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1 ed the ratio of gamma2L/S transcripts in the basolateral nucleus of the amygdala.
2 transduction mechanisms, potentially in the basolateral nucleus of the amygdala.
3 , in area CA1 of the hippocampus, and in the basolateral nucleus of the amygdala.
4 elective vulnerability of neurons within the basolateral nucleus of the amygdala.
5 acid (1S,3R-ACPD) were characterized in the basolateral nucleus of the amygdala.
6 grams) was bilaterally administered into the basolateral nucleus of the amygdala and evoked clear-cut
7 %) and alpha1 transcript levels (63%) in the basolateral nucleus of the amygdala, and [(3)H]Ro 15-178
10 orted that these effects depend on an intact basolateral nucleus of the amygdala (BLA) and efferents
11 nd spine density of pyramidal neurons in the basolateral nucleus of the amygdala (BLA) and increased
15 nal-regulated kinase (ERK) activation in the basolateral nucleus of the amygdala (BLA) and was necess
16 nucleus of the solitary tract; NTS) and the basolateral nucleus of the amygdala (BLA) are active ear
18 tage-gated calcium channels (L-VGCCs) in the basolateral nucleus of the amygdala (BLA) are necessary
19 ed that bilateral excitotoxic lesions of the basolateral nucleus of the amygdala (BLA) block the enha
20 atergic inputs onto pyramidal neurons in the basolateral nucleus of the amygdala (BLA) contribute to
23 in releasing factor (CRF), injected into the basolateral nucleus of the amygdala (BLA) in male Wistar
24 y of pharmaco-behavioral data implicates the basolateral nucleus of the amygdala (BLA) in the facilit
26 ntagonist) administered bilaterally into the basolateral nucleus of the amygdala (BLA) of male Spragu
27 cally attenuating the activity of either the basolateral nucleus of the amygdala (BLA) or the ventral
28 ed whether blocking of NMDA receptors in the basolateral nucleus of the amygdala (BLA) prevents new f
29 ated administration of NPY directly into the basolateral nucleus of the amygdala (BLA) produces selec
30 tween the orbitofrontal cortex (OFC) and the basolateral nucleus of the amygdala (BLA) provide a crit
34 ynaptic connectivity of the pathway from the basolateral nucleus of the amygdala (BLA) to the medial
35 e central nucleus of the amygdala (CeA), the basolateral nucleus of the amygdala (BlA), or the bed nu
36 smission in projections from the mPFC to the basolateral nucleus of the amygdala (BLA), whereas inhib
42 planted with bilateral cannulae aimed at the basolateral nucleus of the amygdala complex and infused
45 ion of post-synaptic 5-HT1A receptors in the basolateral nucleus of the amygdala may produce anxiogen
46 ons between the orbitofrontal cortex and the basolateral nucleus of the amygdala may provide a critic
47 stereotaxically placed within the posterior basolateral nucleus of the amygdala of rats at successiv
48 ents examining the effects of lesions of the basolateral nucleus of the amygdala on the glucocorticoi
50 region of the hippocampus, as well as in the basolateral nucleus of the amygdala, than did ovariectom
52 sites in the CA1 region of the hippocampus, basolateral nucleus of the amygdala, ventral tegmental a
53 BA receptor agonist muscimol centered in the basolateral nucleus of the amygdala was given to inactiv
54 Phaseolus vulgaris leucoagglutinin into the basolateral nucleus of the amygdala), with the dendritic
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