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1 t negative clonal selection pathway that has been co-opted by a common bacterial pathogen to induce s
2 erful cell puncturing nanomachines that have been co-opted by bacteria for self-defense against both
3 fibroblast growth factor (FGF) signaling may be co-opted by breast cancer cells.
4 pically involved in immune cell homeostasis, are co-opted by cancer cells to thwart the development o
5 int in maintaining self-tolerance, which can be co-opted by cancer to evade immune attack.
6  during anti-microbial immune responses, can be co-opted by cancer to evade immune destruction.
7 f epithelial-to-mesenchymal transition (EMT) is co-opted by cancer cells to metastasize to distant si
8              Some elements of this machinery are co-opted by certain pathogens to gain entry into hos
9 mponents of regulatory landscapes, which can be co-opted by chance mutations in cis-regulatory elemen
10 the view that the karilysin catalytic domain was co-opted by competent bacteria through horizontal ge
11                   During infection, Alix can be co-opted by enveloped retroviruses, including HIV, pr
12 he ability of RISC to locate target RNAs has been co-opted by evolution many times to generate a broa
13 e genes that mediate immune response or that are co-opted by HIV-1 for completion of its lifecycle sh
14  fate choice in the innate immune system has been co-opted by Ikaros to orchestrate B lymphocyte gene
15  of allergic inflammatory circuitry that can be co-opted by metastatic cancer cells to facilitate lun
16  virulence factors for the pathogen but have been co-opted by plants as triggers for active resistanc
17                                 This pathway is co-opted by some viruses.
18 uggest a single molecular mechanism may have been co-opted by synaptic partners to ensure joint lamin
19      Thus, the T(FH) differentiation pathway is co-opted by T(reg) cells to control the germinal cent
20 ult system for lysosome homeostasis that has been co-opted by the autonomous defense program to clear
21 appears that the flagellar AS transcript has been co-opted by the Rcs system to regulate virulence.
22 e possibility is that this feedback response is co-opted by the learning system and used as a templat
23 ncy of the allosteric site for the cell wall is co-opted by these antibiotics, and under these condit
24 tural motility-stimulating activity that has been co-opted by tumor cells.
25 lin action through Irs2 induction, which can be co-opted by Vegf inhibitors to modulate glucose metab

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