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1 l as other disorders in which endothelia may be damaged.
2 narios it is likely that these systems would be damaged.
3 aling pathways in conditions where the liver is damaged.
4 cause a G(2) cell cycle arrest when cell DNA is damaged.
5 tuations in which the endothelial glycocalyx is damaged.
6 rate NHEJ when the terminal bases of the DSB are damaged.
7 undergoes regeneration only when hair cells are damaged.
8 he DNA repair machinery because both strands are damaged.
9 fferents are activated when outer hair cells are damaged.
10 overy after various injuries, in which AEC I are damaged.
11 arB, but the consequences of the interaction are damaged.
12 motor function when primary cortical regions are damaged.
13 oximately 10% of the oligonucleotide strands are damaged.
14 s were detected where the epithelial surface was damaged.
15 and its SWV peaks at 0.04 V decreased as DNA was damaged.
16 super-oxide at pH 6.5, cytosolic fumarase B was damaged.
17 ted DNA from nematodes exposed to the nickel was damaged.
18 in the remaining cells because the sites had been damaged.
19 long-term viability, since mitochondria had been damaged.
20 even after as long as 60 h after the DNA has been damaged.
21 sharing the same milieu with cells that have been damaged.
22 helial tissue whose resident stem cells have been damaged.
23 , but the choroid, tapetum, and outer retina were damaged.
24 were used to detect which retinal cell types were damaged.
25 cal integrity and additional functions after being damaged.
36 of eukaryote chromosomes slows down when DNA is damaged and the proteins that work at the fork (the r
38 ion (allo-HSCT), the gastrointestinal mucosa is damaged, and colonizing bacteria are impacted, leadin
39 lon in experimental colitis where the mucosa is damaged, and this is likely to contribute to altered
40 takes place almost immediately after the DNA is damaged (approximately 2 min post-UV irradiation), wh
41 Polymers that can easily be repaired after being damaged are attractive as this characteristic can
42 roximately 1 helical turn, 10 bp) of the DNA is damaged, are lesions of enhanced biological significa
45 support the view that in glaucoma RGC axons are damaged at the optic nerve head and degenerate withi
48 otection cannot improve function if the axon is damaged, because the soma is still disconnected from
51 -zone cells die when their antigen receptors are damaged by activation-induced cytidine deaminase.
53 collapse ensues when these specialized cells are damaged by disease, a situation that currently neces
58 is not yet established whether HDL proteins are damaged by reactive nitrogen species in the circulat
61 However, it's unclear how many cells would be damaged by a particular bursting bubble, or more prec
62 ntial to present antigen to T cells and thus be damaged by direct antigen-specific interactions with
66 s one of the cellular constitutents that can be damaged by the interaction of A2E and blue light.
68 pace of albino rats whose photoreceptors had been damaged by continuous exposure to blue light for 3
70 how that splenocytes from lpr mice that have been damaged by gamma-irradiation expressed Fas protein.
71 tion but was downregulated in axons that had been damaged by glaucomatous injury and exhibited aberra
73 ian cells, the repair of DNA bases that have been damaged by reactive oxygen species is primarily ini
77 lly cueing in to the odor profile of a plant being damaged by a parasitized herbivore that contains t
83 steps in the synthesis of secreted proteins is damaged by heat shock, resulting in a selective halt
85 Chk1 is required to prevent mitosis when DNA is damaged by ionizing radiation (IR), whereas either ki
86 es acting on DNA during ES indicate that DNA is damaged by mechanical forces, and the damage takes pl
88 his observation confirms that retinal tissue is damaged by the development of a myopic crescent, rath
89 he basis of our data, the top graphene layer is damaged by the sputtering process, and the acid treat
92 n addition, DNA in buffered aqueous solution was damaged by ionizing radiation at different radiation
94 generated to visualize how the cell membrane was damaged by phage lysis, antibiotics attack, or dehyd
95 , well-differentiated colon carcinoma cells, were damaged by aspiration and the ensuing response was
97 ion mass spectrometry and DNA samples, which were damaged by gamma-irradiation or by H(2)O(2)/Fe(III)
99 ly, we used three Arabidopsis genotypes that were damaged by mechanical wounding or by insects of var
101 eted of Mirk became less viable because they were damaged by ROS, and had increased levels of G(1) cy
103 block cell cycle progression when their DNA is damaged, cells still undergo frequent genetic rearran
105 snout or the sensory nerves innervating them are damaged during a critical period in development, whi
109 To discover whether islet sympathetic nerves are damaged during the autoimmune destruction of islet B
113 ripheral branch of the trigeminal nerve, can be damaged during the surgical removal of lower third mo
116 These results suggest that if complex III is damaged during ischemia, PTP opening may result in su
120 response properties of striate neurons that were damaged (e.g., high spatial-frequency tuning, low c
124 s observed when primary auditory cortex (A1) is damaged have led to the widespread view that A1 may h
125 oped rosettes if host pigment epithelium had been damaged, if trauma to the donor tissue occurred dur
126 to understanding mechanisms by which tissues are damaged, illuminating mechanisms of repair and regen
132 ing functionally related areas of the brain, are damaged in PSP, leading to specific clinical deficit
138 of ecological systems around the world have been damaged in recent years, either by natural factors
141 teraction is disrupted when the frontal lobe is damaged in disease, and in extreme cases patients are
144 The structural integrity of tissue proteins is damaged in processes ranging from remodeling of the e
145 hat the ubiquinol oxidation site at center P is damaged in the bc(1) complexes with Rieske proteins l
154 lusions that (i) renewal mechanisms have not been damaged irreparably even at relatively advanced sta
157 to ensure that when a sensory cortical area is damaged, its basic sensory functions are 'taken over'
159 e mechanisms by which retinal ganglion cells are damaged may provide information required to develop
160 icate that regardless of which nuclear layer was damaged, MG respond by generating multipotent progen
161 ructure accounting for punctate labeling may be damaged mitochondria, lipofuscin or amyloid deposits.
162 liomyelitis-like disease where motor neurons are damaged, MUNE may also be a sensitive clinical or th
164 cal production are disrupted when hair cells are damaged or lost but that these behaviors return to n
165 for ammonia assimilation, when mitochondria are damaged or non-functional because of glucose ferment
168 ow-timescale disturbances occur when muscles are damaged or when limb dynamics change as a result of
169 in both types of disease, and helminths can be damaged or killed by toxic products released by eosin
171 ly useful in generating cell types that have been damaged or depleted by a variety of human diseases.
173 rin expression is altered when the epidermis is damaged or diseased, and there is good evidence that
175 s and suggest that when the occipital cortex is damaged or inhibited, and the visual processing for e
178 DNA replication when either the template DNA is damaged or the replication machinery malfunctions.
180 Checkpoint mechanisms delay mitosis when DNA is damaged or upon replication stress, but little is kno
182 th the checkpoint arrest of mitosis when DNA is damaged or when mitotic spindles have formed aberrant
186 astrointestinal tract, and peripheral nerves are damaged; Pompe's disease, in which the heart, skelet
187 cted to gentle purification procedures, they were damaged, preventing generation of reliable structur
192 in an individual, the same retinal location is damaged, regardless of visual function under test; gl
193 containing at least one copy of the Bax gene were damaged severely by exposure to excitotoxins or by
197 anoparticles rolls across a surface that has been damaged, stopping to repair any defects it encounte
199 ct their phenotype; however, when the retina is damaged, the Ascl1-expressing glia initiate a respons
201 When the human primary visual cortex (V1) is damaged, the dominant geniculo-striate pathway can no
203 axons of short thoracic propriospinal cells are damaged, their cell bodies of origin remain intact 2
205 ' to find a compensatory behaviour when they are damaged: they are limited to their pre-specified sel
206 of AT residues is essential and the A sites are damaged to a greater extent than they are in DNA.
208 was dramatically decreased when plasmid DNA was damaged to contain multiple types of DNA adducts, al
211 cal tubules (90%), and most cortical tubules were damaged, whereas few glomerular cells expressed MCP
212 line-treated laminin-alpha2-deficient muscle was damaged with cardiotoxin and muscle regeneration qua
213 DNA, contrasted with the multiple sites that were damaged within the observed segment of the collagen
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