ライフサイエンスコーパス: basilar artery

1 roximal occlusions (internal carotid, M1, or basilar arteries).

2 ebral arteries and 5 days (+/- 2.5 d) in the basilar artery.

3 d one patient (1.02%) had an aneurysm of the basilar artery.

4 s coexisting with a fusiform aneurysm of the basilar artery.

5 keel immediately adjacent to the assembling basilar artery.

6 -type Ca2+ channels in native VSMCs from rat basilar artery.

7 tery, the posterior cerebral artery, and the basilar artery.

8 ht account for the effects of alcohol on the basilar artery.

9 e in modulating constrictor responses of the basilar artery.

10 days (+/- 2 d) and 1.5 days (+/- 1 d) in the basilar artery.

11 ormed over the ventral medulla to expose the basilar artery.

12 er normal values for the middle cerebral and basilar arteries.

13 asal cisterns and subsequent invasion of the basilar arteries.

14 It involves mostly vertebral and basilar arteries.

15 d wide middle cerebral, internal carotid and basilar arteries.

16 l arteries, posterior cerebral arteries, and basilar arteries.

17 junction conductances in SMC pairs from rat basilar arteries.

18 Unlike basilar artery, 12 weeks of a high-fat diet was not suff

19 osterior cerebral arteries, the VAs, and the basilar artery; 4) intracranial atheromatous branch dise

20 ertebral artery (40 patients, 12 bilateral), basilar artery (46 patients).

21 h muscle cells were isolated from guinea-pig basilar artery and conventional whole-cell recordings of

22 c oxide synthase-dependent reactivity of the basilar artery and to determine a potential mechanism wh

23 One of the patients with a basilar artery aneurysm also had coarctation of the aort

24 Two had basilar artery aneurysms, 7 had internal carotid artery

25 We examined the vascular responses of the basilar artery (BA) and its side branches through a cran

26 on in endovascular stroke treatment (EVT) of basilar artery (BA) occlusion.

27 lood vessels [internal carotid artery (ICA), basilar artery (BA), middle cerebral artery (MCA)], the

28 of this study was to measure the response of basilar artery blood flow to hyperventilation in patient

29 e is augmented in hypertension was tested in basilar artery cells from Wistar rats exhibiting stable

30 ad wider posterior communicating but smaller basilar artery diameters.

31 ired acetylcholine-induced dilatation of the basilar artery during diabetes mellitus can be restored

32 c oxide synthase-dependent dilatation of the basilar artery during diabetes mellitus may be related,

33 ic oxide synthase-mediated dilatation of the basilar artery during diabetes mellitus.

34 Doppler ultrasonography was used to measure basilar artery flow during rest and after hyperventilati

35 ther cxcl12b or cxcr4a results in defects in basilar artery formation, showing that the assembly and

36 The basilar artery geometry strongly influenced both skewing

37 e examined whether impaired responses of the basilar artery in alcohol-fed rats in response to acetyl

38 itric oxide synthase-dependent dilatation of basilar artery in alcohol-fed rats.

39 n of L-NMMA did not affect dilatation of the basilar artery in diabetic rats in response to acetylcho

40 c oxide synthase-dependent dilatation of the basilar artery in diabetic rats towards that observed in

41 a direct acidifier of the cell, dilates rat basilar artery in K(ATP)-dependent fashion.

42 nM) produced dose-related dilatation of the basilar artery in non-alcohol-fed and alcohol-fed rats.

43 We measured the diameter of the basilar artery in non-diabetic rats, diabetic (streptozo

44 roM) produced dose-related dilatation of the basilar artery in non-diabetic rats, which was inhibited

45 ) did not alter constrictor responses of the basilar artery in nondiabetic and diabetic rats.

46 duced similar dose-related dilatation of the basilar artery in nondiabetic and diabetic rats.

47 U/ml) did not alter baseline diameter of the basilar artery in nondiabetic and diabetic rats.

48 roM) produced dose-related dilatation of the basilar artery in nondiabetic and diabetic rats.

49 de dismutase did not alter dilatation of the basilar artery in nondiabetic rats.

50 A only partially inhibited dilatation of the basilar artery in response to acetylcholine in diabetic

51 ynthase, may contribute to dilatation of the basilar artery in response to acetylcholine in rats trea

52 lin treated diabetic rats, dilatation of the basilar artery in response to acetylcholine was signific

53 ital microscopy, we measured the diameter of basilar artery in response to nitric oxide synthase-depe

54 In contrast, dilatation of the basilar artery in response to nitroglycerin was similar

55 Dilatation of the basilar artery in response to nitroglycerin was similar

56 c oxide played a role in constriction of the basilar artery in response to the agonists.

57 ine were not affected in either aorta or the basilar artery in vitro.

58 We measured the diameter of the basilar artery in vivo in nondiabetic and diabetic rats

59 c oxide synthase-dependent reactivity of the basilar artery in vivo.

60 tes mellitus on constrictor responses of the basilar artery in vivo.

61 Measurements of the basilar artery indicated a considerable hypertrophy, ind

62 Basilar artery maximal diameter was greater in SF mice (

63 corrected p=0.010) and decreased flux in the basilar artery (mean difference -0.9 mL/s, 95% CI -1.5 t

64 itive results also apply to the patient with basilar artery occlusion (BAO).

65 le to reperfusion therapy; 11 patients had a basilar artery occlusion and were excluded, leaving 100

66 ic mechanism, distal territory location, and basilar artery occlusive disease carried the poorest pro

67 d dilation that was markedly impaired in the basilar artery of mice expressing dominant-negative muta

68 e smooth muscle cells were isolated from the basilar artery of the guinea-pig and, within 10 h, inwar

69 ial recanalization of the middle cerebral or basilar artery on TCD were studied.

70 he major artery supplying the hindbrain, the basilar artery, runs along the ventral keel of the hindb

71 The greater basilar artery sensitivity to hyperventilation shown by

72 Our data indicate that basilar artery SMCs are coupled in vivo in a richly comp

73 ry effects on voltage-gated Ca2+ channels of basilar artery smooth muscle cells.

74 nce to that molecule and markedly suppressed basilar artery spasm after subarachnoid hemorrhage.

75 any benefit in the treatment of vertebral or basilar artery stenosis.

76 rculation with emphasis on the vertebral and basilar arteries (the posterior cerebral circulation).

77 A 6-year-old male with vertebral-basilar artery thrombosis was recognized to have high-mo

78 g posterior fossa lesions, the management of basilar artery thrombosis, which may have a longer time

79 oxide modulates constrictor responses of the basilar artery to angiotensin II, arginine vasopressin,

80 Thus, responses of the basilar artery to important vasoactive agonists are not

81 fed rats, and did not alter responses of the basilar artery to nitric oxide synthase-dependent or -in

82 ide dismutase did not alter responses of the basilar artery to nitroglycerin in alcohol-fed rats, and

83 ic stenosis or occlusion in vertebral and/or basilar arteries underwent large-vessel flow measurement

84 We examined NT in basilar artery vascular smooth muscle cells (VSMCs) from

85 The prevalence of basilar artery vasospasm in children with moderate traum

86 Reactivity of the basilar artery was measured 2-3 months after injection o

87 The diameter of the basilar artery was measured using intravital microscopy

88 f freshly isolated contractile VSMC from rat basilar artery, we found that EGF (100 ng ml(-1)) caused

89 Rabbit basilar arteries were exposed to autologous EPCs ( appro

90 Changes of middle cerebral arteries and basilar arteries were extremely rare, thus we can say th

91 intracisternal delivery of autologous EPCs, basilar arteries were isolated and expression of vasoreg

92 Elongated, ectatic, tortuous vertebral and basilar arteries were the most common angiographic and p

93 Rings of canine basilar artery were incubated with increasing titers of

94 The responses to acetylcholine in basilar artery were restored to normal after treatment w

95 uced marked dose-related constriction of the basilar artery which also was similar in nondiabetic and

96 uced only minimal changes in diameter of the basilar artery which were similar in nondiabetic and dia

97 In vitro or in vivo treatment of basilar artery with conditioned media from EPCs also cau