戻る
「早戻しボタン」を押すと検索画面に戻ります。

今後説明を表示しない

[OK]

コーパス検索結果 (1語後でソート)

通し番号をクリックするとPubMedの該当ページを表示します
1 e provide evidence that the hedgehog pathway is activated in 20% of T-ALL samples.
2              The same PF3D7_1255200 var gene was activated in 4 different experimental infections.
3          The mobile-phone positioning system was activated in 667 out-of-hospital cardiac arrests: 46
4              In particular, these components are activated in a sequential manner in order to success
5         Here, we show that although caspases are activated in a well-studied neoplastic tumor model i
6                              This system can be activated in a convertase-independent manner from int
7 vocal evidence that naive CD4(+) T cells can be activated in a nonlymphoid organ.
8            The purified protein was shown to be activated in a pH-dependent manner.
9 and involves approximately 10% of the genome being activated in a highly dynamic pattern.
10 e, could be used to change the way a prodrug is activated in a "programmable" manner.
11 vation has been proposed, in which PKGIalpha is activated in a cGMP-independent fashion via oxidation
12  polarity-responsive enhancer in upd3, which is activated in a coincident manner by both JNK-dependen
13  galactose, the galactose regulatory pathway is activated in a fraction of the cell population hours
14                     The innate immune system is activated in a number of degenerative and inflammator
15  RIM pathway, and, accordingly, pH signaling is activated in a phosphodeficient Rim8 mutant and impai
16                             Moreover, Notch1 is activated in a small subset of SCC cells at the invas
17 roblasts, in which constitutive Gli activity is activated in a Smoothened (Smo)-independent fashion,
18  chemically-induced injury, Ascl3 expression is activated in a subset of horizontal basal cells (HBCs
19 ke the other phospholipase C family members, is activated in a sustained manner.
20 ysical studies have shown that alpha-catenin is activated in a tension-dependent manner that in turn
21 em2)(-/-) Tg-AD reveals that the DAM program is activated in a two-step process.
22                        The complement system is activated in a wide spectrum of CNS diseases and is s
23 lones expressing an array of Vbeta receptors were activated in a dose-, time-, and processing-depende
24  Jag1 are expressed in, and Notch2 signaling is activated in, a subset of human HCC samples.
25 oves glucose tolerance and insulin signaling is activated in AAV mice.
26 eroceptive insular cortex (a region known to be activated in acute MWD) also prevented the establishm
27            Toll-like receptor 2 was shown to be activated in AD lesional skin (immediately proximal t
28 both vascular pathology and inflammation and is activated in AD patients and AD mice.
29                       A third IL17 subfamily is activated in adult immune cells indicating that expre
30              The JAK-STAT pathway appears to be activated in all myeloproliferative neoplasms, regard
31                             The mTOR pathway was activated in all 6 TCL cell lines tested and everoli
32                 After this visit, both leads were activated in all patients for an additional 3 month
33 ts signals from cytokines IL-4 and IL-13 and is activated in allergic airway disease.
34                       The complement pathway is activated in Alzheimer's disease.
35 alleles have not been identified and T cells are activated in an allele unrestricted manner.
36 er spinal cord injury (SCI), meningeal ILC2s are activated in an IL-33-dependent manner, producing ty
37                                   MAIT cells are activated in an MR1-restricted manner by diverse str
38                   RhoA, cdc42, Rac1, and FAK were activated in an EPAC1/2-dependent manner, and there
39 ents where it was unknown whether the target was activated in any individual patient.
40                    The WNT signaling pathway is activated in APAs.
41 they have restricted mobility but reside and are activated in apposition to CX3CR1(+) macrophages.
42 rget for the transcription factor ERG, which is activated in approximately 50% of human prostate canc
43                             The PI3K pathway is activated in approximately 70% of breast cancers.
44 post infection, indicating that ET signaling was activated in Arabidopsis early by SCN infection.
45 trast, no new cohesion is detected when Rec8 is activated in arrested oocytes by tamoxifen despite co
46 over PI3K/AKT2 as a direct PDGFA target that is activated in ASCs during WAT hyperplasia and is funct
47                                     NK cells are activated in asthma; however, their role in allergic
48 report in this study that TGF-beta signaling is activated in astrocytes during toxoplasmic encephalit
49                        The PERK-elF2 pathway was activated in astrocytes of the spinal dorsal horn in
50                   We conclude that HIF1alpha is activated in ATII cells after lung injury and promote
51 , a cytosolic sensor of double-stranded DNA, is activated in autoinflammatory disorders and by DNA da
52                                        Stat3 is activated in basal cells and their daughters early in
53     We establish that beta-catenin signaling is activated in basal cells during early pregnancy, and
54 howed that nuclear factor kappaB (NF-kappaB) is activated in BCR-ABL1-expressing cells, but the mecha
55 gene encoding the nuclear receptor PPARgamma is activated in bladder cancer, either directly by gene
56      In the infarcted heart, Smad3 signaling is activated in both cardiomyocytes and interstitial cel
57 ike endoplasmic reticulum (ER) kinase (PERK) was activated in both intact and Gcn2(-/-) mice.
58        The autophagy and proteasome pathways were activated in both muscles at varying intensities ov
59                Several inflammatory pathways were activated in both subtypes.
60 cancer samples, indicating that this pathway is activated in breast cancers that are clinically resis
61                               Its expression is activated in breast cancers, where high tumor levels
62     We report here a mechanism by which they are activated in cancer cells, involving release of the
63                                      EMT may be activated in cancer cells by reactive oxygen species
64             These results show that PP2A can be activated in cancer cells to inhibit proliferation.
65                    Furthermore, this pathway is activated in cancer cells, and correlates with pro-in
66            We show that the MRTF-SRF pathway is activated in cancer-associated fibroblasts (CAFs).
67                                          NF2 was activated in cardiomyocytes subjected to H2O2 and in
68  expression demonstrated that the Hh pathway is activated in CD34(+) CP-CML stem/progenitor cells.
69   The mechanistic target of rapamycin (mTOR) is activated in CD4(-)CD8(-) double-negative (DN) T cell
70 edia, whereas transcription of MDH1 and MDH2 is activated in cells cultured in YNB + Glu but not in Y
71               Although nuclear factor-kappaB is activated in cells from patients with inflammatory di
72                                 Many enzymes are activated in chemotransduction to hydrolyze ATP.
73              Here we show that both pathways are activated in chondrocytes after treatment with TGF-b
74                                      RET can be activated in cis or trans by its co-receptors and lig
75 , we evaluate whether the kynurenine pathway is activated in clinical HAT and associated with CNS inf
76 o1-CIN tissues upregulated pathways known to be activated in colon cancer, including lipid metabolism
77 rget of rapamycin complex 1 (mTORC1) pathway was activated in Cpt2M(-/-) hearts; however, daily rapam
78  by several 2'-O-methyl-modified nucleotides was activated in culture supernatants of S. aureus but n
79     Subsequently, Wnt/beta-catenin signaling is activated in cushion mesenchymal cells where it suppo
80                             Villin-CreER(T2) was activated in developed tumors by administration of t
81 e time, the BMP signal transducers SMAD1/5/8 were activated in developing coronary arteries.
82 ings provide the first evidence that the UPR is activated in DTC in the bone marrow from cancer patie
83 y of colonic muscles; different conductances are activated in each cell type of the SMC-ICC-PDGFRalph
84                Differential genetic programs are activated in each cell type to construct functionall
85                                 DDK, despite being activated in early S phase, does not phosphorylate
86                             Once the program is activated in early embryos, Fli1 then takes over to s
87                        The finding that MAPK was activated in early differentiating and mature D2J os
88 levels are increased in ECs, stress pathways are activated in ECs, and ISCs overproliferate.
89 sponsive reporter, we demonstrate that Notch is activated in endothelial progenitors during vasculoge
90                Under normal conditions, S1P1 was activated in endothelial cells of lymphoid tissues a
91 showed that the steroid biosynthesis pathway is activated in enzalutamide-resistant prostate cancer c
92                        Both Pdgfrb and Igf1r were activated in ex vivo T-ALL cells, and coculture wit
93 numerous genes that control inflammation and is activated in glomerular cells in glomerulonephritis (
94  the present study, we showed that ER stress was activated in granulosa cells of PCOS patients as wel
95           The renin-angiotensin system (RAS) is activated in heart failure (HF) and inhibition of RAS
96 Hypoxia-inducible factor-1alpha (HIF-1alpha) is activated in hepatic stellate cells (HSCs) by hypoxia
97  pegIFN-alpha in serum, the Jak/STAT pathway was activated in hepatocytes only on the first day after
98 endent HIF1alpha/VEGF-A/FOXO1/TWIST1 pathway was activated in high-grade PCa specimens, and knockdown
99  silent in the genome, this group of viruses is activated in HIV-1-infected and cancer cells.
100       After carbon tetrachloride, HIF-1alpha was activated in HSCs.
101       Importantly, BCAT1 expression not only is activated in human blast crisis CML and de novo acute
102 n patients with AAV indicated that NF-kappaB is activated in human NCGN also.
103 es have shown that homologous cortical areas are activated in humans during tongue movement tasks, th
104 scle cells of the human intestinal tract and is activated in inflamed and fibrotic tissue.
105  epithelial A1-adenosine receptors (A1-AdoR) are activated in influenza A virus-infected mice.
106                     We found that Ikkepsilon was activated in intestinal tumors forming upon loss of
107 bited by the Hippo tumor suppressor pathway, are activated in KSHV-infected cells in vitro, KS-like m
108       In this study, we found that caspase-3 is activated in L88 bone marrow stroma cell-derived exos
109 nal-regulated kinase (ERK) signaling pathway is activated in Langerhans cell histiocytosis (LCH) hist
110         Hence, type I cytokine receptors may be activated in leukemia through 2 types of transmembran
111 nuclease inositol-requiring enzyme 1 (IRE1), is activated in lipid-laden macrophages that infiltrate
112                     A bidirectional response is activated in <5 min of training, in which phosphoryla
113 hown to be an eat-me signal on cancer cells, is activated in macrophages for secretion and cell-surfa
114                Here we report that caspase-1 is activated in macrophages under hypertonic conditions.
115                               SCF E3 ligases are activated in many cancers and inhibitors could have
116          The Hedgehog (Hh) signaling pathway is activated in many cancers and is a promising target f
117 sis of epithelial barrier tissues; how MASPs are activated in mature epithelia is unknown.
118              Here, we show that, when ERK1/2 are activated in mature quiescent oligodendrocytes durin
119                        Abl kinases (Abl/Arg) are activated in melanomas and drive progression; howeve
120 cal adhesion formation and FAK/SRC signaling is activated in mesenchymal tumor cells by crosslinked c
121    We previously showed that beta1 integrins are activated in metastatic prostate cancer (PCa) cells,
122               We show that CGRP(PBN) neurons are activated in mice implanted with Lewis lung carcinom
123 e propose that the noncanonical inflammasome is activated in mice during acute intestinal inflammatio
124 egulation of critical cellular processes and is activated in more than 30% of human cancers.
125             Oncogenic NOTCH signaling, which is activated in more than 65% of T-ALL patients by activ
126  In these studies, we show that RA signaling is activated in mouse and zebrafish models of AKI, and t
127             We found that Jak/Stat-signaling is activated in Muller glia in response to NMDA-induced
128 1 knockouts, indicating that the endocardium is activated in multiple cases where sinus venosus angio
129  data demonstrate that the NOD2-RIP2 pathway is activated in murine and human VL and plays a role in
130 TS whereby p53 is deleted and oncogenic Kras is activated in muscle satellite cells via a Pax7(CreER)
131 on; (2) beta1 Integrin/FAK signaling pathway was activated in myeloma cells under hypoxic condition.
132 activated kinase (AMPK), a metabolic sensor, was activated in myotubes exposed to high CO2, and loss-
133 quires intact TLR9, but how the TLR9 pathway is activated in NASH is not clear.
134 d and are tightly linked, since beta-catenin is activated in nearly all of the CAR-dependent tumours
135                                       Lin28B is activated in neuroblastoma (NB), a childhood tumor in
136                                     SPOT can be activated in neuron cell bodies or uncaged in dendrit
137             We found that these channels can be activated in neurons of the medial nucleus of the tra
138                         We found that Cdc25A is activated in neurons undergoing ischemic stress media
139 oot cap regulators SOMBRERO and BEARSKIN1/2, are activated in nlp7-1 seedlings.
140 m of the large, membrane fission GTPase, can be activated in nonneuronal cells downstream of cancer-r
141                                          AEP is activated in normal mice in an age-dependent manner,
142     For example, the murine protein p204 can be activated in numerous tissues by distinct transcripti
143 lays a vital role in cell cycle progression, is activated in numerous human malignancies, and is link
144                              The AKT pathway was activated in one subtype while MAP kinase pathway wa
145 , and the ErbB3-binding protein-1 (Ebp1) can be activated in oral tumorigenesis and can serve as a tr
146                           Hedgehog signaling is activated in osteoarthritis, where it promotes chondr
147 enhancers are evolutionary conserved and can be activated in other tissues, in contrast to enhancers
148           In addition, proapoptotic pathways are activated in P1 knock-in premegakaryocyte/erythroid
149 SPHK1/Akt/NF-kappaB signaling pathway, which is activated in pancreatic cancer.
150                      We also reveal that ATM is activated in pancreatic precancerous lesions in the c
151 cyclin-dependent kinase 4/6 (CDK4/6) pathway is activated in parallel with renal cell-cycle entry but
152 ormal adult lymphatic endothelial cells, but is activated in pathological situations such as inflamma
153                 However, how beta1 integrins are activated in PCa cells is unknown.
154             We found here that although MSKs are activated in peritoneal B cells in response to TLR4
155  found that the WNT/beta-catenin pathway can be activated in PHHs, as assessed by universal beta-cate
156                 In nature, this strategy may be activated in phytochrome-disabling, vegetation-dense
157 gen-specific B lymphocytes and T lymphocytes are activated in piperacillin-hypersensitive patients.
158              Multi-layered defense responses are activated in plants upon recognition of invading pat
159                    We found that necroptosis was activated in postmortem human AD brains, positively
160                                        NR1D1 was activated in primary mouse and human macrophages by
161                            When beta-catenin was activated in Prom1+ DP cells there was an increase i
162 roblastoma RAS viral oncogene homolog (NRAS) is activated in PSC cholangiocytes.
163 OR), which governs most protein translation, was activated in rat spinal dorsal horn neurons after re
164                        The JAK/STAT3 pathway was activated in reactive astrocytes in two transgenic m
165 istence of enhancer regulatory elements that are activated in regenerating tissue.
166 murine leukemia virus envelope protein (Env) are activated in relation to each other, sequentially or
167            The NLRP3 inflammasome can indeed be activated in resident macrophage/monocyte-like cells
168 rentiating epiblast shows that CDX2 and ELF5 are activated in response to BMP4.
169 eptor potential vanilloid 4 (TRPV4) channels are activated in response to changes in plasma membrane
170           The inflammatory caspases 1 and 11 are activated in response to different agonists and act
171 g IFN-gamma-producing NKG2D(+) NK cells that are activated in response to DS-induced stress signals.
172        We report here that mTORC1 and mTORC2 are activated in response to exogenously supplied fatty
173                                        MAPKs are activated in response to G protein-coupled receptor
174                   Innate type 2 immune cells are activated in response to helminths, allergens, and c
175 em cell population in adult skeletal muscle, are activated in response to hypertrophic stimuli and gi
176 8(MAPK) molecules and 36.5% of MK2 molecules are activated in response to IL-1beta.
177                        In the CNS, microglia are activated in response to injury or infection and in
178 ndicating that the label-retaining (LR) GBCs are activated in response to injury.
179 lammatory caspases (caspases 1, 4, 5 and 11) are activated in response to microbial infection and dan
180                              These promoters are activated in response to unique signals.
181 ptionally repressed in ES cells but ready to be activated in response to differentiation signals.
182 HSCs) are dormant in the bone marrow and can be activated in response to diverse stresses to replenis
183 alateral prefrontal cortex are also shown to be activated in response to INMS.
184            While RTKs are generally known to be activated in response to ligand binding, many of thes
185  A aminoarabinose transferase ArnT, known to be activated in response to limiting magnesium.
186 omponent of the innate immune system and can be activated in response to microbial and endogenous dan
187  a homotetrameric transcription factor which is activated in response to a variety of cellular stress
188 diated proteasome degradation mechanism that is activated in response to abnormal Ca(2+) homeostasis.
189 cocorticoid production in the adrenal cortex is activated in response to an increase in cyclic AMP (c
190           Chaperone-mediated autophagy (CMA) is activated in response to cellular stressors to preven
191          However, the mechanism by which p53 is activated in response to centrosome loss remains unkn
192       The allelochemical DIMBOA gene cluster is activated in response to co-cultivation with rice, wh
193       The non-receptor tyrosine kinase c-Abl is activated in response to DNA damage and induces p73-d
194            The DNA damage checkpoint pathway is activated in response to DNA lesions and replication
195 ine the molecular mechanism by which PKCzeta is activated in response to hyperglycemia and suggest th
196                          In particular, IRF3 is activated in response to infection by a range of viru
197                     Cyclooxygenase-2 (COX-2) is activated in response to ischemia and significantly c
198 ess their own unfolded protein response that is activated in response to mitochondrial protein foldin
199 sive Polycomb complexes in growing cells but is activated in response to oncogenic stress.
200                                    Autophagy is activated in response to stress, but its regulation i
201              Alkaloid accumulation in plants is activated in response to stress, is limited in distri
202                           p38 MAPK signaling is activated in response to stretch, and inhibition of p
203  (UPR) is a cellular adaptive mechanism that is activated in response to the accumulation of unfolded
204                        The complement system is activated in response to tissue injury.
205 cell line and animal models showed that Pak1 is activated in response to UV-B radiation, and this act
206  did not attain the levels observed when p53 was activated in response to etoposide treatment and rem
207 n-activated protein kinase kinase-1 (MEKK-1) were activated in response to TNF-alpha.
208                         Calcium oscillations were activated in rice atrichoblasts by CO4, but not the
209                                  Yap and Taz are activated in Schwann cells by mechanical stimuli and
210 onstrates that oncogenic ErbB2 signaling can be activated in SCs but not impede transdifferentiation
211        The Rho family GTPases Rac1 and Cdc42 were activated in senescent cells, and simvastatin reduc
212                 Here we show that complement is activated in skeletal muscle injury and plays a key r
213 nfiguration and proximal or distal geometry, were activated in solution by ultrasound-generated elong
214 logical pathway caused by epigenetic changes is activated in some forms of congenital neuromuscular d
215 at NRF2 and protumorigenic NRF2 target genes were activated in some actinic keratoses, the major prec
216                      Consistently, NF-kappaB was activated in sorafenib-resistant HCC cells, and this
217 ompeting functional roles, only one of which was activated in speech production.
218 s, and we present evidence that this pathway is activated in sporadic CCM patients.
219   Both ER and mitochondrial stress responses are activated in sqrd-1 mutant animals exposed to H2S, b
220 ulation of the right and left phrenic nerves were activated in synchrony with mechanically ventilated
221      The mechanism by which mast cells (MCs) are activated in T cell-mediated inflammatory processes
222                                    NF-kappaB was activated in T cells by inducing the expression of a
223  pathways linked to epigenetic reprogramming were activated in TBDM above levels observed with DM alo
224 fied a group of CTNNB1/TCF target genes that are activated in the absence of TCF7L1, including EPHB3,
225 ests that serine-aspartyl proteases-caspases are activated in the AD brain.
226 tes are pivotal in this process because they are activated in the attempt to digest Abeta which start
227 DNA damage nor the intra-S phase checkpoints are activated in the first cell cycle, demonstrating tha
228        Indeed, 4 of 11 enzymes of this cycle are activated in the light through dithiol/disulfide int
229 farnesoid X receptor (FXR, encoded by NR1H4) are activated in the liver in the fasted and fed state,
230 ase- and oxidative stress-dependent pathways are activated in the lung tissue of C57/BL6J-betaENaC-Tg
231 al nervous system tumors and found that MNKs are activated in the majority of tumors tested.
232 a, determination of which cells and pathways are activated in the network of APCs remains elusive.
233 mmune encephalitis (EAE), autoimmune T cells are activated in the periphery before they home to the C
234              We examined whether macrophages are activated in the skin of imiquimod (IMQ)-treated mic
235 monstrating that the PGLYRP1/TREM-1 axis can be activated in the absence of bacterial products.
236 signaling pathways (e.g., ERK signaling) can be activated in the absence of nuclear strain transfer,
237          Because Notch signaling is known to be activated in the bleomycin model of pulmonary fibrosi
238  first time that this prominent catalyst can be activated in the dark even when electrically insulate
239 low and it is unclear how Ca(2+) release can be activated in the interior of atrial cells.
240 rently expressed genes and were predicted to be activated in the intestine of insulin-resistant subje
241  investigated whether the NLRP3 inflammasome is activated in the adipose tissue of burned patients, a
242 dings suggest that the CgA-angiogenic switch is activated in the BM of multiple myeloma patients and
243             Our studies demonstrate that p53 is activated in the brain following reovirus infection a
244 onstrate that DR-induced apoptotic signaling is activated in the brain following WNV infection and su
245                                        c-Abl is activated in the brain of Parkinson's disease (PD) pa
246                        Here we show that IIS is activated in the cells surrounding an epidermal wound
247 orkhead boxO family of transcription factors is activated in the diaphragm during mechanical ventilat
248 s, and that the ubiquitin-proteasome pathway is activated in the diaphragm.
249  model, the Yap protein of the Hippo pathway is activated in the eyes, and blood vessels near the les
250                 Here, we demonstrate that Gi is activated in the Golgi by GIV/Girdin, a non-receptor
251 r AMP-dependent enzyme, AMP deaminase (AMPD) is activated in the liver of diabetic mice, which parall
252                             The MAPK pathway is activated in the majority of melanomas and is the tar
253  analyses reveal that miR-200a-3p expression is activated in the pre-ameloblast cells to enhance epit
254                                In yeast, PKA is activated in the presence of high glucose concentrati
255                   On the contrary, Snf1/AMPK is activated in the presence of low glucose or alternati
256  system, the human sex-determining gene hSRY is activated in the single-cell embryos of the mouse.
257                                Tryptophanase is activated in the stationary phase by the stationary-p
258                                         EGFR is activated in the ventral midline and neurogenic ectod
259                              The same region was activated in the aphasic patients as they listened t
260       A mirror right-lateralized FTP network was activated in the Count and Decision trials, but not
261 ly translocate between both compartments and was activated in the cytoplasm.
262 GUS fusion assays we established that miR827 was activated in the initial feeding cells and this acti
263                                         LBOW was activated in the intestinal epithelium, and a year-l
264 ated in one subtype while MAP kinase pathway was activated in the other.
265         Here, we found that the mTOR pathway was activated in the parathyroid of rats with secondary
266                             Neurostimulation was activated in the sham group at 3 months and outcomes
267         Multiple cell death-related pathways were activated in the degenerating D stump, whereas acti
268 tein signaling, such as bmp2, nog, and dlx5, were activated in the extraction wounds of the ALN-treat
269 sical complement pathway associated proteins were activated in the glaucoma samples suggesting an inn
270 ozoon cuniculi infection, CD11b(-) CD8(+) DC were activated in the lamina propria (LP) and acquired t
271 entially expressed and 94 signaling pathways were activated in the topical MSCs transplanted onto the
272 in the hearing condition at those sites that were activated in the two conditions.
273 components of insulin/insulin-like signaling are activated in their discs.
274 egulated in daughters of overweight fathers, was activated in their mammary tissues and tumors.
275 llular signal-regulated kinase (ERK) kinases are activated in these knockout animals.
276 gation in megakaryocytes, an endomitotic SAC is activated in these cells upon Plk1 inhibition.
277           However, the immune receptors that are activated in this context remain elusive.
278 at the level of activation, but how TGF-beta is activated in this disease is unknown.
279                    It is not clear how ARL-8 is activated in this process.
280                         How the inflammasome is activated in this untreatable disease is largely unkn
281                      Here we report that XOD is activated in THP-1 human myeloid cells in response to
282 anaerobe nanovesicle mimic that can not only be activated in THRs but also induce hypoxia in tumors b
283                     Here we report that XBP1 is activated in TNBC and has a pivotal role in the tumor
284 lls and demonstrate that specific mechanisms are activated in tolerant T helper cells to directly rep
285 nd other Treg cell signature genes, began to be activated in Treg precursor cells.
286 use and contains multiple MYC enhancers that are activated in tumor cells.
287                                         RhoA is activated in tumors from neuroblastoma patients, and
288 cytidine deaminase-induced mutagenesis might be activated in tumourigenesis and limited therapeutical
289                                        Cdc42 is activated in two temporal steps in the G1 phase: the
290 iety of DNA repair mechanisms, many of which are activated in vascular disease.
291                                         MKK4 is activated in vitro by reduced Trx but not oxidized Tr
292         Thus, we found that when donor cells were activated in vitro and injected intraperitoneally,
293            Primary pancreatic stellate cells were activated in vitro by C5a.
294                        Human CD19(+) B cells were activated in vitro in the presence or absence of 9c
295      In the current study, human neutrophils were activated in vitro with immobilized extracellular m
296          Finally, we demonstrate that ERK1/2 are activated in vivo in the colon of Stx2-intoxicated i
297 rial diseases, we show that the ATF4 pathway is activated in vivo upon mitochondrial stress.
298  and that in human lung cancers this pathway is activated in vivo.
299  in vitro and injected intraperitoneally, or were activated in vivo, they migrated simultaneously to
300        Previously we demonstrated that FOXO1 was activated in wound healing but to higher levels in d

WebLSDに未収録の専門用語(用法)は "新規対訳" から投稿できます。
 
Page Top