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1 e ortholog of alpha9 in the chick's cochlea (basilar papilla).
2 auditory subdivision, the cochlear duct, or basilar papilla.
3 r cell ejection from the proximal end of the basilar papilla.
4 bryonic day 11 (E11) of the developing chick basilar papilla.
5 is expressed early in the development of the basilar papilla.
6 cells from the frog sacculus and the turtle basilar papilla.
7 f cell proliferation in both the utricle and basilar papilla.
8 lectrically tuned auditory organ, the turtle basilar papilla.
9 lication of ACh to hair cells in the chicken basilar papilla.
10 s caused by the acoustic injury to the chick basilar papilla.
11 tributions of KCa channel isoforms along the basilar papilla.
12 oximal (mid-frequency) region of the chicken basilar papilla.
13 sectional anatomical pathology of the BWC's basilar papilla.
15 rn blot analysis of total RNA from the chick basilar papilla after noise trauma revealed increased le
18 o the apical or low frequency portion of the basilar papilla and coincided with maximal expression of
19 tage 20, the lateral crista at stage 22, the basilar papilla and lagena at stage 23, the macula utric
21 mately characteristic frequency) between the basilar papilla and NM is established as cochlear nerve
22 those studies, the surface anatomy of BWC's basilar papilla and sacculus was examined utilizing scan
23 ons and in peripheral fibers innervating the basilar papilla and synapsing at the base of hair cells.
24 r cells and support cells in the utricle and basilar papilla, and its expression does not change duri
27 f the substructural alterations in the chick basilar papilla at the earliest signs of hair cell degen
29 the hearing organ of the chicken, called the basilar papilla (BP), after cellular differentiation.
30 ly phases of cell orientation in the chicken basilar papilla (BP), Vangl2 is present at supporting ce
33 ed in most support cells in the mature chick basilar papilla but not in vestibular organs of the chic
35 ment sufficient to destroy hair cells in the basilar papilla causes a rapid, transient downregulation
36 ll ears tested, even in non-TM species whose basilar papilla contained as few as 50-60 hair cells.
39 cells that reenter the mitotic cycle in the basilar papilla do not express detectable levels of FGFR
41 the position of their cell bodies along the basilar papilla, foreshadowing the tonotopic mapping obs
42 o the cochlear nerve, cochlear ganglion, and basilar papilla (i.e., avian cochlea) in fixed tissue an
43 s after drug-induced hair cell damage to the basilar papilla in an opposite way to that found in the
44 ations within the apical half of the chicken basilar papilla in vivo and found broadly-tuned travelli
45 in the middle region along the length of the basilar papilla in which, in one cell, the terminals occ
46 junctions connecting supporting cells of the basilar papilla, in which its immunofluorescence colocal
47 ecursor during HC regeneration in the mature basilar papilla is consistent with their developmental h
48 re, frequency tuning within the apical avian basilar papilla is not mechanical, and likely derives fr
52 ry brainstem, the cochlear ganglion, and the basilar papilla of chicks from embryonic (E) day 5 to E2
53 eural may express neurofilament and that the basilar papilla of the neonatal chicken is not morpholog
55 m 1 to 8-20 weeks, whereas hair cells in the basilar papilla remained morphologically intact out to 2
56 uditory sensory organ (the lagena macula and basilar papilla, respectively), which each have a distin
57 ut patches from hair cells along the chicken basilar papilla revealed 'tonotopic' gradations in calci
60 tion density among hair cells of the chick's basilar papilla (the avian analog of the mammalian Organ
61 ontribute to the structural integrity of the basilar papilla, the maintenance of the ionic barrier at
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