ライフサイエンスコーパス: be depressed

1 specially children whose mothers continue to be depressed.

2 three times the odds that a nonsmoker would be depressed.

3 are, if anything, more likely than girls to be depressed.

4 d less belonging support were more likely to be depressed.

5 puts with high release probability tended to be depressed.

6 GAP knockout mice, whereas P38 MAPK function is depressed.

7 F cells discharge and sensory responsiveness is depressed.

8 eart failure, especially when renal function is depressed.

9 of interventions for youth with diabetes who are depressed.

10 terviews would help to identify patients who are depressed.

11 by NMDA receptors that include NR2A subunits are depressed.

12 ncreased with Ni exposure and calcium influx was depressed.

13 tured RPE, the mitotic activity of the cells was depressed.

14 and brachial artery flow-mediated dilatation was depressed.

15 or shock whether or not ventricular function was depressed.

16 ence of calsyntenin-1, synaptic transmission was depressed.

17 nts were anxious and stressed than when they were depressed.

18 ion, sarcoplasmic reticulum (SR) Ca2+ stores were depressed.

19 led receptors into the postsynaptic membrane were depressed.

20 intracellularly, inspiratory drive currents were depressed.

21 was accelerated, whereas HF ventricular CaTs were depressed.

22 low: 11.9% had heightened anxiety, and 9.8% were depressed.

23 f sickness per depressed worker per month of being depressed.

24 did not see the infant were least likely to be depressed (13 of 33, 39%, vs three of 14, 21%, vs one

25 e that senescent cardiac angiogenic activity was depressed (2 of 17 allografts were viable in 18-mont

26 Contractile reserve to dobutamine was depressed (62.3 +/- 0.9 FA versus 49.2 +/- 1.5 PM, F

27 ,S)-SAM to ACC and methylthioadenosine (MTA) are depressed 630- and 25-fold for the E47Q and E47D enz

28 , especially at night when net calcification was depressed ~78% compared to ambient pCO2 .

29 lated by exogenous application of 5-HT: they are depressed after 5-HT exposure.

30 LV diastolic and systolic function are depressed after TOF repair.

31 AM), a newly discovered vasodilator peptide, is depressed after hemorrhage and resuscitation.

32 by activation of CRF1 receptors with CRF but was depressed after activation of CRF2 receptors with Uc

33 e excitatory neurotransmission in LA neurons was depressed after ischemia via presynaptic mechanisms.

34 s not involved, because AMPAR-mediated EPSCs were depressed after a train.

35 both resting-state field potentials and CBF were depressed after cocaine administration (19.8+/-4.7%

36 At rest, the second H-reflexes were depressed an average of 73% relative to the first.

37 ll unclear because some DA neurons appear to be depressed and others excited by aversive stimuli.

38 fferentiation, because synaptic transmission is depressed and fails entirely, in a cyclical manner, a

39 at the contractility of failing human hearts is depressed and that abnormalities of basal Ca2+ regula

40 Basal left ventricular (LV) contractility is depressed and the force-frequency relationship is blu

41 , synaptic transmission in autonomic ganglia is depressed and the periphery becomes deregulated.

42 were placed under anoxia, protein synthesis was depressed and no "new" proteins were detected.

43 Contractile function of isolated myocytes was depressed and preceded left ventricular dilation and

44 ion closure, peak oxygen consumption (VO(2)) was depressed and there was systemic desaturation at res

45 cyclooxygenase-2 (Cox-2) and IL-6 expression were depressed and delayed.

46 ts remained significant both in patients who were depressed and in those who were nondepressed at enr

47 y synaptic responses, responses in two cells were depressed and in two they were enhanced.

48 nd motor output curves in small hand muscles were depressed and motor thresholds were elevated compar

49 Calcium transient amplitudes were depressed and the decay was slowed in DHF, especial

50 e work performance among service workers who were depressed and those who were not depressed.

51 ines in response to an inflammatory stimulus is depressed, and this may contribute to delayed recover

52 okine release from FLAP knockout macrophages was depressed, and their restricted ability to induce VS

53 Membrane expression of CD71 and HLA-DR were depressed, and monocytes were anergic to lipopolysa

54 The steady active tension was depressed approximately 45 % with 25 mM added P(i).

55 timulation of L-type Ca(2+) channel currents was depressed approximately 75% in the G(alphaq) mice.

56 CD8(+)-T-cell response in the knockout mice was depressed, as determined by pCTL and IFN-gamma assay

57 ress disorder were 3.93 times more likely to be depressed at 7 months.

58 4 %, Li 1.9 +/- 0.3 % in controls, P < 0.05) was depressed at 2 days by dorsal root section (Lc 4.8 +

59 xtracellular matrix immediately after injury was depressed at 20 and 50 mM peroxide, but by 12 h was

60 y was elevated and the L-type Ca(2+) channel was depressed at 8 months only.

61 lthough the chemoreceptor reflex bradycardia was depressed at a low dose of ANGII (5 fmol), all highe

62 rotein kinase A (PKA) activity in ob/ob mice was depressed at baseline and correctable towards the ac

63 001), as LVES stress/LVES volume index ratio was depressed at baseline and following MV repair versus

64 ncy excitation of BA interneurons (INs) that was depressed at higher frequencies.

65 ntractility fell significantly over time and was depressed at last follow-up in patients receiving mo

66 at frequencies below 10 kHz and above 20 kHz was depressed at low intensities, but was greatly enhanc

67 In both peas and soybeans, the oxygen uptake was depressed at low temperatures (<16 degrees C) and lo

68 Those who were depressed at 13 months were 7.96 times more likely

69 Similarly, those who were depressed at 7 months were 9.5 times more likely to

70 idents with a GDS-15 score, 374 of 765 (49%) were depressed at baseline; 484 of 765 (63%) provided 12

71 d b-wave amplitudes of the electroretinogram were depressed at the first postoperative week testing b

72 ording to the calculations, the pK(a) values are depressed because of unfavorable self-energies and r

73 The cardiac index was depressed below 3.0 L/min/m2 in 65 of 98 patients.

74 (P < 0.05), and blood glucose concentrations were depressed below the control during recovery (P < 0.

75 mong the major side effects of this strategy are depressed beneficial immunity and increased rates of

76 After CFPI alone CA1 ATP levels were depressed bilaterally only at 2 h (p < 0.05).

77 , and physical impairment are more likely to be depressed but may be less able to seek appropriate ca

78 onosynaptic potentials in Renshaw cells that are depressed, but not abolished, by cholinergic antagon

79 Resting ventricular function was depressed, but responsiveness to beta-agonist was no

80 cardiomyocyte mechanics and calcium kinetics were depressed, but isoproterenol reversed the inhibitor

81 time, constrictions to angiotensin I and II were depressed, but there was no loss of NTG vasodilator

82 tes of POPC; lipids in the surrounding shell are depressed by 0.5-0.8 A, but otherwise the phosphoino

83 Acetylcholine synapses are depressed by high-frequency stimulation, in part due

84 We found that hypocretin neurons are depressed by opiates, drugs of abuse that reduce cog

85 inary excretion of its 11-dehydro metabolite are depressed by the COX inhibitors.

86 er serious adverse events, and tended to not be depressed by the end of treatment.

87 atalytic activity of K165M and K165C mutants is depressed by >10(3)-fold.

88 he Curie temperature, based on Arrott plots, is depressed by 1-2 K from the bulk value.

89 ha-I, an index of actual generation in vivo, is depressed by aspirin or indomethacin.

90 Hypothalamic TRH is depressed by at least 75% compared with wild-type con

91 vesicular refilling with acetylcholine (ACh) is depressed by inhibitors, two sizes of quanta should b

92 High-frequency synaptic transmission is depressed by moderate rises in the extracellular pota

93 that of pathway refinement and LTD magnitude is depressed by nitrendipine, an L-type Ca(2+) channel b

94 oprotein P in human plasma has shown that it is depressed by selenium deficiency and by cirrhosis.

95 ndependent recombination in Escherichia coli is depressed by the redundant action of single-strand ex

96 promise: left ventricular developed pressure was depressed by 20%, and cardiac phosphocreatine was de

97 sarcomere-length shortening (nanometers, nm) was depressed by 42% (151.0+/-10.7 [control] versus 87.0

98 The amplitude of the second EPSC was depressed by 60% at 10 msec and recovered with tau a

99 state level of TPP+ accumulation in the lens was depressed by 90% when the lenses were cultured in a

100 The SR Ca(2+) fractional release was depressed by Ac-10C at relatively low SR Ca(2+) cont

101 In SC-CA1, it was depressed by agonists to levels below control, where

102 r than those for wild-type, whereas the kcat was depressed by an order of magnitude.

103 ia except in the absence of glucose, when it was depressed by approximately 50 %.

104 terleukin-6 by Mphi from ovariectomized mice was depressed by approximately 50%.

105 failed to bind FGF-2 unless heparan sulfate was depressed by chlorate or heparinase treatment.

106 It was not affected by hypoxia but was depressed by cobalt.

107 ly when the excitatory synaptic transmission was depressed by further lowering [Ca(2+)](o) with EGTA.

108 r for PGI(2), but not for other prostanoids, was depressed by HSD in WT and even more so in both muta

109 ,5-tetranor-prostane-1,20-dioic acid (PGE-M) was depressed by mPGES-1 deletion.

110 f the GHR, JAK2, and STAT5 in response to GH was depressed by nearly half in CRF (P < 0.05), and nucl

111 Since it was depressed by prior incorporation of the Ca(2+) buffe

112 Depolarisation-induced constriction was depressed by rotenone during hypoxia by 23 +/- 4 %;

113 naptic input from the somatosensory thalamus was depressed by sensory stimulation, and this depressio

114 Left ventricular ejection fraction was depressed by septic shock (p = .01) and further decr

115 response evoked by chemoreceptor stimulation was depressed by suramin, but convergent excitatory inpu

116 PKCdelta specific activity was depressed by the addition of VEGF (by 41+/-8% [P<0.0

117 dinor 6-keto PGF(1alpha) (PGI-M), only PGI-M was depressed by the COX-2 inhibitor.

118 FS to pyramidal cell unitary IPSCs were depressed by (RS)-S-amino-3-(3-hydroxy-5-tert-butyl

119 measure of excitation-contraction coupling, were depressed by 50% in both the inflow and outflow por

120 (P < 0.05), and nuclear phospho-STAT5 levels were depressed by approximately one third (P < 0.01).

121 ) and pentobarbitone sodium (41 %, 1:1), and were depressed by bicuculline (-41 +/- 5.7 %, 5:8) and p

122 elevation of Ca(2+) and release of proteins were depressed by cytokines.

123 d by D1LRs whereas pallidal inputs to the EP were depressed by D2LRs.

124 atory synapses onto hippocampal interneurons were depressed by either capsaicin, a potent TRPV1 chann

125 ither release of Ca(2+) from stores nor SOCI were depressed by high glucose levels.

126 When the discharges were depressed by severe hypoxia, exposure to light exci

127 mission of MF origin, MF GABAergic responses were depressed by the activation of metabotropic glutama

128 rminals by endogenously released substances, were depressed by the GABAA receptor antagonist SR95531

129 erpolarizations evoked by cyclopiazonic acid were depressed by the gap junction inhibitor 2-aminoetho

130 tom third at age 9 years were more likely to be depressed (CIS-R) at age 18 years (adjusted odds rati

131 iferation by multiple FasL- murine CTL lines is depressed compared to that of FasL+ CTL lines.

132 de evidence that net community calcification is depressed compared with values expected for pre-indus

133 ral blood mononuclear cells from TB patients was depressed, compared with that of healthy control sub

134 Synaptic transmission is depressed due to a reduction of cytoplasmic and readi

135 rictor effects of sympathetic nerve activity are depressed during hypoxia.

136 Women who are depressed during pregnancy have been found to have a

137 es and 13% of elderly beneficiaries reported being depressed during the previous year.

138 egulated via an AKT-dependent pathway, which is depressed during prolonged hypoxia.

139 ediated by endothelium-derived nitric oxide) is depressed during the early, hyperdynamic stage of sep

140 mains controversial whether adrenal function is depressed during the late stage of polymicrobial seps

141 torpid animals, indicating that translation is depressed during torpor.

142 rising from a single cholinergic interneuron was depressed during repetitive 2 Hz firing, intrastriat

143 Twenty-one percent were depressed during at least 80% of the follow-up peri

144 ison group was all mothers or only those who were depressed during pregnancy but not exposed to antid

145 diabetes and 8.5% (n = 886) without diabetes were depressed during pregnancy or postpartum.

146 the AMR dynamics only when bacterial numbers were depressed during therapy.

147 e occasions: during winter when the patients were depressed, during winter following 2 weeks of light

148 epression, more frequently agreed that "if I were depressed, fellow medical students would respect my

149 VSCCs in spines of rat CA1 pyramidal neurons are depressed for at least 30 min after brief trains of

150 m flashes showed directly that synaptic gain was depressed for 100-200 ms following transmission of a

151 sured by the end-systolic stiffness constant was depressed from 3.66 +/- 0.20 to 2.65 +/- 0.12 (p < 0

152 actin sliding velocity of the mutant myosin were depressed >80% compared with wild-type myosin.

153 p had evidence of IHD; 10 of 15 patients who were depressed had 1 or more traditional IHD risk factor

154 way in which the wt transposition frequency is depressed, i.e. by keeping the end sequences suboptim

155 Spouses were more likely to be depressed if they were older, less well educated, mor

156 oteins to maintain health, but its functions are depressed in aging and obesity.

157 pid species containing hexadecatrienoic acid are depressed in all of the sfd ssi2 npr1 plants, we pro

158 undance and sarcoplasmic reticulum Ca uptake are depressed in both sarcomere mutation-positive and -n

159 Cells lacking Coa4 are depressed in CcO activity but show no impairment in

160 Although endothelial NOS responses are depressed in congestive heart failure (CHF), inducib

161 the resultant neutrophil trafficking in vivo are depressed in G6PT-/- ascites during an inflammatory

162 Bradykinin responses are depressed in hypertension, and endothelial dysfuncti

163 ally deficient in the PGI2 receptor (IP) but are depressed in mice genetically deficient in the TxA2

164 s in the bone marrow, but not in the spleen, are depressed in mice treated with BQR.

165 ex of a mouse model of Alzheimer disease and are depressed in mouse brain cortex by deletion of p47(p

166 tuberculosis (MTB)-induced T cell responses are depressed in peripheral blood mononuclear cells of p

167 Resting energy expenditure (REE) can be depressed in affected individuals for undefined reaso

168 studies that, although calcification may not be depressed in all species, acidification will produce

169 escent conduct disorder, were more likely to be depressed in young adulthood compared to their counte

170 Exercise capacity is depressed in ACHD patients (even in allegedly asympto

171 e hypotheses that striatal dopamine activity is depressed in ADHD and that this contributes to sympto

172 like growth factor 1 receptor (IGF1R), which is depressed in atrial tissue samples from patients with

173 ic neurons from Nckx4 knock-out animals, and is depressed in cells in which NCKX4 is overexpressed.

174 Because mitochondrial function is depressed in failing hearts and iron accumulation can

175 monstrate, for the first time, that RCAN1-1L is depressed in Huntington disease.

176 Because cytosolic Ca(2+) signaling is depressed in injured dorsal root ganglia (DRG) neuron

177 Calcium-dependent contractile reserve is depressed in myocytes from rats with postinfarction h

178 that are not affected by hypertrophy, and it is depressed in patients who have a mutation for hypertr

179 ity between a predictive stimulus and reward is depressed in proportion to predicted reward.

180 ed the hypothesis that RV functional reserve is depressed in SSc-PAH patients.

181 RV contractile reserve is depressed in SSc-PAH versus IPAH subjects, associated

182 n 64.7 percent and left ventricular function was depressed in 26.0 percent.

183 Left ventricular systolic function was depressed in 30 patients (83%), with a median ejecti

184 ts (74.1%), while right ventricular function was depressed in 32 (59.3%).

185 f excitatory amino acids, such as glutamate, was depressed in 6 of 40 cells (none were enhanced).

186 a(2+)-ATPase to phospholamban protein levels was depressed in 7-week AS compared with controls but no

187 (i) increased in controls, but this response was depressed in 7-week AS.

188 Peak power was depressed in all fibre types with the greatest loss

189 CD4(+) T-cell expression of CD40L was depressed in B7KO mice in vivo and in vitro.

190 LV contractility was depressed in both groups (for bolus patients, median

191 RV SE was depressed in both groups to a similar degree to that

192 Phosphorylation of the MPM-2 epitope was depressed in both X-ray and etoposide-treated cells,

193 splenic macrophage IL-6 productive capacity was depressed in cells from animals subjected to CL/ETX

194 We found that synaptic transmission was depressed in corticostriatal slices after perfusion

195 Bradykinin, but not ACH-mediated vasomotion, was depressed in epicardial segments that constricted wi

196 IRES activity was depressed in H ferritin-deficient mouse embryonic fi

197 At week 1, %S was depressed in HYPER, COMB, and HYPO (9+/-8%, 7+/-6%,

198 In contrast, in vivo LV systolic pressure was depressed in L-arginine treated versus untreated rat

199 percent decrease in flow induced by L-NMMA, was depressed in male compared with female patients (-17

200 or-mediated inhibitory postsynaptic currents was depressed in medium spiny neurons of ethanol drinkin

201 Insulin receptor tyrosine kinase activity was depressed in muscle from obese insulin-resistant pat

202 The response to increased pacing frequency was depressed in NOS1(-/-) compared with wild type myocy

203 the muscle protein fractional synthetic rate was depressed in patients on day 1 (0.035%/hour; 95% CI,

204 Cardiac output was depressed in rats exposed to 20 cm H2O of PEEP by 32

205 Mesenteric blood flow was depressed in rats ventilated with 20 cm H2O of PEEP

206 e intact experiments, the slope of the PRSWR was depressed in the cardioplegia group compared with ba

207 esults indicate that hepatocellular function was depressed in the early stage of sepsis (i.e., 5 hrs

208 on of vascular cell adhesion molecule-1 mRNA was depressed in the IL-1R1 null mice, and corresponding

209 ereas the SITS-sensitive component of phi(E) was depressed in the low pH(i) range.

210 Epimerase activity was depressed in the mutant, but increased upon restorat

211 role in the differentiation of osteoclasts, was depressed in these cells.

212 vation of B cell-specific gene transcription was depressed in these cells.

213 d myofilament-Ca(2)(+) responsiveness, which was depressed in untreated Tm-E54K mice.

214 -old mice although the responses to both Ags were depressed in 2-year-old mice.

215 ols; however, erbB2 and erbB4 protein levels were depressed in 22-week aortic stenosis animals (48% d

216 in contrast, erbB2 and erbB4 message levels were depressed in 22-week aortic stenosis animals at the

217 GABA concentration-response curves were depressed in a mixed/non-competitive fashion.

218 mmunoglobulin G against C. difficile toxin A were depressed in aged mice, and vancomycin treatment re

219 ker for the high-affinity choline transport, were depressed in aged rats and GM1 corrected the defici

220 of this synchronously regulated gene cluster were depressed in both ventral and dorsal horn neurons w

221 ron, IL-12, and tumor necrosis factor alpha) were depressed in cells from MAM-injected wild-type C3H/

222 aspase-3 activation and TUNEL-positive cells were depressed in COX-2(-/-) livers.

223 f breathing at rest and during gas exposures were depressed in EH but not HH.

224 ssure and left ventricular ejection fraction were depressed in GhOEs.

225 Levels of TRH-[Gly(4)] were depressed in homozygotes.

226 rcoplasmic reticulum Ca2+-ATPase mRNA levels were depressed in male LVH but not in female LVH compare

227 viral and intracellular bacterial pathogens were depressed in mice in vivo and in humans to phorbol

228 Finally, epicardial BK responses were depressed in patients with high ACE levels and in t

229 Mechanical bone properties of strength were depressed in Pb-exposed bones, but HFD had no signi

230 ed with increased expression of SERCA2a, and were depressed in Pim-1-deficient hearts.

231 hough transitional 2 (T2) B cell populations were depressed in the absence of Zfp318.

232 ctional shortening and peak systolic [Ca2+]i were depressed in the MI compared with the control group

233 as by the ecto-ATPase inhibitor ARL67156 and were depressed in the presence of P2 receptor antagonist

234 SERCA2a protein levels were depressed in WT 7-week AS mice, but were preserved

235 diated slow inhibitory postsynaptic currents were depressed in wt but not betaarr-2 ko LC neurons aft

236 tension is pronounced during shortening, but is depressed/inhibited during lengthening, is consistent

237 temperature of the chain melting transition is depressed less for t18:1-18:0PC (31.5 degrees C) than

238 asal excitatory synaptic transmission in CA1 was depressed, long-term potentiation (LTP) was enhanced

239 rastriatally evoked fast EPSCs in LA neurons were depressed < or =24 hr after ischemia.

240 to model the probability that an individual was depressed, manic, or euthymic.

241 tocol that can be effective for patients who are depressed, markedly suppresses synaptic drive onto V

242 ich apolipoprotein B-containing lipoproteins were depressed may be via alterations in LDL receptor qu

243 The positive-well-being, depressed-mood, and composite scores of the Psych

244 In CLL specimens, E-cadherin expression was depressed more than 50% in 62% cases (relative to no

245 at 12 hours reperfusion wall thickening (WT) was depressed more, P<0.05, in CD (-46+/-5%) as compared

246 CD4+ cells were depressed more than CD8+ cells.

247 The 3 outcome states were depressed, nondepressed, and death, with scores of

248 Cholinergic effects were depressed (not obliterated) by D-tubocurarine (D-TC

249 vivors who were disabled were more likely to be depressed (odds ratio 1.35 per activities of daily li

250 ely with iNOS inhibitors, pacemaker activity was depressed only at the anastomosis site.

251 lly predicted youth depression if the mother was depressed only once.

252 ilure (CHF) doubles either when CHF patients are depressed or when their plasma norepinephrine (NE) l

253 e victims of violence or victimization or to be depressed or suicidal.

254 sk of corneal graft rejection when IFN-gamma is depressed or absent.

255 d in C3H/HeJ cell supernatants, whereas they were depressed or remained low in BALB/c cell supernatan

256 In HF, dobutamine +dP/dt response was depressed (P<0.001 versus control), and increased co

257 patients undergoing major abdominal surgery are depressed preoperatively.

258 One defect of CD21/35(-/-) animals is depressed production of Ag-specific IgG3 which we sho

259 after cardioversion from atrial fibrillation are depressed relative to left atrial appendage emptying

260 rall response rates in core-lesioned animals were depressed relative to that in the shell- or sham-le

261 tation in response to repetitive activation, are depressed significantly by baclofen, and do not expr

262 The baroreceptor reflex bradycardia was depressed significantly by NTS microinjections of AN

263 taining synthetic peptide, contractile force was depressed significantly by, 28% at 4 Hz, 37.7% at 5

264 Furthermore, cerebral metabolism was depressed significantly in a time-dependent manner w

265 rs (ie, V(max), CT, DTE, and S(1)-S(1)) also were depressed significantly by vernakalant and ranolazi

266 Calcineurin phosphatase activity was depressed similarly in both cyclosporine-treated gro

267 Populations of channels in single spines are depressed stochastically and synchronously, independ

268 Women were more likely (64%) to be depressed than men (44%).

269 authors studied 49 young people who had not been depressed themselves but who had a parent with a hi

270 When men are depressed they may experience symptoms that are diff

271 vels of PIF1- and PIF2-deficient ODV binding were depressed threefold compared to control levels.

272 ine-treated animals, putative BLA-mPFC EPSCs were depressed through a presynaptic mechanism.

273 greatly impaired in factor VIII deficiency, being depressed to less than 1/29 (<1.9 nmol TAT/L/min)

274 xidation of exogenous long-chain fatty acids is depressed to 30% wild-type levels.

275 eart allografts 5 days after transplantation is depressed to 40% of that of syngenic controls, and th

276 The pKa value of K312 is depressed to 9.58, a value indicating that at physiol

277 Testis selenium was depressed to 19% in mice fed an 0.1 mg selenium/kg d

278 Brain selenium was depressed to 43%, but feeding 2 mg selenium/kg diet

279 Kidney was depressed to 76% and reached Sepp(+/+) levels on an

280 Uptake of both tracers was depressed to a greater extent in areas of severe isc

281 eld of viruses from one of the parental RNAs was depressed to levels comparable to or less than the y

282 nucleotide exchange and the level of Ras-GTP were depressed to near basal values on the time scale of

283 eric myosin exhibited ATPase activities that were depressed to the levels observed in hearts expressi

284 erminating a line of first-order transitions is depressed toward zero temperature.

285 n the floccular complex of the cerebellum to be depressed transiently at the time of the instruction

286 EEG background activity is depressed transiently after moderate hypoxia-ischemia

287 the gene for inducible nitric oxide synthase was depressed transiently.

288 Regional LAD wall thickening was depressed under baseline conditions (LAD 3.7+/-0.3 v

289 1992, however, the odds that a smoker would be depressed were three times the odds that a nonsmoker

290 nts joining after randomisation, the odds of being depressed were 0.76 (95% CI 0.53 to 1.09) for the

291 hic measures whereas their systolic function is depressed when measured by the two-dimensional echoca

292 Urinary PGE-M was depressed, whereas urinary 2,3-dinor 6-keto PGF(1alp

293 , and metal assimilation efficiency (for Zn) was depressed, while the metal efflux rate (for Zn) was

294 nses to beta-adrenergic receptor stimulation are depressed with pressure overload-induced cardiac hyp

295 tractile reserve in response to rapid pacing was depressed with impaired augmentation of both peak-sy

296 The tension was depressed with increased [P(i)] in a concentration-d

297 direction (GFAP and NF-kappaB), while others were depressed with age (PDI and intensity of Nissl stai

298 s: when one input was potentiated, the other was depressed, with the direction and magnitude of chang

299 Genetic diversity was depressed within esterase 5 within Sex-ratio chromos

300 and K(+)-induced increases in IPSC frequency were depressed without a change in IPSC amplitude.