ライフサイエンスコーパス: basilar

1 y examination revealed wheezes and decreased basilar air entry.

2 phase-contrast magnetic resonance imaging of basilar and carotid arteries to measure cerebral blood f

3 Basilar and posterior communicating arteries (PCAs) were

4 e for the cochlear resonance, presumably the basilar and tectorial membranes, move together in phase

5 s specific to a compartment either apical or basilar and that an additional mechanism may be required

6 occlusion (defined as intracranial carotid, basilar, and M1 segment of middle cerebral artery occlus

7 oss-sectional areas of the internal carotid, basilar, and middle cerebral arteries on the first day a

8 sham controls, with reductions of 33% in the basilar arbor and 28% in the apical arbor.

9 n lesioned and sham rats in either apical or basilar arbors; however, the proportion of immature to m

10 ive fungi, with extensive involvement of the basilar arterial circulation of the brain.

11 ChR agonists induced exclusive relaxation of basilar arterial rings without endothelium.

12 etic-dependent nitrergic dilation of porcine basilar arterial rings.

13 rculation with emphasis on the vertebral and basilar arteries (the posterior cerebral circulation).

14 ic stenosis or occlusion in vertebral and/or basilar arteries underwent large-vessel flow measurement

15 Rabbit basilar arteries were exposed to autologous EPCs ( appro

16 Changes of middle cerebral arteries and basilar arteries were extremely rare, thus we can say th

17 intracisternal delivery of autologous EPCs, basilar arteries were isolated and expression of vasoreg

18 roximal occlusions (internal carotid, M1, or basilar arteries).

19 It involves mostly vertebral and basilar arteries.

20 d wide middle cerebral, internal carotid and basilar arteries.

21 l arteries, posterior cerebral arteries, and basilar arteries.

22 er normal values for the middle cerebral and basilar arteries.

23 asal cisterns and subsequent invasion of the basilar arteries.

24 ertebral artery (40 patients, 12 bilateral), basilar artery (46 patients).

25 We examined the vascular responses of the basilar artery (BA) and its side branches through a cran

26 on in endovascular stroke treatment (EVT) of basilar artery (BA) occlusion.

27 lood vessels [internal carotid artery (ICA), basilar artery (BA), middle cerebral artery (MCA)], the

28 corrected p=0.010) and decreased flux in the basilar artery (mean difference -0.9 mL/s, 95% CI -1.5 t

29 c oxide synthase-dependent reactivity of the basilar artery and to determine a potential mechanism wh

30 One of the patients with a basilar artery aneurysm also had coarctation of the aort

31 Two had basilar artery aneurysms, 7 had internal carotid artery

32 ired acetylcholine-induced dilatation of the basilar artery during diabetes mellitus can be restored

33 ther cxcl12b or cxcr4a results in defects in basilar artery formation, showing that the assembly and

34 The basilar artery geometry strongly influenced both skewing

35 e examined whether impaired responses of the basilar artery in alcohol-fed rats in response to acetyl

36 itric oxide synthase-dependent dilatation of basilar artery in alcohol-fed rats.

37 n of L-NMMA did not affect dilatation of the basilar artery in diabetic rats in response to acetylcho

38 a direct acidifier of the cell, dilates rat basilar artery in K(ATP)-dependent fashion.

39 nM) produced dose-related dilatation of the basilar artery in non-alcohol-fed and alcohol-fed rats.

40 We measured the diameter of the basilar artery in non-diabetic rats, diabetic (streptozo

41 roM) produced dose-related dilatation of the basilar artery in non-diabetic rats, which was inhibited

42 A only partially inhibited dilatation of the basilar artery in response to acetylcholine in diabetic

43 ynthase, may contribute to dilatation of the basilar artery in response to acetylcholine in rats trea

44 lin treated diabetic rats, dilatation of the basilar artery in response to acetylcholine was signific

45 ital microscopy, we measured the diameter of basilar artery in response to nitric oxide synthase-depe

46 In contrast, dilatation of the basilar artery in response to nitroglycerin was similar

47 Dilatation of the basilar artery in response to nitroglycerin was similar

48 ine were not affected in either aorta or the basilar artery in vitro.

49 c oxide synthase-dependent reactivity of the basilar artery in vivo.

50 Measurements of the basilar artery indicated a considerable hypertrophy, ind

51 Basilar artery maximal diameter was greater in SF mice (

52 itive results also apply to the patient with basilar artery occlusion (BAO).

53 le to reperfusion therapy; 11 patients had a basilar artery occlusion and were excluded, leaving 100

54 ic mechanism, distal territory location, and basilar artery occlusive disease carried the poorest pro

55 d dilation that was markedly impaired in the basilar artery of mice expressing dominant-negative muta

56 ial recanalization of the middle cerebral or basilar artery on TCD were studied.

57 nce to that molecule and markedly suppressed basilar artery spasm after subarachnoid hemorrhage.

58 any benefit in the treatment of vertebral or basilar artery stenosis.

59 A 6-year-old male with vertebral-basilar artery thrombosis was recognized to have high-mo

60 g posterior fossa lesions, the management of basilar artery thrombosis, which may have a longer time

61 fed rats, and did not alter responses of the basilar artery to nitric oxide synthase-dependent or -in

62 ide dismutase did not alter responses of the basilar artery to nitroglycerin in alcohol-fed rats, and

63 We examined NT in basilar artery vascular smooth muscle cells (VSMCs) from

64 The prevalence of basilar artery vasospasm in children with moderate traum

65 Reactivity of the basilar artery was measured 2-3 months after injection o

66 The responses to acetylcholine in basilar artery were restored to normal after treatment w

67 In vitro or in vivo treatment of basilar artery with conditioned media from EPCs also cau

68 Unlike basilar artery, 12 weeks of a high-fat diet was not suff

69 he major artery supplying the hindbrain, the basilar artery, runs along the ventral keel of the hindb

70 f freshly isolated contractile VSMC from rat basilar artery, we found that EGF (100 ng ml(-1)) caused

71 s coexisting with a fusiform aneurysm of the basilar artery.

72 keel immediately adjacent to the assembling basilar artery.

73 -type Ca2+ channels in native VSMCs from rat basilar artery.

74 tery, the posterior cerebral artery, and the basilar artery.

75 ht account for the effects of alcohol on the basilar artery.

76 days (+/- 2 d) and 1.5 days (+/- 1 d) in the basilar artery.

77 ebral arteries and 5 days (+/- 2.5 d) in the basilar artery.

78 d one patient (1.02%) had an aneurysm of the basilar artery.

79 Animals underwent a basilar cistern inoculation of group B Streptococci to i

80 Animals underwent a basilar cistern tap receiving either sterile saline as a

81 y partially or completely disappeared in the basilar cisterns (P <.001) and cerebral sulcal subarachn

82 pines, and a previously unreported cell with basilar dendrites and complex, spiny apical processes (t

83 Basilar dendrites exhibited idiosyncratic geometry acros

84 1 region of the hippocampus pyramidal neuron basilar dendrites extend into the stratum oriens-alveus

85 ule cells were hypertrophic and formed spiny basilar dendrites from which the principal axon emerged.

86 spines, markers of excitatory inputs, on the basilar dendrites of Golgi-impregnated pyramidal neurons

87 C (Brodmann area 46) were Golgi-stained, and basilar dendrites of pyramidal cells in the deep half of

88 ntified in second-, third-, and fourth-order basilar dendrites of pyramidal neurons in layer II-III o

89 across compartments, between the apical and basilar dendrites, follows different rules and requires

90 characterized by large somata and extensive basilar dendrites.

91 pyramidal neurons with elongated "tap root" basilar dendrites.

92 and was specific to the apical, but not the basilar, dendrites of CA1.

93 tion of terminal branches of apical, but not basilar, dendrites of IL neurons.

94 t capture of L-LTP can also occur within the basilar dendritic compartment and that the tagging signa

95 compartment, can also take place within the basilar dendritic compartment and, if so, whether captur

96 After Golgi impregnation, basilar dendritic field size was estimated for layer V p

97 Smaller basilar dendritic field size was evident in proximal and

98 lation delivered in a restricted part of the basilar dendritic tree invariably produced sustained pla

99 ior circulation events and had vertebral and basilar imaging, of whom 37 (26.2%) had > or = 50% verte

100 s, severe and diffuse in nine, posterior and basilar in three, and limited to the apices in one (not

101 increased the wall thickness in the apex and basilar infarct regions compared with the control infarc

102 ne anomalies seen in paediatric patients are basilar invagination, C1-C2 instability, atlantoaxial ro

103 th zones were positioned periodically in the basilar layer to enhance branching of barb ridges.

104 to a constant suppressor displacement on the basilar membrane (as in experiments with wild-type anima

105 body vibrations normal to the surface of the basilar membrane (BM) at 0.8 (d(1)), 5.8 (d(2)), 15.6 (d

106 The mammalian basilar membrane (BM) consists of two collagen-fiber lay

107 Sound-evoked vibrations of the basilar membrane (BM) in anaesthetised guinea-pigs are s

108 Using a laser velocimeter, basilar membrane (BM) responses to tones were measured i

109 -otoacoustic-emissions (DPOAEs), and passive basilar membrane (BM) responses.

110 ounts of energy as passive, pressure-driven, basilar membrane (BM) traveling waves.

111 served to propagate longitudinally along the basilar membrane (BM) ultimately stimulate the mechano-s

112 h in vivo physiological measurements for the basilar membrane (BM) velocity, V(BM), frequency tuning

113 IHCs respond to basilar membrane (BM) vibration by producing a transduce

114 rimentally tested in this study by measuring basilar membrane (BM) vibrations at the cubic distortion

115 ty, harmonic distortion, and DC shift on the basilar membrane (BM), tectorial membrane (TM), and OHC

116 m to be observed in vivo at the level of the basilar membrane (BM).

117 rwise damped sound-induced vibrations of the basilar membrane [2-4], a mechanism known as negative da

118 erilymphatic chloride level manipulations of basilar membrane amplification in the living guinea pig.

119 by the passive mechanical properties of the basilar membrane and active feedback from the outer hair

120 ound stimuli from hair cell receptors in the basilar membrane and are arranged tonotopically.

121 ncy ratio, and measured as vibrations at the basilar membrane and at the stapes, and as sound pressur

122 of OHC electrical activity, pressure at the basilar membrane and basilar membrane displacement gave

123 ed by measuring the phase difference between basilar membrane and stapes vibrations.

124 ugh the fluid-tissue interaction between the basilar membrane and the fluid in scala tympani (ST) has

125 pled fluid-structure interaction between the basilar membrane and the scala fluids.

126 partition and active frequency tuning of the basilar membrane are enhanced in the cochleae of CD-1Cx3

127 ibited considerable re-growth of PAFs in the basilar membrane area.

128 s as compression waves rather than along the basilar membrane as backward-traveling waves.

129 timulation of the cochlear bone vibrates the basilar membrane as well.

130 isplacements of the reticular lamina and the basilar membrane at the 19 kHz characteristic place in g

131 nds on not only the passive mechanics of the basilar membrane but also an active amplification of the

132 duced comparable amplitudes of motion of the basilar membrane but differed in the polarity of their f

133 fferences in wave propagation time along the basilar membrane can provide the necessary delays, if th

134 gy is that tuned mechanical vibration of the basilar membrane defines the frequency response of the i

135 tivity, pressure at the basilar membrane and basilar membrane displacement gave direct evidence for p

136 as been considered to be proportional to the basilar membrane displacement or velocity.

137 ve relations between transducer currents and basilar membrane displacements are lacking, as well as t

138 sured acoustically and electrically elicited basilar membrane displacements from the cochleae of wild

139 Such faint sounds produce 0.1-nm basilar membrane displacements, a distance smaller than

140 to cochlear filtering that is independent of basilar membrane filtering.

141 hick fibers that coursed radially across the basilar membrane in small fascicles, gave off small bran

142 on (reflecting the nonlinear response of the basilar membrane in the cochlea), followed by linear sum

143 A hearing sensation arises when the elastic basilar membrane inside the cochlea vibrates.

144 A simple monophasic vibratory mode of the basilar membrane is found at both ends of the cochlea.

145 OHCs at various cochlear locations while the basilar membrane is mechanically stimulated.

146 The basilar membrane is typically set into motion through ai

147 s at the stapes earlier than at the measured basilar membrane location.

148 man speech, is not principally determined by basilar membrane mechanics.

149 are characterized here through recordings of basilar membrane motion and hair cell extracellular rece

150 was found by taking the phase difference of basilar membrane motion between two longitudinally space

151 sducer currents (or receptor potentials) and basilar membrane motion in an excised and bisected cochl

152 istortion product otoacoustic emissions, and basilar membrane motion indicate that the TM remains fun

153 The model is fit to in vivo basilar membrane motion with one free parameter for the

154 energy at the appropriate moment to enhance basilar membrane motion.

155 cells that detect and amplify sound-induced basilar membrane motions.

156 odel could be tested by measuring TTS on the basilar membrane of the Otoa(EGFP/EGFP) mice to improve

157 The detectable basilar membrane response to a low-level 16-kHz tone occ

158 In a sensitive cochlea, the basilar membrane response to transient excitation of any

159 he amplification and frequency tuning of the basilar membrane responses to sounds are almost normal.

160 compared with that at the commonly measured basilar membrane side.

161 tion result from longitudinal differences in basilar membrane stiffness and numerous individual grada

162 lfrog's amphibian papilla lacks the flexible basilar membrane that effects tuning in mammals, its aff

163 ane in hearing: it enables the motion of the basilar membrane to optimally drive the inner hair cells

164 also found in the response of the mammalian basilar membrane to sound, signals the operation of an a

165 vibration collaboratively interacts with the basilar membrane traveling wave primarily through the co

166 hlear outer hair cells (OHCs) to amplify the basilar membrane traveling wave; however, it is unclear

167 Compared with basilar membrane traveling waves, tectorial membrane tra

168 sms within outer hair cells that amplify the basilar membrane travelling wave.

169 peaked at a higher frequency than transverse basilar membrane tuning in the passive, postmortem condi

170 apical ends of outer hair cells and from the basilar membrane using a custom-built heterodyne low-coh

171 in the cochlea, as shown by measurements of basilar membrane velocity and auditory nerve responses t

172 The basilar membrane velocity was measured through the trans

173 hlear responses are amplified during maximum basilar membrane velocity.

174 n discrepancies between previously published basilar membrane vibration and auditory nerve single uni

175 Data also show that basilar membrane vibration at the emission frequency is

176 The phase relation of reticular lamina to basilar membrane vibration changes with frequency by up

177 ate that outer hair cells do not amplify the basilar membrane vibration directly through a local feed

178 ear amplifier from available measurements of basilar membrane vibration in sensitive mammalian cochle

179 best to different sound frequencies because basilar membrane vibration is mechanically tuned to diff

180 sions showed no significant change while the basilar membrane vibration nearly disappeared.

181 a vibration is substantially larger than the basilar membrane vibration not only at the best frequenc

182 The current data indicate that basilar membrane vibration was not involved in the backw

183 ve yielded ever-better descriptions of gross basilar membrane vibration, the internal workings of the

184 ically been limited to point measurements of basilar membrane vibration.

185 ase differences between reticular lamina and basilar membrane vibrations are absent in postmortem coc

186 electrically in the second turn and measured basilar membrane vibrations at two longitudinal location

187 n longitudinal location, electrically evoked basilar membrane vibrations showed the same tuning and p

188 generate forces for amplifying sound-induced basilar membrane vibrations, yet how cellular forces amp

189 on the enhanced mechanical properties of the basilar membrane within the cochlear duct.

190 al coupling between adjacent sections of the basilar membrane, and such coupling may be critical for

191 o Deiters' cells, the sulcus epithelium, the basilar membrane, and the surface of the spiral limbus.

192 ratchet: Sound-evoked forces, acting on the basilar membrane, are transmitted to the hair bundles, w

193 h constrain outer hair cells standing on the basilar membrane, causes a leftward shift in outer hair

194 efrom, may not involve the usual wave on the basilar membrane, suggesting that additional cochlear st

195 zing their responses to the vibration of the basilar membrane, the radial vibrations of the tectorial

196 n physical and geometrical properties of the basilar membrane, the sensitivity or gain of the hearing

197 ning which OHCs enhance the vibration of the basilar membrane, thereby tuning the gain of cochlear am

198 uter hair cells that generates forces on the basilar membrane, we demonstrate that these forces inter

199 bing amplification in narrow segments of the basilar membrane, we further show that a cochlear travel

200 echanical filters analogous to the cochlea's basilar membrane, which deconstructs complex sounds into

201 cells that effectively couple energy to the basilar membrane, which reduces sensitivity.

202 interferometry to measure vibrations of the basilar membrane.

203 and were enhanced compared with those at the basilar membrane.

204 f sensory and supporting cells riding on the basilar membrane.

205 ng the organ of Corti than in displacing the basilar membrane.

206 nge well below the resonant frequency of the basilar membrane.

207 decouples active hair-bundle forces from the basilar membrane.

208 ar to be related in part to the width of the basilar membrane.

209 amina, but only a slow tuned response of the basilar membrane.

210 ts along the cochlea, similar to that of the basilar membrane.

211 ously measured close to the sensory tissue's basilar membrane.

212 traveling wave to amplify the motion of the basilar membrane.

213 increases the sound-evoked vibrations of the basilar membrane.

214 nd that the stapes vibrates earlier than the basilar membrane.

215 line in motion amplitude occurred across the basilar membrane.

216 ular lamina, to the transverse motion of the basilar membrane.

217 on caused motion of a minimal portion of the basilar membrane.

218 seen when the beam was directed towards the basilar membrane.

219 her than an immediate local vibration of the basilar membrane; this travelling wave vibrates in phase

220 hese mice have a low-frequency hearing loss, basilar-membrane and neural tuning are both significantl

221 ries are very similar to the trajectories of basilar-membrane peak velocity toward scala tympani.

222 10], we show that the nonlinear component of basilar-membrane responses to sound stimulation leads th

223 Whether measured by basilar-membrane vibration, nerve-fiber activity, or per

224 rvating different loci on the left and right basilar membranes.

225 g the developing tonotopic axis of the chick basilar papilla (BP) identified a gradient of Bmp7.

226 ly phases of cell orientation in the chicken basilar papilla (BP), Vangl2 is present at supporting ce

227 of efferent fibers that innervate the chick basilar papilla (BP).

228 o the cochlear nerve, cochlear ganglion, and basilar papilla (i.e., avian cochlea) in fixed tissue an

229 a macula, whereas SFRP2 is maintained in the basilar papilla along with Fzd10 and Wnt7b.

230 ista, the utricle, the saccule, and both the basilar papilla and lagenar macula form.

231 mately characteristic frequency) between the basilar papilla and NM is established as cochlear nerve

232 Flanking the basilar papilla are Wnt7a, Wnt9a, Wnt11, and SFRP2 on th

233 f the substructural alterations in the chick basilar papilla at the earliest signs of hair cell degen

234 ation pathways among supporting cells in the basilar papilla but not in the utricular macula.

235 ed in most support cells in the mature chick basilar papilla but not in vestibular organs of the chic

236 ment sufficient to destroy hair cells in the basilar papilla causes a rapid, transient downregulation

237 ll ears tested, even in non-TM species whose basilar papilla contained as few as 50-60 hair cells.

238 The avian basilar papilla contains tall and short hair cells, with

239 Interestingly, hair cells in the avian basilar papilla demonstrate both electrical resonance an

240 cells that reenter the mitotic cycle in the basilar papilla do not express detectable levels of FGFR

241 s after drug-induced hair cell damage to the basilar papilla in an opposite way to that found in the

242 ations within the apical half of the chicken basilar papilla in vivo and found broadly-tuned travelli

243 in the middle region along the length of the basilar papilla in which, in one cell, the terminals occ

244 re, frequency tuning within the apical avian basilar papilla is not mechanical, and likely derives fr

245 onotopic mapping observed between NM and the basilar papilla later in development.

246 In the chicken cochlea (the basilar papilla or BP), dying hair cells are extruded fr

247 m 1 to 8-20 weeks, whereas hair cells in the basilar papilla remained morphologically intact out to 2

248 ut patches from hair cells along the chicken basilar papilla revealed 'tonotopic' gradations in calci

249 A semi-intact preparation of the chick basilar papilla was developed to study calcium-dependent

250 Most notably, the basilar papilla, an auditory organ, contained infected s

251 neural edge of the avian auditory organ, the basilar papilla, by embryonic day 5 (E5).

252 Finally, in contrast to the chick basilar papilla, ectopic activation of Notch signaling d

253 the position of their cell bodies along the basilar papilla, foreshadowing the tonotopic mapping obs

254 junctions connecting supporting cells of the basilar papilla, in which its immunofluorescence colocal

255 In the basilar papilla, nuclear cProx1 expression is down-regul

256 uditory sensory organ (the lagena macula and basilar papilla, respectively), which each have a distin

257 ontribute to the structural integrity of the basilar papilla, the maintenance of the ionic barrier at

258 auditory subdivision, the cochlear duct, or basilar papilla.

259 r cell ejection from the proximal end of the basilar papilla.

260 bryonic day 11 (E11) of the developing chick basilar papilla.

261 is expressed early in the development of the basilar papilla.

262 cells from the frog sacculus and the turtle basilar papilla.

263 lication of ACh to hair cells in the chicken basilar papilla.

264 The basilar papillae were studied by conventional transmissi

265 Similarly, in cultured chicken basilar papillae, supporting cell proliferation in respo

266 hout HNSCC specimens, when compared with the basilar pattern of protein expression and minimal inflam

267 plasia, these gradients were dissipated, and basilar plaques were formed within a subset of basal cry

268 r the organization of projections within the basilar pons and spinal cord.

269 addition, loss of Nfib results in defects in basilar pons formation and hippocampus development that

270 In addition, the basilar pons forms normally in dystrophin-deficient mice

271 n in the Large glycosyltransferase gene, the basilar pons is absent.

272 ts from lesions involving the neurons of the basilar pons that link the ipsilateral cerebral cortex w

273 ed in 25 patients with focal infarcts in the basilar pons to determine whether pontine lacunar syndro

274 e required for the normal development of the basilar pons, one of several precerebellar nuclei of the

275 aw region projects mainly to the ipsilateral basilar pons, the MI whisker region has significantly mo

276 culation are localized to rostral and medial basilar pons; hand coordination is medial and ventral in

277 se but not dystrophin is required for normal basilar pontine development.

278 dbrain indicates that the development of the basilar pontine nuclei is delayed, with pontine neurons

279 One such population, the neurons of the basilar pontine nuclei, expresses high levels of Nfi pro

280 increased axonal growth in the deafferented basilar pontine nuclei.

281 rpositus nucleus (IPN) of the cerebellum and basilar pontine nucleus (PN) during different phases of

282 terograde tracing experiments identified the basilar pontine nucleus at the confluence of outputs fro

283 ction from the medial central nucleus to the basilar pontine nucleus.

284 We also provide evidence that the convergent basilar pontine pathways carry corollary discharges from

285 rate sensory (upper body proprioceptive) and basilar pontine pathways onto individual granule cells a

286 esions (pure motor hemiplegia) to incomplete basilar pontine syndrome and restricted deficits after s

287 There was a basilar predominance of interstitial lung disease at CT.

288 n the stria vascularis, spiral ligament, sub-basilar region, stromal tissue, and the spiral and vesti

289 ng, non-severe injury mechanism, no signs of basilar skull fracture, and no severe headache) had a ne

290 The prevalence of > or = 50% vertebral and basilar stenosis in posterior circulation transient isch

291 r = 50% apparently symptomatic vertebral and basilar stenosis using contrast-enhanced MRA in consecut

292 circulation events, > or = 50% vertebral and basilar stenosis was associated multiple transient ischa

293 Presence of > or = 50% vertebral and basilar stenosis was unrelated to age, sex or vascular r

294 whom 37 (26.2%) had > or = 50% vertebral and basilar stenosis, compared with 41 (11.5%) patients with

295 are) CA1 spine density, as well as increased basilar (stratum oriens) CA3 spine density.

296 s, manifested initially as meningitis and/or basilar stroke.

297 he basilar to apical than from the apical to basilar surface of PMC.

298 Each participant was asked to identify a basilar tip aneurysm in a validated model head.

299 PMNL migration was higher from the basilar to apical than from the apical to basilar surfac

300 white matter tracts and lower volume in the basilar (ventral) pons, cerebellar white matter and visu