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1 expression induced RPE cell apoptosis, which was abrogated by 100 nm docosahexaenoic acid, 10 ng/ml p
2                        This repopulation can be abrogated by a PGE2-neutralizing antibody and celecox
3            The autophagy response to ammonia is abrogated by a cell-permeable form of pyruvate result
4 ic function for a wild-type polyQ tract that is abrogated by a competing longer polyQ mutation in a d
5        Interestingly, SMN-Gemin2 interaction is abrogated by a spinal muscular atrophy (SMA)-causing
6                                 This binding is abrogated by a Tyr(253) --> Ala substitution.
7 naling was found when GLP-1R internalization was abrogated by a dominant-negative version of dynamin
8                                  This effect was abrogated by a GABAA-R antagonist.
9              Chaperone-mediated dissociation was abrogated by a non-hydrolysable ATP analogue, indica
10 st differentiation in vitro, and this effect was abrogated by a physiological BMP-2 inhibitor, noggin
11                            This augmentation was abrogated by a PI3K inhibitor.
12 NO synthesis in a dose-dependent manner that was abrogated by a specific miR-939 inhibitor.
13                                      Effects were abrogated by a vascular endothelial growth factor (
14 m binding to Bcl-2/Bcl-xL; the latter effect was abrogated by ABT-737.
15 masome assembly and activation, effects that were abrogated by acetylation mimic-modified SOD2.
16 ydroethidium fluorescence, an elevation that was abrogated by administration of nitro-l-arginine meth
17                                This increase was abrogated by administration of the NAD(P)H oxidase i
18  activation, as CNS IL-1beta-induced atrophy is abrogated by adrenalectomy.
19 and interleukin 6 responses to infection and was abrogated by alveolar macrophage depletion, using cl
20                                These effects were abrogated by AMPK silencing.
21                                These effects are abrogated by an intra-arterial infusion of corticost
22 ion of human platelets in vitro, which could be abrogated by an antipodoplanin antibody.
23 Cs/ALDH1(-)CD44(-)-HNC and this effect could be abrogated by an IL-6-neutralizing antibody.
24 and activity in HLA-DR/TLR-transfected cells is abrogated by an anti-HLA-DR Ab.
25                         PMA-induced shedding was abrogated by an ADAM (A disintegrin and metalloprote
26    Analogue potentiation of the GABA current was abrogated by an alpha(1) subunit Q241L mutation, ind
27 the immune activation of cardiac fibroblasts was abrogated by an IL-1R antagonist and an IL-1alpha-bl
28 d expression of M2 genes by TonEBP knockdown was abrogated by antagonism of IL-10 by either neutraliz
29 as associated with ICOS/ICOSL engagement and was abrogated by antagonist anti-ICOS and anti-ICOSL ant
30 s, and that virus infectivity in HT-29 cells is abrogated by anti-A-type antibodies as well as signif
31 eptide concentration-dependent manner, which is abrogated by anti-defensin alpha-1 IgG.
32 ration in vitro toward tumor extracts, which was abrogated by anti-CCL2 neutralizing Abs.
33 KO mice and that increased IL-17A expression was abrogated by anti-IL-10 Ab treatment.
34 mmatory cytokine production in a manner that was abrogated by anti-PD-L1 blocking antibody.
35         Furthermore, activation of NF-kappaB was abrogated by anti-TLR2, anti-EBV-encoded dUTPase blo
36                     The induction of M cells was abrogated by anti-TNF-alpha blockade, suggesting tha
37                              These responses were abrogated by anti-CD4 antibodies and mutagenesis of
38 eased AHR and higher leukotriene levels that were abrogated by anti-PlGF Ab or the 5-lipoxygenase inh
39 ) had the same effect, and stimulation could be abrogated by antibodies neutralizing alpha interferon
40 late-phase decrease in body temperature that was abrogated by antibody-mediated depletion of neutroph
41  a chemoprotective tumor cell niche that can be abrogated by Axl-targeting approaches.
42 ere similarly induced by LMP-400 and CPT and were abrogated by AZD7762.
43 d activation of the neutrophil NADPH oxidase was abrogated by bacterium-derived proteolysis of galect
44 genic capacity of Th17 cells to transfer EAU was abrogated by BET inhibitors in an adoptive transfer
45 n but not G protein-dependent signaling, and are abrogated by beta-arrestin-2 small interfering RNA.
46 une cytokine elicitation by all Ad serotypes was abrogated by blockade of endosomal acidification, ca
47 wling behavior in CD14(dim)CD16(+) monocytes was abrogated by blockade of ICAM1, VCAM1, or CX3CL1, in
48                                  This effect was abrogated by blockade of TIGIT's complementary costi
49  stress-induced worsening of retinal NV that were abrogated by blockade of VCAM-1 included increases
50   A component of FIV that is Kir independent is abrogated by blocking Ca(2+) -sensitive K(+) channels
51                                     Adhesion is abrogated by blocking with anti alpha(v)beta(5) and a
52 onstrated increased adhesion to suPAR, which was abrogated by blocking of low-density lipoprotein rel
53                      This latter interaction was abrogated by blocking protein kinase C activity, res
54 ngiogenic response in endothelial cells that was abrogated by blocking VEGF, whereas peripheral blood
55          The antileukemic activity of CB-839 was abrogated by both the expression of a hyperactive GA
56                  These profibrotic responses were abrogated by both genetic and pharmacological disru
57 that MtRS also induces p53 expression, which was abrogated by Ca(2+) chelators and short hairpin RNA-
58 on 2 hr after the intake of CsA, which might be abrogated by calcium channel blockers.
59 roliferation caused by blue/green could also be abrogated by capsazepine, and by the antioxidant, N-a
60  (5uM) stimulated proliferation, which could be abrogated by capsazepine.
61                         This enhanced uptake was abrogated by carbogen breathing, indicating that in
62   This chemoreflex anti-inflammatory network was abrogated by carotid chemoreceptor denervation and b
63  prevented by stop codons or activity of D10 was abrogated by catalytic site mutations, without other
64 h this epitope suppressed ongoing EAE, which was abrogated by CD8(+) T cell depletion.
65 tional proinflammatory functions in NTN that were abrogated by cell-specific deletion of RORgammat.
66 nhibit translation, and that this activation is abrogated by certain modifications present in cellula
67 man villous trophoblasts, and this induction was abrogated by CH223191, an aryl hydrocarbon receptor
68 gy-initiation proteins Atg3 and Atg5, and it is abrogated by chelating cytoplasmic calcium or inhibit
69                  IL-34 binding to U251 cells was abrogated by chondroitinase ABC treatment, and CS co
70 se loss triggered by the LRRTM/NL deficiency was abrogated by chronic blockade of synaptic activity a
71 ibitory effects of PRH on cell proliferation are abrogated by CK2 and that CK2 inhibits the binding o
72 mulation, whereas proliferation of non-Tregs is abrogated by CN inhibitors.
73 ese detrimental effects of diabetes mellitus were abrogated by co-administration of SEP and L-Cit.
74 lasma glucagon concentrations; these effects were abrogated by coinfusion of corticosterone.
75 ce to endocrine therapy in breast cancer may be abrogated by combination therapies targeting both ER
76 naling and stroma-mediated resistance, which is abrogated by combination therapy with gamma secretase
77 y responses against pathogenic organisms can be abrogated by commensals; however, the molecular mecha
78 idney allograft rejection phenotype that can be abrogated by complement inhibition.
79 ocytes caused increased levels of HGF, which were abrogated by concomitant knockdown of Smad3 and Sma
80 sis-specific manner in pulmonary lesions and are abrogated by concurrent re-expression of integrin-al
81 te growth promotion and myelin disinhibition were abrogated by CRMP2 inhibition and largely mimicked
82 rophic-like disease in STIM1 transgenic mice was abrogated by crossing in a transgene expressing a do
83 ulation caused by these hypertensive stimuli were abrogated by CTLA4-Ig.
84 w effects were CXCR4-dependent, because they were abrogated by CXCR4 inhibition.
85                             This interaction is abrogated by cyclin A-CDK2-mediated phosphorylation o
86                                These effects are abrogated by DDX58 knockdown.
87              In addition, maturation of OCLs was abrogated by defective cell fusion of pre-OCLs deple
88 tion of E-Cadherin and upregulation of Snail were abrogated by deguelin treatment.
89                PMA-induced shedding of Tim-3 was abrogated by deletion of amino acids Glu(181)-Asp(19
90 ibitor aphidicolin, and active demethylation was abrogated by deletion of Tet3 in both pronuclei.
91 he later phase (and development of fibrosis) was abrogated by deletion of TNFR1.
92 ggesting a role in tumor growth; this effect was abrogated by deletion of two hydrophilic domains.
93 -type S. aureus compared with a lukED mutant was abrogated by depleting neutrophils.
94 as confined to the splenic marginal zone and was abrogated by depletion of CD169(+) cells.
95                           This amplification was abrogated by depletion of donor CD4 T cells.
96  responsive, whereas their enduring activity is abrogated by different microenvironmental immunosuppr
97 imicrobial peptide human beta-defensin 2 and is abrogated by digestion of milk HA with a specific hya
98              Suppression of CGG RNA toxicity was abrogated by disease-associated mutations in TDP-43.
99 n be opposed by HDAC1, but not by HDAC3, and is abrogated by disrupting HIF1alpha-p300 interaction.
100 ER stress observed in hLrp1(-/-) hepatocytes were abrogated by E64D treatment, whereas inhibiting ER
101 -inducible factor-1alpha (HIF-1alpha), which was abrogated by early growth response-1 (EGR-1) small i
102  confers resistance to bile salts, which can be abrogated by efflux pump inhibitors.
103 smin induces neuronal death, and this effect is abrogated by either neuroserpin or the NMDA receptor
104 ctivated T cells (NFAT) nuclear accumulation was abrogated by either antioxidant supplementation or C
105                             These activities were abrogated by either knocking down PIG3 or mutating
106 f NMC cells induced by knockdown of BRD4-NUT is abrogated by enforced expression of MYC.
107                 This constitutive activation was abrogated by ex vivo treatment with the janus kinase
108 e neurotoxicities, and this protection could be abrogated by exogenous tPA.
109         Vgamma9Vdelta2 T-cell activation can be abrogated by exposing susceptible cells (tumor and my
110                       The actions of miR-10b were abrogated by expressing a modified TIP30 cDNA resis
111  This effect of Asef2 on spines and synapses is abrogated by expression of GEF activity-deficient Ase
112 er was activated by E2F1, and the activation was abrogated by expression of SHP and its co-repressor
113                               This shielding was abrogated by fragment N2.
114 wal defect in transplanted recipients, which was abrogated by genetic ablation or pharmacologic inhib
115             Intracellular antibody signaling was abrogated by genetic deletion of TRIM21 and was rest
116                         All of these effects are abrogated by GPR120 knockdown.
117 ression of microglial BV-2 activation by ASI was abrogated by GR inhibitor, RU486 or GR siRNA.
118 n rats subjected to coronary ligation, which was abrogated by GR-1.
119 (PAF) causes wheal and flare responses which are abrogated by H1-antihistamines giving rise to the hy
120 mino acids 22-27 of H3, and this interaction is abrogated by H3K27 modification.
121 ol induced S-phase accumulation; this effect was abrogated by HDACI coadministration, which suggests
122 eta mRNA upregulation in chicken macrophages was abrogated by heat inactivation or pre-exposure of th
123 nd AZD-2281 both in vitro and in vivo, which was abrogated by HER2 reduction.
124 d insulin sensitivity in obese M4K4 iKO mice was abrogated by high exogenous insulin over the course
125 iated pro-apoptotic pathway, and this effect is abrogated by histidine-rich glycoprotein (HRGP).
126  HeH and recombinant P. falciparum H3 (PfH3) was abrogated by histone-specific antibodies.
127 nto giant unilamellar vesicles, effects that are abrogated by hydrophobic mutations.
128 gressive 4T1 breast cancer cells in mice can be abrogated by inactivation of tumor-evoked regulatory
129 restricts virus replication, and infectivity is abrogated by inactivation (e.g., irradiation, heating
130 e of binding ligands at the active site, and is abrogated by inactivation of the catalytic residue Se
131             However, the increase in EHT can be abrogated by increased Notch signalling.
132  inhibition of B6.K(d) heart graft rejection was abrogated by increasing T cell help through transfer
133 of CM-Plin5 mice or Plin5-transfected cells, is abrogated by incubation with PKA.
134  necrosis factor-alpha-activated macrophages was abrogated by incubation with MC.
135 clusively within RV-infected cells and could be abrogated by inhibiting the lysosomal-endosomal degra
136                                This increase was abrogated by inhibiting inositol trisphosphate-media
137 t increase in angiogenic chemokines that can be abrogated by inhibition of NF-kappaB signaling.
138 s and that the efficacy of this strategy can be abrogated by inhibition of TGFbetaR on BM-hMSCs.
139 njury induced sickness behavior, which could be abrogated by inhibition of the HMGB1-RAGE pathway or
140 or HSF1-mediated neuroprotection, protection is abrogated by inhibition of classical histone deacetyl
141 y controlled by hypoxia and how this control is abrogated by inhibition of insulin-like growth factor
142                                This turnover is abrogated by inhibition of the ATG system, indicating
143 ty in mesenchymal progenitor cells, and this was abrogated by inhibition of ataxia-telangiectasia-mut
144 3beta (GSK-3beta) at serine 9, as the latter was abrogated by inhibition of either PI3K or Akt.
145 th is dependent on actin reorganization, can be abrogated by inhibitors of actin polymerization, and
146                                This response was abrogated by inhibitors of ASM or in ASM-deficient m
147 lization of the centrosome by Akt inhibition was abrogated by inhibitors of glycogen synthase kinase
148   Increases in VEGF and NADPH oxidase levels were abrogated by inhibitors of NADPH oxidase and MAPK,
149                              This impairment is abrogated by iNOS inhibition or c-Jun deficiency, but
150 ed with expression of TNF receptor TNFR1 and was abrogated by interfering with the TNFalpha/TNFR1 int
151                 These effects of FeTMPyP(5+) were abrogated by intra-RVM naloxone, implicating potent
152 bly, this function is unique to Hb alpha and is abrogated by its genetic depletion.
153                                These effects were abrogated by JD5037 or the SPT inhibitor myriocin.
154               However, the enhanced activity is abrogated by kinase-dead, dominant negative mutants o
155 ive oxygen species generation and cell death were abrogated by knock down of uncoupling protein 3.
156 )O(2) and other oxidants, and the protection was abrogated by knockdown of BRCA1.
157            ANG II-induced activation of NHE3 was abrogated by knockdown of IRBIT, whereas overexpress
158 ased production of superoxide, both of which were abrogated by knockdown or antibody blockade of SIRP
159  inflammatory actions by activated platelets were abrogated by lack of CD40 on injected platelets.
160  centriole cohesion and basal foot formation are abrogated by loss of Kif3a.
161 dac2-mediated deacetylation, and this effect is abrogated by loss of Hdac2-Gata4 interaction.
162 ral endothelial alterations induced by Abeta were abrogated by lovastatin, consistent with its anti-i
163                        Thus, the disease can be abrogated by lowering serum suPAR concentrations thro
164 sed endothelial cell apoptosis, all of which were abrogated by maternal administration of intravenous
165 CPC to isoproterenol-induced cell death that is abrogated by metoprolol.
166 nflammatory molecules and EMMPRIN, and these were abrogated by minocycline.
167  increases lung metastasis in vivo, and this is abrogated by miR-452 overexpression.
168                                Such activity was abrogated by modification of plasminogen structure a
169          Left ventricular adverse remodeling was abrogated by MPC therapy, resulting in a marked redu
170                                   The effect is abrogated by mutation of the catalytic Ser-525 and re
171 -loop region in the C-type lectin domain and is abrogated by mutation within the domain.
172 that LpSpl possesses S1P lyase activity that was abrogated by mutation of the catalytic site residues
173 using wild type AChE 3'-UTR RNA segment that was abrogated by mutation of the consensus recognition s
174               Such arm-dependent folding can be abrogated by mutations associated with beta-cell dysf
175 ct did not depend on enzymatic activity, but was abrogated by mutations disrupting the hexameric stru
176 tion of high glucose-induced NOX4 expression was abrogated by N(omega)-nitro-l-arginine methyl ester,
177 yperplasia in colonic crypts of Fabp-PG mice was abrogated by NEMO treatment, suggesting a cross-talk
178                                These effects were abrogated by NET inhibition with DNAse or a neutrop
179                                This activity was abrogated by neutralization of IL-1beta.
180 uced virus replication in PAR2ko fibroblasts was abrogated by neutralizing IFNbeta antibody.
181 n of metabolic disorders via brain ER stress was abrogated by NF-kappaB inhibition in the hypothalamu
182 e chemoattractant receptor for chemerin, and was abrogated by NK cell depletion.
183  Furthermore, neuroendocrine gene expression was abrogated by Notch activity in SCLC cells.
184  Notch1 trafficking and signaling in ECs and was abrogated by Notch1 deletion in ECs in vivo.
185                                         This was abrogated by NOX4 small interfering RNA knockdown an
186 HNE-induced increases in GSH and NQO1 levels were abrogated by Nrf2 small interfering RNA in normal c
187 mented AHR to MCh in the OVA/OVA mice, which was abrogated by NVP-QAB-205.
188 persensitive to amino acid feeding, and this is abrogated by oral administration of EGCG prior to cha
189                 This enhanced chemotaxis can be abrogated by osteoprotegerin (soluble decoy receptor
190  while in the remaining cases, TP53 activity is abrogated by other oncogenic events, such as hyperact
191 weeks, and endogenous sex hormone production was abrogated by ovariectomy.
192 itation analysis and furthermore found it to be abrogated by panr2.
193 giotensin-II-promoted MARCKS phosphorylation is abrogated by PEG-catalase, implicating endogenous H(2
194 l of organ transplantation with CD40Ig could be abrogated by permanent depletion of CD8(+) cells that
195 led that the biological effects of MND could be abrogated by pertussis toxin.
196  lysosomal damage and cytotoxicity of Dp44mT were abrogated by Pgp inhibitors, Pgp silencing, or incr
197                                 This process was abrogated by pharmacologic inhibition of PI3Kbeta, b
198 thelial de-differentiation observed in vitro was abrogated by pharmacological blockade of the ALK-5 c
199                        This mitogenic effect was abrogated by pharmacological inhibition of type I TG
200 spiration and ATP production, an effect that was abrogated by phosphocreatine-mediated reactivation o
201  line expressing endogenous mutated p53, can be abrogated by pre-miR-205 or silencing of mutated p53.
202 lated induction of miR146a and miR155, which is abrogated by pre-treatment with triclosan.
203 en-induced acute liver failure plasma, which was abrogated by preincubation with deoxyribonuclease-I.
204 e oxygen species (ROS) generation because it is abrogated by pretreatment of PC cells with the free r
205 r, FXIIa stimulated IPF HLF migration, which was abrogated by pretreatment of cells with heparinase I
206 MOG-induced models of EAE, respectively, and was abrogated by pretreatment with a depleting anti-CD25
207                                These changes were abrogated by pretreatment with lovastatin or NADPH
208 induced BBB disruption and neuroinflammation were abrogated by pretreatment with the rho kinase inhib
209                  Moreover, NK-cell expansion was abrogated by prior injection of clodronate-loaded li
210 V gp120-stimulated production of neurotoxins was abrogated by prior introduction into the cells of a
211 n of miR-31 repressed Fzd9 expression, which was abrogated by prostacyclin.
212 d transendothelial migration capacity, which was abrogated by RAC inhibition.
213 s RANK and undergo hyperproliferation, which is abrogated by RANKL neutralization.
214                            This effect could be abrogated by rapamycin suggesting that mTOR signaling
215                        Ancestry associations were abrogated by recruitment site but not region of ori
216 fic effects of the inhibitory peptide region were abrogated by replacements of 4-10 residues with Gly
217 astasis whose elaboration and tissue effects are abrogated by RLH innate immune responses.
218       Conversely, actin severing by Gelsolin was abrogated by RNA interference-mediated silencing of
219        The relaxin-like actions of CGEN25009 were abrogated by RXFP1 silencing in vitro, and, in comp
220 ation of nPKCs (PKCepsilon) and PKD that can be abrogated by selective inhibitors or by expression of
221      The adverse immunostimulatory responses were abrogated by selective B cell-targeted delivery and
222 show that fusion of macropinosomes/endosomes is abrogated by septin immunodepletion and function-bloc
223                                ATR signaling was abrogated by shRNAs to ATR and TopBP1, but shRNAs to
224     S100P elevation under hypoxic conditions was abrogated by silencing of Cav1 or NF-kappaB function
225 ar signal-related kinase 1/2 and Snail1 that was abrogated by silencing of TrkA or CCN2 using small i
226  CCL5 Such infiltration and tumor regression were abrogated by silencing CCL5 in BECN1-defective tumo
227 uced neurite-like projections, both of which were abrogated by silencing EWS-WT1.
228 gnificantly enhanced level of apoptosis that was abrogated by siRNA silencing of DR5.
229  LNCaP and C4-2 prostate cancer cells, which is abrogated by small hairpin RNA (shRNA) or the dominan
230 nvasion, and the enhancement of invasiveness was abrogated by small interfering RNA (siRNA)-mediated
231                              These reactions were abrogated by SNP.
232 ll without any loss of specificity, as entry was abrogated by soluble ephrinB2.
233 tion was driven by the ERK1/2 pathway, as it was abrogated by specific inhibitors of MEK.
234 nduced increase in VEGF expression in WT EPC was abrogated by STAT3 inhibitor, suggesting IL-10 signa
235 levels were maintained, whereas IL-13 levels were abrogated by steroid treatment in neonatal HDM-expo
236  recombinant HCMV in which pUL21a expression was abrogated by stop codon mutations, and the defect wa
237 g PD-1 ligand (CD274), and these differences were abrogated by sustained CD274 blockade.
238 ion of NK1-expressing PC, with these effects being abrogated by systemic administration of the opioid
239  transcription and protein expression, which were abrogated by Tat heat inactivation, immunodepletion
240 lly, Treg-mediated resolution of lung injury was abrogated by TGF-beta inhibition.
241                                These effects are abrogated by the addition of MAB228 (a monoclonal an
242                       The inhibitory effects are abrogated by the introduction of a function-blocking
243 nuclein via elevations in MAO-B levels could be abrogated by the addition of deprenyl, a specific MAO
244 -) dams provoked pregnancy loss, which could be abrogated by the adoptive transfer of IL-10(+/+) NK c
245                This essential function could be abrogated by the dominant-negative effect of HOXD10 a
246                      Mediator production can be abrogated by the use of inhibitors directed against k
247 the nucleus to the cytoplasm in Teffs, which is abrogated by the addition of exogenous cysteine.
248      In Ewing sarcoma cells, NOTCH signaling is abrogated by the driver oncogene EWS-FLI1.
249 prognostic impact of chemotherapy resistance is abrogated by the implementation of early response-bas
250                 However, the tumor phenotype is abrogated by the introduction of a hypomorphic mutant
251 the angiogenic receptor neuropilin-1 (NRP-1) is abrogated by the liver kinase B1 (LKB1), a serine-thr
252                 This tumor suppressor effect is abrogated by the PC-associated SPOP mutations.
253 poI-induced contact between homologous genes is abrogated by the transcriptional inhibitors actinomyc
254 nd to lower concentrations of eotaxin, which was abrogated by the above-mentioned PKCbetaII inhibitor
255 ; and queuine-stimulated in vivo methylation was abrogated by the absence of the enzyme that inserts
256 omprised of Th1, Th17, and Th22 cells, which was abrogated by the addition of anakinra.
257 ssing the couAB promoter region, and binding was abrogated by the addition of nanomolar quantities of
258                            Furthermore, this was abrogated by the addition of recombinant OA to cultu
259          The toxicity of IL-36beta + TT/Alum was abrogated by the administration of a neutralizing an
260 f S100A9KO mice, but not wild-type mice, and was abrogated by the administration of anti-CD8 Ab or ad
261 ile recruitment to the oxidative DNA lesions was abrogated by the anti-oxidant N-acetylcysteine, this
262 s into network structures induced by Ad-LKB1 was abrogated by the co-transduction of a dominant negat
263 gmented by dominant-negative RhoA(T19N), but was abrogated by the constitutive-active RhoA(G14V).
264 uclear localization of P. pastoris MS (PpMS) was abrogated by the deletion of 107 C-terminal amino ac
265 e, the suppressive effect of oxo-m on PF-LTP was abrogated by the GDP analog GDP-beta-S (applied intr
266 in response to interleukin-7, an effect that was abrogated by the JAK1/2 inhibitor ruxolitinib.
267 nced KLF6-mediated growth suppression, which was abrogated by the KLF6-4A phosphomutant.
268                               The inhibition was abrogated by the LOXL2 function-blocking monoclonal
269         Induction of Gadd45-beta by LY379268 was abrogated by the mGlu2/3 receptor antagonist (2S)-2-
270 ulin autoantibodies by TRAV5D-4 alpha-chains was abrogated by the mutation of insulin peptide B:9-23
271                                  This effect was abrogated by the PDGFRalpha-specific inhibitor creno
272 or-specific transfusion (DST), respectively, was abrogated by the presence of anti-K(d) mAbs, and all
273 ated KLF6 degradation in the presence of SV1 was abrogated by the proteasome inhibitor MG132.
274 s revealed that TNF-alpha-induced cell death was abrogated by the simultaneous inhibition of caspases
275 ypt organoid proliferation and crypt budding was abrogated by the Wnt inhibitor IWP2.
276 ncrease in the DPAGT1 transcript levels that was abrogated by the Wnt inhibitor, Dickkopf-1.
277 Ancestry-related differences in relapse risk were abrogated by the addition of a single extra phase o
278 d STAT3 phosphorylation and astrogliogenesis were abrogated by the addition of neutralizing antibody
279 l of the effects of TNF-alpha on SDC4 and HS were abrogated by the metalloproteinase (MMP) inhibitor
280                       These Ca(2+) increases were abrogated by the MS channel inhibitor Grammostola s
281 s-specific CTLs, and its therapeutic effects were abrogated by the mTOR inhibitor rapamycin.
282                     The effects of uPA-PAI-1 were abrogated by the nitric-oxide synthase (NOS) inhibi
283  Spontaneous and TGF-beta1-stimulated EndoMT were abrogated by the restoration of functional CAV1 dom
284  mice with burn injury and this effect could be abrogated by TNF-alpha or caspase-3 inhibitors.
285  or high glucose in vitro, and this increase was abrogated by transgenic overexpression of superoxide
286 e single binding sites in vitro, and binding was abrogated by transversions in the binding sites that
287 ta indicate that inflammatory osteolysis can be abrogated by treatment with a molecule composed of th
288  transcription 5 (STAT5) phosphorylation and is abrogated by treatment with C188-9, a STAT3/5 inhibit
289 mia cells correlated with dCK expression and was abrogated by treatment with a dCK inhibitor.
290                                   Resistance was abrogated by treatment with agents that scavenged RO
291  of enzymatically active and inactive AMCase was abrogated by treatment with allosamidin.
292 mice suppressed melanoma growth; this effect was abrogated by treatment with neutralizing antibodies
293 ce by the ERBB2 extracellular domain mutants was abrogated by treatment with small-molecule inhibitor
294 inflammation and goblet cell differentiation were abrogated by treatment of neonatal Foxa2(Delta/Delt
295                       The effects of HI diet were abrogated by treatment of the mice with N-acetyl cy
296 ng term barrier-promoting effects of HDL-S1P were abrogated by treatment with the recycling blocker,
297                                 However, VNC was abrogated by TRPV4 channel, GABAB, as well as an ade
298                    The effects of mast cells were abrogated by type I transforming growth factor beta
299      To ascertain if this interference could be abrogated by using a synthetic non-acetylated oligosa
300                 All these beneficial effects were abrogated by vagotomy.

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