1 ddition of palmitate to lipid A, appeared to be alone sufficient to activate transcription at sigma(E

2 n of miR-155 in DCs is a critical event that is alone sufficient to break self-tolerance and promote

3 lf-templating change in protein conformation is alone sufficient to create a stable change in CPEB ac

4              The histone domain of macroH2A1 is alone sufficient to direct enrichment on the inactive

5 urogenin3 under control of the Pdx1 promoter is alone sufficient to drive early and ectopic different

6 ; H3K9 methylation-dependent heterochromatin is alone sufficient to form functional centromeres.

7 f the catalytic subunit of telomerase, TERT, was alone sufficient to increase the proliferative capac

8 t acute infection of mice with live bacteria is alone sufficient to induce acute pancreatitis.

9                                         RelA is alone sufficient to induce RelB gene transcription an

10 mad4 in the head and neck epithelium of mice is alone sufficient to initiate spontaneous HNSCC, in co

11 ted to determine whether hbeta(c) activation is alone sufficient to promote differentiation.

12 ng two CR domains), appropriately separated, being alone sufficient to provide the low nanomolar affi

13            Furthermore, BH4 deficiency alone is alone sufficient to reduce intracellular GSH:GSSG rat

14 DNA, we hypothesize that tightly looping DNA is alone sufficient to repress transcription.

15 t form side chain-intramodule hydrogen bonds are alone sufficient to specify the fold of an LA module

16 ted cells; 2) activation of an inducible Akt was alone sufficient to stimulate the phosphorylation of