor-induced endothelial-mesen
243 monstrate that SPARC significantly increases basic fibroblast growth factor-induced fibroblast migrat
244 on and apoptosis but significantly inhibited basic fibroblast growth factor-induced fibroblast migrat
246 hich vascular endothelial growth factor- and basic fibroblast growth factor-induced neovacularization
247 r endothelial cell growth factor-induced and basic fibroblast growth factor-induced phosphorylation o
248 r endothelial cell growth factor-induced and basic fibroblast growth factor-induced proliferation, as
249 both vascular endothelial growth factor- and basic fibroblast growth factor-induced tissue plasminoge
250 ial growth factor, hepatocyte growth factor, basic fibroblast growth factor), inflammatory factors (i
252 cular endothelial cells (HMVECs) mediated by basic fibroblast growth factor, insulin-like growth fact
254 nd in response to angiogenic factors such as basic fibroblast growth factor, interleukin-8, tumor nec
255 emonstrates that the truncated form of 24-kd basic fibroblast growth factor is effective in suppressi
256 LD22-4, an 86-amino acid fragment of the basic fibroblast growth factor, is an inhibitor of cell
257 n 6, vascular endothelial growth factor, and basic fibroblast growth factor, known to be highly expre
258 ormal vascular endothelial growth factor and basic fibroblast growth factor levels, implying an essen
259 ll-vascular endothelial growth factor (VEGF)/basic fibroblast growth factor Matrigel plug assay, in w
260 porcine aortic endothelial cell (PAEC)-VEGF/basic fibroblast growth factor-Matrigel implants in nude
261 necessary to down-regulate the expression of basic fibroblast growth factor, matrix metalloprotease-9
262 Changes in plasma placental growth factor, basic fibroblast growth factor, matrix metalloproteinase
263 tube formation in response to both VEGF- and basic fibroblast growth factor-mediated angiogenic stimu
264 y of EphA2/Fc soluble receptors against VEGF/basic fibroblast growth factor-mediated neovascularizati
265 partially scrambled mutant aptamer inhibited basic fibroblast growth factor-mediated neovascularizati
266 ic neuronal responses after a combination of basic fibroblast growth factor, neurotrophin-3 and brain
268 g purified angiogenic growth factors such as basic fibroblast growth factor or vascular endothelial g
269 stromal cell-derived factor-1alpha, but not basic fibroblast growth factor or vascular endothelial g
270 s impregnated with an angiogenic stimulator (basic fibroblast growth factor or vascular endothelial g
271 3 at nanomolar concentrations inhibit either basic fibroblast-growth-factor or sphingosine-1-phosphat
272 nt increased compared to baseline, including basic fibroblast growth factor (P = .046), hepatocyte gr
273 uced by the profibrotic growth factors (GFs) basic fibroblast growth factor, platelet-derived growth
275 ofibrotic ontologies associated with reduced basic fibroblast growth factor production, which was con
276 In cultured endothelial cells, VEGF, but not basic fibroblast growth factor, promotes the Src-mediate
277 ctors (brain-derived neurotrophic factor and basic fibroblast growth factor), protein chaperones (hea
278 factor (VEGF), VEGF receptor 1 (Flt-1), and basic fibroblast growth factor proteins were higher in t
279 expression of nerve growth factor (NGF) and basic fibroblast growth factor, sensory axons were able
280 roteins (eg, platelet-derived growth factor, basic fibroblast growth factor) sequestered by platelets
281 esulted in an increased and sustained bFGF2 (basic fibroblast growth factor) signaling and promoted c
282 In vitro analyses suggest that Shp2 mediates basic fibroblast growth factor signals in stimulating se
283 on of a combination of three growth factors, basic fibroblast growth factor, stem cell factor, and en
284 noma cell adhesion, migration, invasion, and basic fibroblast growth factor stimulated proliferation
285 imulated chick chorioallantoic membranes and basic fibroblast growth factor-stimulated mouse corneas.
286 alternative pro-angiogenic factors, such as basic fibroblast growth factor, stromal-derived factor-1
288 we studied MC/Mph migration and assembly in basic fibroblast growth factor-supplemented Matrigel plu
289 rotrophic factor-family receptor alpha-1 and basic fibroblast growth factor supports proliferation of
290 dothelial cells was up-regulated by VEGF and basic fibroblast growth factor synergistically, and by h
291 TAT3 signalling, but they respond to activin/basic fibroblast growth factor to form self-renewing epi
292 growth in the absence of serum, but require basic fibroblast growth factor to remain undifferentiate
293 h vascular endothelial growth factor or with basic fibroblast growth factor to stimulate DNA synthesi
294 of soluble GDNF-family receptor alpha-1 and basic fibroblast growth factor to support replication.
295 growth factors (hepatocyte growth factor and basic fibroblast growth factor) to induce mammary acini
296 mers of epidermal growth factor receptor and basic fibroblast growth factor, two glycoproteins known
298 ation and decreased migration in response to basic fibroblast growth factor when compared with WT cel
299 inases (MMP)-2, MMP-3, MMP-7, and MMP-9, and basic fibroblast growth factor, which play crucial roles
300 um [termed hESC Cocktail (HESCO)] containing basic fibroblast growth factor, Wnt3a, April (a prolifer
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