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1 y transmission in the PFC at inputs from the basolateral amygdala.
2 gulation of AMPA-receptor endocytosis in the basolateral amygdala.
3 in emotion-related circuits anchored in the basolateral amygdala.
4 d striatum and remained intact in cortex and basolateral amygdala.
5 eceptors on output projection neurons of the basolateral amygdala.
6 enlargement is localized specifically to the basolateral amygdala.
7 ses and theta oscillations recorded from the basolateral amygdala.
8 ion, and these effects are most prominent in basolateral amygdala.
9 ering protein expression within the adjacent basolateral amygdala.
10 rentially modulated through the vSub and the basolateral amygdala.
11 a direct pathway between anterior insula and basolateral amygdala.
12 ocial interaction-specific expression in the basolateral amygdala.
13 mmunohistochemical staining for c-Fos in the basolateral amygdala.
14 of NPS to modulate neuronal activity in the basolateral amygdala.
15 contralateral cortex, nucleus accumbens, and basolateral amygdala.
16 e in initiating asymmetric activation of the basolateral amygdala.
17 put sampled from only the right but not left basolateral amygdala.
18 rected actions has been found to involve the basolateral amygdala.
19 and complexity of neurites of neurons in the basolateral amygdala.
20 e medial prefrontal cortex, hippocampus, and basolateral amygdala.
21 alcium/calmodulin-dependent kinase II in the basolateral amygdala.
22 mmunofluorescence, and RNA sequencing of the basolateral amygdala.
23 cortex via the paraventricular thalamus and basolateral amygdala.
24 ippocampal-entorhinal cortex network and the basolateral amygdala.
25 INs, and principal excitatory neurons in the basolateral amygdala.
26 exclusively within excitatory neurons of the basolateral amygdala.
27 mbens, the medial prefrontal cortex, and the basolateral amygdala.
28 lective potentiator CIQ bilaterally into the basolateral amygdala (3, 10, or 30 mug/side) following e
29 focused on the primary output neurons of the basolateral amygdala, a brain region that plays a key ro
30 chronized theta oscillations between V2L and basolateral amygdala, a physiological correlate of succe
31 dal neurons such as hippocampal area CA1 and basolateral amygdala, a slow afterhyperpolarization (sAH
32 eported neural correlates of salience in the basolateral amygdala (ABL) of rats during learning in an
36 norepinephrine (NE) administration into the basolateral amygdala after training on an inhibitory avo
37 cingulate, thalamus, nucleus accumbens, and basolateral amygdala-all regions involved in motivated b
40 ess-mediated increases in GABA efflux in the basolateral amygdala, an event accompanied by no changes
42 42 had increased c-Fos expression within the basolateral amygdala and in serotonergic neurons in the
43 et, PTEN, colocalized with miR-144-3p in the basolateral amygdala and showed functional downregulatio
45 s (accumbens shell, ventral hippocampus, and basolateral amygdala) and differences (medial prefrontal
46 268 expression in the infralimbic cortex and basolateral amygdala, and elevated c-Fos or Zif268 expre
47 terior cingulate cortex, infralimbic cortex, basolateral amygdala, and habenula inhibited social play
49 synaptic properties of ventral hippocampus, basolateral amygdala, and prefrontal cortex input to the
51 revealed that functional Hcn channels in the basolateral amygdala are necessary for conditioned fear
52 macology, revealing that Hcn channels in the basolateral amygdala are required for fear acquisition a
56 valbumin-expressing (PV) interneurons in the basolateral amygdala (BLA) a dedicated role in the selec
57 he dorsomedial prefrontal cortex (dmPFC) and basolateral amygdala (BLA) and altered synaptic transmis
59 ing the intrinsic functional connectivity of basolateral amygdala (BLA) and centromedial amygdala (CM
60 question, we recorded activity in the mPFC, basolateral amygdala (BLA) and dorsal and ventral hippoc
62 this hypothesis directly, as neurons in both basolateral amygdala (BLA) and gustatory cortex (GC)-ana
63 aversive unconditioned stimuli (USs) in the basolateral amygdala (BLA) and have examined their role
64 that NgR1 expression is required in both the basolateral amygdala (BLA) and infralimbic (IL) cortex t
66 itioned fear requires neural activity in the basolateral amygdala (BLA) and medial prefrontal cortex
67 ation of specific brain regions, such as the basolateral amygdala (BLA) and nucleus accumbens (NAc),
68 , we investigate causal contributions of the basolateral amygdala (BLA) and orbitofrontal cortex (OFC
69 ered activity in brain systems including the basolateral amygdala (BLA) and orbitofrontal cortex (OFC
71 e examined the role of a pathway linking the basolateral amygdala (BLA) and pDMS in such goal-directe
72 agonistic relationship may exist between the basolateral amygdala (BLA) and PFC during emotional proc
73 gion of the dorsal hippocampus (CA1) and the basolateral amygdala (BLA) and that of dopaminergic, nor
75 nderlie learning and memory deficits via the basolateral amygdala (BLA) and the hippocampus; however,
76 in the higher-order auditory cortex Te2 and basolateral amygdala (BLA) and their crosstalk during th
78 We therefore evaluated the contribution of basolateral amygdala (BLA) and ventral hippocampus (vHPC
80 l cortex (OFC) and its interactions with the basolateral amygdala (BLA) are critical for goal-directe
82 (vHPC), medial prefrontal cortex (mPFC), and basolateral amygdala (BLA) are each required for the exp
83 Reciprocal circuits between the mPFC and basolateral amygdala (BLA) are particularly important fo
84 depression (LTD) in principal neurons of the basolateral amygdala (BLA) are thought to underlie the a
85 eir synaptic connections with neurons in the basolateral amygdala (BLA) at neonatal to adult developm
86 had stronger functional connectivity of the basolateral amygdala (BLA) complex with the pregenual an
90 Neuropeptide Y (NPY) administration into the basolateral amygdala (BLA) decreases anxiety-like behavi
91 s-Tyr-d-Trp-Orn-Thr-Pen-Thr-NH(2)), into the basolateral amygdala (BLA) during posttraining, nonconti
92 a dampening of neuronal excitability in the basolateral amygdala (BLA) ex vivo and in vivo, and were
93 ts with bilateral excitotoxic lesions of the basolateral amygdala (BLA) failed to display such cue-po
94 by miR-19b, Adrb1 levels were reduced in the basolateral amygdala (BLA) following chronic stress.
98 imbic medial prefrontal cortex (PL-mPFC) and basolateral amygdala (BLA) have been implicated in the e
99 By contrast, inhibitory Gi-DREADDs in the basolateral amygdala (BLA) impaired the acquisition of b
100 rtance of the orbitofrontal cortex (OFC) and basolateral amygdala (BLA) in acquisition of the rGT and
101 udies implicate cholinergic signaling in the basolateral amygdala (BLA) in behaviors related to stres
102 the FAAH inhibitor URB597, directly into the basolateral amygdala (BLA) in conjunction with memory re
103 n addition, we infused mifepristone into the basolateral amygdala (BLA) in ethanol-seeking animals as
104 we identified a role for miR-34a within the basolateral amygdala (BLA) in fear memory consolidation.
105 n between the roles of the central (CeA) and basolateral amygdala (BLA) in regulating social behavior
106 nts has demonstrated a critical role for the basolateral amygdala (BLA) in such reward-seeking action
108 from the ventral hippocampus (vHipp) and the basolateral amygdala (BLA) in the medial prefrontal cort
110 e 5-HT(2C)R agonist CP-809101 in the lateral/basolateral amygdala (BLA) increases shock-elicited fear
111 minalis (jcBNST) is activated in response to basolateral amygdala (BLA) inputs through the stria term
113 is structure, and the insular cortex and the basolateral amygdala (BLA) interact during CTA formation
117 s of E2 signaling.SIGNIFICANCE STATEMENT The basolateral amygdala (BLA) is a key structure of the fea
128 tual meaning of the LFH call in responses of basolateral amygdala (BLA) neurons from awake, freely mo
129 ted regions, it is reasonable to expect that basolateral amygdala (BLA) neurons should produce a rich
130 dator) induces long-lasting sensitization of basolateral amygdala (BLA) noradrenergic (NE) receptors
131 either central nucleus of amygdala (CeA) or basolateral amygdala (BLA) of female rats with one parti
132 ve onto excitatory projection neurons in the basolateral amygdala (BLA) of juvenile Fmr1(-/y) knockou
134 nist actions on synaptic transmission in the basolateral amygdala (BLA) of the rat were examined usin
135 by specifically overexpressing FKBP51 in the basolateral amygdala (BLA) or central amygdala resulted
136 divergent downstream structures such as the basolateral amygdala (BLA) or nucleus accumbens (NAc).
137 memory but was ineffective when given in the basolateral amygdala (BLA) or the ventral medial prefron
138 ore, this effect was not mediated via the LC-basolateral amygdala (BLA) pathway, because BLA inactiva
139 ow that neuropeptide-receptor systems in the basolateral amygdala (BLA) play an important role in the
145 The lateral orbitofrontal cortex (lOFC) and basolateral amygdala (BLA) promote cocaine-seeking behav
147 as negatively related to both aggression and basolateral amygdala (BLA) reactivity to angry faces, wh
149 ions between the prefrontal cortex (PFC) and basolateral amygdala (BLA) regulate emotional behaviors.
153 that an increase in calcineurin (CaN) in the basolateral amygdala (BLA) supports the shift from fear
154 emporally precise optogenetic stimulation of basolateral amygdala (BLA) terminals in the central nucl
155 tified distinct neuronal circuits within the basolateral amygdala (BLA) that differentially mediate f
156 ing measuring freezing have demonstrated the basolateral amygdala (BLA) to be critical to both fear l
157 f the medial prefrontal cortex (mPFC) or the basolateral amygdala (BLA) to examine the effects of bet
158 supports unidirectional gustatory input from basolateral amygdala (BLA) to hypothalamus in sated subj
159 AMPAR)-silent excitatory synapses within the basolateral amygdala (BLA) to nucleus accumbens (NAc) pr
160 t involve activation of projections from the basolateral amygdala (BLA) to the medial prefrontal cort
163 cannabinoid 1 receptor (Cnr1) and CCK in the basolateral amygdala (BLA), a brain region critical for
164 at activation of efferent projections of the basolateral amygdala (BLA), a brain region implicated in
165 the BLA evokes norepinephrine release in the basolateral amygdala (BLA), alters BLA neuronal activity
166 fy the roles of the dorsal hippocampus (DH), basolateral amygdala (BLA), and medial prefrontal cortex
167 king the medial prefrontal cortex (PFC), the basolateral amygdala (BLA), and nucleus accumbens (NAc)
168 ginate from medial prefrontal cortex (mPFC), basolateral amygdala (BLA), and ventral subiculum of the
169 ehavior, with a focus on the NACc and on the basolateral amygdala (BLA), another important locus for
170 gonist) of pain-related hyperactivity in the basolateral amygdala (BLA), but not central amygdala (Ce
171 ommunication between the hippocampus and the basolateral amygdala (BLA), but the mechanisms of this p
173 egion of the hippocampus, dentate gyrus, and basolateral amygdala (BLA), GFP immunofluorescence coinc
174 s, including those examining the role of the basolateral amygdala (BLA), have traditionally used tech
175 with changes in the synaptic circuits of the basolateral amygdala (BLA), in which emotional learning
177 excitatory input from pyramidal cells of the basolateral amygdala (BLA), neurons that are activated b
178 ue-guided alcohol seeking is mediated by the basolateral amygdala (BLA), nucleus accumbens (NAc), and
179 omous receptor of neuregulin 1 (NRG1) in the basolateral amygdala (BLA), was expressed in GABAergic n
180 we visualized robust c-fos expression in the basolateral amygdala (BLA), which was reduced in mice pr
181 ronal activity in one key limbic region, the basolateral amygdala (BLA), whose activity fluctuates ac
198 ronic stress contributes to hyperactivity of basolateral amygdala (BLA; comprised of basal, lateral,
199 e, selectively control PCs projecting to the basolateral amygdala (BLAPC) compared to those projectin
206 gests that these changes limited only to the basolateral amygdala complex may not be sufficient to in
207 , including the lateral nucleus (LAT) of the basolateral amygdala complex, is sensitive to stress.
208 ere we tested whether neural activity in rat basolateral amygdala conforms to this pattern by recordi
209 ur results indicate that anterior insula and basolateral amygdala constitute a network part that is p
210 on how distinct interneuron subtypes in the basolateral amygdala contribute to the acquisition and e
211 ese regions, the prefrontal cortex (PFC) and basolateral amygdala, convey executive control and affec
212 ion in the nucleus accumbens and central and basolateral amygdala correlated with genotype-related di
213 NAc core DA as being important for allowing basolateral amygdala-dependent information to gain contr
215 lutamatergic signaling mechanisms within the basolateral amygdala differentially and dissociably medi
216 d abolition of the signal by inactivation of basolateral amygdala disrupted this change in orienting
217 o the nucleus accumbens core, but not in the basolateral amygdala, dorsal hippocampus, infralimbic or
218 intracellular signaling pathways within the basolateral amygdala during stress-induced reinstatement
219 insic functional connectivity, with the left basolateral amygdala emerging as the strongest predictor
220 ions of the core (but not the shell) and the basolateral amygdala enhanced context-specific locomotor
222 ctify this, we monitored, in near-real time, basolateral amygdala glutamate concentration changes dur
223 e we combined chemogenetic excitation of rat basolateral amygdala glutamatergic neurons with a variet
224 ndamide and CRF1 signaling exhibited blunted basolateral amygdala habituation, which further mediated
228 ere discussed in the context of roles of the basolateral amygdala in coding and using reward predicti
229 st this, we recorded single unit activity in basolateral amygdala in rats with 6-hydroxydopamine or s
230 ning and GluN2A-containing NMDARs within the basolateral amygdala in the memory destabilization and r
233 anyl induced similar oxygen decreases in the basolateral amygdala, indicating that brain hypoxia coul
236 tral amygdala, but MJN110 infusions into the basolateral amygdala, interfered with the naloxone-preci
238 as not examined whether neuron number in the basolateral amygdala is stable through this period.
239 nduction of beta-adrenergic signaling in the basolateral amygdala is sufficient to induce acute and s
241 Activation of noradrenergic circuitry in the basolateral amygdala is thought to have a role in stress
243 ate gyrus, we find that knockdown of PTEN in basolateral amygdala leads to a significant decrease in
244 nnection of the medial prefrontal cortex and basolateral amygdala led rats to become more impulsive b
245 D1 neuron projections implicates the medial basolateral amygdala (mBLA) as a downstream target of th
247 fect of acute stress occurred independent of basolateral amygdala neural input and was mimicked by tr
248 ircuit revealed enlarged dendritic arbors in basolateral amygdala neurons in S1, but normal infralimb
249 ed the effects of shRNA knockdown of PTEN in basolateral amygdala neurons on synaptic spine density a
250 nsic excitability and synaptic plasticity in basolateral amygdala neurons that give rise to temporall
253 re) produces a long-lasting sensitization of basolateral amygdala noradrenergic substrates [via a cor
255 In contrast, FosB remained elevated in the basolateral amygdala of mice with resolved nociception a
256 was also seen in the infralimbic cortex and basolateral amygdala of stress-susceptible male mice aft
257 sions of the NAc core, NAc medial shell, and basolateral amygdala on context-specific locomotor sensi
258 e, we examined the effects of lesions of the basolateral amygdala on the occurrence of unblocking whe
259 togenetically stimulated mPFC projections to basolateral amygdala or nucleus accumbens, two subcortic
262 statory neocortex, medial prefrontal cortex, basolateral amygdala, or the central nucleus of the amyg
263 umber of Fos-positive neurons in central and basolateral amygdala, orbitofrontal cortex, ventral and
265 s (NAc) shell from ventral subiculum (vSub), basolateral amygdala, paraventricular thalamus, and vent
267 complex, and local plasticity in excitatory basolateral amygdala principal neurons is considered to
268 nstrate that C1ql3-expressing neurons in the basolateral amygdala project to the medial prefrontal co
269 we found that local injection of NPS in the basolateral amygdala promotes anxiolysis after chronic e
273 contrast with hippocampus, cell grafts into basolateral amygdala rescued the hyperactivity deficit,
274 DA synthesis to DA neurons projecting to the basolateral amygdala restored short-term memory but not
275 and pharmacological manipulations of GC and basolateral amygdala revealed the role of top-down input
278 tent with findings that the NAc core and the basolateral amygdala share a variety of behavioral funct
279 idate, miR-144-3p, robustly expressed in the basolateral amygdala, showed specific extinction-induced
280 per side) into the CeA, but not the adjacent basolateral amygdala, significantly attenuated binge-lik
281 ences in monoamine levels were observed (the basolateral amygdala, subregions of the hippocampus and
282 tions from the midbrain dopamine system into basolateral amygdala, suggests that the PKH signal in am
283 an anxiety-related dorsal raphe nucleus (DR)-basolateral amygdala system to pharmacological challenge
284 ial isolation of female rats sensitizes a DR-basolateral amygdala system to stress-related stimuli, w
285 malian brain, such as the hippocampus or the basolateral amygdala, the clustering of the scaffolding
286 ns of a protein synthesis inhibitor into the basolateral amygdala, the subsequent fear response was a
287 nsfers conditioned information formed in the basolateral amygdala to brain structures that generate e
288 thesized that the mPFC communicates with the basolateral amygdala to disrupt learning in females afte
290 ogical investigation of projections from the basolateral amygdala to the medial prefrontal cortex and
291 absent or labile, in the projection from the basolateral amygdala to the NAc in incubation of cocaine
292 olateral prefrontal cortex, hippocampus, and basolateral amygdala) to identify imaging predictors of
293 , we report that transient inhibition of the basolateral amygdala triggered a profound increase in so
294 strocytes depressed excitatory synapses from basolateral amygdala via A1 adenosine receptor activatio
295 ectivity between the anterior insula and the basolateral amygdala was strongly related to state anxie
296 sing paired recordings obtained in the mouse basolateral amygdala, we found that AACs powerfully inhi
297 ine concentrations in the left but not right basolateral amygdala were elevated in groups presented w
299 estraint stress increased GABA efflux in the basolateral amygdala, whereas central amygdala efflux re
300 became silent with time, engram cells in the basolateral amygdala, which were necessary for fear memo
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