ライフサイエンスコーパス: be abolished by

1 Transducer currents are abolished by a homozygous Tmie-null mutation, and su

2 sensitivity to ruthenium red inhibition, can be abolished by a point mutation in the putative pore do

3 Notably, the FMRP-Top3beta interaction is abolished by a disease-associated mutation of FMRP, s

4 Notably, it is abolished by a mutation in gap junctions in projectio

5 its nuclear localization, and this activity is abolished by a phosphomimetic mutation on Ser-137.

6 ve a robust hydration shell, whose stability is abolished by a single G18V mutation.

7 This augmentation was abolished by a blocker of the Na(+)-Ca(2+) exchanger

8 The MFNG effect was abolished by a DDD to DDA mutation that inactivates

9 The effect of CCh on VFT was abolished by a muscarinic (atropine, 0.1 mumol l(-1)

10 n embryonic kidney 293 (HEK293) cells, which was abolished by a point mutation at the C terminal of A

11 induced increase in GLT-1 protein expression was abolished by a protein kinase A inhibitor and an NF-

12 excess of purified trehalose dimycolate and was abolished by a S124A mutation affecting the catalyti

13 the antiviral activity of both ALA1 and ALA2 was abolished by a single amino acid substitution known

14 ccus furiosus (PfFtn) in which self-assembly was abolished by a single amino acid substitution.

15 responses were purely glutamatergic, as they were abolished by a combination of AMPA and NMDA glutama

16 The effects of increased plasma prolactin were abolished by a genetic absence of eNOS.

17 Both of these activities were abolished by a S513A mutation.

18 rostatic epithelial proliferation, which can be abolished by ablating IL-1 signaling or depleting its

19 odeling and heart failure, and these effects were abolished by ablation of tumor necrosis factor rece

20 ed near the active zone and this segregation is abolished by actin stabilization, slowing the rate of

21 t manner and that the effects of cyclopamine were abolished by adding SHH protein.

22 of the FTO obesity locus with leptin levels is abolished by adjustment for BMI, associations of the

23 antly, tumor promotion by p53-deficient MSCs was abolished by administration of S-methylisothiourea,

24 ated level of serum creatinine in CNI groups was abolished by Aliskiren.

25 H-induced improvement in sustained attention was abolished by alpha(1)-receptor blockade.

26 All effects of ketamine were abolished by AMPA receptor antagonists and mimicked

27 oagulation in nude mice, and this activation was abolished by an anti-human TF Ab.

28 MMP8 knockdown, and this compensatory effect was abolished by an antibody against soluble E-cadherin.

29 which induced ER stress in JEG-3 cells that was abolished by an ET-1-neutralizing antibody.

30 hrough ERK1/2 and PKA pathway; these effects were abolished by an alpha2A-adrenoreceptor antagonist.

31 responses scaled with stimulus intensity and were abolished by anesthesia.

32 This potentiation was abolished by antagonism of purinergic receptors (sur

33 This effect was abolished by anti-VEGF-A antibodies.

34 Epileptic impairments were abolished by antibodies to IL-6 or IL-1beta.

35 depends on nitric oxide (NO) as the response is abolished by application of either N(G)-nitro-l-argin

36 ype seedlings, but the greater PA inhibition was abolished by application of zinc to the seedling.

37 is effect decays progressively over time and is abolished by approximately 600 ms.

38 n of vascular ring from mouse thoracic aorta was abolished by ARB or genetic deletion of AT1.

39 This production was abolished by AT-56, a specific competitive L-PGDS in

40 ISO+I(TO) decrease-induced CADs were abolished by beta(1)-antagonism with atenolol at th

41 nicotine-induced increase in EPSC frequency was abolished by blockade of alpha4beta2 nAChRs, voltage

42 potential firing in HA cells, an effect that was abolished by blockade of AMPA receptors.

43 re produces locomotor sensitization that can be abolished by blocking calcium (Ca(2+))-permeable alph

44 These promoting effects of Klotho can be abolished by blocking integrin beta1.

45 ted potassium channels with iberiotoxin, and is abolished by blocking small conductance calcium-activ

46 This gain was abolished by blocking Gln synthetase, an enzyme that

47 This was abolished by blocking mEPCR and was absent in ex viv

48 These changes were abolished by blocking beta1-integrins and mimicked

49 uptake and foam cell formation, all of which were abolished by blocking SRA.

50 This effect was abolished by both a point mutation that inhibits cla

51 Importantly, this bias was abolished by both electrolytic lesions of the LHb an

52 These beneficial effects were abolished by C-16, an ACE2 inhibitor.

53 These EADs were abolished by caffeine and tetrodotoxin (but not ran

54 and anti-alpha-Gal antibody to both proteins was abolished by carbohydrate oxidation.

55 mouse partially ligated left carotid artery were abolished by caspase-1 inhibition, local delivery o

56 This rolling was abolished by CD29 blockade on hMSCs and vascular cel

57 g that hippocampal learning-related activity is abolished by cerebellar inactivation.

58 Learning was abolished by cerebellar ablation.

59 sed spike activity during the trace interval were abolished by cerebellar inactivation, persistent re

60 The activation of calcineurin by TLCS was abolished by chelating intracellular Ca(2+).

61 on occurred with sub-second lifetimes, which was abolished by cholesterol.

62 aine seeking in GTs than STs and this effect was abolished by cholinergic losses despite the fact tha

63 /6 mice induced slowly developing HTN, which was abolished by co-infusion of the ER stress inhibitor

64 ped astrocytes, the GABA transporter current was abolished by combined application of the selective G

65 Binding was abolished by competition with free ligands, demonstr

66 strictly C3-dependent manner, as the binding was abolished by compstatin.

67 nduced neurogenesis and behavioral responses are abolished by constitutive knockout of SMARCA3.

68 This protection was abolished by cotreatment with the SERCA inhibitor cy

69 The interaction can be abolished by cTnI phosphorylation at Ser22 and Ser23,

70 early checkpoint in ILC2 development, which was abolished by deficiency in the zinc-finger protein B

71 Furthermore, this correlation is abolished by delaying spindle organization or prevent

72 ause both RNA binding and enzymatic activity are abolished by deletion of the SAS-specific C-terminal

73 HR is abolished by deletion of RecA and reduced in the abse

74 Centromeric localization of KNL2 is abolished by deletion of the CENPC-k motif and by mut

75 ocytes, required chemokine receptor CCR2 and was abolished by deletion of IL-1beta in CCR2(+) blood m

76 Thus, cocaine-associated autophagy is abolished by depleting GAPDH via shRNA; by the drug C

77 Migration of CD4(+) T cells toward dLN cells was abolished by depleting the CD11b(+) cells and was al

78 reases in secreted and intracellular Abeta40 were abolished by depletion of presenilin 2 (PSEN2), rec

79 orepressor activity of Tip60 at the ERE site is abolished by diarylpropionitrile, genistein, equol, a

80 tion by 10-fold (P<0.0001); this natriuresis was abolished by direct renal interstitial infusion of s

81 ons are enhanced when host circadian rhythms are abolished by disrupting the key clock gene transcrip

82 essed psychomotor response to cocaine, which was abolished by disruption of the DC pathway, but not t

83 tabilization of inner-membrane-protein mRNAs is abolished by dissociating the RNA degradosome from th

84 ne phosphatase expression, and activity that was abolished by DKK-1, a blocker of the Wnt/beta-cateni

85 d receptor heterodimerization, both of which are abolished by EGFR inhibitors.

86 It was abolished by EGTA, which preferentially blocked endo

87 cumulation induced by mechanical stimulation are abolished by either pharmacologic inhibition of ERKs

88 These effects were abolished by either beta-adrenoreceptor blockade wi

89 This antiproliferative effect was abolished by elevating [Ca(2+)]i.

90 stemic circulating IL-13 and TGF-beta1; this was abolished by endothelial-specific deletion of IL33 b

91 Dimerization could be abolished by engineering glycosylation sites into the

92 These effects are abolished by enhancing mitochondrial Ca(2+) with acu

93 ly, these protective effects produced by XMJ were abolished by eNOS inhibitor L-NAME or specific eNOS

94 D-Ile effects on synaptic plasticity are abolished by enzymatically depleting D-serine or by

95 he regenerative effects of nitrate treatment were abolished by eradication of the nitrate-reducing ba

96 re unaffected by acyclovir pre-treatment but were abolished by exposure to Tetrodotoxin (TTX) which b

97 oliferation between NG2KO and control islets was abolished by ganglionic blockade or by weaning the m

98 These effects were abolished by gefitinib, an EGFR tyrosine kinase inh

99 These effects are abolished by genetic deletion of GPR55 or by the GPR

100 hanced acid-induced muscle pain in mice that was abolished by genetic inactivation of ASIC3.

101 ity of neurons, and the remaining inhibition was abolished by group II/III metabotropic glutamate rec

102 This EC-derived protection was abolished by HG.

103 d cell survival or graft functional recovery was abolished by HIF-1alpha siRNA.

104 iate evoked EPSCs, and its inhibitory effect was abolished by himbacine.

105 oxygen-glucose deprivation, and this effect was abolished by histamine receptor type I antagonist.

106 agellin receptor FLS2, whose gene expression was abolished by HopQ1 in a promoter-dependent manner.

107 n human proximal tubular (HK-2) cells, which was abolished by HSP-70 or HO-1 siRNA.

108 (P-bodies); the translocation of stim1 mRNA was abolished by HuR overexpression.

109 by JNJ 16259685, and neuroprotection by DHPG was abolished by ICI 182,780.

110 PIC-induced behavioral deficits were abolished by IL-6 antibodies and were mimicked by r

111 The conditioning effect of dPAG, however, was abolished by inactivation of the BLA.

112 The ATP effect was abolished by inhibiting DGK and in the rdgA mutant,

113 Cleavage of the beta1 integrin can be abolished by inhibition of MMP-2 activity; it can be

114 Importantly, this effect of XPLN depletion is abolished by inhibition of Akt or mTOR kinase activit

115 fect of TOR inactivation on BZR1 degradation is abolished by inhibition of autophagy and by the bzr1-

116 to mimic GTPase-induced structural changes, is abolished by inhibition of S223 phosphorylation.

117 potentiating effect of PNU-282987 on eEPSCs was abolished by inhibition of protein kinase A (PKA).

118 by aberrant Ca(2+) releases and EADs, which were abolished by inhibition of Ca(2+)/calmodulin-depend

119 o insulin in HFD male rats and these actions were abolished by inhibition of either AMPK or nitric ox

120 iR-126 level and SMC turnover; these effects were abolished by inhibition of endothelial miR-126 and

121 All of these changes were abolished by inhibition of mitochondrial processing

122 platelet activation and Syk phosphorylation were abolished by inhibition of PI3K, which demonstrates

123 The HBV-promoting effect was abolished by inhibitors of phosphatidylinositol 3-ki

124 ediated by glutamate and not 5-HT because it was abolished by ionotropic glutamate receptor antagonis

125 tic muscle and spinal cord, an activity that is abolished by IS-associated SNPs.

126 TSH effects on MTCO1, TFAM, and porin could be abolished by K1-70, a TSH-receptor antagonist, sugges

127 manner, because its junctional localization is abolished by knock-out of PLEKHA7, and is rescued by

128 lial cells on the promotion of wound healing was abolished by knockdown of CTCF with CTCF-specific sh

129 g events in human vascular endothelial cells were abolished by knockdown of AT1 and inhibited by AT1

130 ted in the absence of extracellular Ca(2+) , was abolished by LaCl3 and was attenuated approximately

131 dance behaviors in a runway operant paradigm were abolished by lesions of LHb efferents, lesions of t

132 timulation, and Delay cell persistent firing was abolished by local NR2B NMDAR blockade or by systemi

133 ong-term recognition memory, and this effect was abolished by loss of the photopigment melanopsin.

134 blocker isradipine; however, depolarization was abolished by lowering extracellular Na(+).

135 hosphorylation and intercellular gaps, which were abolished by lysophosphatidic acid receptor 1/2 inh

136 ed visceral hypersensitivity, an effect that is abolished by microglial depletion.

137 erived EVs could trigger EC migration, which was abolished by miR-150 knockdown in SMCs, suggesting m

138 This activity is abolished by mismatches in the upstream DNA and is in

139 The IL-15-mediated growth advantage is abolished by mitogen-activated protein kinase kinase/

140 Responses to nerve stimulation were abolished by MRS-2500 and not observed in muscles w

141 liferation in HepG2 cells; and these effects were abolished by mTORC1 inhibition, demonstrating an es

142 subtilis cells and that this activity could be abolished by multiple structural alterations.

143 ogenous peptide substrate, and this activity is abolished by mutagenesis of lysine 723 in the catalyt

144 n the NH(2)-lobe of subunit troponin C (TnC) was abolished by mutagenesis, and effects on cardiac tro

145 mediate inactivated state, but these effects were abolished by mutating either of these sites to non-

146 athicity within the TMDs as the interactions were abolished by mutating residues on the polar faces t

147 sults in autonomous kinase activation, which is abolished by mutation of the Cys-290 site.

148 s not required, but phosphorylation in trans is abolished by mutation of the transmembrane domain.

149 hibited partial apical-lateral polarity that was abolished by mutation of its ankyrin-binding site bu

150 insensitive to TRPV4 channel modulation and was abolished by Na(+) /Ca(2+) exchanger blockade.

151 This effect is abolished by naris closure.

152 This redox response was abolished by NCLX overexpression and restored by the

153 d vessel formation in matrigel plugs in mice was abolished by Ned-19 and, most notably, failed to occ

154 PTH and FGF23-sensitive phosphate transport were abolished by NHERF1 shRNA knockdown.

155 Constitutive normoxic NO formation was abolished by NOS inhibition and intracellular NO sca

156 ing of Bim to Bcl-2 and Bcl-xL, effects that were abolished by obatoclax coadministration.

157 Dentate hyperactivity was abolished by optogenetic stimulation of cholinergic

158 This delay was abolished by ouabain or zero K(+) saline, which elim

159 Ubiquitination of DNMT1K142me1 was abolished by overexpression of PHF20L1, suggesting t

160 so depends on the concentration of ssRNA and is abolished by overstretching of the dsDNA.

161 ting action potentials in LHb neurons, which are abolished by p11 knockdown.

162 dily triggered [Ca(2+)]i oscillations, which were abolished by P2 receptor antagonists.

163 ity of pro-inflammatory cytokines/chemokines are abolished by PARP1 ablation or inhibition, or blocke

164 salt appetite mediated by mineralocorticoids is abolished by PBN lesions.

165 sults in oncogenic toxicity; this effect can be abolished by pharmacological inhibition of Mek to res

166 These effects were abolished by pharmacological or molecular inhibitio

167 P2Y6-activated responses were abolished by phospholipase C (PLC) and inositol tri

168 analogous effects of FXYD1 on K0.5Na, which are abolished by phosphomimetic mutants and also by incr

169 while these inhibitory effects of sildenafil were abolished by PKG inhibitor Rp-8Br-cGMPs.

170 in wild-type and P2Y(12) deficient platelets was abolished by PP2, and AYPGKF-mediated P-selectin exp

171 e, the effect of adenosine on cell migration was abolished by pre-exposure to TRAM-34.

172 by IBH1, but the inhibitory effects of IBH1 were abolished by PRE1.

173 in ATP concentration in culture medium that was abolished by preincubation with TRP channel antagoni

174 strongly reduced when speech intelligibility is abolished by presenting speech/noise stimuli in rever

175 dependent on GPR55 activation, because they are abolished by pretreatment with ML-193 [N-(4-(N-(3,4-

176 he enhancement of both responses by cysteine was abolished by pretreatment with 1 mm PAG.

177 bition of TNF-alpha secretion by macrophages was abolished by pretreatment with 1-methyl-D-tryptophan

178 al neurons of nucleus ambiguus; the response was abolished by pretreatment with the GPER antagonist G

179 of phospho-Stat3 to the Bv8 promoter, which was abolished by pretreatment with the Stat3 inhibitor W

180 duced pressor and sympathoexcitatory effects were abolished by pretreatment of the RVLM with the vaso

181 increase in ventilation and CO2 sensitivity is abolished by preventing adrenaline release or blockin

182 cell surface synaptic GABA(A) receptors and is abolished by preventing CaMKII phosphorylation of GAB

183 Unexpectedly, sequestration is abolished by preventing substrate ubiquitylation.

184 nding of the antibody to all three receptors was abolished by prior incubation of the receptors with

185 ed to sympathoexcitation since these effects were abolished by propranolol.

186 ion, and morphological changes, all of which are abolished by R458A mutation.

187 and the induction of ventricular tachycardia was abolished by RAGE silencing.

188 ceptor ligands at the glucagon receptor that is abolished by RAMP2 interaction.

189 s evoked by laryngeal citric acid challenges were abolished by recurrent laryngeal nerve (RLN) transe

190 tihyperalgesic effects of sigma-1 antagonism were abolished by reducing the neutrophil load with in v

191 Production of Nrf1gamma is abolished by removal of SDS1 or PEST2 degrons, wherea

192 asurements, we show that conductance by Bot1 is abolished by removal of the Na(+) ion binding site.

193 rosis predominantly in stellate cells, which was abolished by removal of extracellular Ca(2+) and sig

194 sodium excretion and renal interstitial cGMP were abolished by renal interstitial nitric oxide syntha

195 sion of pohr driven GFP in this strain could be abolished by replacing the intact ohrR gene with a mu

196 ts show escalation of ethanol seeking, which was abolished by restoring mGluR(2) expression in the in

197 ostate cancer cells in which PTEN expression was abolished by RNA interference-mediated attenuation.

198 -8220, PP2, dimethyl-BAPTA, or LY294002, but were abolished by Ro-31-8220 plus PP2, dimethyl-BAPTA, o

199 ited biases toward food-associated cues that were abolished by satiety.

200 f the 5-HT1A receptor antagonist WAY-100635, was abolished by SB-269970 (5-HT7 receptor antagonist),

201 e chemoattractive activity of Nur77(-/-) BMM is abolished by SDF-1alpha inhibiting antibodies.

202 criminating LSD from saline, and this effect is abolished by selective blockade of the 5-HT(2A) recep

203 dopamine D3 receptors (D3R), as its effects were abolished by selective D3R antagonists and absent i

204 lencing induced greater ADSC survival, which was abolished by short hairpin RNA against hypoxia-induc

205 d ventilated rats, moderate AIH-induced pLTF was abolished by siBDNF and UO126, but not siTrkB or PI8

206 Hypoxia-induced DCK expression was abolished by silencing hypoxia-inducible factor 1alp

207 obustly than humid heat, and this modulation was abolished by silencing of dry-air receptors.

208 akefulness caused by cholinergic stimulation was abolished by simultaneous reverse microdialysis of c

209 The inhibitory effects of IL-28B could be abolished by siRNA or antagonistic peptides against t

210 hosphorylation and XBP1 mRNA splicing, which was abolished by siRNA-mediated knockdown of kinase inse

211 f HIF-1alpha increased Angptl4 levels, which was abolished by siRNA.

212 teraction with human SLX4 (huSLX4) and could be abolished by small interfering RNA (siRNA) depletion

213 collagen I led to TGF-beta expression, which was abolished by small interfering RNA (siRNA) Hic-5 kno

214 ha-mediated suppression of virus replication was abolished by small interfering RNA (siRNA) knockdown

215 y effects of Ang II on STAT3 phosphorylation were abolished by small-interfering RNA knockdown of TRP

216 ith increased resistance to apoptosis, which is abolished by Smurf2 silencing.

217 ostate cancer cell invasiveness, which could be abolished by SNAI2 silencing.

218 This endoglin-dependent adhesion was abolished by soluble endoglin, RGD peptides, the ant

219 These effects were abolished by specific inhibitors of both pathways,

220 vasion up to fivefold, whereas cell mobility was abolished by STAT3-DN (>85%).

221 nstressed cells, but SET-mediated repression is abolished by stress-induced acetylation of p53 CTD.

222 This conformational activation is abolished by substituting Glu295 with lysine, thereby

223 This regulatory effect was abolished by targeted disruption of the interactions

224 The beneficial effects were abolished by TBCA while pretreatment with apocynin

225 ed both behavior and learning, learning that was abolished by temporally specific inhibition of orbit

226 exclusively in female mice, and this effect was abolished by testosterone administration.

227 ells fire spontaneous action potentials that are abolished by tetrodotoxin, and all display spontaneo

228 cts of ghrelin on AMPK in peripheral tissues are abolished by the lack of functional CB1 receptor via

229 -apoptosis induced by mechanical stimulation are abolished by the Wnt antagonist Dkk1 and the beta-ca

230 The toxicity of RelE2sca could be abolished by the co-expression of its cognate RelB2sc

231 y effects of JAM-A-deficient platelets could be abolished by the inhibition of alphaIIbbeta3 signalin

232 n over production of type I IFN, which could be abolished by the interruption of RIG-I.

233 Colocalization of PDI and beta-actin could be abolished by the membrane-permeable sulfhydryl blocke

234 osynthetic gene expression in a pathway that is abolished by the herbicide norflurazon.

235 s the induction of LTP in DMS neurons, which is abolished by the inhibition of NR2B-NMDARs.

236 ystem, and that the ability to cause disease is abolished by the natural absence or experimental dele

237 aling pathways as the high-voltage component is abolished by the phosphoinositide 3-kinase blocker wo

238 Ankyrin-G interaction with E-cadherin is abolished by the same mutations resulting in increase

239 xpression in adjacent cells, and this effect is abolished by the T164A mutation.

240 eotide-binding domain 1 (NBD1) pore loop and is abolished by the Y662A mutation that disrupts substra

241 AcnB-pgdA transcript binding was abolished by the addition of iron.

242 uction by PAMPs in primary human hepatocytes was abolished by the addition of neutralizing IL-6 antib

243 This residual was abolished by the addition of quinine.

244 rnal QX-222 created by mutations of Ile-1575 was abolished by the additional mutation K1237E, support

245 NO formation was abolished by the C273A mutation in mARC-1.

246 m-induced deposition of properdin by zymosan was abolished by the C3-inhibiting peptide compstatin.

247 This specific modulation of coupled RyRs was abolished by the Ca(2+)/calmodulin-dependent kinase

248 This was abolished by the Ca(2+)/calmodulin-dependent protein

249 omycin, in control and FHL2 knockdown cells, was abolished by the calcineurin inhibitor cyclosporine,

250 The PA and GAPC binding was abolished by the cation zinc whereas oxidation of GA

251 ATP release was abolished by the channel inhibitor carbenoxolone and

252 mpairment to recognize the familiar juvenile was abolished by the coinfusion of agonist plus antagoni

253 e maternal defense test, maternal aggression was abolished by the CRF-R2, but not the CRF-R1, agonist

254 Furthermore recombinant GST-theta activity was abolished by the denaturants triton X-100, Gua-HCl,

255 solic NADH, and NADH-induced INCX inhibition was abolished by the H2O2 scavenger catalase.

256 nhanced glycerol release, a dual effect that was abolished by the inhibition of cAMP-dependent protei

257 terial EC differentiation, and this increase was abolished by the inhibition of ROS generation or by

258 the effect of TX on GLT-1 promoter activity was abolished by the inhibition of the NF-kappaB pathway

259 n of NAD(+) significantly increased JO2, and was abolished by the inhibitor of mitochondrial pyruvate

260 EPO-induced pMF was abolished by the MEK/ERK inhibitor U0126 [1,4-diamin

261 ive against amyloid toxicity, and its action was abolished by the mGlu1 receptor antagonist, (3,4-dih

262 Prdx6 for aiPLA2 activity, and this activity was abolished by the mutation of serine 32 to alanine (S

263 bitor trichostatin A, an effect that in turn was abolished by the NF-kappaB inhibitor N(4)-[2-(4-phen

264 preconditioning; this cytoprotective effect was abolished by the NO scavenger.

265 on in the presence of fibronectin aggregates was abolished by the PKA inhibitor H89, whereas the effe

266 Galactolipase activity was abolished by the PNLIP beta5-loop and decreased by t

267 at Thr(345)-Leu(346) and Asn(347)-Leu(348), was abolished by the presence of Asn(347) glycosylation

268 The inhibition of the infectivity was abolished by the presence of mannose.

269 fect of high glucose on Wnt signaling, which was abolished by the SOD1 mimetic, tempol.

270 a rapid and sustained influx of Ca(2+) that was abolished by the TRPV channel inhibitor, ruthenium r

271 The effects of CIH were abolished by the antibody.

272 not depend on the loss of noradrenaline but were abolished by the antiparkinsonian drug, L-DOPA, or

273 f 32 kDa), most of the behavioral phenotypes were abolished by the D1R antagonist, SCH23390, and the

274 perties and decussating enamel prisms, which were abolished by the loss of MMP20 in the M180Tg/DKO mi

275 the mineralocorticoid receptor (MR) as they were abolished by the MR antagonist, spironolactone, and

276 d AF susceptibility in miR-106b-25(-/-) mice were abolished by the RyR2 blocker K201.

277 , typical of P2X1 homomeric receptors, which were abolished by the selective P2X1 antagonist NF449 (1

278 lammatory effects of water-induced hemolysis were abolished by the simultaneous administration of DEA

279 Responses to GSK1016790A were abolished by the TRPV4 antagonist HC067047 and were

280 Both effects were abolished by the Y1R antagonist, PD160170.

281 ivation, in HC11 cells, whereas such effects were abolished by TNFalpha neutralizing antibody treatme

282 1-EBIO enhanced whole cell currents which were abolished by TRAM-34 and reduced by apamin indicati

283 Minocycline-induced neuroprotection was abolished by transfecting the NSCs with Nrf2-small i

284 Remarkably, these deficits were abolished by transient, post hoc interference with

285 This effect was abolished by treating the mice with U0126, a specifi

286 The inhibitory actions of ghrelin were abolished by treating the vagal ganglia neurons wit

287 Glucocorticoid Klf13 gene expression was abolished by treatment with a GR antagonist (RU486)

288 ermore, the protective effects of isoflurane were abolished by treatment with a TGF-beta1 neutralizin

289 These effects were abolished by treatment with two specific inhibitors

290 Hyperosmotic responses were abolished by TRPM8 antagonists, and were absent in

291 hus, we hypothesize that NSs function(s) can be abolished by truncation of specific domains, and co-e

292 Inhibition was abolished by truncation of the phosphoprotein-bindin

293 demonstrated by proximity ligation assay and was abolished by U0126, a MAP kinase inhibitor.

294 44/42 MAPK and increased MAPK activity which was abolished by UO126.

295 These effects were abolished by vagotomy.

296 Activity is abolished by vanadate (10 mM), but is less sensitive

297 The NNNP NMT was abolished by VIP6-28 (30 mum), absent in the VIP(-/-

298 These paracrine effects were abolished by VPC23019.

299 roughout the axon, entered mitochondria, and was abolished by WldS expression.

300 Furthermore, increased cancellous bone is abolished by Wnt inhibition but further increased by